1 Next, we
discuss how (
18)F-FDG PET studies have advanced understa
2 ds that could be used for authentication and
discuss how a combination of methods could be used in a
3 ght recent advances in our understanding and
discuss how a consideration of the interfaces between th
4 icrobiota and the immune system, and we will
discuss how a dietary-induced disruption of the intestin
5 Here, we
discuss how a family of protein kinases that phosphoryla
6 Finally, we
discuss how a greater knowledge of the genetics of droug
7 We
discuss how a novel methodology that iteratively combine
8 In this Review, we
discuss how a recent increase in observational data for
9 We
discuss how aberrant epigenetic pathways identified in b
10 ural circuits implicated in the disorder and
discuss how abnormalities in circuitry relate to symptom
11 processes of learning and memory, the review
discusses how aging affects the perception and integrati
12 We
discuss how amyloid materials exemplify the emergence of
13 We
discuss how and why relevant neuropsychological studies
14 We
discuss how associated genetic variants can lead to unde
15 classification is considered and the authors
discuss how atrial cardiomyopathic properties might guid
16 We also
discuss how autophagy contributes to differentiation of
17 We also
discuss how bacterial pathogens can alter host gene expr
18 seases, describe the challenges involved and
discuss how basic developmental studies have contributed
19 We also
discuss how biomarkers targeting these axes can be used
20 f obesity with cardiovascular disease (CVD),
discussing how both the degree and the duration of obesi
21 This review will
discuss how CD4 T cell responses directed against an exo
22 In this Minireview, we will
discuss how cells adapt to mitochondrial stress through
23 e on the biology of scleractinian corals and
discuss how cellular processes of the host and symbionts
24 We then
discuss how central circuits integrate and transform mec
25 Finally, we will
discuss how changes in the expression of regulation of C
26 In addition, it
discusses how claims that statins commonly cause adverse
27 We also
discuss how CLASH differs from other recently developed
28 In this review we
discuss how commensal or pathogenic properties of organi
29 tomy of explore/exploit decision making, and
discuss how computational psychiatry can benefit from fo
30 We
discuss how conflicting foraging rules could explain why
31 Lastly, we
discuss how conformational changes under the presence of
32 underlying pathophysiological mechanisms, we
discuss how congenital disorders of autophagy inform our
33 hods for measuring costs in health care, and
discusses how cost management strategies can either impr
34 This Perspective
discusses how current physiological data could potential
35 Further, we
discuss how DC functions empowered by specific delivery
36 Here we
discuss how decision-making organizes experiences accord
37 In this review, we
discuss how DENV modulates the innate immune DNA-sensing
38 In this review we
discuss how derangements in these developmental pathways
39 pithelial transport and barrier function, we
discuss how diarrhea can result from a decrease in lumin
40 Here we
discuss how differences in susceptibility to infection r
41 We also
discuss how different nitrophenol absorption profiles al
42 In this minireview, we
discuss how discovery of the role of the mammalian cytos
43 We also
discuss how disruption of these PTP regulatory mechanism
44 systems approach to theory of mind (ToM) and
discuss how drawing upon perspectives from bilingualism
45 Here, we
discuss how drug delivery and therapeutic efficacy are g
46 etic interplay of astrocytes and neurons and
discuss how dysregulation of these pathways may contribu
47 In this review, we
discuss how each of these aspects may contribute to the
48 Here, we
discuss how each parasite species adapts to this tissue
49 Here, we
discuss how early learning experiences are beyond the co
50 ross species with different brain sizes, and
discuss how early regional specification of progenitor c
51 We will also
discuss how early-life events can influence the subseque
52 We also
discuss how eIF2 phosphorylation contributes to the main
53 We then
discuss how engineered immune cells are being designed t
54 We
discuss how evolutionary birth-death processes can provi
55 Here we
discuss how EVs released by cancer or stromal cells impa
56 We
discuss how exploiting these differences and taking adva
57 c roles of EGLN enzymes and HIF isoforms and
discuss how feedback loops based on recently identified
58 Furthermore, we
discuss how FGLK motifs are required for efficient precu
59 tic control of mating in C. albicans We also
discuss how fitness advantages could have driven the evo
60 herapeutic landmark for SMA therapeutics and
discuss how future developments will need to address the
61 investigating various types of cannabis and
discuss how future research can help to better understan
62 We further
discuss how future research might solve the remaining op
63 n this review, we summarize new evidence and
discuss how future studies may address the role of neura
64 Finally, we
discuss how GABABR activation of mTORC1 helps resolve ke
65 Finally, we
discuss how gaining a deeper understanding of GxEs may t
66 Then, we
discuss how genome editing techniques enable a radically
67 In this review, we
discuss how genome-wide association studies (GWASs) and
