ライフサイエンスコーパス: dioxygenation

1 ative distinction of CH3-group oxidation and dioxygenation.

2 actone from the ring fission product of 5NSA dioxygenation.

3 itrosyl hemoglobin rather than destroy it by dioxygenation.

4 ccurs most effectively via O(2)-dependent NO dioxygenation.

5 m in which peroxidative cleavage precedes AA dioxygenation.

6 were kinetically competent to participate in dioxygenation.

7 tive cleavage is not the initiating event in dioxygenation.

8 m in which peroxidative cleavage precedes AA dioxygenation.

9 urement of oxidase and myeloperoxidase (MPO) dioxygenation activities in whole blood.

10 g the molecular mechanisms involved in thiol dioxygenation and sets the stage for exploration of the

11 anism by which HbN recycles itself during NO dioxygenation and the reductase that participates in thi

12 mes are remarkable in that they catalyze two dioxygenations and two cyclizations of the native substr

13 n experiment and theory showed that rates of dioxygenation are determined by the enzymatic O2 activat

14 he oxidation of fluoranthene is initiated by dioxygenation at the C-1,2, C-2,3, and C-7,8 positions.

15 isomers, 2,6-dinitrotoluene, and naphthalene dioxygenation by NBDO varied considerably, the correlati

16 Because aromatic dioxygenation by nonheme iron dioxygenases is frequently

17 netically competent to participate in the 15-dioxygenation event.

18 or biodegradation pathways initiated via (i) dioxygenation, (ii) reduction, and (iii) CH3-group oxida

19 ysis of OPP's inhibition of arachidonic acid dioxygenation indicated mixed inhibition toward the ferr

20 The major site of dioxygenation is the C-2,3 dioxygenation route, which co

21 Thiol dioxygenation is the initial oxidation step that commits

22 and CO binding kinetics, and steady-state NO dioxygenation kinetics.

23 om(-1) s(-1) indicate an extremely efficient dioxygenation mechanism.

24 utes a novel approach to the challenging 1,6-dioxygenation motif.

25 oboglobin models to promote the nitric oxide dioxygenation (NOD) reaction similar to oxy-hemes.

26 Since it is unlikely that 11- and 15-dioxygenation occurs via different mechanisms, our findi

27 poxygenase-1 (15-hLO-1), which catalyzes the dioxygenation of 1,4-cis,cis-pentadiene-containing polyu

28 We investigated the dioxygenation of a series of nitroaromatic compounds to

29 Two in two: Dioxygenation of alkenyl boronic acids has been achieved

30 Dioxygenation of aromatic rings is frequently the initia

31 -formation reaction, while also preventing a dioxygenation of its cysteinate substrate?

32 orresponding isotope effects, especially for dioxygenation of N-substituted, aromatic contaminants, a

33 The dioxygenation of nitric oxide by oxyheme in globin prote

34 rtmentalization of hemoglobin and subsequent dioxygenation of nitric oxide may explain the vascular c

35 Our study illustrates that dioxygenation of nitroaromatic contaminants exhibits a l

36 The corresponding (13)C-KIEs for the dioxygenation of nitrobenzene and 2-nitrotoluene were 1.

37 Dioxygenation of nitrobenzene to catechol and 2-nitrotol

38 periment and theory also agreed well for the dioxygenation of nitrobenzene, which was associated with

39 os for the competing CH3-group oxidation and dioxygenation of nitrotoluenes by MnO4(-) were obtained

40 ansport and uptake processes and larger than dioxygenation of other aromatic hydrocarbons.

41 15-lipoxygenase (15-LO) participates in the dioxygenation of polyenoic fatty acids.

42 ant and mammalian lipoxygenases catalyze the dioxygenation of polyunsaturated fatty acids, which cont

43 iquitous family of enzymes that catalyze the dioxygenation of polyunsaturated fatty acids.

44 LOXs) catalyze the regio- and stereospecific dioxygenation of polyunsaturated membrane-embedded fatty

45 alpha,beta-unsaturated lactone derived from dioxygenation of pyrene at an apical ring, 2H-naphtho[2,

46 ae alkaloids, all based on the key enzymatic dioxygenation of suitable aromatic precursors.

47 Similar observations were made for the dioxygenation of these substrates by 2NTDO.

48 Here, we systematically investigated the dioxygenation of two nitroaromatic compounds (nitrobenze

49 ed in the plastid envelope and catalyzed the dioxygenation of unsaturated membrane fatty acids, leadi

50 19Z-hexaenoic acid (isomer IV), and a double dioxygenation product 10S,17S-dihydroxy-docosa-4Z,7Z,11E

51 observed consistently in the substrates and dioxygenation products.

52 Haptoglobin does not change NO dioxygenation rates of Hb; rather, the large size of the

53 The structure indicates that the dioxygenation reaction is initiated by a direct attack o

54 olysis occurs secondarily to the accelerated dioxygenation reaction of plasma oxyhemoglobin with endo

55 is inactivated by cell-free hemoglobin in a dioxygenation reaction that also oxidizes hemoglobin to

56 Moreover, HMS catalyzes a very similar dioxygenation reaction to that of HPPD, adding the secon

57 ially met-R state Hb that arises from the NO dioxygenation reaction.

58 Individual reduction, ligand binding, and NO dioxygenation reactions were examined at 20 degrees C, w

59 he endothelium, via accelerated nitric oxide dioxygenation reactions with free plasma hemoglobin.

60 The major site of dioxygenation is the C-2,3 dioxygenation route, which consists of 18 enzymatic step

61 The C-1,2 and C-2,3 dioxygenation routes degrade fluoranthene via fluorene-t

62 te, thereby preventing the possible thiolate dioxygenation side reaction.

63 s the rate law for 3,5-di-tert-butylcatechol dioxygenation when one begins with Pierpont's [VO(DBSQ)(

64 86-89% of 2,4-DNT transformation was due to dioxygenation while TNT was mostly reduced and 2,6-DNT r

65 sotope effects for aniline and diphenylamine dioxygenation with those from abiotic oxidation by manga

66 a framework to explain cofactor-independent dioxygenation within a protein architecture generally em