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1  were sensitised on the irradiated site with dinitrochlorobenzene.
2 ey were exposed to a sensitizing dose of 2,4-dinitrochlorobenzene.
3  sunscreen, 48 h prior to sensitization with dinitrochlorobenzene.
4 od of individuals previously sensitized with dinitrochlorobenzene.
5 mmune T cells from individuals sensitized to dinitrochlorobenzene.
6 stimulating factor and then derivatized with dinitrochlorobenzene.
7 asuring the strength of sensitization to 2,4-dinitrochlorobenzene after UVR exposure, and to diphenyl
8 jects were challenged with four doses of 2,4-dinitrochlorobenzene and four doses of diphenylcycloprop
9 eoxy-Delta-((12,14))-prostaglandin J(2), 2,4-dinitrochlorobenzene, and iodoacetamide] correlates with
10 crease in skin fold thickness of five graded dinitrochlorobenzene challenge sites on the arm, 2 wk af
11 terleukin-4, when restimulated in vitro with dinitrochlorobenzene-derivatized fresh peripheral blood
12                                We found that dinitrochlorobenzene-derivatized fresh peripheral blood
13  by investigating why similar chemicals, 2,4-dinitrochlorobenzene (DNCB) and 2,4-dinitrothiocyanobenz
14                                          2,4-Dinitrochlorobenzene (DNCB) is widely used in human clin
15 sis fungoides and Sezary syndrome) using 2,4-dinitrochlorobenzene (DNCB) skin testing as part of the
16 xperimental contact sensitization model with dinitrochlorobenzene (DNCB) to gain insight into the uni
17 igated the T-cell response to the hapten 2,4-dinitrochlorobenzene (DNCB) which can sensitize all immu
18 e sensitized by cutaneous application of 2,4-dinitrochlorobenzene (DNCB), a small, highly lipophilic
19 ion phase of contact hypersensitivity to 2,4-dinitrochlorobenzene (DNCB), in ten PLE patients and 11
20 VA)-induced allergic airway inflammation and dinitrochlorobenzene (DNCB)-induced CHS models.
21 nd 1% ABT-281 ointments profoundly inhibited dinitrochlorobenzene-induced contact hypersensitivity in
22                                          2,4-Dinitrochlorobenzene is a potent immunogen capable of in
23 clude that induction of sensitization by 2,4-dinitrochlorobenzene is suppressed less by UVR in patien
24 hapten-specific T cells were stimulated with dinitrochlorobenzene-modified, cultured autologous perip
25          The strength of the reaction to 2,4-dinitrochlorobenzene relative to the reaction to dipheny
26 senting the reactivity of the subject to 2,4-dinitrochlorobenzene (Sigma DN) and diphenylcyclopropeno
27 in the fluoride displacement reaction of 2,4-dinitrochlorobenzene using fluorous solid-phase extracti
28  Contact hypersensitivity responses (CHS) to dinitrochlorobenzene were quantified after challenge usi

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