ライフサイエンスコーパス: dilated

1 the auditory tubes can become pathologically dilated.

2 Adjacent intrahepatic bile ducts were dilated.

3 st, the ventricles are collapsed rather than dilated.

4 ons in FLNC and the development of high-risk dilated and arrhythmogenic cardiomyopathies.

5 Dilated and hypertrophic cardiomyopathies are the most c

6 As expected, patients also had dilated and impaired left atria compared with control su

7 s in FLNC caused an overlapping phenotype of dilated and left-dominant arrhythmogenic cardiomyopathie

8 f the demarcation membrane system, which was dilated and poorly developed.

9 who found FLNC variants among patients with dilated and restrictive cardiomyopathies, pointed to thi

10 edema, diffuse intraretinal hemorrhages, and dilated and tortuous retinal veins.

11 tients and families, including hypertrophic, dilated, and arrhythmogenic cardiomyopathies and inherit

12 lumens in ducts and secretory portions were dilated, and blisters and papules formed on the skin sur

13 I) or those with mild to moderate TR with a dilated annulus (>/=40 or >/=21 mm(2), Class IIa).

14 pt knockout mice were viable but with mildly dilated aortas that had a 43% reduction in NAD(+) in the

15 Incompetent bicuspid aortic valves with dilated aortic annuli are also satisfactorily treated wi

16 ts presenting with aortic insufficiency or a dilated aortic annulus or ascending aorta were at greate

17 We studied consecutive patients with a dilated aortic root (>/=4 cm) that underwent echocardiog

18 In patients with dilated aortic root and trileaflet aortic valve, a ratio

19 HODS AND Ascending aortas from patients with dilated aortopathy were immunostained for NAMPT, reveali

20 ied in 28 probands previously diagnosed with dilated, arrhythmogenic, or restrictive cardiomyopathies

21 In the patient population with dilated ascending aorta, both peak TKE and total TKEsys

22 s associated with bicuspid aortic valves and dilated ascending aortic geometries that are not assessa

23 MRI revealed dilated atypical Virchow-Robin space (VRS).

24 A left hepatectomy was done and dilated bile ducts filled with caseous necrotic material

25 disseminated tuberculosis, and point-of-care dilated binocular indirect ophthalmoscopy eye examinatio

26 Small, round clear spheres in vitreous on dilated biomicroscopic retinal examination.

27 ereoscopic fundus photography (method 1) and dilated biomicroscopy in combination with optical cohere

28 e surgery when the tricuspid annulus (TA) is dilated but methodology for the measurement of TA size a

29 hether patterns are passively, homogeneously dilated by growth or whether they remodel during tissue

30 T images showed a striking detailed Y-shaped dilated capillary correlating with the clinical finding.

31 ing of the glomerular basement membrane, and dilated capillary loops, with a similar but later onset

32 ension development predicts hypertrophic and dilated cardiomyopathies in mice associated with essenti

33 c insert are linked to heart disease such as dilated cardiomyopathies.

34 re detected in about one-third of idiopathic dilated cardiomyopathies.

35 Brugada syndrome (7), long QT syndromes (5), dilated cardiomyopathy (2), and hypertrophic cardiomyopa

36 arrhythmias (69% and 52%, respectively), and dilated cardiomyopathy (74% and 14%, respectively).

37 otential of silencing miR-34a in settings of dilated cardiomyopathy (DCM) and atrial fibrillation (AF

38 mains debilitating heart conditions, such as dilated cardiomyopathy (DCM) and hypertrophic cardiomyop

39 ompared them with samples from patients with dilated cardiomyopathy (DCM) and inflammatory cardiomyop

40 phospholamban (PLN) gene are associated with dilated cardiomyopathy (DCM) and severe heart failure.

41 rect targets of FXR1 in human left ventricle dilated cardiomyopathy (DCM) biopsy samples and mouse mo

42 ABSTRACT: Dilated cardiomyopathy (DCM) can be caused by mutations

43 c metabolite quantification in patients with dilated cardiomyopathy (DCM) compared with that at 3 T.

