ライフサイエンスコーパス: dimer

1 ium with the corresponding thiuram disulfide dimer.

2 mechanism of formation of the Pd(I) bromide dimer.

3 onnectivity to form the corresponding Ru(2+)-dimer.

4 ger than the previously studied cytochrome c dimer.

5 uminescence resonance energy transfer of APJ dimer.

6 ucture of an amino-terminally truncated EHD4 dimer.

7 n and rotation of a partially dissociated gD dimer.

8 ro-nucleotide loop monomer or an interlocked dimer.

9 ey actually assemble, function and gate as a dimer.

10 lized, antiferromagnetically coupled Co(IV)2 dimer.

11 d only a single "canonical" site per tubulin dimer.

12 t in a polymerization-competent antithrombin dimer.

13 he open "face-back" tunnel through the PD-L1 dimer.

14 experimental systems, the enzyme exists as a dimer.

15 associates, forming an orthogonal homophilic dimer.

16 al significance to the crystal structure NS5 dimer.

17 or to yield mu-oxo and/or mu-hydroxo Mn(III) dimers.

18 occur at protein-protein interfaces of opsin dimers.

19 in the conformational heterogeneity of Hsp27 dimers.

20 lass III nucleotidyl cyclases are functional dimers.

21 sites on the GPCR, and (iii) asymmetric GPCR dimers.

22 olate (pyalk) ligand and robustness of these dimers.

23 t it exists as monomers but also as covalent dimers.

24 probe, due to the formation of TLR-4 protein dimers.

25 t and to prevent formation of inactive Ni(I) dimers.

26 of ATP hydrolysis, forcibly disrupting IRE1 dimers.

27 emble to form a stochastic repertoire of cis-dimers.

28 us an inbuilt SSu mimic that concentrates L2 dimers.

29 correlates with increased propensity to form dimers.

30 uman and mouse Siglecs form disulfide-linked dimers.

31 Monomers (5 Si atoms/molecule) and dimers (10 Si atoms/molecule) dominated the mass spectra

32 transcription factors operate as monomers or dimers, a few, including the E26 transformation-specific

33 The remarkable difference observed in the dimer abundance under O3- versus OH-dominant environment

34 ting experiments suggest a model where a PC4 dimer accommodates the DNA with one monomer on the G4 st

35 ng effects, the EPOR chains in the different dimers adopt very similar conformations with no clear di

36 d rhDNase, which only forms His-His covalent dimers after light treatment to determine the factors th

37 ions that allow the formation of an inactive dimer, alter substrate/coenzyme binding, or impair struc

38 The CTD dimer and CTD trimer interfaces are also intrinsically v

39 identified conditions that destabilized the dimer and measured the effects of these conditions on cl

40 We found here that Leu-27 is buried in the dimer and that the L27A mutation promotes monomerization

41 While Pb(IV) ions affected mainly Abeta dimer and trimer formation, hydrophobic toluene mainly a

42 UV-induced DNA lesion cyclobutane pyrimidine dimer and was recently found to incorporate ribonucleoti

43 hat NapA and NHA2, but not NhaA, form stable dimers and do not selectively retain membrane lipids.

44 The transactivation of RAF dimers and ERK signaling promotes HCC cell survival, pre

45 G12V K-Ras exists as a mixture of monomers, dimers and larger oligomers, while the K101E mutant is p

46 ies are much less prevalent than monomers or dimers and the aerosol yield is higher.

47 psis UVR8, but M. polymorpha UVR8 has weaker dimers and the proteins appear more constitutively nucle

48 ters interactions that stabilize coiled-coil dimers and thick filaments, causing disruption in ordere

49 of this photochemistry are covalently bonded dimers and trimers of the starting oxoacids, many of whi

50 A mainly occurs as a monomer, but also forms dimers and trimers, potentially via a coiled-coil alpha-

51 sortase A to form crosslinked SCRII, mainly dimers and trimers.

52 CRP, TNF, sIL-6R, I-FABP, sCD14, D-dimer, and HA levels were elevated in acute HIV infectio

53 iproteins bind to the Ras effector domain as dimers, and high-resolution crystal structures revealed

54 Motile DDB complexes bound up to two LIS1 dimers, and mutational analysis suggested that LIS1 bind

55 ity is intrinsic to chemoreceptor trimers-of-dimers, and results from conformational entropy within t

56 phosphorylated filaments to folded monomers, dimers, and small oligomers, followed by diffusion of th

57 The data for the dimer are compared to two different ZnTPP-PDI monomer re

58 Dimers are asymmetrical and display a bent "S-shape" arc

59 ange of non-natural cytokine receptor hetero-dimers are competent to elicit a signaling output.