68 I also
discuss how genomic and functional approaches such as BH
69 We first
discuss how,
given plants' high levels of morphological
70 We
discuss how growing understanding of key anatomic sanctu
71 We
discuss how host specialization can enable aphids to co-
72 Finally, we
discuss how human noninvasive neuroimaging can benefit f
73 We then
discuss how,
if and when organismal responses (acclimate
74 ells in the intestine, airways, and skin and
discuss how immune communications with hematopoietic cel
75 Herein, we
discuss how implementation of NIH guidelines will help i
76 ped by the aging T cell system fail and also
discuss how,
in some settings, the programs associated w
77 We
discuss how incorporating biological knowledge from mode
78 We also
discuss how increasing our understanding of intestinal m
79 Using this model, we
discuss how individual or group variations in parental i
80 In this Perspective, Jose Florez
discusses how information from genetics and genomics may
81 izes key components of NASH pathogenesis and
discusses how inherent and acquired variations in regula
82 Here we
discuss how integrating physical approaches and engineer
83 e goal-directedness of voluntary action, and
discuss how internal generation of action can be linked
84 We outline each ethical issue and
discuss how it can be conceptualized and managed so that
85 approach toward treating these data, and we
discuss how it can be fruitfully employed to infer physi
86 We
discuss how LINEs and SINEs have expanded in eukaryotic
87 Here, we
discuss how lipid oxidation creates important chemical a
88 We will also
discuss how mechanistic analysis of these pathways has l
89 We
discuss how meta-atoms can be assembled into unique plas
90 We
discuss how metabolic workload can modulate immunologica
91 Symposium 'Metabolism in Time and Space' to
discuss how metabolism influences cellular and developme
92 In this Tutorial Review, we
discuss how metal oxides with designed defects can be sy
93 We
discuss how MFNs could be applied to diverse migratory t
94 Finally, we
discuss how microtubule-based engineered systems can ser
95 Lastly, we
discuss how miRNAs may challenge and extend current theo
96 In particular, we
discuss how mitochondria contribute to specific aspects
97 Finally, we
discuss how modern detection and diagnostic technologies
98 es that have reached clinical evaluation and
discuss how molecular vaccine approaches can make AIT mo
99 Here, we
discuss how multiplex assays of variant effect (MAVEs) c
100 Overall, we present and
discuss how mycorrhizal fungi can affect the feeding beh
101 In this review we
discuss how native antigen is presented to B cells and i
102 We
discuss how ncRNAs inhibit spurious recombination among
103 In this review, we
discuss how networks can be used to help understand pati
104 We
discuss how networks regulating apoptotic cell clearance
105 Giza et al.,
discuss how neuroscience can provide balance between phy
106 In this Update, we
discuss how new advancements in genomic and genetic tool
107 Furthermore, we
discuss how new genetic study designs are starting to ex
108 tudies of somatic and germline variants, and
discuss how non-coding variants can be interpreted on a
109 We
discuss how nutritional and current therapeutic approach
110 Here, however, we
discuss how object and spatial information are in fact c
111 We also
discuss how oncogenes and tumour suppressors promote nut
112 We
discuss how optical mapping can be used as a validation
113 Finally, we
discuss how optogenetic functional magnetic resonance im
114 In this Review, we
discuss how organoids have been used to identify and cha
115 We cover these topics herein and
discuss how other properties of the lymphatic vasculatur
116 analyses of real transmission histories and
discuss how our findings should aid in interpreting phyl
117 ent 4 patient cases from our institution and
discuss how our management reflects what we have learned
118 We
discuss how our methods can be used for the rapid determ
119 riming, consider alternative approaches, and
discuss how our specific account can be extended to new
120 In this Review, we
discuss how over a century of study of the readily cultu
121 In addition, we
discuss how patient selection from differing phases of H
122 ch phagocytes sense apoptotic cell death and
discuss how phagocytosis is integrated with environmenta
123 Here, we
discuss how phase separation, in which a component of on
124 r data sets from The Cancer Genome Atlas and
discuss how predictions from these algorithms can be int
125 We also
discuss how progress in human genetics has led to the ge
126 We also
discuss how progress in stem cell biology and cellular r
127 In this review, we
discuss how prominent stratification theories might be e
128 erience at NeuroMorpho.Org as an example, we
discuss how publicly available repositories may benefit
129 We
discuss how quantum computers can augment classical comp
130 ew the epigenetic basis of human disease and
discuss how recent discoveries in this field could be tr
131 In this review, we
discuss how recent efforts delineating rewired metabolic
132 We also
discuss how recent insights into the pathogenesis of ALD
133 e review proposed roles for facilitation and
discuss how recent progress in uncovering the underlying
134 riments performed in UHV environments and to
discuss how recent reports will guide future endeavors.
