ライフサイエンスコーパス: disease resistance

1 responsiveness and broad-spectrum bacterial disease resistance.

2 roles of HCT1806 and HCT4918 in Rp1-mediated disease resistance.

3 inhibition of H2O2 degradation and enhanced disease resistance.

4 in expression did not compensate for loss of disease resistance.

5 nes may be enriched in functions involved in disease resistance.

6 o essential for sugars and glycerol-mediated disease resistance.

7 rly life and with implications for metabolic disease resistance.

8 (Hordeum vulgare) MORCs also are involved in disease resistance.

9 logous repair function may be a mechanism of disease resistance.

10 xity of AML biology and of the mechanisms of disease resistance.

11 sitive and eliminate the negative aspects of disease resistance.

12 carcass and meat quality, reproduction, and disease resistance.

13 bition of host signaling networks, restoring disease resistance.

14 echanisms underlying MAPK functions in plant disease resistance.

15 25 is required for maintaining XA21-mediated disease resistance.

16 ogen-induced salicylic acid accumulation and disease resistance.

17 volved in antioxidation, detoxification, and disease resistance.

18 sion of salicylic acid (SA) accumulation and disease resistance.

19 iggering of the hypersensitive response from disease resistance.

20 n the appearance of a unique plant phenotype disease resistance.

21 velopment, responses to abiotic stresses and disease resistance.

22 , therefore, provide a link between GNOM and disease resistance.

23 and to a lesser extent IL-17, contribute to disease resistance.

24 uggesting a function of the protein in basal disease resistance.

25 e presence of a regulatory cascade affecting disease resistance.

26 y (ETI) has been implicated in race-specific disease resistance.

27 defense-related gene expression and enhanced disease resistance.

28 eceptors to recognize pathogens and initiate disease resistance.

29 susceptibility to vaccine therapy, and human disease resistance.

30 ther signaling molecules in regulating plant disease resistance.

31 nes previously shown to be involved in plant disease resistance.

32 enes that underpin quantitative variation in disease resistance.

33 l responses appear to play a crucial role in disease resistance.

34 nteractions, with elevated levels increasing disease resistance.

35 eeding programs aiming to improve growth and disease resistance.

36 t have been linked with stress tolerance and disease resistance.

37 phenotypic variance contributing to whirling disease resistance.

38 ant for generating vascular sap that confers disease resistance.

39 coinciding with the kinetics of DCA-mediated disease resistance.

40 that will vary in heritable traits, such as disease resistance.

41 compromised salicylic acid accumulation and disease resistance.

42 ng terminal differentiation, senescence, and disease resistance.

43 igase, HISTONE MONOUBIQUITINATION1 (HUB1) in disease resistance.

44 for mutations that suppressed edr1-mediated disease resistance.

45 ifying specific genes implicated in whirling disease resistance.

46 drug development and genetic improvement of disease resistance.

47 reduced in NOD-Idd22 mice, correlating with disease resistance.

48 growth has resulted in temperature-resilient disease resistance.

49 genome editing technologies for engineering disease resistance.

50 nsporters play a role in sugar signaling for disease resistance.

51 tissues and may therefore be involved in the disease resistance.

52 ene silencing in pathogenic fungi and confer disease resistance.

53 necdotally linked to enhanced broad-spectrum disease resistance.

54 rm interfering TALEs, or iTALEs) to overcome disease resistance.

55 prediction models and genomic selection for disease resistance.

56 of BAK1 leads to enhanced virulence, but not disease resistance.

57 rograms trying to incorporate broad-spectrum disease resistance.

58 e thought to have an important role in plant disease resistance.

59 rs autophagy in the host that coincides with disease resistance.

