ライフサイエンスコーパス: disequilibria

1 co-metabolic interactions to maximize energy disequilibria.

2 uns and at higher magnitudes than homozygote disequilibria.

3 commonly linked to the occurrence of genetic disequilibria.

4 a, with D. magna showing the least amount of disequilibria.

5 e and genetic drift on cytonuclear genotypic disequilibria.

6 ing of inferences from marker Hardy-Weinberg disequilibria.

7 en the sexes on cytonuclear polymorphism and disequilibria.

8 mates; however, the additional dicytonuclear disequilibria allow more accurate estimates of both form

9 ctions of nucleotide variability and linkage disequilibria among conversion-mediated sites in hsp70Ab

10 versity observed within populations, linkage disequilibria analyses and association indices calculate

11 are fundamental to our understanding of such disequilibria and ascent dynamics.

12 al selection regimes more likely to generate disequilibria and maintain cytonuclear polymorphism and

13 ons provide evidence for substantial genetic disequilibria and nonadditive genetic effects underlying

14 Here we use 210Pb-226Ra-230Th radioactive disequilibria and other geochemical attributes in oceani

15 r's exact test for the genotypic cytonuclear disequilibria and some approximations of the exact test.

16 s of genetic variation (for example, linkage disequilibria) and tests of hypotheses (using simulation

17 tensive genetic diversity, a lack of linkage disequilibria, and little phylogenetic structure, demons

18 mmetry, both nuclear-nuclear and cytonuclear disequilibria are equivalent.

19 Cytonuclear disequilibria are generated de novo in both sexes when b

20 onfirms the prediction that pairwise linkage disequilibria are predominantly generated by migration.

21 Di-genic disequilibria are significant for four of 74 di-locus pa

22 that disease susceptibilityweighted linkage disequilibria are zero, given disease heterogeneity, it

23 btain the dynamics of the sample cytonuclear disequilibria assuming random drift alone as the source

24 a simultaneously that are (1) Hardy-Weinberg disequilibria at each locus, (2) gametic disequilibrium

25 imentally, this study shows that cytonuclear disequilibria at life stages with sex differences can be

26 Where the disequilibria attained by seed and pollen flow are signi

27 Where the disequilibria between cytoplasmic and nuclear genes are

28 nal or paternal parents, and zygotic linkage disequilibria between different loci after the mutation

29 encies, recombination fractions, and linkage disequilibria between different markers.

30 If selection promotes coupling disequilibria between genes of similar effect, recombina

31 but we also derive some results for linkage disequilibria between neutral sites.

32 Linkage disequilibria between TNF and HLA alleles were calculate

33 However, linkage disequilibria built up by correlational selection are ex

34 A mantle-melting model can simulate observed disequilibria but preservation requires a subsequent mec

35 Additionally, three-locus disequilibria can be generated by higher-order intermigr

36 We show analytically that cytonuclear disequilibria can be generated de novo if the cytoplasmi

37 ial levels of permanent two- and three-locus disequilibria can be generated in adults by (i) nonzero

38 ure type individuals and nonzero cytonuclear disequilibria can be maintained within a hybrid zone if

39 and small in magnitude, measurable permanent disequilibria can result from selective differences both

40 Disequilibria cannot be created or maintained, and heter

41 w patterns of allele frequencies and linkage disequilibria change over time.

42 ining timescales derived from uranium-series disequilibria, crystal sizes and trace-element zoning in

43 iii) readily creates permanent host-symbiont disequilibria de novo, whereas uniform transmission can

44 missense variant (S1370A), but these linkage disequilibria did not differ between the NIDDM and contr

45 icates that stochastically generated linkage disequilibria do select for increased recombination, a r

46 expected values of the cytonuclear genotypic disequilibria for both the homozygotes and heterozygotes

47 The large (238)U-(234)U-(230)Th disequilibria for some of the glasses, along with the wi

48 ate between loci associations due to linkage disequilibria from those caused in other ways can render

49 Although previous work suggests that the disequilibria generated by cytonuclear selection may be

50 age are also taken into account, the gametic disequilibria generated by the Bulmer and Hill-Robertson

51 We also find that patterns of linkage-disequilibria in admixed Hispanic/Latino populations are

52 and seeds coupled with nuclear-mitochondrial disequilibria in migrant seeds, or different nuclear fre

53 seeds coupled with mitochondrial-chloroplast disequilibria in migrant seeds.

