ライフサイエンスコーパス: dimethylsulfoxide

1 d out by isothermal titration calorimeter in dimethylsulfoxide.

2 rom the framework under acidic conditions in dimethylsulfoxide.

3 thylacetamide approximately propionic acid < dimethylsulfoxide.

4 benign solvents such as water, formamide, or dimethylsulfoxide.

5 ogues (3, 18, 19, and 21) was carried out in dimethylsulfoxide.

6 ed with CLT compared with those treated with dimethylsulfoxide.

7 the antioxidant agents Desferal, Tempol, and dimethylsulfoxide.

8 ed air or were injected with the NNK solvent dimethylsulfoxide.

9 eal delivery of 50 mg/kg AG1296 or AG1478 in dimethylsulfoxide 1 hour before V2O5 instillation and ag

10 Vehicle (dimethylsulfoxide, 2.0 ml/kg, n=6) was administered at 3

11 Dimethylsulfoxide, a chemical scavenger for reactive oxy

12 The titration results in dimethylsulfoxide, acetonitrile, nitromethane, 1,2-dichl

13 incorporation and cell counts compared with dimethylsulfoxide alone.

14 he prototypical inducers of differentiation, dimethylsulfoxide and hexamethylene bisacetamide (HMBA),

15 Nuclear run-on analyses indicated that dimethylsulfoxide and HMBA enhanced transcriptional init

16 c acid or an acidic ion-exchange resin) with dimethylsulfoxide and/or poly(1-vinyl-2-pyrrolidinone) a

17 lar concentration of a cryoprotectant agent (dimethylsulfoxide), and the distribution of organic mate

18 s in the molecular solvents dichloromethane, dimethylsulfoxide, and methanol.

19 fiers (DMF, thiodiglycol, dimethylacetamide, dimethylsulfoxide, and N-methylpyrrolidone) can signific

20 The 3D NMR structures of the analogues in dimethylsulfoxide are consistent with the observed bindi

21 hylviologen as electron donor but it reduced dimethylsulfoxide at a very low rate.

22 we show superior preparations of antigens in dimethylsulfoxide, avoiding their rapid decomposition in

23 Compared with dimethylsulfoxide, bexarotene blocked angiotensin II-ind

24 highly denaturing PCR conditions (i.e., with dimethylsulfoxide), both low- and high-stability paralog

25 been measured in solution (acetonitrile and dimethylsulfoxide) by using the EPR radical equilibratio

26 with our studies designed to understand why dimethylsulfoxide causes terminal differentiation of the

27 hloride, dimethylformamide, tetrahydrofuran, dimethylsulfoxide, chloroform, and hexamethylphosphorami

28 ing (1 degrees C/min); and 2), intracellular dimethylsulfoxide concentration is lower (by as much as

29 pid content, cholesterol content, and buffer dimethylsulfoxide concentration.

30 ring solution ionic strength in concert with dimethylsulfoxide content is sufficient to disrupt the p

31 methyl sulfoxides, dibenzyl sulfoxides, and dimethylsulfoxide could be utilized to generate diaryl s

32 udy, we showed that hepatocytes in long-term dimethylsulfoxide culture have definite advantages over

33 HMBA and dimethylsulfoxide did not change the acetylation, phosph

34 oxidase caused recognition and ingestion of dimethylsulfoxide-differentiated HL-60 cells by J774A.1

35 We used dimethylsulfoxide-differentiated HL-60 cells showing ind

36 reduction of trimethylamine oxide (torA) and dimethylsulfoxide (dmsA).

37 Culturing pluripotent stem cells in dimethylsulfoxide (DMSO) activates the retinoblastoma pr

38 material at room temperature are described: dimethylsulfoxide (DMSO) and tetrabutylammonium fluoride

39 l the oxo transfer reaction coordinate using dimethylsulfoxide (DMSO) as a substrate.

40 er systems were performed in the presence of dimethylsulfoxide (DMSO) at 2, 5, 10, and 100 mol % DMSO

41 emically-defined medium supplemented with 2% dimethylsulfoxide (DMSO) can be maintained in a well-dif

42 eviously shown that hepatocytes in long-term dimethylsulfoxide (DMSO) culture, fed a chemically defin

43 Forty canines were divided into dimethylsulfoxide (DMSO) group and 10 mM, 15 mM, and 20

44 Dimethylsulfoxide (DMSO) is used to cryopreserve hematop

45 f these reactions reveal the key role of the dimethylsulfoxide (DMSO) ligand in controlling this chem

46 e of this study was to analyze the effect of dimethylsulfoxide (DMSO) on skin flap viability.

