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1 rgans per year, despite the very low risk of disease transmission.
2  against higher morbidity and a real risk of disease transmission.
3 in order to mitigate the risk of human prion disease transmission.
4 evaluations for donors at increased risk for disease transmission.
5 s of pandemics, particularly animal-to-human disease transmission.
6 or climate change to have a marked impact on disease transmission.
7 ycle, ensuring both infection chronicity and disease transmission.
8 r secretory tissues is necessary for natural disease transmission.
9 understanding of how floral traits influence disease transmission.
10 his host trafficking factor in microsporidia disease transmission.
11 blood meal; if there is no bite, there is no disease transmission.
12  dynamics affect local patterns of diarrheal disease transmission.
13 they wish to screen based on the dynamics of disease transmission.
14 e potential to improve cure rates and reduce disease transmission.
15  the fecal-oral route being a common mode of disease transmission.
16 of current and future temperature regimes on disease transmission.
17 linking resistance selection with changes in disease transmission.
18 estock, and pets from pest insect attack and disease transmission.
19 lar aggregates believed to play key roles in disease transmission.
20 vironmental change will increase or decrease disease transmission.
21 eractions and develop interventions to block disease transmission.
22 om the development of mathematical models of disease transmission.
23 pulation mixing, gene flow, and pathogen and disease transmission.
24 petitors can reduce vector density and hence disease transmission.
25 ounting for rainfall as a driver of enhanced disease transmission.
26 ito control and prevention of mosquito-borne disease transmission.
27 ble on the incidence of allograft-associated disease transmission.
28 arries some, largely unquantifiable, risk of disease transmission.
29 oice, parental care, territoriality and even disease transmission.
30 vity that are likely to contribute to facile disease transmission.
31 odiversity could either increase or decrease disease transmission.
32  the need for alternative methods to prevent disease transmission.
33 f the social network relevant for infectious disease transmission.
34  that biodiversity loss frequently increases disease transmission.
35 ctions suitable for modelling mosquito-borne disease transmission.
36 e of the possible approaches for controlling disease transmission.
37 to intercohort interactions, leading to more disease transmission.
38 ty in secreta is a crucial concern for prion disease transmission.
39 spersal has a particularly important role in disease transmission.
40 spital environments and facilitate efficient disease transmission.
41 urden and is therefore unlikely to interrupt disease transmission.
42 or trained personnel, and risk of infectious disease transmission.
43 ve minimized, but not eliminated, infectious disease transmission.
44 athogen interactions, which are important to disease transmission.
45 nced the dominant anticontagionist school of disease transmission.
46 le interventions was associated with reduced disease transmission.
47 ities are important predictors of infectious disease transmission.
48 obiota factors that contribute to infectious disease transmission.
49 ase prevention but also the factors enabling disease transmission.
50  also provide epidemiological information on disease transmission.
51 XI mutations may be associated with dominant disease transmission.
52  by a population with a reduced capacity for disease transmission.
53 y likely accounts for many cases of dominant disease transmission.
54 mal and family histories indicating dominant disease transmission.
55 tor's biology, to discover the weak links in disease transmission.
56 btained using an autosomal dominant model of disease transmission.
57 ts and should be incorporated into models of disease transmission.
58 ir hosts is important for ensuring continued disease transmission.
59 t effective route of between-farm infectious disease transmission.
60  behaviour, and limiting freedom to diminish disease transmission.
61 a novel means of controlling arthropod-borne disease transmission.
62 by glycan site occupancy) to strain type and disease transmission.
63 gs have impact on our current concept of CWD disease transmission.
64 fected at an early age and with male-to-male disease transmission.
65 nformation to develop new methods to prevent disease transmission.
66  problems, such as antibiotic resistance and disease transmission.
67 y be reflected in a species barrier to prion disease transmission.
68 erved were related to the species barrier of disease transmission.
69 be exploited to gain additional insight into disease transmission.
70  view toward identifying features related to disease transmission.
71  by social structure such as information and disease transmission.
72 imely public health interventions to prevent disease transmission.
73 mally the limited information into models of disease transmission.
74 hite blood cells (WBC) resulted in efficient disease transmission.
75 s to generalizable scientific insights about disease transmission.
