ライフサイエンスコーパス: disc disease

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1 avenue for treating advanced intervertebral disc disease.

2 D3 may serve a critical role in degenerative disc disease.

3 toration of cell numbers during degenerative disc disease.

4 ADAMTS-5 protein expression with severity of disc disease.

5 loping biological therapies for degenerative disc disease.

6 ge of a new form of therapy for degenerative disc disease.

7 that may be important in the pathogenesis of disc disease.

8 ed expression of NOTCH genes in degenerative disc disease.

9 To correlate these observations with disc disease, a condition characterized by tissue vascul

10 e studies show an association between lumbar disc disease and mutations of genes encoding the alpha-2

11 er inflammatory conditions that characterize disc disease are not yet known.

12 s a key role in pathogenesis of degenerative disc disease by promoting aggrecan degradation by ADAMTS

13 Treatments for degenerative disc diseases continue to be a challenge.

14 enetic mutations in disorders such as lumbar disc disease, further investigation of the interaction b

15 udy 10-year follow-up who did not have optic disc disease, including glaucoma, were included.

16 Low back pain associated with degenerative disc disease is optimally treated with distraction of th

17 reed (PBonferroni = 0.01) and intervertebral disc disease (IVDD) across breeds (PBonferroni = 4.0 x 1

18 a positive association between SN and lumbar disc disease (LDD).

19 ing IL-1beta and TNF-alpha activities during disc disease may restore NOTCH signaling and nucleus pul

20 g this transcription factor for treatment of disc disease must spare osmoprotective, prosurvival, and

21 ere procured from patients with degenerative disc disease (n = 25) or herniated IVDs (n = 12); nondeg

22 Degenerative disc disease often causes severe low-back pain, a public

23 of the Th17 lymphocyte lineage in mediating disc disease remains uninvestigated.

24 lternative for the treatment of degenerative disc disease that is more logistically convenient than t

25 provide potential therapies for degenerative disc disease, which is the major cause of lower back pai

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