ライフサイエンスコーパス: disequilibrium

1 ccounting for shared variance due to linkage disequilibrium).

2 le when some of the SNPs are in high linkage disequilibrium.

3 heir effect sizes in the presence of linkage disequilibrium.

4 an anti-histamine agent in the management of disequilibrium.

5 limited dissolution of calcite under extreme disequilibrium.

6 confounding associations as well as linkage disequilibrium.

7 d the thousands of surrogate SNPs in linkage disequilibrium.

8 c loci do not systematically exploit linkage disequilibrium.

9 lele frequencies and a fast decay of linkage disequilibrium.

10 allele combinations co-occur in high linkage disequilibrium.

11 type values across samples, known as linkage disequilibrium.

12 tromeric KIR region and are in tight linkage disequilibrium.

13 and were independent with respect to linkage disequilibrium.

14 iplicity of infection (2.7), and low linkage disequilibrium (500-bp) were observed in Chikhwawa Distr

15 also displays consistent patterns of linkage disequilibrium across African populations and has signal

16 ion is often prevented by the strong linkage disequilibrium across the entire MHC complex.

17 nto distinct strains and substantial linkage disequilibrium across the genome(1,2).

18 it leads to strong correlations and linkage disequilibrium across very distant sites in the genome.

19 m the Hardy-Weinberg equilibrium and linkage disequilibriums after applying Bonferroni correction.

20 with methods based on the admixture linkage disequilibrium (ALD) is to remove the effect of source L

21 is dependent on many factors (e.g., linkage disequilibrium, allele frequencies, genetic architecture

22 Complex structure of linkage disequilibrium also makes it challenging to separate cau

23 S) summary data while accounting for linkage disequilibrium among variants.

24 nd inherently takes into account the linkage disequilibrium among variants.

25 ic, nonparametric, joint linkage and linkage disequilibrium analyses using a microsatellite marker pa

26 This result was supported by transmission disequilibrium analyses using a subset of 541 case famil

27 Local linkage disequilibrium analysis and allele mining identified pos

28 kage that performs joint linkage and linkage disequilibrium analysis between a marker and a putative

29 In particular, the differences in linkage disequilibrium and allele frequency patterns across ethn

30 enetic estimates of Ne computed from linkage disequilibrium and approximate Bayesian computation were

31 six novel SNPs in PRPF6 were in high linkage disequilibrium and associated with PRPF6 mRNA expression

32 otide polymorphisms (SNPs) in strong linkage disequilibrium and comprising a novel G3 haplotype.

33 Linkage disequilibrium and conditional analyses indicate that si

34 fectionate bonds may contribute to emotional disequilibrium and confer elevated risk for the onset of

35 on, Manhattan plot visualization for linkage disequilibrium and eQTL data, and an ontology search for

36 de Polymorphisms (SNPs) according to linkage disequilibrium and P-value or use all SNPs, handles geno

37 ased on flux assessments from (238)U:(234)Th disequilibrium and sediment traps, we found 2 to 3 times

38 By taking into account linkage disequilibrium and sparseness of the data, the proposed

39 ecause of the uncertainty induced by linkage disequilibrium and the fact that some loci harbor multip

40 near SLC2A9 that were not driven by linkage disequilibrium and were replicated in FHS.

41 sal could be explained by breakup of linkage disequilibrium, and direct selection on wing shape is al

42 ed polymorphism, unusual patterns of linkage disequilibrium, and lower levels of population different

43 ects, single variants tagged through linkage disequilibrium, and population stratification.

44 s, hence confirming previous data of linkage disequilibrium as a cause for disease association.

45 mating type; IA(s) values show high linkage disequilibrium as is expected in clonal reproduction.

46 four other SNPs in high-to-moderate linkage disequilibrium as the most likely causal SNPs.

47 -Gly85-Pro86-Met87) in near-complete linkage disequilibrium at the edge of the peptide-binding groove

48 tion, we also detected long-distance linkage disequilibrium at two underlying loci, GS-OH and GS-ELON

49 Using a linkage disequilibrium-based analysis, individual sampling (i.e.

50 siderable reduction in power for all linkage disequilibrium-based statistics.

51 e examine various mechanisms that may induce disequilibrium behaviour in modern seep carbonates, and

52 ignal may be delayed due to the geographical disequilibrium between climate and tree populations.

53 eraged differences in the pattern of linkage disequilibrium between diverse populations to fine-map t

54 ging differences in the structure of linkage disequilibrium between diverse populations, and increase

55 ts and variation in the structure of linkage disequilibrium between ethnicities.