135 e node to subcircuit to large-scale GRNs and
discuss how regulatory design features such as network a
136 We
discuss how relational values differ from moral values a
137 We
discuss how repeat proteins may differ due to not just u
138 We
discuss how ritual serves these social functions.
139 noma, and pancreatic and ovarian cancer, and
discuss how scientific advances may help overcome these
140 ntext of constantly growing genomic data, we
discuss how screening strategies must be improved when a
141 This Minireview
discusses how selected combinations of the MS, MS(2) , L
142 G or CWA/CAW motifs (where W is A or T), and
discuss how self-reinforcing feedback loops between DNA
143 We
discuss how sensory inputs may be combined with self-mot
144 tory inputs in healthy cortical circuits and
discuss how shifts in excitation/inhibition balance may
145 We also
discuss how single-molecule approaches have increased ou
146 eometric analysis of M2nLn complexes, and we
discuss how size and geometry of the ligand is expected
147 We
discuss how small-molecule inhibitors of the tryptophan
148 We
discuss how some of these genetic alterations in brain t
149 derlying host defense in the very young, and
discuss how specific developmental demands increase the
150 In this review, we
discuss how studies are beginning to address the nature
151 Here we
discuss how studies of the human T cell response to micr
152 I
discuss how successful (or not) we have been in addressi
153 Finally, we
discuss how such approaches are providing important insi
154 We
discuss how such findings from the neural field can prov
155 We also
discuss how such intricate measurements of spatial and t
156 these particles within biological media, and
discuss how surface chemistry presentation may affect th
157 We conclude by
discussing how systems biology approaches are a fruitful
158 We also review and
discuss how targeting TFEB and lysosomes may offer innov
159 In connection with this, we also
discuss how technical advances facilitate a new roadmap
160 Here we
discuss how technology can be applied effectively to bet
161 Finally, we will also
discuss how the advances in cancer genomics and cancer m
162 cancer disparities in the United States and
discuss how the analysis of tumor biology can advance he
163 We
discuss how the autophagy machinery controls the burden
164 Herein we
discuss how the band edge positions in zeolitic imidazol
165 We
discuss how the biochemical and mechanical properties of
166 In this Primer, we
discuss how the brain monitors the water content of the
167 We
discuss how the CDW symmetry may be related to the '1/8-
168 We
discuss how the chemical features of the high-energy pho
169 We
discuss how the combined AEP-AOP construct helps to maxi
170 We
discuss how the concept of "random" differences is conte
171 In this review, we
discuss how the concept of CSCs has been defined, what a
172 We
discuss how the conditions required for the identificati
173 We
discuss how the counter-regulatory and tolerogenic funct
174 e summarize recent progress in the field and
discuss how the emerging knowledge in Arabidopsis may be
175 In this Review, we
discuss how the experience and data generated from that
176 At last we
discuss how the formation of magnetite-based organic-ino
177 In this Review, we
discuss how the genomics of non-human organisms can prov
178 Herein, we
discuss how the hematopoietic GATA factors (GATA-1-3) fu
179 More importantly, we
discuss how the higher resolution structures now attaina
180 le link between malaria and hypertension and
discuss how the hypothesis could be confirmed or refuted
181 We
discuss how the ideas proposed in the target article cou
182 We then
discuss how the involvement of multiple cell types, each
183 We
discuss how the issue of polymorphism needs to be incorp