47 ), washed, and cultured (3 days) with either dimethylsulfoxide (DMSO) or hexamethylene bisacetamide (

48 Using the microperfusion chamber, water and dimethylsulfoxide (DMSO) permeability coefficients of mo

49 The molybdoenzyme dimethylsulfoxide (DMSO) reductase contributes to the re

50 form of the cofactor that is required by the dimethylsulfoxide (DMSO) reductase family of enzymes, wh

51 Microbial oxidation of DMS to dimethylsulfoxide (DMSO) represents a major sink of DMS

52 ons on dipalmitoylphosphatidylcholine (DPPC)/dimethylsulfoxide (DMSO) system that has the same lipid:

53 s: the control group (group 1) received only dimethylsulfoxide (DMSO) which is the solvent for CMX-13

54 olic acidosis and liver and kidney toxicity; dimethylsulfoxide (DMSO), a free radical-scavenging solv

55 ulation, we tested the effects of capsaicin, dimethylsulfoxide (DMSO), and morphine on withdrawal lat

56 ia (MEL) cells induced to differentiate with dimethylsulfoxide (DMSO), and p300 and Tal1 were observe

57 tities of certain organic chemicals, such as dimethylsulfoxide (DMSO), betaine, polyethylene glycol a

58 er mobility than the backbone labels, and in dimethylsulfoxide (DMSO), the backbone end labels were s

59 amine N-oxide (TMAO) and the organic solvent dimethylsulfoxide (DMSO), which we refer to as 'chemical

60 In this report, we analyze GJIC in a dimethylsulfoxide (DMSO)-containing long-term primary mo

61 The solvent dimethylsulfoxide (DMSO)-d6 containing 1-ethyl-3-methyli

62 to biologically-driven conversion of DMS to dimethylsulfoxide (DMSO).

63 ng peptide during hydrogelation in different dimethylsulfoxide (DMSO)/H(2)O solutions.

64 fatigue trial; (2) a group given intravenous dimethylsulfoxide (DMSO, 0.5 ml/kg of a 50% solution) be

65 were treated with sophorolipids or vehicle (dimethylsulfoxide [DMSO]/physiologic saline) by intraven

66 Deferoxamine, alpha-tocopherol, and dimethylsulfoxide each inhibited hyperglycemia-induced f

67 urine erythroleukemia (MEL) cells induced by dimethylsulfoxide, expression of TfR1 increased, whereas

68 t with antioxidants, N-ACETYL-L-CYSTEINE, or dimethylsulfoxide, failed to attenuate plasma endotoxin

69 xarotene or 9-cis retinoic acid) or vehicle (dimethylsulfoxide) for 24 hours before stimulation with

70 Dimethylsulfoxide has been shown to decrease necrosis of

71 on, whereas a scavenger of hydroxyl radical (dimethylsulfoxide), hydrogen peroxide (catalase), and su

72 The action of HMBA and dimethylsulfoxide in the transient transfection system r

73 y the hydroxyl radical scavengers, including dimethylsulfoxide, mannitol, and 5,5-dimethyl-1-pyrrolin

74 The findings imply that HMBA and dimethylsulfoxide modulate transcription by a mechanism

75 ion (mDP) and different solvents (ethanol or dimethylsulfoxide)] on this interaction by fluorescence

76 in organic solvents, such as isopropanol and dimethylsulfoxide or by enzymatic cleavage of the pyroph

77 In a Sla1(-) background, [PSI] curing by dimethylsulfoxide or excess Hsp104 is increased, while t

78 t of ischemia; vehicle-treated rats received dimethylsulfoxide or saline.

79 Treatment with dimethylsulfoxide or sodium butyrate induces a rapid hep

80 oups were studied: phosphate-buffered saline/dimethylsulfoxide (PBS/DMSO) (vehicles), PBS/ANTU, and K

81 Dimethylsulfoxide reductase (DMSOR) and sulfite oxidase

82 Enzymes belonging to the dimethylsulfoxide reductase (DMSOR) family of pyranopter

83 The bis-molybdopterin enzyme dimethylsulfoxide reductase (DMSOR) from Rhodobacter cap

84 ductase (PcrAB), a specialized member of the dimethylsulfoxide reductase superfamily, catalyzes the f

85 tein-bound sites of DMSOR and TMAOR (DMSOR = dimethylsulfoxide reductase, TMAOR = trimethylamine N-ox

86 Neither treatment with TNF-alpha alone or dimethylsulfoxide removal alone induced apoptosis.

87 ly in hepatocytes that had been subjected to dimethylsulfoxide removal.

88 eference electrophiles) have been studied in dimethylsulfoxide solution by UV-vis spectroscopy.

89 of these peptides were determined by NMR in dimethylsulfoxide solution.

90 mation of POBN-CH3 adduct in the presence of dimethylsulfoxide, suggesting the production of hydroxyl

91 ed from 20 to 72 wt% with the addition of 2% dimethylsulfoxide to distilled water used as a medium.

92 enes, we discovered the capacity of HMBA and dimethylsulfoxide to enhance the expression of transient

93 upplemented with epidermal growth factor and dimethylsulfoxide to maintain rat hepatocytes in a highl

94 s showed a similar tolerance to methanol and dimethylsulfoxide up to 50% in assay buffer.

95 Elevated urinary excretion of dimethylsulfoxide was associated with MTO deficiency.

96 eutic window: STAZN (0.6 mg/kg) dissolved in dimethylsulfoxide was given intra-peritoneally at 2 and

97 Experiments were performed in a fluid dimethylsulfoxide/water cryosolvent system at 240 K, und

98 rom Salmonella typhimurium at 234-248 K in a dimethylsulfoxide/water cryosolvent system.

99 ron generation, an organogel was obtained in dimethylsulfoxide, while nanorods and micellar aggregate

100 hed with mixtures of aqueous formic acid and dimethylsulfoxide with increasing concentrations.