60 Nonrace specific disease resistance 1 (NDR1) is a conserved downstream re

61 ransfer DNA insertion lines exhibit enhanced disease resistance after inoculation with virulent Pseud

62 ptor kinase-VI.2 (LecRK-VI.2) contributes to disease resistance against the hemibiotrophic Pseudomona

63 SmD3b, or PRMT5 function results in enhanced disease resistance against the virulent oomycete pathoge

64 ust have the complete package of high yield, disease resistance, agronomic performance, and end-use q

65 ed in the crop, particularly for transfer of disease resistance alleles.

66 naturally occurring performance-enhancing or disease- resistance alleles, including alteration of sin

67 4 containing disease-resistance alleles that decrease the pathogenic

68 ve pressure driving the local persistence of disease resistance among its wildlife hosts in endemic a

69 of RING E3 ligases that contribute to plant disease resistance and abiotic stress tolerance through

70 oligodendrocyte glycoprotein (MOG) promotes disease resistance and CD4(+) T cell deletion within the

71 ere positively correlated with the degree of disease resistance and defense gene expression.

72 expected critical regulatory role of eATP in disease resistance and defensive signalling.

73 rsity, with demonstrated benefits for colony disease resistance and division of labor.

74 Constitutive disease resistance and elevated levels of salicylic acid

75 r and fruit pigmentation, fruit astringency, disease resistance and forage quality.

76 sedimentation and promotes oyster survival, disease resistance and growth, in contrast to low-relief

77 s well understood as qualitative (monogenic) disease resistance and has not been used as extensively

78 strong effects on plant growth, development, disease resistance and heat tolerance.

79 impact of cruciferous phytoalexins on plant disease resistance and human health.

80 tion mutants wrky22-1 and wrky22-2 had lower disease resistance and lower induction of innate immunit

81 We identified several genes involved in disease resistance and pathogen defense.

82 ties can affect key agronomic traits such as disease resistance and plant growth.

83 ock-defense cross talk could help to improve disease resistance and productivity in economically impo

84 n an explosion of information regarding both disease resistance and susceptibility to pathogens.

85 -presenting cells (APCs) correlated with the disease resistance and T-cell-type response.

86 has focused on the molecular basis of plant disease resistance and the role of secreted effector pro

87 rn more about the role of NK cells in cattle disease resistance and vaccination.

88 phological and physiological traits, such as disease resistance and yield that need to be maintained

89 roduction of floral structures, induction of disease resistance, and adaptation to stress.

90 otentially beneficial for plant improvement, disease resistance, and biotechnological advances, such

91 istics, flowering time, response to gravity, disease resistance, and ethylene production.

92 ecking, stronger defense response, increased disease resistance, and increased pest resistance.

93 scope of implementing genomics selection for disease resistance, and specifically for quantitative re

94 s in plant growth regulation, morphogenesis, disease resistance, and stress responses.

95 ed to analyse how the characteristics of the disease resistance, and the method of deployment, affect

96 ggest that breeding for traits involved with disease resistance, and tolerance to cold- and heat-indu

97 requires genetic improvement of persistency, disease resistance, and tolerance to grazing.

98 ty may be functionally important in terms of disease-resistance, and that Bd prevalence and/or host s

99 fficient breeding process in which trials of disease resistance are integrated with other traits.

100 capture all the relevant genetic variance in disease resistance, as genetic analysis currently is not

101 In vivo disease resistance assays, using ZmTps21 and Zmtps21 nea

102 levels of XA21 and compromised XA21-mediated disease resistance at the adult stage.

103 0, which was critical for maintenance of the disease resistance because its neutralization with an IL

104 begin development of durable (long-lasting) disease resistance beyond the limits imposed by conventi

105 e altered under diet-restriction to increase disease resistance, but our findings suggest that increa

106 cesses including endoreduplication and plant disease resistance, but the molecular mechanism underlyi

107 ion patterns correlate with high stress- and disease-resistance, but proximate mechanisms for this tr

108 unctions for NRT2.1 that may influence plant disease resistance by down-regulating biotic stress defe

109 Gaining disease resistance by elevating the expression of host r

110 er protective MHC class II molecules, afford disease resistance by engaging a naturally occurring con

111 mbers execute their function and spectrum of disease resistance by recognizing the cognate TALEs in p

112 mbers execute their function and spectrum of disease resistance by recognizing the cognate TALEs in t

113 MAPKs or MPKs), play critical roles in plant disease resistance by regulating multiple defense respon

114 In general, whole-genome models for disease resistance can produce prediction accuracy suita

115 pv. tomato (Pst) DC3000, including increased disease resistance, cell death and ROS production.