60 Three-dimensional (3D) models of dimers are generated by threading a target amino acid se

61 the stabilization is such that hydrogenated dimers are observable with FTIR at low overpotentials.

62 ect receptor function, we show that receiver dimers are phosphorylated in trans by the donor; this pr

63 amantyl (C28H38) and oxadiamantyl (C26H34O2) dimers are stabilized by London dispersion attractions b

64 These locked dimers are strong immunogen candidates for a next-genera

65 Here we show that STAT5 dimers are sufficient for NK cell development, whereas S

66 The Ir(IV,V) dimers are unprecedented and exhibit remarkable stabilit

67 This dimer arrangement is different from any oligomeric state

68 use of preformed and isolated Pd(I) bromide dimer as a precatalyst provided superior results, in ter

69 ethyldiethylenetriamine and diglyme bind the dimer as bidentate rather than tridentate ligands.

70 r and a perylene-3,4:9,10-bis(dicarboximide) dimer as the acceptor (PDI2).

71 avivirus particle structure, definition of E dimers as the key antigenic target, and deep understandi

72 Two Cas1 dimers assemble on a Cas2 domain dimeric core, which is

73 rgoes conformational changes stabilizing the dimer assembly.

74 in solution, consistent with the pentamer-of-dimers assembly seen crystallographically.

75 SEC-MALLS) experiments showed that CodY is a dimer at concentrations found in bacterial cells.

76 We test the method on the driven Hubbard dimer at half filling, and compare exact and approximate

77 188 and that chimpanzee and gorilla A3C form dimers at the same interface as human A3C S188I, but thr

78 We have constructed models of the LBD dimers based on the GluK2 LBD crystal structures and inv

79 DS monomer was the dominant observable form, dimer-based metalation was significant.

80 high selectivity exclusively to monosolvated-dimer-based transition structures.

81 l structures revealed that these miniprotein dimers bind Ras in an unprecedented mode in which the Ra

82 The NFkappaB dimers bind to a myriad of genomic sites and switch the

83 llowed us to simultaneously measure FcgammaR dimer-binding Abs to 32 different HIV Ags, providing a m

84 embly of the full-length RagA(GTP):RagC(GDP) dimer bound to Ragulator at 16 A resolution, revealing t

85 seen for other organisms, and within tubulin dimers, but binds mammalian tubulin only at interdimer c

86 ophilia, and inflammation (LDH, bilirubin, D-dimer, C-reactive protein [CRP]) improved.

87 ind that under these conditions, the kinesin dimer can attach to the microtubule with either one or t

88 e how harnessing a conformationally adaptive dimer can drive large-scale cargo movement without the r

89 A new study finds that archaeal histone dimers can multimerize into extended superhelical struct

90 in the tubules, which develops when tubulin dimers change shape, triggered by a hydrolysis event.

91 monomers (class 1) or constitutively active dimers (class 2).

92 er CLC channels and transporters form stable dimers, ClC-4 was mostly observed as monomer, with ClC-3

93 Here we show that macrocyclic cinnamate dimers combine these productive stress-responsive modes.

94 nitial reports of NECD from the cytochrome c dimer complex, no further evidence of the effect has bee

95 DNA-bound ParA-AMPPNP adopts a dimer conformation distinct from the ATP sandwich dimer,

96 mer is intrinsically disordered and that the dimer conformation is very similar to that of the tetram

97 The particle is a dimer consisting of two ping-pong balls connected by a r

98 sequencing of short, cyclobutane pyrimidine dimer-containing ssDNA oligos generated during repair of

99 wever, the mechanism by which the PAR4-P2Y12 dimer controls beta-arrestin-dependent Akt signaling is

100 including UV-induced cyclobutane pyrimidine dimers (CPDs) and BaP diol epoxide-deoxyguanosine (BPDE-

101 Cyclobutane pyrimidine dimers (CPDs) are DNA photoproducts linked to skin cance

102 When molecular dimers, crystalline films or molecular aggregates absorb

103 omers upon GTP binding, leading to a monomer-dimer cycle during GTP hydrolysis.