76  in species interaction and so a lowering of disease transmission.
77 he tsetse vector is being explored to reduce disease transmission.
78 hole blood has an apparent 100% efficacy for disease transmission.
79 a in order to evaluate intra- and interclass disease transmission.
80 cted a multistate investigation to interrupt disease transmission.
81 rtunities to reduce mosquito populations and disease transmission.
82 to reduce the tsetse's vector competence and disease transmission.
83 prevent and control MERS-CoV or new emerging disease transmission.
84  sources have provided new means of studying disease transmission.
85  the population-scale dynamics of infectious disease transmission.
86 f environmental and physiological drivers of disease transmission.
87 ective treatment and subsequent reduction in disease transmission.
88 to their hosts, and are thus responsible for disease transmission.
89 roorganisms in streams enables long-distance disease transmission.
90 ble approach toward understanding infectious disease transmission.
91  host protein, PrP(C), plays a major role in disease transmission.
92 ome infections by changing the landscape for disease transmission.
93 r bioaerosols in order to reduce the risk of disease transmission.
94 -infected people as a new risk of increasing disease transmission.
95 addition of these glycans may play a role in disease transmission.
96 d therapeutic intervention and prevention of disease transmission.
97  the relative roles of badgers and cattle in disease transmission.
98 ariation by linking multiple host species to disease transmission.
99 erations and Safety Committee, the OPTN/UNOS Disease Transmission Advisory Committee (DTAC) and the c
100                                   The Ad Hoc Disease Transmission Advisory Committee (DTAC) reviewed
101     The Organ Procurement Transplant Network Disease Transmission Advisory Committee (DTAC), a multid
102        Within the families with male-to-male disease transmission, alpha increased with the early mea
103  high vaccination rates can potentially halt disease transmission altogether.
104 cribe sterile injection equipment to prevent disease transmission among drug-using patients and that
105 ghlights the importance of host behaviour in disease transmission among natural populations.
106                                              Disease transmission and barrier techniques during denta
107 is discussed in relation to short coat hair, disease transmission and blood loss.
108             We analyse two models describing disease transmission and control on regular and small-wo
109 igm shift in our understanding of infectious disease transmission and control.
110 equate treatment leads to worsening disease, disease transmission and drug resistance.
111 itical to understanding the linkages between disease transmission and environmental factors.
112  understanding the role of prion shedding in disease transmission and for diagnosis.
113 for the risk assessment of blood-borne prion disease transmission and for refining the target perform
114 for the risk assessment of blood-borne prion disease transmission and for refining the target perform
115 red from mobile phone data are predictive of disease transmission and improve significantly on standa
116  is nowadays important for the prevention of disease transmission and in the future - hopefully - for
117 ide range of environmental conditions during disease transmission and infection.
118                                      Risk of disease transmission and limitations in the ability to t
119 tic variation among parasites to patterns of disease transmission and manifestations has been the goa
120 fluenza mortality are associated with higher disease transmission and more rapid spread than are mild
121 ased on a parsimonious mathematical model of disease transmission and only requires data collected th
122                                              Disease transmission and outbreak intensity, measured as
123 ckness parasite, Trypanosoma brucei, impacts disease transmission and pathogenesis.
124 rP(Sc) core of 27-30 kDa implicated in prion disease transmission and pathogenesis.
125 ental reservoirs are important to infectious disease transmission and persistence, but empirical anal
126 mechanisms accounting for the differences in disease transmission and phenotype between affected fema
127 s made identifying the mechanisms underlying disease transmission and progression extremely difficult
128 l surveillance can be a sensitive measure of disease transmission and provide a more objective testin
129 ass gatherings, which may act as hotspots of disease transmission and spread.
130                     From the perspectives of disease transmission and sterility maintenance, the worl
131 dels, which may represent a risk for further disease transmission and thus a significant public-healt
132 th respect to the intrinsic stochasticity of disease transmission and traffic flows.
133 Multiple pathogenic infections can influence disease transmission and virulence, and have important c
134                          Differences between disease transmission and waterfowl migration directions
135 h (for example, in the detection of possible disease transmission), and as part of divide-and-conquer
136 ase pathogen development and survival rates, disease transmission, and host susceptibility.