56 networks in an NMD mutant could be linked to disequilibrium between functional and nonsense mRNAs.

57 how hexokinase and mTORC1 cooperate to sense disequilibrium between glucose uptake and utilization an

58 that have no shared markers based on linkage disequilibrium between loci appearing in different datas

59 ce computational burden and to limit linkage disequilibrium between single-nucleotide polymorphisms (

60 lincRNAs, we examined the pattern of linkage disequilibrium between SNPs in the lincRNAs and SNPs tha

61 Restricted recombination may promote linkage disequilibrium between the colour locus and incompatibil

62 We also find linkage disequilibrium between the inverted region and the early

63 rong enough to establish significant linkage disequilibrium between the mitochondrial and nuclear gen

64 iatric PTSD is accompanied by a connectivity disequilibrium between the salience and default-mode net

65 rm a high-resolution genome scan for linkage disequilibrium between unlinked genomic regions in natur

66 ial/ethnic groups creates long-range linkage disequilibrium between variants with different allelic f

67 onservative and fails to account for linkage disequilibrium between variants.

68 and detected an approximately 33-kb linkage disequilibrium block (containing the lead SNP rs965513)

69 all six environments and tagged to a linkage disequilibrium block comprising two promising candidate

70 e chromosome 12E signal to a 1.95 Mb linkage disequilibrium block containing only one gene, sel-1 sup

71 Only 1 gene is found in the linkage disequilibrium block containing rs9828519: SLC9A9.

72 itive sites overlapping the rs874040 linkage disequilibrium block in human memory, but not in naive C

73 A linkage disequilibrium block of polymorphisms located in the HLA

74 nd that are also sole members of the linkage disequilibrium block surrounding a PGC-SCZ GWAS hit.

75 ity) a priori candidate genes within linkage disequilibrium blocks for these loci.

76 tially methylated regions within the linkage disequilibrium blocks of the single nucleotide polymorph

77 e same pH (3.3) and comparable thermodynamic disequilibrium but different equilibrated pressures of C

78 ave low genetic variability and high linkage disequilibrium, but relatively few autozygous segments a

79 o and uses a model that accounts for linkage disequilibrium by explicitly modeling haplotype frequenc

80 e through colonization of habitats in severe disequilibrium by pre-adapted individuals, rather than b

81 ve sweep, characteristic patterns of linkage disequilibrium can arise in the genomic region surroundi

82 from 3 breeds to compare the SNP and linkage disequilibrium characteristics together with the power a

83 these findings indicate the predominance of disequilibrium clumped isotope behaviour in modern cold

84 iate most of the risk, but extensive linkage disequilibrium complicates the localization of independe

85 bit a clear gradient of short--range linkage disequilibrium consistent with a Central Asian domestica

86 trong relation between magma ascent rate and disequilibrium crystallization and exsolution plays a ke

87 VDR SNPs (rs7968585 and rs731236) in linkage disequilibrium (D' = 0.98; r2 = 0.6).

88 We estimate, using linkage-disequilibrium decay patterns, that admixture occurred 6

89 e in southern Chad and estimate from linkage-disequilibrium decay that this occurred 4,750-7,200 year

90 ication which, combined with a rapid linkage disequilibrium decay, makes it difficult to perform geno

91 We found that linkage-disequilibrium decays at 100 Kb in this collection and

92 Linkage disequilibrium decays within 10 kb (based on the provisi

93 Genome-wide linkage disequilibrium declined more slowly for the commercial p

94 re selected based on the short-range linkage disequilibrium distance, which is inherent with long bre

95 rring hard selective sweeps based on linkage disequilibrium distortions under different conditions, i

96 Two patients had a rapid progression with disequilibrium, dysarthria, dysphagia, and central hypov

97 mple lag hypotheses have become prominent in disequilibrium ecology, proposing that communities track

98 functional annotations and allow for linkage disequilibrium estimated from reference genotype data.

99 global array of ocean-atmosphere radiocarbon disequilibrium estimates to demonstrate a approximately

100 eristics such as allele frequencies, linkage disequilibrium etc., is an integral component of many st

101 ed 3 common genetic variants in high linkage disequilibrium for severe pre-treatment pain, representi

102 We used two methods (patterns of linkage disequilibrium from whole-genome SNPs and MSMC estimates

103 because it prevents the breakdown of linkage disequilibrium generated by migration; the selective adv

104 and colouration, such as fine-scale linkage disequilibrium, genomic rearrangements and pleiotropy, t

105 Patterns of linkage disequilibrium, homoplasy, and incompatibility are diffi

106 gulatory variants in regions of high linkage disequilibrium identified by expression quantitative tra

107 49694C>T) exhibited a high degree of linkage disequilibrium in all test populations.