44 Improved understanding of dilated cardiomyopathy (DCM) due to titin truncation (TT

45 Non-ischemic dilated cardiomyopathy (DCM) has been recognized as a he

46 Studies of children with dilated cardiomyopathy (DCM) have suggested that improve

47 esidue 9 in PLN to cysteine (R9C) results in dilated cardiomyopathy (DCM) in humans and transgenic mi

48 Dilated cardiomyopathy (DCM) is a common form of cardiom

49 Dilated cardiomyopathy (DCM) is a condition of abnormal

50 Dilated cardiomyopathy (DCM) is a leading cause of heart

51 Dilated cardiomyopathy (DCM) is an important cause of he

52 Dilated cardiomyopathy (DCM) is best understood as the f

53 The cause of idiopathic dilated cardiomyopathy (DCM) is unknown by definition, b

54 Patients with nonischemic dilated cardiomyopathy (DCM) may be at lower risk for ve

55 ix hypertrophic cardiomyopathy (HCM) and two dilated cardiomyopathy (DCM) mutants were studied by bio

56 Nonischemic dilated cardiomyopathy (DCM) often has a genetic pathoge

57 tudies reveal reduced force generation and a dilated cardiomyopathy (DCM) phenotype.

58 Peripartum cardiomyopathy (PPCM) and dilated cardiomyopathy (DCM) show similarities in clinic

59 g heart and in the heart of a mouse model of dilated cardiomyopathy (DCM) triggered by Serum Response

60 mode of inheritance, are important causes of dilated cardiomyopathy (DCM), a disease characterized by

61 Ntvs)] are the most common genetic cause for dilated cardiomyopathy (DCM), a major cause of heart fai

62 rotein, is often associated with arrhythmia, dilated cardiomyopathy (DCM), and heart failure.

63 rtality) suggest that for many patients with dilated cardiomyopathy (DCM), implantable cardioverter-d

64 disease (CCD) and, after six months of age, dilated cardiomyopathy (DCM), most noticeably in the mal

65 mice count among the first mouse models for dilated cardiomyopathy (DCM), yet the exact role of MLP

66 tions of CPET with outcomes in children with dilated cardiomyopathy (DCM).

67 reifuss muscular dystrophy (EDMD) as well as dilated cardiomyopathy (DCM).

68 n-truncating variants (TTNtv) commonly cause dilated cardiomyopathy (DCM).

69 ing variants (TTNtvs) are the major cause of dilated cardiomyopathy (DCM); however, allelic heterogen

70 patients with aortic stenosis (AS, n=9) and dilated cardiomyopathy (DCM, n=6).

71 Inflammatory dilated cardiomyopathy (DCMi) is a major cause of heart

72 loci that are significantly associated with dilated cardiomyopathy (false discovery corrected P</=0.

73 aring the survival of children with familial dilated cardiomyopathy (FDCM) to that of children with i

74 tients (n = 154) with documented chronic and dilated cardiomyopathy (ICM, n = 61; NICM, n = 93) requi

75 y (FDCM) to that of children with idiopathic dilated cardiomyopathy (IDCM) has produced conflicting r

76 nction and myocardial injuries in idiopathic dilated cardiomyopathy (IDCM) using cardiac magnetic res

77 Forty-seven patients with idiopathic dilated cardiomyopathy (left ventricular ejection fracti

78 was examined in human explanted hearts with dilated cardiomyopathy (n = 25).

79 Dilated cardiomyopathy (n=27), myocarditis or sarcoidosi

80 findings, patients were classified as having dilated cardiomyopathy (n=40) or iCMP (n=75).

81 achycardia (VT) in patients with nonischemic dilated cardiomyopathy (NIDCM) are insufficient.

82 tic information in patients with nonischemic dilated cardiomyopathy (NIDCM).

83 hy, few studies exist in chronic nonischemic dilated cardiomyopathy (NIDCM).