137 ionary time, due to chance, changes in local disease transmission, and parasite population structurin
138 rces required for food security, patterns of disease transmission, and processes of carbon sequestrat
139 nsity and activity level, the probability of disease transmission, and the structural organization of
140                      Snail abundance fosters disease transmission, and thus the dynamics of snail pop
141 esults suggest that anthropogenic effects on disease transmission are complex, and highlight the need
142 f the complex interplay of factors affecting disease transmission are needed to assist with efforts a
143  well as their direct effect on vector-borne disease transmission are needed to evaluate its potentia
144 r, the aphid-virus interactions required for disease transmission are poorly understood.
145 .g., dispersal of offspring, competition, or disease transmission) are assumed to operate over a sing
146 a vaccination programmes result in continued disease transmission, as evidenced by recent large outbr
147 imulations of susceptible-infected-recovered disease transmission, as well as traditional non-network
148 cape factors, as well as forces facilitating disease transmission at fine scales.
149                                     Wildlife disease transmission, at a local scale, can occur from i
150  data in settings with high, medium, and low disease transmission based on clinical disease.
151 reduce some of the potential for contact and disease transmission between badgers and cattle.
152 es that influence diverse ecology, including disease transmission between conspecifics and courtship
153 her global warming will increase or decrease disease transmission between individuals remains far fro
154                                Cross-species disease transmission between wildlife, domestic animals
155 ation resulted in almost complete absence of disease transmission but elicited striking PrP-amyloid d
156                                 Evidence for disease transmission by bed bugs is lacking.
157 e management must be to minimize the risk of disease transmission by finding new ways to reduce the c
158    Technologies and practices that interrupt disease transmission by flies need to be developed and p
159 ng how host immune defenses indirectly alter disease transmission by influencing vector behavior has
160 ce and blood feeding, integral behaviors for disease transmission by the malaria vector mosquito Anop
161 , in which the relationship of sociality and disease transmission can be comparatively and experiment
162 n of the role of individual heterogeneity in disease transmission can contribute further in this rega
163 ue highlight how differences in the route of disease transmission can enhance the lethality of an alr
164 e is stronger, since we assume that enhanced disease transmission can only be achieved at the cost of
165 d out of jail and sexual contacts (including disease transmission) can provide useful information.
166 ication of natural stochastic differences in disease transmission, can give rise to persistent oscill
167 tus of the host can have profound effects on disease transmission, changing host susceptibility and i
168 mics and the development of novel vector and disease transmission control strategies, but also will e
169 ill be small, the risk of future blood-borne disease transmissions could be entirely eliminated by a
170  within populations, thus directly affecting disease transmission cycles.
171 d meal, a key event in both reproduction and disease transmission cycles.
172          The bottleneck governing infectious disease transmission describes the size of the pathogen
173  those aspects of midge biology facilitating disease transmission, describes the factors controlling
174 hropod-vertebrate interactions and potential disease transmission during the Mesozoic.
175 of individual variation of infection load on disease transmission dynamics and how this influences th
176  Linkage analysis is useful in investigating disease transmission dynamics and the effect of interven
177                                Understanding disease transmission dynamics in multihost parasite syst
178                    Host extinction can alter disease transmission dynamics, influence parasite extinc
179 more likely to have contact, which may drive disease transmission dynamics.
180 systematically evaluate their effects on the disease transmission dynamics.
181 he thermostability of PrP(Sc) aggregates and disease transmission efficiency makes inconsistent the p
182     Establishment of a donation service area disease transmission evaluation service is a valuable pr
183 iewed the records of potential donor-derived disease transmission events (PDDTE) to describe donor ch
184 g dynamic feedbacks involving the ecology of disease transmission, evolutionary processes, and their
185 ative perspective on plague's ecology (i.e., disease transmission exacerbated by alternative hosts) a
186  performed to assess the possibility of such disease transmission from a commercially available bone
187 thogens, originate from animals, and ongoing disease transmission from animals to people presents a s
188 egies to reduce both the biting nuisance and disease transmission from bed bugs.
189 xenografts is tempered by the possibility of disease transmission from cattle to humans.
