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1  or the NADPH oxidase inhibitors apocynin or diphenylene iodonium.
2 uction was prevented by N-acetylcysteine and diphenylene iodonium.
3 acetylcysteine and a flavoprotein inhibitor, diphenylene iodonium.
4 ocarbamate, and the NADPH oxidase inhibitor, diphenylene iodonium.
5 but was inhibited by the flavoenzyme blocker diphenylene iodonium.
6 NO), and by a nonspecific oxidase inhibitor, diphenylene iodonium.
7  alone, 5.0+/-1.1-fold; NAC, 1.8+/-0.2-fold; diphenylene iodonium, 1.4+/-0.3-fold migration; and ebse
8 th ebselen (3.6 +/- 0.2 versus 1.1 +/- 0.2), diphenylene iodonium (3.6 +/- 0.2 versus 1.0 +/- 0.1), a
9 with a farnesyltransferase inhibitor or with diphenylene iodonium, a flavoprotein inhibitor.
10 was effectively blocked by pretreatment with diphenylene iodonium, a NADPH oxidase inhibitor.
11 ss, ERL-induced cytotoxicity was reversed by diphenylene iodonium, a NOX complex inhibitor.
12 ial cells with the NAD(P)H oxidase inhibitor diphenylene iodonium abolishes RSV-induced STAT activati
13      Scavenging reactive oxygen species with diphenylene iodonium also blocks both priming and ERK ph
14                                 Furthermore, diphenylene iodonium, an inhibitor of flavin-containing
15  (ROS) dependent because it was inhibited by diphenylene iodonium, an inhibitor of NADPH oxidase, and
16 and was inhibited by 1-methylcyclopropene or diphenylene iodonium, an inhibitor of NADPH oxidase.
17 s H2O2 activates only p38MAPK (14-fold), and diphenylene iodonium, an NADH/NADPH oxidase inhibitor, a
18 ects were attenuated by N-acetylcysteine and diphenylene iodonium, an NADPH oxidase inhibitor.
19 as suppressed by the NADPH oxidase inhibitor diphenylene iodonium and a DUOX2-specific small interfer
20 vious studies with NADPH oxidase inhibitors, diphenylene iodonium and apocynin, suggested that NADPH
21 hils treated with the flavoprotein inhibitor diphenylene iodonium and in neutrophils from patients wi
22 gnificantly retarded in the presence of both diphenylene iodonium and MnTMPyP.
23 trast, inhibitors of NADPH oxidase activity, diphenylene iodonium and staurosporine, as well as antio
24                          The NADPH inhibitor diphenylene iodonium and the superoxide dismutase mimeti
25          Inhibitors of Rac1 (NSC23766), NOX (diphenylene iodonium), and NOX1 (small interfering RNA [
26  inhibited by the NAD(P)H oxidase inhibitor, diphenylene iodonium, as well as by overexpression of do
27 t ascorbate, or the ROS-generation inhibitor diphenylene iodonium attenuated gravitropism while enhan
28       Antioxidants and the oxidase inhibitor diphenylene iodonium blocked ERK phosphorylation, but sp
29 ocalized near nectary pores and inhibited by diphenylene iodonium but not by cyanide or azide, sugges
30 nhibitor of flavoprotein-dependent oxidases, diphenylene iodonium, but not by inhibitors of various o
31 re treated with the NADPH oxidase inhibitors diphenylene iodonium chloride and apocynin.
32                  The NADPH oxidase inhibitor diphenylene iodonium chloride did not affect the calcium
33  Cu) and inhibitors of NADPH oxidases (e.g., diphenylene iodonium chloride), enzymes responsible for
34 s, and in wild-type plants by treatment with diphenylene iodonium chloride, an inhibitor of hydrogen
35 endent production of hydrogen peroxide via a diphenylene iodonium chloride-sensitive calcium-dependen
36 ion was inhibited by NADPH oxidase inhibitor diphenylene iodonium (DPI) and mitochondria electron cha
37 s via quenching by the flavoenzyme inhibitor diphenylene iodonium (DPI) and oxidized glutathione, the
38                  Addition of the antioxidant diphenylene iodonium (DPI) diminished DMXAA-induced IFN-
39                              The presence of diphenylene iodonium (DPI) inhibited ROS production by U
40 cking the activity of the NADPH oxidase with diphenylene iodonium (DPI) inhibits ROS formation and ph
41 cells could be attenuated in the presence of diphenylene iodonium (DPI), an inhibitor of the NADPH ox
42  inhibited by an inhibitor of NADPH oxidase, diphenylene iodonium (DPI), and by antioxidants, N-acety
43 ed by NADPH oxidase inhibitors, apocynin and diphenylene iodonium (DPI), but not the xanthine/xanthin
44 as inhibited by the NADPH oxidase inhibitors diphenylene iodonium (DPI), imidazole, and pyridine.
45 gene expression was inhibited by antioxidant diphenylene iodonium (DPI), which is consistent with the
46 nd Tiron (5 mmol/L) and the flavin-inhibitor diphenylene iodonium (DPI, 20 micromol/L) abolished nore
47 nd mouse HSCs by the NADPH oxidase inhibitor diphenylene-iodonium (DPI) and in HSCs lacking the NADPH
48 en, catalase, and the flavoprotein inhibitor diphenylene iodonium each attenuated PDGF- and Rac1-medi
49 dies and inhibition experiments using CO and diphenylene iodonium indicate that only the low spin hem
50 wever, the antioxidants N-acetylcysteine and diphenylene iodonium inhibited both AA- and Ang II-induc
51                      Treatment of cells with diphenylene iodonium inhibited virus-induced oxidative s
52  an Arabidopsis thaliana azide-sensitive but diphenylene iodonium-insensitive apoplastic oxidative bu
53 etylcysteine or the flavin protein inhibitor diphenylene iodonium is demonstrated to reduce oxidant l
54 tion in LPS-stimulated HMs were inhibited by diphenylene iodonium (nonspecific inhibitor for flavin-c
55 e, which generates superoxide and H2O2, with diphenylene iodonium or apocynin decreased Ang II-induce
56 reatment of rat VSMCs with the NOX inhibitor diphenylene iodonium or the antioxidants N-acetylcystein
57 eine, the flavin-containing enzyme inhibitor diphenylene iodonium, or the glutathione peroxidase mime
58 njury correlated with the acute induction of diphenylene iodonium-sensitive NADPH-dependent superoxid
59 n (IL)-1beta stimulation of SMCs resulted in diphenylene iodonium-sensitive ROS production within int
60         In addition, NADPH oxidase inhibitor diphenylene iodonium significantly inhibited the TDCA-in
61 NADPH oxidase function through the inhibitor diphenylene iodonium, the superoxide dismutase mimetic M
62  two inhibitors of ROS generation, azide and diphenylene iodonium, we found that perxoxidases generat
63 as abrogated by N-acetyl cysteine but not by diphenylene iodonium, which displayed prooxidant activit