184 From this perspective, we will also
discuss how the knowledge discovered from FA research se
185 We
discuss how the lability of the species that make up the
186 In addition, we
discuss how the life history of facultative pathogens li
187 Thus, we
discuss how the long channel does not hinder catalysis u
188 In particular, we will
discuss how the low feasibility of conducting an adequat
189 Finally, we
discuss how the model provides a means to establish natu
190 Conversely, we
discuss how the molecular similarities of DLBCL and FL h
191 Here, we
discuss how the naive state is inherently linked to prei
192 In this Review, we
discuss how the nanoscale protein organization in the ki
193 ry, review recent findings in this area, and
discuss how the pattern of connectivity between striatal
194 ssemblies generated by various pipelines and
discuss how the platform associated data characteristics
195 In this Feature, we
discuss how the properties of paper determine the perfor
196 ert on mammalian developmental processes and
discuss how the somatic activity of TEs can influence ge
197 We
discuss how the speed changes we observe can lead to ste
198 We
discuss how the sperm interacts with the female reproduc
199 We
discuss how the statistical analysis can guide further d
200 Here, we
discuss how the subunit structure, stoichiometry, and au
201 This article will also
discuss how the technique has been employed in cutaneous
202 Here we
discuss how the tumor and its microenvironment influence
203 Based on this study, we
discuss how the unique features of trypanosomatid riboso
204 he entry into sporulation in B. subtilis and
discuss how the use of regulated cell death pathways dur
205 linking hyperkalemia with poor outcomes and
discusses how the efficacy of certain treatments might d
206 This Perspective
discusses how the US Food and Drug Administration (FDA),
207 I
discuss how their approach can be used to help determine
208 ated to interrogate the role of DCs and will
discuss how their use has progressively clarified our un
209 this pedagogical review and perspective, we
discuss how thermodynamics determines both the overall p
210 iagnostic and therapeutic aspects of AD, and
discuss how these achievements may improve patient progn
211 sp60, interact with folding proteins, and we
discuss how these chaperones may guide the folding proce
212 Here I
discuss how these complexes are recruited across the yea
213 Here, we
discuss how these CRISPR-Cas inhibitors were discovered
214 uish coding from noncoding transcription and
discuss how these differences might have important impli
215 Here I
discuss how these discoveries, reported in Cell Host & M
216 he control of focal adhesion remodeling, and
discuss how these emerging interconnections between inte
217 We
discuss how these exciton and carrier dynamics are contr
218 eview evidence from different organisms, and
discuss how these experiments have broadened our mechani
219 We
discuss how these experiments provide a new understandin
220 Finally, we will
discuss how these findings can be applied to improve the
221 I
discuss how these findings have important implications f
222 We
discuss how these findings may explain the inconsistent
223 Finally, we
discuss how these improvements may help direct future HM
224 abolites interact with the immune system and
discuss how these interactions could impact acute GVHD.
225 connections between CheD, CheX, and MCPs and
discuss how these interactions play critical roles durin
226 We
discuss how these marked differences in the biology of v
227 erlie effective lung immunity in health, and
discuss how these may be affected by external environmen
228 or reshaping the pancreatic tumor stroma and
discuss how these might improve patient outcomes.