190 tter, as an efficacious measure to interrupt disease transmission from uncontrolled spills in Ebola o
191   With the rise in global travel, preventing disease transmission has become paramount to avoid the s
192  importance of the environment in infectious disease transmission has grown, so too has interest in p
193 he dynamics of within- and between-community disease transmission have distinct components but are al
194 intravenous access, and risks of blood-borne disease transmission, have fueled an interest in develop
195 etween worker density and the probability of disease transmission: high levels of both factors intera
196 istically modelling individual-to-individual disease transmission in a landscape with heterogeneous p
197                   Analysis and simulation of disease transmission in a symmetric meta-population sugg
198 offer a reproductive option to prevent mtDNA disease transmission in affected families.
199 ormed experimental studies of foot-and-mouth disease transmission in cattle and estimated this fracti
200 lication of social network analysis to study disease transmission in healthcare settings.
201                   This increases the risk of disease transmission in highly vaccinated populations.
202 n effect with a general trait-based model of disease transmission in multi-host communities.
203  review the models that have been applied to disease transmission in social insects and elucidate are
204     To examine the effects of temperature on disease transmission in the field, we manipulated baculo
205                                 The risk for disease transmission in the United States is partly the
206 tana fleas were implicated as the vector for disease transmission in this case.
207 e present a mathematical model of infectious disease transmission in which people can engage in publi
208 e the dynamics of post-disaster vector-borne disease transmission, in the context of conducive/favour
209 ent data have unmasked an oligogenic mode of disease transmission, in which mutations at different BB
210 pares to other treatment options relative to disease transmission, including its influence on antibio
211 l incorporates an SIS compartmental model of disease transmission into a game theoretic model of stra
212                                   To prevent disease transmission into commercial pig herds, it is th
213 es (MRT) circumventing mother-to-child mtDNA disease transmission involve replacement of oocyte mater
214 o accept a kidney labeled increased risk for disease transmission (IRD) accept a low risk of window p
215 , particularly in those attending CCCs where disease transmission is common.
216 ironmental heterogeneities involved in local disease transmission is crucial to capturing the spatial
217                              When infectious disease transmission is density-dependent, the risk of i
218                      The most common mode of disease transmission is ingestion of contaminated bovine
219     Identifying the major sources of risk in disease transmission is key to designing effective contr
220 c virus genomes, yet evidence for waterborne disease transmission is lacking.
221 patially explicit agent-based model in which disease transmission is sensitive to population density
222       Selection favors lower resistance when disease transmission is spatially local and the damage t
223                     A major concern in prion disease transmission is the spread of the disease agent
224 t structure, a critical driver of infectious disease transmission, is not completely understood and c
225 ppropriate approach to identify critical STI disease transmission locations.
226   Rapid availability, low risk of infectious disease transmission, lower risk of graft-versus-host di
227 ing, and transfer, and inherent freedom from disease transmission, make it a promising substrate for
228                     This triallelic model of disease transmission may be important in the study of bo
229 'i Plateau suggest that predicted changes in disease transmission may be occurring.
230 e role of mental health in the prevention of disease transmission may help fight continuing and futur
231  hosts) has transformed our understanding of disease transmission mechanisms and capacity to predict
232 thogen's; Infection Systems (Pathogen, Host, Disease, Transmission Method and Anatomy) and Incidents
233 an epidemic by employing an individual-based disease transmission model and a coalescent process taki
234 g outbreaks, the authors developed a dynamic disease transmission model that can simulate many aspect
235 this traced vaccination policy in a smallpox disease transmission model to estimate the number of cas
236   Here we introduce an SIR-type multi-strain disease transmission model with perfect cross immunity w
237 ad, which we demonstrated using a stochastic disease transmission model.
238 analysis, economic analysis, statistical and disease transmission modelling - we aimed to explore the
239          Environmentally mediated infectious disease transmission models provide a mechanistic approa
240 tor-borne disease and be applicable to other disease transmission models.
241                                 Although the disease transmission normally requires direct interactio
242                     Where genetic change and disease transmission occur on comparable timescales addi
243                                              Disease transmission occurs when their habitats overlap.
244  for effective treatment and interruption of disease transmission of tuberculosis (TB).