116 dentification of genes controlling QTL-based disease resistance contributes to breeding for cultivars

117 and their impact on prevention of late-onset disease, resistance, cost, and toxicity.

118 microconidia and lesion length, a measure of disease resistance, data were collected 4, 8, and 12 wee

119 d high-quality diets did not differ in their disease resistance, despite differing in their body cond

120 The suppression of cell death and disease resistance did not require a physical associatio

121 dopsis thaliana compromises host and nonhost disease resistance due to open stomata during pathogen i

122 ad positive effects on enhancing anthracnose disease resistance during storage and also gave a residu

123 uncouple SA-dependent defense signaling from disease resistance execution.

124 ants, potentially indicating the presence of disease-resistance factors.

125 related to major agronomic traits, including disease resistance, flowering time, glucosinolate metabo

126 persensitive-response (HR) cell death or its disease resistance functions, further suggesting that nu

127 enes, nucleotide-binding-leucine-rich repeat disease resistance gene clusters have undergone dramatic

128 sistance in plants by means of an endogenous disease resistance gene from Arabidopsis thaliana named

129 redicted protein-coding genes, including 292 disease resistance gene homologs, and nine genes determi

130 a TE inserted into the Arabidopsis thaliana disease resistance gene RPP7 recruited the histone mark

131 as caused by a partially dominant autoactive disease resistance gene, Rp1-D21, which caused HR lesion

132 the cloning of Tsn1, which was found to have disease resistance gene-like features, including S/TPK a

133 ucine-rich-repeat proteins (NLRs), the major disease-resistance gene families, has been unexplored in

134 this region identified three Rpp4 candidate disease resistance genes (Rpp4C1-Rpp4C3 [Wm82]) with gre

135 In particular, disease resistance genes and receptor-like kinases commo

136 Disease resistance genes are valuable natural resources

137 eotide-binding, leucine-rich repeat (NB-LRR) disease resistance genes by small RNAs is particularly u

138 In plants, most disease resistance genes encode nucleotide binding Leu-r

139 The repetitive nature of plant disease resistance genes encoding for nucleotide-binding

140 However, few recessive disease resistance genes have been characterized.

141 his work that artificial evolution of NB-LRR disease resistance genes in crops can be enhanced by mod

142 nucleotide-binding site-leucine-rich repeat disease resistance genes were annotated, suggesting the

143 e, and showed that new genes, exemplified by disease resistance genes, are preferentially located in

144 ubgenomes and use them to identify candidate disease resistance genes, to guide tetraploid transcript

145 Some activate disease resistance genes.

146 -nucleotide binding site-leucine rich repeat disease resistance genes.

147 ng the classical MHC molecules and few other disease resistance genes.

148 e binding site-leucine-rich repeat (NBS-LRR) disease resistance genes.

149 emergence of NLR genes including functional disease-resistance genes in pepper plants.

150 composed largely of transposed genes: NB-LRR disease-resistance genes, genes encoding MADS-box and B3

151 protein-coding regions, with enrichment for disease-resistance genes.

152 Plant disease resistance governed by quantitative trait loci (

153 d in agriculture, but the mechanisms of Rhg1 disease resistance have remained unclear.

154 nd provides an important mechanism for their disease resistance; however, many aspects of the cell wa

155 tion, there has been success in breeding for disease resistance; however, much of this work and resea

156 sduction pathways in cancer progression, and disease resistance; (ii) intratumoral heterogeneity and

157 lopment of crop plants with enhanced pest or disease resistance, improved nutritional qualities and/o

158 positive association between spleen size and disease resistance in a teleost fish.