84 jection Delivery Effects on Neomyogenesis in Dilated Cardiomyopathy [PoseidonDCM]; NCT01392625).

85 ion using RNA-sequencing in 97 patients with dilated cardiomyopathy and 108 non-diseased controls.

86 .5 years; 64 patients (65.9%) had idiopathic dilated cardiomyopathy and 21 patients (21.6%) had ische

87 wall LGE identifies a group of patients with dilated cardiomyopathy and an LVEF >/=40% at increased r

88 aborted SCD among consecutive referrals with dilated cardiomyopathy and an LVEF >/=40% to our center

89 Here, we studied a family with dilated cardiomyopathy and associated conduction system

90 Mutation in DnaJC19 results in dilated cardiomyopathy and ataxia syndrome, whereas expr

91 tem cells were used to examine a family with dilated cardiomyopathy and atrial and ventricular arrhyt

92 we link a subset of 517 epigenetic loci with dilated cardiomyopathy and cardiac gene expression.

93 compared with subjects with noninflammatory dilated cardiomyopathy and controls.

94 myocardial tissue and blood of patients with dilated cardiomyopathy and controls.

95 obal developmental delay, failure to thrive, dilated cardiomyopathy and epilepsy, ultimately leading

96 l mutations within HSPB7 are associated with dilated cardiomyopathy and heart failure in human patien

97 ocytes, notably in ageing hearts, leading to dilated cardiomyopathy and heart failure in LGM2B patien

98 (2)(+) responsiveness in a model of familial dilated cardiomyopathy and improve cardiac function and

99 nd inflammation in patients with nonischemic dilated cardiomyopathy and inflammatory cardiomyopathy (

100 f Titin truncation variants in patients with dilated cardiomyopathy and population controls.

101 se of ivabradine in paediatric patients with dilated cardiomyopathy and symptomatic chronic heart fai

102 relationship of ivabradine in children with dilated cardiomyopathy and symptomatic chronic HF.

103 In patients with nonischemic dilated cardiomyopathy and VT, endocardial and adjuvant

104 No CEs occurred in patients without dilated cardiomyopathy and/or LGE.

105 ed cardiomyopathies allowed establishment of dilated cardiomyopathy as mostly cytoskeleton, force tra

106 t for either sex in a setting of more severe dilated cardiomyopathy associated with atrial fibrillati

107 these circRNAs are dynamically regulated in dilated cardiomyopathy but not in hypertrophic cardiomyo

108 day 6.5-9.5 developed a phenotype similar to dilated cardiomyopathy by 1 year of age.

109 may be a viable approach to the treatment of dilated cardiomyopathy by not only preventing maladaptiv

110 achycardia (VT) in patients with nonischemic dilated cardiomyopathy can be challenging because of the

111 ncation mutation information from 1714 human dilated cardiomyopathy cases and >69 000 controls and fo

112 Patients with myocarditis or dilated cardiomyopathy develop autoantibodies to SERCA2a

113 in quality control, we used a mouse model of dilated cardiomyopathy driven by cardiac restricted over

114 cardioverter defibrillator in patients with dilated cardiomyopathy for the primary prevention of sud

115 d controls, affecting known as well as novel dilated cardiomyopathy genes.

116 und that eQTL variants are also enriched for dilated cardiomyopathy genome-wide association signals i

117 lume Variability) in pediatric patients with dilated cardiomyopathy has reported reproducibility of s

118 Most known genes for human dilated cardiomyopathy have a corresponding zebrafish or

119 ommon cause of heart failure was nonischemic dilated cardiomyopathy in 27.5% (whites, 19.9%; P<0.001)

120 and metabolic compromise in the etiology of dilated cardiomyopathy in DMD and identify a window of o

121 iated with features of both hypertrophic and dilated cardiomyopathy in mice.

122 ns presented with severe hypertrophic and/or dilated cardiomyopathy in utero, at birth, or in early c

123 Because a common finding in dilated cardiomyopathy is a reduction in the myofilament

124 Up to 40% of dilated cardiomyopathy is associated with inflammation o

125 involved in viral RNA replication.IMPORTANCE Dilated cardiomyopathy is the most common indication for

126 tes into the spectrum of hypertrophic versus dilated cardiomyopathy is unknown.