245                                   Infectious disease transmission often depends on the contact struct
246            Vaccination dramatically disrupts disease transmission on a contact network, and indeed, h
247                        We present a model of disease transmission on a regular and small world networ
248  in reproductive mode associated with either disease transmission or hybridization.
249 ng mosquitoes with individuals refractory to disease transmission, or bringing about population suppr
250 ens themselves but also the immune response, disease transmission, or even just the symptoms.
251 et, number of affected members, male-to-male disease transmission, or race.
252 roving statistical methodologies to estimate disease transmission parameters from these data.
253 mic in Asia, with live bird trade as a major disease transmission pathway.
254 ates empirically that small perturbations in disease transmission patterns can fundamentally alter th
255  3) infer from these similarities infectious disease transmission patterns.
256  capture early reports of unknown infectious disease transmission prior to official laboratory confir
257 which explore vector population genetics and disease transmission probabilities and show that using e
258 show that it is possible to characterize the disease transmission process under these conditions.
259                                The theory of disease transmission provides a consistent framework wit
260  the host reproduction rate, or the baseline disease transmission rate, is reduced, and the parasite
261 These new Wolbachia strains may be affecting disease transmission rates of infected mosquito species,
262 nd services, making them a natural basis for disease transmission rates over distance.
263 equence of this aggregation can be increased disease transmission rates.
264 pendent on effective vector control reducing disease transmission rates.
265 netic factors includes complex distortion of disease transmission seen in aunt/uncle-niece/nephew (AU
266                  Past patterns of infectious disease transmission set the stage on which modern epide
267 period parameter have a non-linear effect on disease transmission, so a greater understanding of the
268 o a lack of knowledge over the mechanisms of disease transmission; some strains of TSE are able to cr
269 ng our ability to understand high-resolution disease transmission, sometimes even down to the host-to
270 ich also takes into account other drivers of disease transmission such as rainfall, is applied to the
271 ltiple infectious disease traits influencing disease transmission, such as the frequently modeled pro
272 used to parameterize a mathematical model of disease transmission that captures the differing spatial
273 athematical model was adapted for infectious disease transmission that estimated a distribution for o
274 ission model of HIV and sexually transmitted disease transmission that was parameterised and fitted t
275 ereas this strategy is important for driving disease transmission, the molecular mechanisms underlyin
276 . difficile spore formation is essential for disease transmission, the regulatory pathways that contr
277  in infectivity substantially contributes to disease transmission, then breeding designs which explic
278  standards over the last decade, the risk of disease transmission through allogeneic blood transfusio
279  can be induced by a prion-like mechanism of disease transmission through propagation of protein misf
280 tor of infectivity in terms of prevention of disease transmission through selective isolation policy
281 elop a mathematical framework for predicting disease transmission through semi-directed contact netwo
282 l contact networks and for the simulation of disease transmission through such networks.
283 ance systems to assess and analyze risks for disease transmission through the transfer of organs, tis
284 d data have been proven useful for inferring disease transmission to a more refined level than previo
285 is paper we use an individual-based model of disease transmission to assess how an epidemic is influe
286  disposal of feces by one household prevents disease transmission to households nearby.
287 ecently used for the modelling of infectious disease transmission to model evolutionary game dynamics
288 (Tg) mice expressing cognate PrP(C) Although disease transmission to only a subset of infected TgEq i
289 -like system restores oocyst development and disease transmission to rodent hosts.
290 to examine how increased temperatures affect disease transmission using the crop-defoliating pest, th
291  of the risks of (variant) Creutzfeldt-Jakob disease transmission via dental practice, and whether th
292                               The pattern of disease transmission was compatible with an autosomal-do
293                                              Disease transmission was concentrated along rivers in th
294 ay play an important role in climate change, disease transmission, water and soil contaminants, and g
295 ng a modified Wells-Riley model for airborne disease transmission, we estimated the risk of tuberculo
296 hamsters, which were observed for 10 months; disease transmissions were verified by Western blot test
297 ch we present allows the study of infectious disease transmission when data linking cases to each oth
298 V infection, it is unlikely that subclinical disease transmission will occur.
299                       The risk of infectious disease transmission with FWB transfusion can be minimiz
300 icant effect of the presence of male-to-male disease transmission within the families.

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