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1 t they are the only cells that progress into diplotene.
2 ed to mouse meiotic chromosomes at pachytene/diplotene.
3 at the E18.5, when most oocytes are entering diplotene.
4 ytes, while staining intensity diminished in diplotene and meiotically dividing spermatocytes, the ce
5 ower percentages at later stages (pachytene, diplotene and metaphase I) providing evidence that delet
6 9-1 basidia enter the diffuse stage of early diplotene, and then 50% of these cells enter metaphase I
8 scripts are stripped away, the oval shape of diplotene bivalents between chiasmata, and the rigidity
9 mpanied by increased cell death in pachytene/diplotene cells with markedly elevated levels of phospho
11 ath during early prophase; oocytes reach the diplotene/dictyate stage in nearly normal numbers, but m
13 NH3-Ser50 phosphorylation begins in prophase/diplotene, increases to a maximum at prometaphase-metaph
14 terochromatin, which normally occurs in late diplotene, is reduced in spermatocytes from heterozygous
15 pachytene SCs, as compared to more condensed diplotene-metaphase I bivalents, makes mapping crossover
18 lin A1 in the pericentromeric region in late diplotene of meiosis, perhaps in assembly or function of
21 ually reproducing animals, oocytes arrest at diplotene or diakinesis and resume meiosis (meiotic matu
24 th spermatogenic arrest predominately at the diplotene premeiotic stage and almost no sperm detected
27 granules are prominent in late pachytene and diplotene spermatocytes, and in elongating spermatids.
28 combination nodule, was delayed in Cul4a -/- diplotene spermatocytes, which potentially led to subseq
29 re localized to the XY body in pachytene and diplotene spermatocytes, while only SUMO2/3 and UBE2I we
32 nesis was arrested around the late pachytene-diplotene stages of prophase I; surprisingly, without an
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