159 hosts is critical for rationally engineered disease resistance in agricultural systems.

160 genes useful for improving fruit quality and disease resistance in C. annuum sweet bell and hot chile

161 silencing in pathogens for generating novel disease resistance in crop plants.

162 mmunity and may have potential for enhancing disease resistance in crops.

163 trategies for breeding temperature-resilient disease resistance in crops.

164 ove broad-spectrum, and potentially durable, disease resistance in crops.

165 atic industrial products as well as increase disease resistance in energy and agricultural crops.

166 Hence, the quantitative Htn1 disease resistance in maize is encoded by an unusual inn

167 ested the condition-dependence of growth and disease resistance in male and female Gryllus texensis f

168 nase (MAPK) cascades play important roles in disease resistance in model plant species such as Arabid

169 of disease incidence and severity, a lack of disease resistance in planting materials, shortages of l

170 We describe a method to engineer disease resistance in plants by means of an endogenous d

171 istent with the hypothesis that quantitative disease resistance in plants is conditioned by a range o

172 ascination with epidemiology and genetics of disease resistance in plants, particularly coevolution o

173 is a new promising candidate for engineering disease resistance in plants.

174 d (SA), which plays a key role in activating disease resistance in plants.

175 To assess the genetic basis of whirling disease resistance in rainbow trout, genome-wide mapping

176 breeding targets for improved broad-spectrum disease resistance in rice.

177 n N. benthamiana and HeLa cells also abolish disease resistance in rice.

178 stand the role of MAPK signaling pathways in disease resistance in soybean (Glycine max), 13, nine, a

179 omain-containing GmHSP40.1 in cell death and disease resistance in soybean.

180 gulating SA accumulation, cell death, and/or disease resistance in the Arabidopsis (Arabidopsis thali

181 Studies often consider disease resistance in the context of life-history theory

182 could induce PR1 gene expression and restore disease resistance in the eps1 mutant.

183 results also indicate that studying extreme disease resistance in the face of extensive exposure can

184 ustrate how bacterial effectors that trigger disease resistance in the host can evolve to interfere w

185 to elicit hypersensitive (HR) cell death and disease resistance in tobacco.

186 four DEMs regulating three DEGs involved in disease resistance, including miR156, miR172f, miR172g,

187 se efficiency, abiotic stress tolerance, and disease resistance-into agricultural production.

188 Breeding for disease resistance is a central focus of plant breeding

189 ization of the genetic components underlying disease resistance is a major research area in maize whi

190 ndertaken to uncover the mechanisms by which disease resistance is achieved.

191 s cleavage and inhibits autophagy in plants; disease resistance is also compromised.

192 Disease resistance is commonly based on resistance genes

193 standing the fundamental mechanisms of plant disease resistance is of central importance to sustainab

194 Plant disease resistance is often conferred by genes with nucl

195 The underlying mechanism of the disease resistance is unknown.

196 Endophyte-mediated disease resistance is well-established in the fine fescu

197 ght into molecular mechanisms that determine disease resistance levels in Vitis species native to the

198 gene set associated with chemically induced disease resistance, many of which appear to encode compo

199 c groups, such that genes providing multiple disease resistance (MDR) are expected to be under positi

200 Alleles that confer multiple disease resistance (MDR) are valuable in crop improvemen

201 e relatively nonspecific, providing multiple disease resistance (MDR).

202 mplications for the understanding of natural disease resistance mechanisms at the species level and f

203 KEY MESSAGE: We report plausible disease resistance mechanisms induced by barley resistan

204 We report plausible disease resistance mechanisms induced by barley resistan

205 the suppression of programmed cell death and disease resistance mediated by several CC-NB-LRR protein

206 b are transcriptionally up-regulated during disease resistance mediated by the R gene Mi-1.

207 iously shown to be specifically required for disease resistance mediated by the R protein RPP7.