104 D-dimer (DD) is highly sensitive for AAS but is inadequate

105 - and CSP-NMR-guided HADDOCK modeling of the dimer-dimer interface of the heterotetrameric complex ex

106 erminal extension that structurally mimics a dimer-dimer interface of these enzymes that are canonica

107 ientation of a flexible loop proximal to the dimer-dimer interface that is essential for catalysis (i

108 The highly thermally stable dimers dissociate on the sub-second timescale when subje

109 Nanoparticles comparable in size with IgG dimers do not permeate into it.

110 models that posit, at least in part, a SecA dimer-driven translocation mechanism.

111 High concentrations of D-dimer early after start of NMP can be considered a marke

112 i+4 positions at the center of the TM domain dimer eliminates the barrier and stabilizes the open con

113 ection in mice, those recognizing the DENV E-dimer epitope (EDE) can neutralize ZIKV infection in cel

114 st a segregation mechanism by which ParA-ATP dimers equilibrate to HDRs shown to be localized near ce

115 Moreover, the monomer-dimer equilibrium affinity constant was determined using

116 has been shown to exist in a dynamic monomer-dimer equilibrium modulated by translocation ligands, an

117 IV RNA is regulated independently of monomer:dimer equilibrium of the 5'UTR.

118 were used to generate a model of the low-pH dimer, establishing the presence of residue E73 at the i

119 change the global allosteric behavior of the dimer even when one subunit was wild type.

120 Here we show that the Tie2 ECR forms strong dimers even in the absence of bound ligand.

121 replication to take place, the core protein dimers, existing in several different quaternary structu

122 AR4 and P2Y12 specifically interact and form dimers expressed at the cell surface.

123 V) species, which are usually mu-oxo-bridged dimers (Fe(IV)Fe(IV)), and this allows for the reactivit

124 ests including ESR, CBC with differential, D-dimer, fibrinogen, C3, C4, IL-6, etc.

125 ferent oligomeric state (monomer compared to dimer for OCP1) and lower fluorescence quenching of the

126 characterize a natural propensity of the LBD dimers for various configurational states.

127 The amino termini of nine Sas-6 dimers form a central hub of approximately 12 nm radius

128 eins and carries the potential risk of mixed dimer formation giving rise to a new TCR with unpredicta

129 which was attributable to cysteine-mediated dimer formation.

130 nd long-range recognition mechanism for this dimer formation.

131 uptake, consistent with a role for alphaB in dimer formation.

132 ted stability compared with stability of the dimer formed by Abeta(14-23) hairpin.

133 opes bind with higher affinity to alpha/beta dimers formed by risk haplotypes, supporting molecular m

134 Dimers formed when crystals were grown in the presence o

135 Mixed dimers, formed by mispairing between the endogenous and

136 like dimer was the dominant if not exclusive dimer found in vivo, whether SecA was cytosolic or in li

137 talyzed by GTP hydrolysis, that converts the dimer from a "prefusion" to "postfusion" state.

138 nomer (sP-selectin) or as a disulfide-linked dimer fused to the Fc portion of mouse immunoglobulin G

139 ed, surface expression of HLA-E single-chain dimers (fused to B2M) or trimers (fused to B2M and a pep

140 TF ELISA, soluble P-selectin, d-dimer, FVIII, and C-reactive protein were assayed.

141 ach we immobilize sRecE to enable subsequent dimer generation.

142 conformational entropy within the trimer-of-dimers geometry.

143 igands, and multiple structural forms of the dimer have been crystallized.

144 ture reveals that YbAnbu subunits form tight dimers, held together in part by the Anbu specific C-ter

145 plex is scaffolded by a specific SMC protein dimer (heterodimer in eukaryotes) held together via thei

146 tion, the reaction of the NHC-iodosilicon(I) dimer [IAr (I)Si:]2 (IAr =:C{N(Ar)CH}2 ) with 4 equiv of

147 nal residues and the EH domain keep the EHD2 dimer in an autoinhibited conformation in solution.

148 the crystal structure, whereas BiFae1B is a dimer in solution based on size exclusion chromatography

149 the PCM subunits in the full-length protein dimer in solution differ significantly from that in the

150 electronegative cleft on the histone H2A-H2B dimer in the nucleosome.

151 SP and ASF1A are capable of folding an H3-H4 dimer in vitro under native conditions.

152 howed that leptospiral PerR assembles into a dimer in which a metal-induced conformational switch can

153 -10H12-18O4-9 monomers and C16-20H24-36O8-14 dimers in alpha-pinene derived secondary organic aerosol

154 mall-angle X-ray scattering studies of sTie2 dimers in solution and modeling based on crystal structu

155 ribe the use of human mAbs to stabilize homo-dimers in solution.

