ライフサイエンスコーパス: disease

1 Treatment after disease onset potently blocks progression of disease and furt

2 relation between 11C-PK-11195 and 18F-AV-1451 uptake in all disease groups, across widespread cortical regions.

3 this difference likely results from distinct biological and disease initiation and progression events associated with Chr

4 together with BIN1, to ameliorate synaptic dysfunction and disease progression.

5 perfusion but the involved vascular-sensing mechanisms and disease consequences remain unclear.

6 ALS cohort, truncated STMN2 RNA was confined to tissues and disease subtypes marked by TDP-43 inclusions.

7 rved to improve the health of patients with coronary artery disease (CAD).

8 Myasthenia gravis (MG) is a neuromuscular, autoimmune disease caused by autoantibodies that target postsynaptic pro

9 found that a more extensive lymphadenectomy leads to better disease-specific survival in patients treated with surgery al

10 No deaths or cases of inflammatory bowel disease were reported.

11 There is a growing burden of cardiometabolic disease in many parts of the world.

12 singly prevalent modifiable risk factors for cardiovascular disease.

13 Inflammation plays an important role in cardiovascular disease (CVD) development.

14 impaired glucose metabolism, contributes to cardiovascular disease.

15 ut have villous atrophy and genetic risk factors for celiac disease must undergo endoscopic evaluation after 1-3 years on

16 BEST PRACTICE ADVICE 6: Patients with suspected celiac disease who are seronegative but have villous atrophy and gen

17 mation that is not resolved in due course becomes a chronic disease.

18 The core disease signature revealed remarkably strong connections to g

19 onitored every 6 months for signs and symptoms of diarrheal disease, acute respiratory illness, and anemia.

20 hase clinical trials in children with high-risk MYCN-driven disease, with limited ability to evaluate conventional pharma

21 as well as arthralgias in acute and convalescent filovirus disease.

22 e surveillance, to aggressive and invasive tumors with high disease-specific mortality.

23 d morbidity and mortality during antiretroviral-treated HIV disease.

24 Variation in risk factors across host-disease pairs suggests that either different pathogens cause

25 macrophage homeostasis, lung inflammation and immunological disease.

26 the similarities and differences between MIS-C and Kawasaki disease, focusing on their epidemiology, aetiology and pathop

27 Current models only partially recapitulate key disease features, and pathophysiology is poorly understood.

28 are effective for the treatment of anemia in chronic kidney disease patients and may also be beneficial for the treatment

29 licensed to protect against invasive group B meningococcal disease.

30 With the categorical probabilistic forecasts of disease incidences, this early health warning system is found

31 the improved downstream gene expression-based prediction of disease outcome.

32 reatment after disease onset potently blocks progression of disease and further alpha-motoneuron degeneration.

33 of educational attainment (schooling years) on a variety of disease and life-expectancy outcomes.

34 Anti-IL-10-treated nonhuman primates had similar overall disease outcomes compared with untreated control nonhuman pri

35 ng how variants of IFI16 and AIM2 contribute to periodontal disease pathogenesis may lead to treatment options that addre

36 skill to provide the climate-health outlook about possible disease incidence at least 2 weeks in advance for any locatio

37 ngs of epigenetic alterations in schizophrenia and relevant disease models and discuss their putative origin.

38 nitive Impairment and Imaging Correlates in End Stage Renal Disease, NCT01883349.

39 stimated glomerular filtration rate (eGFR), end-stage renal disease, or death from renal causes), the individual componen

40 Parkinson's disease (PD) pathogenesis may involve the epigenetic control

41 multiple brain disorders and injuries, e.g., in Parkinson's disease or traumatic brain injury (TBI), and hence it will be

42 he same gross lesions in different species or that the same disease may arise in different species under different ecolog

43 The disease is very likely to be underdiagnosed.

44 In particular, the central role of B cells in the disease has been demonstrated by both the robust clinical res

45 function and suggested novel roles of EGR1 and SGK1 in the disease state.

46 lable datasets might help to gain further insights into the disease process.

47 or irritable bowel syndrome are available to those with the disease.

48 Thromboembolic disease was contextualized by premortem AC among consecutive

49 Our study links DNA methylation to disease endpoints via intermediate proteomics phenotypes and

50 on between the virus and innate receptors that may underlie disease pathogenesis.