208 multiple pathogens broadens the spectrum of disease resistances mediated by single plant immune rece

209 In contrast to gene-for-gene disease resistance, nonhost resistance governs defense r

210 DPMP strongly triggers disease resistance of Arabidopsis against bacterial and

211 importance of MAPK signaling pathways in the disease resistance of crops is still largely uninvestiga

212 of sequence, gene organization, and roles in disease resistance of GLP family members in rice and oth

213 e bacteria-binding proteins in prophylaxical disease resistance of gregarious locusts.

214 Disease resistance of IRAK4(-/-) mice paralleled increas

215 BHTC induces disease resistance of plants against bacterial, oomycete

216 h QTL for bacterial blight and sheath blight disease resistance on rice chromosome 2.

217 gene approach constrained by annotation for disease resistance or stress response.

218 with enhanced biomass conversion properties, disease resistance, or nutritional quality.

219 ne is a negative regulator of cell death and disease resistance, possibly through regulating ROS and

220 tectures, including growth, development, and disease-resistance properties, measured in a Pinus taeda

221 ) RESISTANCE TO PSEUDOMONAS SYRINGAE5 (RPS5) disease resistance protein mediates recognition of the P

222 RPS2 is a membrane-associated disease resistance protein of low abundance.

223 ch encodes a Toll-IL-1 receptor (TIR)-NB-LRR disease resistance protein.

224 ssinosteroid signaling, peroxisome function, disease resistance, protein phosphorylation and light pe

225 quired for PCD associated with several other disease resistance proteins and contributed to resistanc

226 temperatures often inhibit the activities of disease resistance proteins and the defense responses th

227 effectors trigger innate immunity via plant disease resistance proteins as described by the "guard h

228 ed families of genes, such as those encoding disease resistance proteins or transcription factors.

229 forms protein complexes with the TIR-NB-LRR disease resistance proteins RPS4 and RPS6 and with the n

230 enzymes involved in rubber biosynthesis and disease resistance proteins that are expanded in the gen

231 miana, that miR482 targets mRNAs for NBS-LRR disease resistance proteins with coiled-coil domains at

232 r the P-loop motif in the mRNA sequences for disease resistance proteins with nucleotide binding site

233 Quantitative disease resistance (QDR) has been observed within many c

234 Quantitative disease resistance (QDR) is the type of resistance most

235 Five disease resistance QTL that colocalized with defense res

236 essor of defense responses controlled by the disease resistance (R) gene homolog SNC1.

237 Disease resistance (R) genes are often clustered in plan

238 The majority of plant disease resistance (R) genes encode proteins that share

239 The majority of plant disease resistance (R) genes encode proteins that share

240 The recognition between disease resistance (R) genes in plants and their cognate

241 us vulgaris) that is associated with several disease resistance (R) genes of known function, includin

242 h is associated with increased expression of disease resistance (R) genes similar to the animal NOD1

243 cated crop species harbor multiple, diverse, disease resistance (R) genes that could be used to engin

244 have evolved a limited repertoire of NB-LRR disease resistance (R) genes to protect themselves again

245 The first plant disease resistance (R) genes were identified and cloned

246 sitica 5) locus, which includes a cluster of disease Resistance (R) genes, many of which show an incr

247 e resistance to viruses is afforded by plant disease resistance (R) genes, which encode proteins with

248 ated with immunity is triggered when a plant disease resistance (R) protein recognizes a correspondin

249 RESISTANCE TO PSEUDOMONAS MACULICOLA1 (RPM1) disease resistance (R) protein.

250 Plant disease resistance (R) proteins recognize potential path

251 gered immunity (ETI) is activated when plant disease resistance (R) proteins recognize the presence o

252 Disease resistance (R) proteins, as central regulators o

253 derived metabolites, and the accumulation of disease resistance-related secondary metabolites and dif

254 s responsible for intraspecific variation in disease resistance remain unclear.