127 ividuals with either the most severe form of dilated cardiomyopathy or whose mutations demonstrated c

128 We enrolled 409 consecutive ischemic and dilated cardiomyopathy patients (mean age: 64+/-12 years

129 nces of gene expression and splicing between dilated cardiomyopathy patients and controls, affecting

130 ) in ischemic cardiomyopathy and nonischemic dilated cardiomyopathy patients evaluated for primary pr

131 ssion are each important determinants of the dilated cardiomyopathy phenotype and are controlled by g

132 d that the knockout strategy ameliorates the dilated cardiomyopathy phenotype in vitro.

133 humanized" telomere lengths, the devastating dilated cardiomyopathy phenotype seen in patients with D

134 Focusing on the dilated cardiomyopathy phenotype we found that eQTL vari

135 en with myocarditis and 1249 with idiopathic dilated cardiomyopathy received HT.

136 during cardiac contraction in patients with dilated cardiomyopathy relative to controls.

137 etween diastole and systole in patients with dilated cardiomyopathy relative to healthy controls usin

138 d reduced ejection fraction due to ischaemic dilated cardiomyopathy resulted in a significant reducti

139 Myocarditis can cause dilated cardiomyopathy resulting in end-stage heart fail

140 signatures and myocardium from subjects with dilated cardiomyopathy showed excessive Parkin and CHOP

141 ffective in females in a setting of moderate dilated cardiomyopathy than in males.

142 BCL2-associated athanogene 3 (BAG3) develop dilated cardiomyopathy that is associated with a destabi

143 r envelope gene SYNE1 in a child with severe dilated cardiomyopathy that underwent transplant, as wel

144 with ischemic cardiomyopathy or nonischemic dilated cardiomyopathy undergoing cardiovascular magneti

145 -nine patients with ischemic or non-ischemic dilated cardiomyopathy undergoing prophylactic ICD impla

146 y shows that different gene mutations induce dilated cardiomyopathy via diverse cellular pathways.

147 Ang II levels in explanted human hearts with dilated cardiomyopathy were elevated despite ACE inhibit

148 d 59+/-15 years, 80% males) with nonischemic dilated cardiomyopathy who underwent CA.

149 en with myocarditis and 1583 with idiopathic dilated cardiomyopathy who were <18 years old and listed

150 in the mouse heart for the first time during dilated cardiomyopathy with heart failure.

151 magnetic resonance identified patients with dilated cardiomyopathy without severe LV systolic dysfun

152 164 ischemic cardiomyopathy, 150 nonischemic dilated cardiomyopathy), the mean left ventricular eject

153 In patients with dilated cardiomyopathy, a more longitudinal orientation

154 is patients, 11 ischemic heart disease, nine dilated cardiomyopathy, and 11 nonfailing donors.

155 n of function occurs in 20% of children with dilated cardiomyopathy, and 40% die or undergo transplan

156 the regulation of development, ischemic and dilated cardiomyopathy, and myocardial infarction.

157 e cardiac Jak2-deleted mice had hypertrophy, dilated cardiomyopathy, and severe left ventricular dysf

158 nd Becker muscular dystrophy (BMD), X-linked dilated cardiomyopathy, as well as DMD and BMD female ca

159 age-dependent cardiac phenotypes, including dilated cardiomyopathy, cardiac conduction disturbance,

160 elanocortin-4 receptor (MC4R) in mice causes dilated cardiomyopathy, characterized by reduced contrac

161 In pediatric patients with dilated cardiomyopathy, compared with dimension and area

162 ith homozygous MYBPC3-null mutations develop dilated cardiomyopathy, coupled with myocyte hyperplasia

163 otypes (hypertrophic cardiomyopathy, HCM and dilated cardiomyopathy, DCM) associated with mutations i

