ライフサイエンスコーパス: dimethylsulfate

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1 ne are clearly protected from methylation by dimethylsulfate.

2 triad was not protected from methylation by dimethylsulfate.

3 lation of the respective diamidoxime 5a with dimethylsulfate.

4 melting, circular dichroism spectroscopy and dimethylsulfate alkylation experiments.

5 expression of H19, we have conducted in vivo dimethylsulfate and DNase I footprinting of regions upst

6 ved the presence of maternal-allele-specific dimethylsulfate and DNase I footprints at the promoter i

7 dification of P RNA-substrate complexes with dimethylsulfate and kethoxal was performed to determine

8 lly reactive toward chemical modification by dimethylsulfate and that methylation of this nucleoside

9 DNA-protein complexes were probed with dimethylsulfate (DMS) and N -ethylnitrosourea (EtNU) to

10 Dimethylsulfate (DMS) methylation-protection footprintin

11 e adjacent A2451 to become hyper-reactive to dimethylsulfate (DMS) modification in a pH-independent m

12 ed with either ultraviolet (UV) radiation or dimethylsulfate (DMS).

13 r dichroism, nuclear magnetic resonance, and dimethylsulfate footprinting analyses.

14 In vivo dimethylsulfate footprinting confirmed that DNA-protein

15 alysis of this 5' flanking region by in vivo dimethylsulfate footprinting in cultured endothelial cel

16 Concordantly, dimethylsulfate footprinting in vivo revealed methylatio

17 We performed in vivo dimethylsulfate footprinting of the 220 bp mouse proxima

18 tic analyses of duplex and bubble templates, dimethylsulfate footprinting, and zero-Angstrom crosslin

19 we find that only in nonexpressing cells are dimethylsulfate footprints and UV photofootprints affect

20 Based on dimethylsulfate genomic footprinting experiments, there

21 howed that two distal kappa B sites, a novel dimethylsulfate-hypersensitive sequence, and a promoter

22 Ultraviolet light and dimethylsulfate in vivo genomic footprinting analyses re

23 The combination of dimethylsulfate, kethoxal, and 1-cyclohexyl-3-(2-morphol

24 e show that classic chemical probes, such as dimethylsulfate, kethoxal, and 1-cyclohexyl-3-(2-morphol

25 Using dimethylsulfate/LMPCR, no detectable proteins bind the T

26 In structural probing experiments with dimethylsulfate, m(1)A is routinely detected by RT-arres

27 acid constant) based on the pH dependence of dimethylsulfate modification.

28 int, we observed a number of strand-specific dimethylsulfate reactivity differences specific to the m

29 Dimethylsulfate reactivity of the dG residues indicates

30 ion of a triplex having the 1-C-G triad with dimethylsulfate resulted in a 50% reduction of methylati

31 Ribosome probing experiments with dimethylsulfate revealed that specific base accessibilit

32 er congener is protected from methylation by dimethylsulfate when the oligomer is 3'-phosphorylated.

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