255 ll studied, how they activate PCD and confer disease resistance remains elusive.

256 e we provide evidence that the RCT1-mediated disease resistance requires the combined presence of the

257 ry biology, with implications for predicting disease resistance, response to environmental change, an

258 Many race- or isolate-specific disease resistance responses of plants toward pathogens

259 ta provide evidence of genetically separable disease resistance responses to AvrB and AvrRpm1 in Arab

260 lecular pattern-triggered immunity (PTI) and disease resistance responses to different types of patho

261 3 are partially required for HopW1-1-induced disease resistance, SA production and HWI1 expression.

262 Efe-AfpA may therefore be a component of the disease resistance seen in endophyte-infected strong cre

263 as the genetic requirement for NDR1 in plant disease resistance signaling has been detailed, our stud

264 suggested that MLA10-mediated cell-death and disease resistance signaling occur independently, in the

265 Initial abiotic stress can interfere with disease-resistance signaling [1-6].

266 is thaliana RPP1 immune receptor activates a disease-resistance signaling pathway that inhibits patho

267 translate pathogen-specific recognition into disease-resistance signaling.

268 It has been proposed to change disease resistance specificity by reprogramming the expr

269 nts can reuse "dead" alleles to generate new disease-resistance specificity, leading to a "death-recy

270 nctional role(s) of occlusions in host plant disease resistance/susceptibility remains controversial.

271 is latter result might be related to Marek's disease resistance/susceptibility.

272 a high-quality diet had significantly poorer disease resistance than females on a low-quality diet an

273 enerally, our work shows that in addition to disease resistance, the costs of immunity vary between i

274 The paradigm of disease resistance through a dysfunctional variant of an

275 y reveals a new intersection between ABA and disease resistance through R protein localization and pr

276 ike receptors (TLRs) perform a vital role in disease resistance through their recognition of pathogen

277 of processes, from cell wall interactions to disease resistance to developmental control.

278 expression of CRK28 in Arabidopsis increased disease resistance to P. syringae Expression of CRK28 in

279 three CYCD3 genes disrupted also compromised disease resistance to P. syringae.

280 lant resistance gene conferring quantitative disease resistance to plants against Fusarium species.

281 to leaf spontaneous cell death and enhanced disease resistance to powdery mildew via the SA-dependen

282 We found that fzl mutants showed enhanced disease resistance to the bacterial pathogen Pseudomonas

283 we report that WIN3 controls broad-spectrum disease resistance to the necrotrophic pathogen Botrytis

284 ld fungus Cladosporium fulvum, also mediates disease resistance to the root parasitic nematode Globod

285 as HER2/Erbb2 can result in more aggressive disease, resistance to chemotherapy and reduced survival

286 But for each disease, resistance to most drugs has appeared quickly a

287 ypes, including susceptibility to autoimmune disease, resistance to pathogens and the risk of adverse

288 e known and hypothesized roles in autoimmune diseases, resistance to viruses, reproductive conditions

289 f multigene segments, using as the example a disease resistance trait of high economic importance.

290 ction of potential biofuels and shuffling of disease-resistance traits between crop species.

291 Interestingly, for fusiform rust disease-resistance traits, Bayes Cpi, Bayes A, and RR-BL

292 s related to milk-production, fertility, and disease-resistance traits.

293 A sex difference in disease resistance under diet-restriction suggests that

294 alicylic acid signaling pathway and enhanced disease resistance upon challenge inoculation with a vir

295 In addition, we show that ERA1 function in disease resistance was independent of its role in stomat

296 Vibrio anguillarum revealed that the larval disease resistance was not influenced by PHB.

297 otein and non-protein coding genes for which disease resistance was the first biological annotation.

298 transduction cascade associated with nonhost disease resistance, we used a virus-induced gene-silenci

299 ta composition, gene expression profiles and disease resistance were assessed.

300 n rice enables us to engineer broad-spectrum disease resistance without compromising plant fitness in