164 c cardiomyopathy, ischemic heart disease, or dilated cardiomyopathy, in comparison to nonfailing hear

165 y bowel disease, infections, cerebral palsy, dilated cardiomyopathy, muscular dystrophy, and schizoph

166 In patients with dilated cardiomyopathy, myocardial Gal-3 expression corr

167 tional mutant mice developed signs of severe dilated cardiomyopathy, myocardial infarctions, and prem

168 emaining cardiac diagnosis groups, including dilated cardiomyopathy, myocarditis, and ischemic and no

169 if concentric hypertrophy does progress to a dilated cardiomyopathy, such a transition would occur ov

170 ted from a patient suffering from idiopathic dilated cardiomyopathy, suggesting that such mutant viru

171 ere pathologies, such as aseptic meningitis, dilated cardiomyopathy, type I diabetes, paralysis, and

172 ve cardiac fibrosis, all features present in dilated cardiomyopathy, were observed in the aged sFRP-1

173 t be an ideal candidate for the treatment of dilated cardiomyopathy, which displays modest microvascu

174 V symptomatic heart failure due to ischaemic dilated cardiomyopathy, who had left ventricular ejectio

175 We treated a dilated cardiomyopathy-linked mouse model expressing a m

176 ponin protein stoichiometry in patients with dilated cardiomyopathy.

177 sease remodelling in patients suffering from dilated cardiomyopathy.

178 he most common type of genetic alteration in dilated cardiomyopathy.

179 , and leiomodin-2-knockout mice present with dilated cardiomyopathy.

180 venting progression of inflammation-mediated dilated cardiomyopathy.

181 to one that leads to chronic remodeling and dilated cardiomyopathy.

182 to identify novel mutations causing familial dilated cardiomyopathy.

183 al and adjuvant epicardial CA in nonischemic dilated cardiomyopathy.

184 scle cell leads to myocyte instability and a dilated cardiomyopathy.

185 children who are transplanted for idiopathic dilated cardiomyopathy.

186 heart failure than children with idiopathic dilated cardiomyopathy.

187 on that ultimately results in a severe fatal dilated cardiomyopathy.

188 patients, and those with either ischemic or dilated cardiomyopathy.

189 many cardiac genes, and loss of RBM20 causes dilated cardiomyopathy.

190 the structural alterations in patients with dilated cardiomyopathy.

191 al human disease phenotypes, hypertrophic or dilated cardiomyopathy.

192 recover spontaneously, some develop chronic dilated cardiomyopathy.

193 patients with heart failure due to ischaemic dilated cardiomyopathy.

194 evated in left ventricles from patients with dilated cardiomyopathy.

195 ises heart function and structure, producing dilated cardiomyopathy.

196 metabolic remodelling and has a key role in dilated cardiomyopathy.

197 ation (Id cDKOs), which develops adult-onset dilated cardiomyopathy.

198 induced hypertrophy and in human hearts with dilated cardiomyopathy.

199 ular arrhythmias in patients with idiopathic dilated cardiomyopathy.

200 t ventricle (LV) ultimately transitions to a dilated cardiomyopathy.

201 ative relatives, and 9 with noncompaction or dilated cardiomyopathy.

202 g heart rate of children with chronic HF and dilated cardiomyopathy.

203 COX and copper deficiency that resulted in a dilated cardiomyopathy.

204 Patients with dilated cardioyopathy had a significantly lower PCr/ATP

205 Chagas disease causes a severe inflammatory dilated chronic cardiomyopathy (CCC).

206 interconnected network of narrow tubules and dilated cisternae that enclose a single lumen.

207 er medical record based on their most recent dilated comprehensive eye examination by a primary eye c

208 roduce a network architecture based on using dilated convolutions to capture features at different im

209 -myosin variants cause hypertrophic (HCM) or dilated (DCM) cardiomyopathy by disrupting sarcomere con

210 CGRP dilated dural, but not pial, vessels and significantly r

211 hypothesis that the right ventricle is more dilated during resuscitation from cardiac arrest caused

212 The right ventricle was dilated during resuscitation from cardiac arrest caused

213 The right ventricle was more dilated during resuscitation when cardiac arrest was cau

214 ay, resulting in a buildup of virions within dilated ER vesicles.IMPORTANCE In humans, symptoms of RV

215 eported an eye care visit and 21% reported a dilated examination in the past 12 months.

