1 t that the mechanism postulated to result in dilutional acidosis is incomplete.

2                               In this study, dilutional acidosis was evaluated as a possible mechanis

3                                 Classically, dilutional acidosis would explain the predominance of th

4 refore, we investigated the effects of acute dilutional anemia on heart, brain, kidneys, liver, small

5                                              Dilutional assays revealed that fluorescence melting cur

6                            We also performed dilutional assays using the MBR/JH-positive cell line SU

7                               Using limiting dilutional bone marrow cultures, we observed an activity

8 (calcifying vascular cells, CVC), derived by dilutional cloning of bovine aortic medial cells, and sh

9                                              Dilutional coagulopathy after resuscitation with crystal

10 reatment leads to increased clot firmness in dilutional coagulopathy as measured with thromboelastome

11  acidosis and provided guidance for limiting dilutional coagulopathy.

12 ombinase is clearly mediated by flow-induced dilutional effects, we find that when factor X is activa

13 y that variations with shear rate arise from dilutional effects.

14  implicated in the renal water retention and dilutional hyponatremia associated with chronic heart fa

15 er excretion, remain under investigation for dilutional hyponatremia in ADHF.

16 onse to osmotic stimuli is a common cause of dilutional hyponatremia in neurological disorders.

17 er than sodium deficiency, is the culprit in dilutional hyponatremia, isotonic saline administration

18 uld differentiate between depletional versus dilutional hyponatremia.

19 umes measured by deuterium oxide and bromine dilutional space determination, respectively.