108 Given the small blocks of linkage disequilibrium in Drosophila, we obtain near base-pair r

109 he adult heart is particularly vulnerable to disequilibrium in homeostasis because its regenerative a

110 sh a link between GSH deficiency and Th1/Th2 disequilibrium in LP-BM5 infection and indicate that I-1

111 isms of selection that maintain high linkage disequilibrium in MHC haplotype blocks.

112 approximately independent blocks of linkage disequilibrium in the human genome.

113 We assessed the ability of histones to cause disequilibrium in the redox status and intracellular [Ca

114 odel, occasional clonality generates linkage disequilibrium in the short term.

115 We found two SNPs in strong linkage disequilibrium in the SLC19A3 locus associated with a re

116 Owing to complex patterns of linkage disequilibrium in this region, it is unclear whether the

117 n hampered by the presence of strong linkage disequilibrium in this region.

118 1980s, through the hypothesized creation of disequilibrium in water-sediment balance following decou

119 d genetic maps that capture detailed linkage disequilibrium information in European and African Ameri

120 Linkage disequilibrium information was used to identify possible

121 In addition, by taking strong linkage disequilibrium into account, another 47 IBD-associated S

122 select important SNPs by taking the linkage disequilibrium into account.

123 d by fire suppression will remain in climate disequilibrium into the foreseeable future.

124 Linkage disequilibrium is positive (Dij'>0) among frequent haplo

125 e number of significant SNPs in high linkage disequilibrium (LD) (r(2) > 0.8) with rs3865444 to just

126 Assessing linkage disequilibrium (LD) across ancestral populations is a po

127 he true biological representation of linkage disequilibrium (LD) among multiple loci.

128 Linkage disequilibrium (LD) analysis indicated strong LD among S

129 Meiotic recombination breaks down linkage disequilibrium (LD) and forms new haplotypes, meaning th

130 s with minor allele frequency (MAF), linkage disequilibrium (LD) and genotype certainty.

131 nt effect sizes while accounting for linkage disequilibrium (LD) and overlapping GWAS samples.

132 The linkage disequilibrium (LD) based quantitative trait loci (QTL)

133 We demonstrate that heterogeneity in linkage disequilibrium (LD) between causal variants and SNPs bia

134 stence of moderate to high levels of linkage disequilibrium (LD) between genetic markers and quantita

135 effective population size (Ne) from linkage disequilibrium (LD) between unlinked pairs of genetic ma

136 oaches require information about the linkage disequilibrium (LD) between variants, there has not been

137 nriched in approximately independent linkage disequilibrium (LD) blocks (e.g. MHC).

138 The presence of conserved linkage disequilibrium (LD) blocks and haplotypes between G. hir

139 et genes and often extend beyond the linkage disequilibrium (LD) blocks containing risk SNPs identifi

140 Length of individual linkage disequilibrium (LD) blocks varied along chromosomes, wit

141 across multiple individuals based on linkage disequilibrium (LD) can facilitate the analysis of low t

142 -wide association studies (GWAS) and linkage disequilibrium (LD) data from a reference sample with in

143 While linkage disequilibrium (LD) decay (r(2) = 0.2) was lowest in the

144 ed strategies that integrate linkage-linkage disequilibrium (LD) mapping in Populus.

145 ublicly available GWAS associations, linkage disequilibrium (LD) measures, functional genomic and var

146 ion size (Ne) can be estimated using linkage disequilibrium (LD) observed across pairs of loci presum

147 sed an algorithm that considers both linkage disequilibrium (LD) patterns and familial transmission i

148 ene statistics are constructed using linkage disequilibrium (LD) patterns from a relevant reference p

149 ethnic comparison revealed different linkage disequilibrium (LD) patterns in HLA-DOA and HLA-DRB1, ex

150 ence requires leveraging the complex linkage disequilibrium (LD) patterns in the cohort to compensate

151 GWAS signals to gene-dense and high linkage disequilibrium (LD) regions, and correlations of gene si

152 This study used the linkage disequilibrium (LD) score regression and genomic profile

153 tations (e.g., exon or 5'UTR), total linkage disequilibrium (LD) scores and heterozygosity to constru

154 With differences in linkage disequilibrium (LD) structure and allele frequencies bet

155 Based on statistical evidence and linkage disequilibrium (LD) structure, we have curated and narro

156 mapping resolution due to extensive linkage disequilibrium (LD) that is characteristic of crosses am