54 utline an exact test for allelic cytonuclear disequilibria in multiallelic systems.

55 rate allele frequency changes or cytonuclear disequilibria in populations with constant viability sel

56 ia can be generated in adults by (i) nonzero disequilibria in the migrant pools or (ii) intermigrant

57 of the statistical properties of cytonuclear disequilibria in two major ways.

58 The full bounds are derived for cytonuclear disequilibria in two-locus systems with an arbitrary num

59 ification of phenotypic traits suggests that disequilibria inherent to viral populations may provide

60 Such significant disequilibria involving the paternally inherited organel

61 This pattern of disequilibria is consistent with the action of genetic d

62 Few significant linkage disequilibria (LDs) occur between the genome-wide CNV lo

63 uclear equilibrium structure, including when disequilibria may be indicators of gene flow.

64 Approximate normality of the disequilibria measures are also demonstrated by Monte Ca

65 double heterozygote, gametic and nongametic disequilibria need to be combined into a composite digen

66 The likelihood of magmatic disequilibria occurring before melt enters shallow crust

67 Second, our method can detect marker-QTL disequilibria of different orders and QTL epistatic inte

68 fitness interactions in males generate male disequilibria only.

69 selfing, or migrant pollen and seeds lacking disequilibria or intermigrant admixture effects.

70 simulations demonstrate that the majority of disequilibria produced by random selection are transient

71 Mutations promoting rotational disequilibria showed catalytic, biochemical and translat

72 quilibrium specifies five different types of disequilibria simultaneously that are (1) Hardy-Weinberg

73 n of DNA marker heterozygosities and linkage disequilibria that are likely to resemble those expected

74 ent, the virus exhibited internal population disequilibria that did not decline with increased adapta

75 ncrease recombination eliminate the negative disequilibria that impede selection and consequently inc

76 zygosities and low within-population linkage disequilibria) that differs qualitatively from the highl

77 investigate the potential magnitude of such disequilibria, their qualitative dynamics, the expected

78 nitude and temporal dynamics of cyto-nuclear disequilibria through time.

79 Our analysis introduces interspecific disequilibria to quantify nonrandom associations between

80 We use host-symbiont disequilibria to quantify the role played by non-random

81 In this paper we use cytonuclear disequilibria to test the neutrality of mtDNA markers.

82 namics, the expected frequency of detectable disequilibria under particular patterns or strengths of

83 The dynamics of the variances of these disequilibria under the random drift model are studied b

84 rns of nuclear and cytonuclear associations (disequilibria) under various models of migration.

85 when nonadditive genetic effects and genetic disequilibria underlie a genetic system, genetic slippag

86 e develop a test statistic using cytonuclear disequilibria via the theory of generalized least square

87 At extreme disequilibria, we observed the onset of reaction front i

88 quilibrium systems initiates parent-daughter disequilibria, which re-equilibrate by the shorter half-

89 to define normalized measures of cytonuclear disequilibria, whose practical utility is illustrated th

90 or reflected their tight linkage respecting disequilibria with other class I variants.

91 lic frequencies of markers and their linkage disequilibria with QTL, because the probabilities of QTL

92 requencies of putative QTL and their linkage disequilibria with the markers are estimated by solving

93 genotypic diversity and evidence of genetic disequilibria, with D. magna showing the least amount of

94 Significant linkage disequilibria within and between these genes were also d