156 Conformational changes in the CT dimer induced by c-di-AMP binding may be the molecular m

157 nts on a doubly tagged tripeptide shows that dimer-induced fluorescence quenching is accompanied by a

158 The pan-RAF dimer inhibitor, LY3009120, could suppress CRAF-fusion o

159 dimerization while the other sequesters the dimer initiation signal preventing dimerization.

160 hat disrupts the BC-loop regions at the P450 dimer interface and results in a CYP126A1 monomeric crys

161 The purified protein was a dimer whose dimer interface involves interactions between the coiled

162 ate, but variation in B-cell epitopes at the dimer interface of DBP leads to induction of strain-limi

163 observation is that lithium ions bind to the dimer interface of GluK2/K5 heteromers and slow their de

164 4I) directly weaken inhibitor binding at the dimer interface of the catalytic core domain but at the

165 CLCNKB mutations in barttin-binding sites, dimer interface or selectivity filter often have severe

166 ease with allosteric inhibitors bound to the dimer interface site.

167 tent with binding of one molecule of 11m per dimer interface, contrary to most benzothiadiazine dioxi

168 This DBD-LBD interaction masked CAR's dimer interface, preventing CAR homodimer formation.

169 located at the surface and/or within the NS5 dimer interface, providing a functional significance to

170 hin or directly adjacent to CDR3 loop at the dimer interface, which remarkably include both destabili

171 Instead, TM2, TM6, and TM11 line the dimer interface.

172 ATPase, positions of the main domains, and a dimer interface.

173 PP2A/RACK1 binding site is buried within its dimer interface.

174 ) in the predicted, tightly packed TM domain dimer interface.

175 ant role in maintaining structure across the dimer interface.

176 immunity are conformational epitopes at the dimer interface.

177 t factors contribute to the stability of the dimer interfaces in the closed conformation and how clam

178 A catalytically competent palladacyclic dimer intermediate has been identified.

179 elices in the periplasmic region of the MotB dimer into a parallel coiled coil.

180 uggest that Ang1 binding may cross-link Tie2 dimers into higher-order oligomers, potentially explaini

181 The coiled-coil dimer is a widespread protein structural motif and, due

182 We propose that NDK5 dimer is an RS structural subunit with an additional mec

183 The assembly of the beta-ladder dimer is concentration dependent.

184 at of the tetramer but the C terminus of the dimer is more flexible.

185 tious virion assembly, ensuring that one RNA dimer is packaged into each nascent virion.

186 dicates that the helicase activity of a UvrD dimer is promoted via direct interactions between UvrD s

187 find that each active site of a transposase dimer is responsible for two hydrolysis and one transest

188 Each dimer is stabilized by interactions of one hydrophobic p

189 ransform in soils to aromatic amines and azo-dimers, it is seldom mineralized by indigenous soil bact

190 One family of regulators, namely Kite dimers (Kleisin interacting winged-helix tandem elements

191 e subunits (LSu) assembled into pentamers of dimers, (L2)5, and differs from Rubiscos from higher pla

192 ients with a Wells score </=4 and a normal d-dimer level or no d-dimer testing) (override group) and

193 with Wells score >4 or </=4 with elevated d-dimer level) (adherent group).

194 =3500 ng/mL), whereas significantly higher D-dimer levels (>3500 ng/mL) were in found in livers with

195 Thus, quantification of D-dimer levels serves an important role in guiding therapy

196 vels but normalization of TNF, sIL-6R, and D-dimer levels.

197 copy to probe singlet fission in a pentacene dimer linked by a non-conjugated spacer.

198 onjugate consisting of pyrrolobenzodiazepine dimers linked to a monoclonal antibody targeting CD33, w

199 copy, demonstrating that two copies of VirD4 dimers locate on both sides of the apparatus, in between

200 press CRAF-fusion oncogenicity by inhibiting dimer-mediated signaling.

201 s, raising the possibility that this unusual dimer might have a biologically relevant role in the reg

202 by London dispersion attractions between the dimer moieties.

203 electrostatic repulsion between NPs when the dimers move from interstitial fluid to cytosol.

204 onding to monosolvated monomers, trisolvated dimers, octasolvated monomers, and octasolvated dimers w

205 3 scFv covalently conjugated to a stabilized dimer of different antitumor Fabs.