157 The analysis of linkage disequilibrium (LD) underpins the development of effecti

158 association studies, are usually in linkage disequilibrium (LD) with each other within a small genom

159 A total of 2255 variants in linkage disequilibrium (LD) with GWAS identified SU/gout associa

160 ants that represent or are in strong linkage disequilibrium (LD) with previously-identified signals a

161 dditional SNP, IL17RA rs41433045, in linkage disequilibrium (LD) with rs41396547, was associated with

162 By exploiting linkage disequilibrium (LD), a real data application demonstrate

163 genetic information sources, namely, linkage disequilibrium (LD), co-segregation (CS) and pedigree re

164 markers, often by pruning markers in Linkage Disequilibrium (LD), ignoring the information contained

165 ssociation studies that benefit from linkage disequilibrium (LD), the main challenge in identifying c

166 genome-wide variants to account for linkage disequilibrium (LD), thus prioritizing associations base

167 dividual-based, population-based and linkage disequilibrium (LD)-aware methods with stringent quality

168 ge sequencing has been combined with linkage-disequilibrium (LD)-based genotype refinement to accurat

169 for calculating risk scores involves linkage disequilibrium (LD)-based marker pruning and applying a

170 Recent work has hinted at the linkage disequilibrium (LD)-dependent architecture of human comp

171 d on SNP prior information including linkage disequilibrium (LD)-weighted genic annotation scores, to

172 1982 measures of between-population linkage disequilibrium (LD).

173 aplotypes) due to the development of linkage disequilibrium (LD).

174 tionship between test statistics and linkage disequilibrium (LD).

175 d to the true causal variant through linkage disequilibrium (LD).

176 can be further reduced based on the linkage disequilibrium (LD).

177 ed on hierarchical representation of linkage disequilibrium (LinDen).

178 Nevertheless, linkage disequilibrium makes it difficult to define, without fun

179 Mapping by admixture linkage disequilibrium (MALD) is a whole genome gene mapping met

180 s method is referred as multi-marker linkage disequilibrium mapping (mmLD).

181 Vegetation-climate disequilibrium may persist for several millennia, relate

182 at rs744373 itself or a variation in linkage disequilibrium may provide a neurogenetic mechanism for

183 The textures mirror those developed by disequilibrium melting induced via rapid heating during

184 ference in adMixed Populations using Linkage Disequilibrium method (LAMP-LD) and evaluated the associ

185 structed and combined with locus ordering by disequilibrium methodology to generate an integrated gen

186 Non-parametric-linkage analysis, linkage-disequilibrium-model analysis, single-SNP analysis, clus

187 L1; the latter is a region with high linkage disequilibrium) nearest to these SNPs has previously bee

188 lation genetic variation, background linkage disequilibrium, number of ancestral populations, between

189 1 traits could be linked through the linkage disequilibrium of 115 associated loci and these links re

190 Because of the strong linkage disequilibrium of A*01 and B*08 alleles with the DR3-DQ2

191 sclerosis (MS) risk, the ubiquitous linkage disequilibrium operating across the genome has stalled e

192 The magma was extracted at low degrees of disequilibrium partial melting of the UPB mantle.

193 user's dataset, as well as visualize linkage disequilibrium pattern, infer haplotypes and design tagS

194 Comparison of linkage disequilibrium patterns between the 13 lead SNPs and six

195 ssess the extent to which the unique linkage disequilibrium patterns in African Americans can contrib

196 iversity, haplotype distribution and linkage disequilibrium patterns in the G. hirsutum and G. barbad

197 cific loci may exist due to distinct linkage disequilibrium patterns.

198 Using these new constraints on disequilibrium plagioclase crystallization we also repro

199 hat basaltic magmas may rise fast enough for disequilibrium processes to play a key role on the ascen

200 entified 44 of these Alu elements in linkage disequilibrium (r(2) > 0.7) with the trait-associated SN

201 First, all known genes in strong linkage disequilibrium (r(2) > 0.8) with the reported single nuc

202 that though these SNPs were in high linkage disequilibrium (r(2) > 0.8), the rare alleles of these S

203 We retrieved all SNPs in linkage disequilibrium (r(2) >/= 0.2) with the glioma-associated

204 ourteen noncoding variants in strong linkage disequilibrium (r(2) >/= 0.8) with rs4888378 were identi

205 tide polymorphism (SNP) is in strong linkage disequilibrium (r(2) = 0.90, D' = 0.96) with the previou

206 SNP rs8041357, which is in complete linkage disequilibrium (r(2) = 1) with rs11543198, was also asso

207 SNPs, which are in moderate to high linkage disequilibrium (r(2)>0.5) with the GWAS SNPs.

208 ngle-nucleotide polymorphism in high linkage disequilibrium (r(2)=0.7) with rs10995, which both could

209 ulation support the view that accounting for disequilibrium range dynamics will be essential for real

210 tive population sizes estimated from linkage disequilibrium ranged from 88 to 825.

