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1 egistration (central-peripheral rivalry-type diplopia).
2 56 patients were excluded for other types of diplopia.
3 table suture was performed to treat vertical diplopia.
4 ure was defined as worsening or no change in diplopia.
5 ltered consciousness followed by symptomatic diplopia.
6 c correction in adults based on the cause of diplopia.
7 a translocated inferiorly, causing binocular diplopia.
8 The patient also complained of diplopia.
9 potential cause of strabismus and binocular diplopia.
10 reased risk for postoperative strabismus and diplopia.
11 disruption of central fusion, and monocular diplopia.
12 report of fusion, until the subject reported diplopia.
13 strabismus, ophthalmoplegia, and paradoxical diplopia.
14 tal septum intact can minimize postoperative diplopia.
15 nics to determine the prevalence of CPR-type diplopia.
16 tion when other causes did not fully explain diplopia.
17 etermine clinical associations with CPR-type diplopia.
18 e clinical findings associated with CPR-type diplopia.
19 M, 25 had symptomatic diplopia and 25 had no diplopia.
20 CI, 38.6%-77.7%) of ED visits, primarily for diplopia.
21 Frequency, type, and cause of diplopia.
22 plopia vs patients with ERM without CPR-type diplopia.
23 an epiretinal membrane (ERM) and presenting diplopia.
24 and where other causes did not fully explain diplopia.
25 mpleted the Diplopia Questionnaire to assess diplopia.
26 CPR-type diplopia and those without CPR-type diplopia.
27 were primarily for acute- or subacute-onset diplopia.
28 st to characterize the type and cause of the diplopia.
29 necessary but is not sufficient for CPR-type diplopia.
30 may explain subjective reports of monocular diplopia.
31 ta) at distance and a phoria at near without diplopia.
32 %) had CPR-type diplopia and 37 (66%) had no diplopia.
33 event long-term sequelae of enophthalmos and diplopia.
34 /12.5-20/50), and 34 patients (81%) reported diplopia.
35 ared to have central-peripheral rivalry-type diplopia), 1 (4%) optical/refractive error (monocular di
36 ad a relatively high incidence of persistent diplopia (12%) and corneal edema (20%), although half of
37 , 1 (4%) optical/refractive error (monocular diplopia), 2 (8%) mixed retinal misregistration (central
38 order of binocular vision (strabismus, 2.3%; diplopia, 2.2%; amblyopia, 0.9%; and nystagmus, 0.2%).
40 sole cause (central-peripheral rivalry-type diplopia), 7 (28%) strabismus (1 of 7 initally appeared
42 onsidered in any patient with vision loss or diplopia accompanied by neurologic symptoms and in the a
51 egistration (central-peripheral rivalry-type diplopia) and strabismus, and for 4 (16%) diplopia cause
55 of binocular vision (strabismus, amblyopia, diplopia, and nystagmus) may have on musculoskeletal inj
57 BFM-90 reduced the recurrence of strabismus, diplopia, and proptosis, but did not correct deficits in
59 c symptoms and signs such as vision loss and diplopia are common in patients with stroke, patients ar
61 symptoms (eg, isolated vertigo, dysarthria, diplopia) are not consistently classified as transient i
63 We defined central-peripheral rivalry-type diplopia as presenting symptomatic diplopia with evidenc
64 noptophore; CPR-type diplopia was defined as diplopia associated with evidence of retinal misregistra
66 0.05 to 1.1, P = .01) than patients without diplopia, but similar aniseikonia (Awaya new aniseikonia
67 e metamorphopsia than those without CPR-type diplopia, but there is considerable individual variabili
68 fy the efficacy of monovision correction for diplopia by measuring the functional impact on vision-sp
73 the highest success rate was for motor plus diplopia criteria (67%) and the lowest success rate was
74 defining success as either meeting motor and diplopia criteria or showing improvement in HRQOL beyond
78 opters by simultaneous prism cover test; (2) diplopia criteria, none or only rare in primary distance
79 18%) were classified as failure by motor and diplopia criteria, with 39 of 40 able to exceed Adult St
81 type of strabismus associated with binocular diplopia due to glaucoma surgery was hypertropia (10/11
83 alysis with prominent bulbar palsies such as diplopia, dysarthria, dysphonia, and dysphagia that woul
84 frequency): hemiparesis, vertigo/dizziness, diplopia, dysarthria, nystagmus, nausea/vomiting, head p
86 tal reconstruction in an effort to alleviate diplopia, enophthalmos, orbital dystopia, and extraocula
89 icle is to report the incidence and cause of diplopia following cataract surgery and laser in-situ ke
93 of dichoptic training with the computer game Diplopia Game (Vivid Vision) run in the Oculus Rift OC D
95 nd orbital blowout fractures associated with diplopia had the lowest satisfaction rates, 55 and 8%, r
99 s with binocular vision will be sensitive to diplopia in any gaze direction; in such cases, the conse
100 To report the prevalence, type, and cause of diplopia in medically and surgically treated patients wi
101 d this suggests a mechanism that can explain diplopia in patients made exotropic after surgery for es
103 omes were as follows: prevalence of CPR-type diplopia in patients with ERM seen in retinal disease cl
105 Ground-in or Fresnel prism may alleviate diplopia in relatively small vertical deviation; however
106 To determine the prevalence of CPR-type diplopia in retinal disease clinic patients with ERM and
107 e most common diagnosis in both settings was diplopia (International Classification of Diseases, Nint
109 d for LASIK procedures, the leading cause of diplopia is decompensation of pre-existing strabismus.