206 zation of actin is induced by a constitutive dimer of MYO6+, indicating that multimerization of MYO6

207 loyed conditions, and is consistent with the dimer of the thiourea being the active catalyst.

208 s FADS (CaFADS) crystal structure predicts a dimer of trimers organization.

209 imental structure-based analysis of GASright dimers of different predicted stabilities, which show th

210 In solution, mass spectrometry shows dimers of G.

211 In the present work we examine three dimers of transcription factors in the NFkappaB family:

212 stair-step infinite chains whereby adjacent dimer-of-trimer units are noncovalently packed via two A

213 dge, and the presence of the histone H2A/H2B dimer on the entry side.

214 or to HJ cleavage: formation of a productive dimer on the HJ.

215 conformation distinct from the ATP sandwich dimer, optimized for DNA association.

216 activated either by self-assembly to form a dimer or by interaction with an accessory protein.

217 imately 60% of APJ molecules were present as dimers or oligomers.

218 -15 activates STAT5 proteins, which can form dimers or tetramers.

219 The identification of the tubulin dimer orientation and membrane-binding domain represents

220 that the oxygen atoms present in the Mn(IV) dimers originate from O2 .

221 meric compounds viz., triglyceride polymers, dimers, oxidized triglyceride monomers, diglycerides and

222 The specificity of coiled-coil dimer pairing is mainly based on hydrophobic and electro

223 s) that drive formation of cytokine receptor dimer pairings that are not formed by endogenous cytokin

224 inactive form of the tetramer, in which the dimer pairs are rotated by 18 degrees relative to each o

225 ucture of the p107 CTD bound to E2F5 and its dimer partner DP1 reveals the molecular basis for pocket

226 imal DVT alone, higher C-reactive protein, D-dimer, peak thrombin, lower Ks, shorter lag phase, decre

227 This dimer placed on a vibrating plate exhibits 3 types of mo

228 c glutamate receptors (mGluRs) are mandatory dimers playing important roles in regulating CNS functio

229 otal polar compounds (TPC), and triglyceride dimers-polymers (TGDP), among others) and e-nose based o

230 ting the importance of the Twist-family bHLH dimer pool in limb morphogenesis.

231 tigations [B98D/6-31+G(d,p)] reveal that the dimers prefer a slip-stacked geometry and feature elonga

232 were designed to individually stabilize the dimer-prone or monomer-prone conformations, validated in

233 These dimers ("protomers") further assemble into a low-rise le

234 energy maps of AMPA and kainate receptor ATD dimers provide a framework for the interpretation of obs

235 ver, values p approximately 12.7 obtained by dimer quantum simulations are preferred for the argon ga

236 The site-specifically bound PU.1 dimer resisted competition from nonspecific DNA and show

237 our recently reported Ir(IV,IV) mono-mu-oxo dimers results in the formation of fully characterized I

238 e, the numerical simulation reveals that the dimer's dynamics in either directed motion mode resemble

239 ls are beyond the thermodynamic reach of the dimer's effective reducing strength.

240 orporated them into our previously developed dimer-scale computational model of MT dynamics.

241 Unexpectedly, we found a coat protein dimer sequestered in the interior of the virion.

242 Numerous studies have shown that D-dimer serves as a valuable marker of activation of coagu

243 An ionic Pd(II)Br3 dimer side product was isolated, characterized, and iden

244 covered in the 1960s, and the best known PBD dimer, SJG-136 (also known as SG2000, NSC 694501 or BN26

245 etwork previously identified in the CD247 TM dimer solution NMR structure.

246 f approximately 12 nm radius from which nine dimer spokes radiate, placing the Sas-6 carboxyl termini

247 The Lamtor2:Lamtor3 dimer stacks upon Lamtor4:Lamtor5 to create a platform f

248 rium uptake of this polymerization-competent dimer strongly supports a beta4A-beta5A beta-hairpin run

249 nt neuronal network toxicity of Abeta, while dimers strongly suppress Abeta fibril formation.

250 insights that help to explain the unusual CC dimer structure and potential sources of stability in In

251 gnificance of packing effects; the gas-phase dimer structure at the same level shows a 1.634 A C-H...