216 in the past 12 months, and ever having had a dilated examination, and odds ratios for factors associa

217 y 25.0% of eyes deemed to be normal based on dilated eye examination by primary eye care physicians h

218 sed as eye care visit in the past 12 months, dilated eye examination in the past 12 months, and ever

219 The odds of reporting receipt of a dilated eye examination in the past year were also highe

220 However, the odds of reported receipt of a dilated eye examination in the past year were lower in c

221 nd Measures: Eye care visit and receipt of a dilated eye examination in the past year.

222 rty-eight percent reported ever having had a dilated eye examination.

223 wer odds of reporting ever having received a dilated eye examination.

224 atients with diabetes reported ever having a dilated eye examination; only 39.62% reported receiving

225 CI] 38.56%-40.67%) reported receiving annual dilated eye examinations, and 90.31% (CI 89.70%-90.91%)

226 gen was inefficient and that ER tubules were dilated, faithfully reproducing the cellular phenotype o

227 erwent a complete eye examination, including dilated fundoscopy and cycloplegic refraction.

228 deemed to have ocular fungal involvement if dilated fundus examination demonstrated evidence of chor

229 ion included Snellen-measured visual acuity, dilated fundus examination, and spectral-domain optical

230 cted visual acuity, slit-lamp biomicroscopy, dilated fundus examination, wide-field photography, fund

231 distance and near visual acuity evaluation; dilated fundus examination; OCT with 12 x 6-mm thickness

232 fraction, clinical findings of slit-lamp and dilated fundus examinations.

233 ent a comprehensive eye examination in which dilated fundus photographs (disc and macula centered) we

234 comprehensive ocular examination, including dilated fundus photography.

235 Dilated funduscopy also showed bone spicule-like pigment

236 d distended duodenum filled with fluid and a dilated gallbladder containing dilute bile with high bic

237 growth of lesions, which exhibited massively dilated glands with a flattened layer of stroma.

238 ell with the number of cross sections of the dilated glandular ducts counted under microscopy.

239 The dilated glandular ducts pushed the uterine horn lumen to

240 Computational modeling revealed that the dilated heart results in reduced HA changes compared wit

241 ve never been analysed in human non-ischemic dilated hearts (DCM).

242 The adult mutant mice developed dilated hearts and showed significantly shorter life spa

243 sis revealed CpG hypermethylation within the dilated human thoracic aorta and in SMCs cultured from t

244 Dilated hyporeflective tubular structures within the cho

245 lic dysfunction is general to all idiopathic dilated (IDCM) and hypertrophic cardiomyopathy (HCM) pat

246 ction of cardiac arrest, the right ventricle dilated in all groups (p < 0.01 for all).

247 mucosa harboring the S631G variant revealed dilated intercellular spaces and reduced desmosomes.

248 g (basal zone hyperplasia, desquamation, and dilated intercellular spaces; P < .0001), lamina propria

249 However, the right ventricle was dilated, irrespective of the cause of arrest, and diagno

250 es in diverse cell types that accumulated as dilated late endosomes.

251 The mice developed hydrocephalus and grossly dilated lateral ventricles, with accumulation of 2-hydro

252 lodged in the aortomesenteric region with a dilated left ovarian vein and pelvic varicose veins.

253 those with an ischemic pathogenesis, a more dilated left ventricle, and a detectable hs-troponin had

254 se with interval cardiovascular events and a dilated LV (increased LV end-diastolic volume [EDV] inde

255 The progression to a dilated LV was uncommon (2%, n=25).

256 as minimal, and few participants developed a dilated LV.

