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1 or the NADPH oxidase inhibitors apocynin or diphenylene iodonium.
2 but was inhibited by the flavoenzyme blocker diphenylene iodonium.
3 uction was prevented by N-acetylcysteine and diphenylene iodonium.
4 acetylcysteine and a flavoprotein inhibitor, diphenylene iodonium.
5 ocarbamate, and the NADPH oxidase inhibitor, diphenylene iodonium.
6 NO), and by a nonspecific oxidase inhibitor, diphenylene iodonium.
7 alone, 5.0+/-1.1-fold; NAC, 1.8+/-0.2-fold; diphenylene iodonium, 1.4+/-0.3-fold migration; and ebse
8 th ebselen (3.6 +/- 0.2 versus 1.1 +/- 0.2), diphenylene iodonium (3.6 +/- 0.2 versus 1.0 +/- 0.1), a
12 ial cells with the NAD(P)H oxidase inhibitor diphenylene iodonium abolishes RSV-induced STAT activati
15 (ROS) dependent because it was inhibited by diphenylene iodonium, an inhibitor of NADPH oxidase, and
16 and was inhibited by 1-methylcyclopropene or diphenylene iodonium, an inhibitor of NADPH oxidase.
17 s H2O2 activates only p38MAPK (14-fold), and diphenylene iodonium, an NADH/NADPH oxidase inhibitor, a
19 vious studies with NADPH oxidase inhibitors, diphenylene iodonium and apocynin, suggested that NADPH
20 hils treated with the flavoprotein inhibitor diphenylene iodonium and in neutrophils from patients wi
22 trast, inhibitors of NADPH oxidase activity, diphenylene iodonium and staurosporine, as well as antio
25 inhibited by the NAD(P)H oxidase inhibitor, diphenylene iodonium, as well as by overexpression of do
26 t ascorbate, or the ROS-generation inhibitor diphenylene iodonium attenuated gravitropism while enhan
28 ocalized near nectary pores and inhibited by diphenylene iodonium but not by cyanide or azide, sugges
29 nhibitor of flavoprotein-dependent oxidases, diphenylene iodonium, but not by inhibitors of various o
32 Cu) and inhibitors of NADPH oxidases (e.g., diphenylene iodonium chloride), enzymes responsible for
33 s, and in wild-type plants by treatment with diphenylene iodonium chloride, an inhibitor of hydrogen
34 endent production of hydrogen peroxide via a diphenylene iodonium chloride-sensitive calcium-dependen
35 ion was inhibited by NADPH oxidase inhibitor diphenylene iodonium (DPI) and mitochondria electron cha
38 cking the activity of the NADPH oxidase with diphenylene iodonium (DPI) inhibits ROS formation and ph
39 cells could be attenuated in the presence of diphenylene iodonium (DPI), an inhibitor of the NADPH ox
40 inhibited by an inhibitor of NADPH oxidase, diphenylene iodonium (DPI), and by antioxidants, N-acety
41 ed by NADPH oxidase inhibitors, apocynin and diphenylene iodonium (DPI), but not the xanthine/xanthin
42 as inhibited by the NADPH oxidase inhibitors diphenylene iodonium (DPI), imidazole, and pyridine.
43 gene expression was inhibited by antioxidant diphenylene iodonium (DPI), which is consistent with the
44 nd Tiron (5 mmol/L) and the flavin-inhibitor diphenylene iodonium (DPI, 20 micromol/L) abolished nore
45 nd mouse HSCs by the NADPH oxidase inhibitor diphenylene-iodonium (DPI) and in HSCs lacking the NADPH
46 en, catalase, and the flavoprotein inhibitor diphenylene iodonium each attenuated PDGF- and Rac1-medi
47 dies and inhibition experiments using CO and diphenylene iodonium indicate that only the low spin hem
48 wever, the antioxidants N-acetylcysteine and diphenylene iodonium inhibited both AA- and Ang II-induc
50 an Arabidopsis thaliana azide-sensitive but diphenylene iodonium-insensitive apoplastic oxidative bu
51 etylcysteine or the flavin protein inhibitor diphenylene iodonium is demonstrated to reduce oxidant l
52 tion in LPS-stimulated HMs were inhibited by diphenylene iodonium (nonspecific inhibitor for flavin-c
53 e, which generates superoxide and H2O2, with diphenylene iodonium or apocynin decreased Ang II-induce
54 reatment of rat VSMCs with the NOX inhibitor diphenylene iodonium or the antioxidants N-acetylcystein
55 eine, the flavin-containing enzyme inhibitor diphenylene iodonium, or the glutathione peroxidase mime
56 njury correlated with the acute induction of diphenylene iodonium-sensitive NADPH-dependent superoxid
57 n (IL)-1beta stimulation of SMCs resulted in diphenylene iodonium-sensitive ROS production within int
59 NADPH oxidase function through the inhibitor diphenylene iodonium, the superoxide dismutase mimetic M
60 two inhibitors of ROS generation, azide and diphenylene iodonium, we found that perxoxidases generat
61 as abrogated by N-acetyl cysteine but not by diphenylene iodonium, which displayed prooxidant activit
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