252 lex that incorporates the high resolution CC dimer structure provides insights that help to explain t

253 lecules located in the conjunction region in dimer structures having interparticle gaps of 7 and 13 n

254 Upon deuteration, this dimer surprisingly yields bimodal isotope distributions

255 core </=4 and a normal d-dimer level or no d-dimer testing) (override group) and those in whom provid

256 oligomeric assemblies ranging from monomers, dimers, tetramers to higher order oligomers to generate

257 from folded monomers to folded antiparallel dimers, tetramers, and hexamers that unfold and polymeri

258 pTyr(705)-STAT3 forms a dimer that avidly binds a consensus target site in the p

259 r the [Co(II)2(bis-salophen)M2] (M = Li, Na) dimers that are present in solution in equilibrium with

260 x, revealing that both proteins form helical dimers that assemble into an unusual tetramer.

261 ers and promoter-proximal elements and forms dimers that facilitate the interaction of these DNA elem

262 P. patens UVR8 proteins form dimers that monomerise and accumulate in the nucleus fol

263 el interactions between fusion domains, into dimers that play a role at some early stage of the fusio

264 The tetramers consist of hydrogen-bonded dimers that sandwich together through hydrophobic intera

265 d: a [Formula: see text]-[Formula: see text] dimer, the [Formula: see text]-[Formula: see text] bond

266 But, for oligomers that are higher than dimer, these distance restraints all have two-fold direc

267 allosteric modulator of D2- and D3-receptor dimers, thus identifying the first allosteric small mole

268 the UbV selective for dimeric XIAP formed a dimer to stimulate E3 activity by stabilizing the closed

269 igomer model compounds (ranging in size from dimers to an octamer) with 5-5 and/or beta-O-4 linkages,

270 utamate receptor ligand-binding domain (LBD) dimers to characterize a natural propensity of the LBD d

271 delivery apparatus and joins 89 coat protein dimers to form a capsid.

272 This allows Kif15 dimers to productively and efficiently generate forces t

273 diminished ability of disulfide-bonded RRM2 dimers to refold and their increased propensity to misfo

274 including Nap1 and Chz1 to deliver H2A.Z-H2B dimers to SWR-C.

275 These complexes range from simple dimers to the respirasome supercomplex consisting of oxi

276 bility of the protein and potentially to the dimer-to-monomer transition.

277 hree synthesized S-lignin model compounds (a dimer, trimer, and tetramer) with beta-O-4 linkages, wer

278 ved from grape seed extract yielded pure OPC dimer, trimer, tetramer, and their gallates (pOPCs).

279 During heating, the increase of dimers, trimers, and oligomers was observed.

280 Remarkably, after the first hydrolysis, the dimer undergoes a flip in the asymmetry while remaining

281 ency and the amplitude of the vibration, the dimer undergoes either a directed motion toward the smal

282 binding domains (NBDs) form a "head-to-tail" dimer upon binding ATP; and the cytoplasmic pathway, fou

283 easurements show a stabilization of the HdeA dimer upon exposure to mildly acidic conditions.

284 eractions including the motion of a myosin-V dimer "walking" on an actin fibre, RNA stem-loop packing

285 Replication of a cyclobutane pyrimidine dimer was accurate, whereas replication of an abasic sit

286 observe it with direct methods, a functional dimer was identified as the active state by following th

287 The crystal structure of the EBD dimer was solved to 2.2 A resolution.

288 A discrete anti-parallel 1M6N-like dimer was the dominant if not exclusive dimer found in v

289 portance of a cellular population of tubulin dimers, we have incomplete information about the mechani

290 ers, octasolvated monomers, and octasolvated dimers were identified.

291 on during NMP, perfusate levels of ALT and D-dimers were low (</=3500 ng/mL), whereas significantly h

292 CYP126A1 dimers were observed in crystal structures of ligand-fre

293 While dimers were virtually unbreakable, dimerization of the m

294 Tpm forms coiled-coil dimers, which assemble into a helical strand that runs a

295 The purified protein was a dimer whose dimer interface involves interactions betwee

296 TMEM16A forms a dimer with two pores.

297 bital (SOMO) to form multiple pancake-bonded dimers with formal bond orders of up to five.

298 Using catalytic site mutants to create Rad50 dimers with only one functional ATPase site, we find tha

299 ned within the central channel of the 14-3-3 dimer, with both 14-3-3 binding motifs simultaneously pa

300 th toluene-d8 and, surprisingly, DCM-d2 into dimers, with significant stabilities, through the format

301 four motors can bind each alphabeta-tubulin dimer within the microtubule lattice.