229 gnition by Rad51/RecA family members, and we
discuss how these models and our results may relate to t
230 We
discuss how these modern psychometric techniques may als
231 We
discuss how these physiological results have motivated t
232 hways regulating dryland N2 O emissions, and
discuss how these processes will respond to emerging glo
233 simple model organisms, mice, and humans we
discuss how these proteins and pathways may potentially
234 We
discuss how these results fit with previous studies that
235 We
discuss how these results may have broad implications fo
236 We
discuss how these signaling networks might be integrated
237 We
discuss how these single-cell tools have uncovered novel
238 Additionally, we
discuss how these systems have been applied in therapeut
239 In particular, we
discuss how these systems work together and provide evid
240 n the ongoing single filament revolution and
discuss how these techniques have greatly contributed to
241 We also
discuss how these three basic elements of cognitive map
242 We
discuss how these tools could be used to design the next
243 We
discuss how these transporters may enhance or restrict d
244 We
discuss how these two properties of the echo signatures
245 ies in dental MSC function and behaviour and
discusses how these and other advances are paving the wa
246 ds on the actin cytoskeleton and conclude by
discussing how these results relate to existing mechanic
247 anscriptional and epigenetic cell states, we
discuss how they are applied to investigate immune cells
248 range of bioprinted neural tissue models and
discuss how they can be used to observe how neurons beha
249 in the diagnostic and discovery process and
discuss how they can inform (and misinform) expert revie
250 at integrates several of these responses and
discuss how they collectively help to preserve cellular,
251 rticle, we review these recent developments,
discuss how they have helped to illuminate PP-InsP struc
252 Here, I describe recent advances and
discuss how they redefine our view of the way in which v
253 Here, we highlight these recent findings and
discuss how they set the stage to interrogate developmen
254 nes, the guideline is reviewed and 2 experts
discuss how they would apply it to a 57-year-old man con
255 ew the data on which the guideline is based,
discuss how they would balance the benefits and harms of
256 Here, an oncologist and an internist
discuss how they would balance these recommendations and
257 We review recent studies of mouse thalamus,
discussing how they revealed general principles that may
258 We
discuss how this approach could be developed and applied
259 We
discuss how this disorder tuned SIT differs from the com
260 Here we
discuss how this emphasis, and the corresponding lack of
261 We also further
discuss how this flood may be related to ancient account
262 We
discuss how this functionality will be applicable to sch
263 We then
discuss how this information can be leveraged to improve
264 to terminally differentiated myofibers, and
discuss how this knowledge has been applied to different
265 We
discuss how this limits their efficacy, uptake and the p
266 We also
discuss how this link could potentially open up some use
267 We
discuss how this method may establish a link between pho
268 We also
discuss how this methodology is currently being used for
269 We
discuss how this model has revealed unique insights into
270 We
discuss how this multiscale design approach can be used
271 We
discuss how this new understanding could improve the the
272 In this context, we
discuss how this plasticity, i.e. the capacity to adapt
273 noids from purified cellular components, and
discuss how this technology can help to address fundamen
274 ner of a bistable toggle switch, and then we
discuss how this toggle switch accounts for the abrupt a
275 We
discuss how this unique property of increasing cell dama
276 Lastly, we
discuss how this work may eventually lead to tangible th
277 r apicomplexans and related organisms, while
discussing how this can be exploited for therapeutic int
278 that can identify such neural codes, and we
discuss how to combine intersection-based analysis of ne
279 We
discuss how to communicate the potential benefit derived
280 In addition, we
discuss how to conduct a power analysis for mediation mo
281 slational Medicine by Flesher et al. (2016),
discuss how to design such a system given current techno
282 Using 5 case vignettes, we
discuss how to prevent and manage the most common nonhem
283 ewed at full resolution in different planes,
discuss how to search the database for a phenotype, and
284 we report on the outcomes of a workshop that
discussed how to integrate structural data from a range
285 viral evasion of the host immune system, and
discusses how to harness the immune system effectively a
286 summarise the current available evidence and
discusses how to integrate scientific knowledge into cli
287 Finally, we
discuss how TP53 alterations should be described by usin
288 We also
discuss how transcription-associated DNA damage might pr
289 Here, we
discuss how transcriptional control is disrupted by gene
290 We also
discuss how TSCM cell stemness could be leveraged therap
291 We will
discuss how tumor microenvironment-driven transient comp
292 In this perspective we
discuss how understanding the cancer epigenome is provid
293 lignment" and "protocol misattribution," and
discuss how understanding these concepts might help impr
294 We
discuss how utilizing bioengineering approaches to manip
295 ontext of neuropsychiatric disorders, and we
discuss how we can gain insight from studies in Drosophi
296 proposals to achieve quantum supremacy, and
discuss how we can reliably compare the power of a class
297 Here I
discuss how we discovered that integrins control mammary
298 We will conclude by
discussing how we can leverage the findings on molecular
299 We
discuss how well-managed marine reserves may help marine
300 of different MLL-FP mouse model systems and
discusses how well they have recapitulated aspects of th