111 in visual field defects, visual acuity, and diplopia is typically observed after emergent applicatio
113 S) as the cause of chronic or acute acquired diplopia may avert neurologic evaluation and imaging in
117 ing diplopia may have 1 of several causes of diplopia, most commonly retinal misregistration (central
118 elevated intraocular pressure, tube erosion, diplopia, motility disturbances, and corneal decompensat
122 ient or persistent monocular ghost images or diplopia occurred in 10 of 178 eyes (5.6%), sometimes re
125 ia occurred in 11.2% of patients, persistent diplopia occurred in only 1.9% of patients and was treat
127 of TED, including soft tissue inflammation, diplopia, ocular motility restriction, and appearance.
128 female, three male) presented with episodic diplopia or facial paresthesias with subsequent brainste
130 All patients had preoperative monocular diplopia or unstable vision attributable to the subluxat
134 ophthalmoplegia and nystagmus, resulting in diplopia, oscillopsia, blurred visual, loss of stereopsi
136 (7 of 31; 95% CI, 10% to 41%), with CPR-type diplopia present in 16% (5 of 31; 95% CI, 5% to 34%).
140 ated right retro-orbital pain and later with diplopia, ptosis, 6th nerve and pupil-sparing partial 3r
142 as diplopia rated "never" or "rarely" on the diplopia questionnaire for reading and straight-ahead di
143 tionnaire (100-0, best to worst HRQOL) and a diplopia questionnaire in a clinical practice before pri
144 n of ascertaining patient symptoms using the Diplopia Questionnaire may be useful in these patients.
146 tically significant 58.6% improvement in the Diplopia Questionnaire score in our patients (P < .0001)
148 primary outcome: Based on the results of the Diplopia Questionnaire, 85% of patients experienced sign
153 ) ambulatory and 49790 (95% CI, 38318-61262) diplopia-related ED visits occurred annually; 12.3% of a
154 ambulatory setting, but approximately 16% of diplopia-related ED visits resulted in a stroke or trans
160 lides at 5 and 10 degrees), and cause of any diplopia (retinal misregistration vs strabismus vs optic
168 ival chemosis, pain on eye movement, minimal diplopia, the usual absence of proptosis, and general pr
169 evalence of monocular diplopia and binocular diplopia unrelated to glaucoma surgery was similar among
171 perceived visual quality and ocular injury, diplopia, visual performance, and blast exposure charact
172 fered between patients with ERM and CPR-type diplopia vs patients with ERM without CPR-type diplopia.
177 totype-frame test and synoptophore; CPR-type diplopia was defined as diplopia associated with evidenc
181 eoperative visual acuity better than 20/400, diplopia was reported at the first postoperative visit b
183 visits in the United States by patients with diplopia were analyzed in this prespecified secondary an
188 However, 10 of 12 patients never experienced diplopia when the nonfoveal face was presented to tempor
189 alry-type diplopia as presenting symptomatic diplopia with evidence of retinal misregistration, and w
190 weeks is recommended in cases of symptomatic diplopia with positive forced ductions and evidence of o
192 acture thought to be in need of repair, with diplopia within 30 degrees of primary gaze, and/or enoph
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