ライフサイエンスコーパス: dilator muscle

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1 ugh alpha-adrenergic stimulation of the iris dilator muscle.

2 neuropil and orthodromically to the pyloric dilator muscles.

3 lung volume, and dysfunctional upper airway dilator muscles.

4 Pharyngeal dilator muscle activation (GGEMG) during wakefulness is

5 of local mechanisms in mediating pharyngeal dilator muscle activation in normal humans during wakefu

6 small upper airway with augmented pharyngeal dilator muscle activation maintaining airway patency awa

7 ortant in mediating the increased pharyngeal dilator muscle activation seen in sleep apnea patients d

8 imulus) were important in driving pharyngeal dilator muscle activation, one would predict that during

9 ich could not be explained by differences in dilator muscle activation.

10 aoral surface electrode to record pharyngeal dilator muscle activity (the genioglossus [EMGgg] normal

11 aoral surface electrode to record pharyngeal dilator muscle activity (the genioglossus [EMGgg]), we e

12 pper airway collapse is decreased pharyngeal dilator muscle activity during sleep.

13 ep apnea (OSA) may have augmented pharyngeal dilator muscle activity during wakefulness, to compensat

14 of two serotonin antagonists on upper airway dilator muscle activity, diaphragm activity, Sao2, and u

15 With reductions in dilator muscle activity, upper airway cross-sectional ar

16 riven primarily by posterior location of the dilator muscle and by dynamic pupillary block, but the e

17 Pharyngeal dilator muscles are clearly important in the pathogenesi

18 Pharyngeal dilator muscles are clearly important in the pathogenesi

19 Pharyngeal dilator muscles are clearly important in the pathophysio

20 Pharyngeal dilator muscles are important in the pathophysiology of

21 Upper airway dilator muscles are phasically activated throughout brea

22 The stimuli controlling pharyngeal dilator muscles are poorly defined.

23 ay have augmented serotonergic control of UA dilator muscles as a mechanism to prevent pharyngeal col

24 ta provide strong evidence that upper airway dilator muscles can be activated throughout inspiration

25 he complex lumen and marked expansion of the dilator muscles characteristic of 50- and 100-day untrea

26 activation is similar to that of pharyngeal dilator muscles during spontaneous and induced apneas, a

27 is, we assessed the relationship between two dilator muscle electromyograms (EMGs, i.e., genioglossus

28 chanics, ventilation, plus activation of two dilator muscles (genioglossus [GG] and tensor palatini [

29 n is in the nucleus ambiguus innervating the dilator muscles of the soft palate, pharynx, and larynx,

30 and collapsibility and enhanced upper-airway dilator muscle responses to avoid OSA.

31 The genioglossus is an upper airway dilator muscle, the length of which is directly related

32 mitigated by highly responsive upper-airway dilator muscles to avoid OSA.

33 way anatomy, (2) the ability of upper airway dilator muscles to respond to rising intrapharyngeal neg

34 increased mechanoreflex-mediated activity of dilator muscles while awake, but this reflex is inhibite

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