257 e constitutively lacking EC Map4k4 displayed dilated lymphatic capillaries, insufficient lymphatic va

258 Cholangioscopy revealed dilated lymphatic vessels obstructing the bile duct and

259 m in diameter, was inserted to the straight, dilated main duct and plastic stent(s) were inserted to

260 multilocular cysts of the pancreas and a non-dilated main pancreatic duct (<5 mm).

261 e because of mechanical interaction with the dilated main pulmonary artery (MPA).

262 n the majority of participants with branched dilated microvessels (11 of 13) (McNemar Test for equal

263 nce of vascular microhemorrhage and branched dilated microvessels in the tissues lining the clinicall

264 plasma gap could be detected in the branched dilated microvessels in tissues lining the GC.

265 sence of microhemorrhage related to branched dilated microvessels.

266 ntraocular pressure measurement, gonioscopy, dilated ophthalmoscopy, and standard automated perimetry

267 ts underwent external ocular examination and dilated ophthalmoscopy.

268 ion in ventricular tissue from patients with dilated or ischemic cardiomyopathy compared to nonfailin

269 electron microscopy of a liver biopsy showed dilated organelles suggestive of the endoplasmic reticul

270 topic sonic hedgehog (Shh) in subsets of non-dilated P7 mutant proximal tubules (likely driving the s

271 ntify white matter lesion load, frequency of dilated perivascular spaces (PVS) and abnormalities in c

272 to facilitate early attendance, and careful dilated postoperative examination for retinal tears is r

273 In patients with a dilated proximal ascending aorta and trileaflet aortic v

274 In bicuspid aortic valve patients with dilated proximal ascending aorta, ascending aortic area/

275 In patients with bicuspid aortic valve and dilated proximal ascending aorta, we sought to assess (1

276 es, kidneys of newborn mutant mice exhibited dilated proximal tubules and immature glomeruli, and the

277 emokine production (e.g. ectopic Ccl2 in non-dilated proximal tubules), and augmented hedgehog signal

278 Kim1 and noted ectopic expression in P7 non-dilated proximal tubules.

279 valence of LMCA extrinsic compression from a dilated pulmonary artery (PA) in patients with PAH and a

280 ed pupil, and one of active locomotion and a dilated pupil, indicative of heightened attention.

281 d one had exposure keratopathy and traumatic dilated pupil.

282 ncreased NO bioavailability that dynamically dilated resistance vessels in vivo under basal condition

283 ecules and pathways that cause hypertrophic, dilated, restrictive, and arrhythmogenic cardiomyopathie

284 ning for diabetic retinopathy include annual dilated retinal examinations to detect proliferative ret

285 the left uterus with evidence of blood in a dilated retort-shaped left fallopian tube and a normal r

286 dard surgical pulmonary valve replacement in dilated right ventricular outflow tracts, permitting low

287 protogranules with paracrystalline contents, dilated rough endoplasmic reticulum, and electron-lucent

288 eurysm and aortic insufficiency secondary to dilated sinotubular junction and a normal aortic annulus

289 Rbpj from the adult endothelium also led to dilated sinusoids, vascular shunts, and necrosis, albeit

290 surface, abnormal vascular architecture, and dilated sinusoids.

291 n initial small-cation conductive state to a dilated state, which allows the passage of larger synthe

292 ates as either a more closed state or a more dilated state.

293 ter is the intentional fracture of maximally dilated stents, which can be accomplished using ultra-hi

294 Participants underwent dilated stereo-digital fundus photography graded accordi

295 tivated the genioglossus muscle and markedly dilated the pharynx, whereas saline had no effect.

296 lso revealed a left dysplastic kidney with a dilated, tortuous ureter.

297 nes recommend TV repair in the presence of a dilated tricuspid annulus at the time of a left-sided va

298 We further demonstrated that dilated vasculature, loss of vascular integrity resultin

299 PR-OCTA disclosed dilated vessels in the intermediate and deep plexuses in

300 ive hamartomas containing abnormal tortuous, dilated vessels prominent in the forelimbs.