211 Linkage disequilibrium rapidly decayed around LanFTc1, suggestin

212 We used linkage disequilibrium regression and polygenic profile scoring

213 Using a new method, linkage disequilibrium regression, we derived genetic correlatio

214 Among them, two SNPs in strong linkage disequilibrium, rs7676822 and rs1911877, located near th

215 Linkage disequilibrium score regression and polygenic profile sc

216 Linkage disequilibrium score regression of 220 cell types implic

217 Linkage disequilibrium score regression was used to calculate ge

218 Using linkage disequilibrium score regression, we estimate the genetic

219 tudy statistics and to the method of linkage disequilibrium score regression.

220 c correlations were calculated using linkage disequilibrium score regression.

221 rbonates studied here also exhibit potential disequilibrium signals.

222 te set and conducted association and linkage disequilibrium (standardized index of association, IA(s)

223 ecreasing over time as niches are filled, or disequilibrium states and progression rules, where richn

224 y affected by selection, calculating linkage disequilibrium statistics, performing haplotype reconstr

225 tes of nucleotide diversity metrics, linkage disequilibrium statistics, recombination rates, a batter

226 oach properly takes into account the linkage-disequilibrium structure among variants, and its time co

227 Differences in linkage disequilibrium structure and heterogeneity in allelic ef

228 llele frequency, effect size and the linkage disequilibrium structure of credible set variants with t

229 We further show that the strong linkage disequilibrium structure within the human MHC that typic

230 nts, which was observed to depend on linkage disequilibrium structure.

231 evident for regions having extensive linkage disequilibrium such as the IBD5 locus.

232 and was not exclusively a result of linkage disequilibrium, suggesting that multiple HLA epitopes ma

233 o cerebellar ataxia, mental retardation, and disequilibrium syndrome 2, cause severe congenital hydro

234 3 single-nucleotide polymorphisms in linkage disequilibrium: T/A at -663, T/C at -470, and C/T at -34

235 Linkage disequilibrium tagging polymorphisms and polymorphisms o

236 developed a RV extension of the generalized disequilibrium test (GDT) to analyze sequence data obtai

237 these, based on significance in the sibship disequilibrium test (P<0.05) or protein-altering propert

238 sed to construct a rare-variant transmission disequilibrium test (rvTDT) in the case-parent data.

239 The transmission disequilibrium test (TDT) is a powerful family-based ass

240 t-child trio data, by using the transmission disequilibrium test (TDT), which is robust to population

241 l approach called the polygenic transmission disequilibrium test and data from 6,454 families with a

242 sion in NSOFC cases through the transmission disequilibrium test and through analyses of the family-b

243 statistical significance in the transmission-disequilibrium test in the full cohort (rs112439957: P =

244 Transmission disequilibrium testing was performed for affection statu

245 Transmission/disequilibrium tests (TDT) based on family designs are r

246 c HLA alleles identified two in high linkage disequilibrium that are associated with fIIP (DRB1*15:01

247 abundance in the plume signals thermodynamic disequilibrium that favors the formation of methane from

248 Frequent recombination limits linkage disequilibrium to about 100 bp in most of the genome, bu

249 selection, haplotype frequencies and linkage disequilibrium to estimate the effects of both selection

250 A GWAS risk loci, and adjustment for linkage disequilibrium to propose target genes of immune cell en

251 sing rapidly and drastically reduced linkage disequilibrium to very low levels even at short map dist

252 In modern seeps, this range of disequilibrium translates into apparent temperatures tha

253 propriately controlling for expected linkage disequilibrium using a genetic map.

254 Strong linkage disequilibrium was observed across a 200 kb region spann

255 We propose that disequilibrium was stronger during the Plio-Pleistocene

256 Dysarthria and disequilibrium were common in patients with bilateral st

257 uring mature mRNA levels and in high linkage disequilibrium with 65 lead type 2 diabetes GWAS SNPs, w

258 usly screen 2,756 variants in strong linkage disequilibrium with 75 sentinel variants associated with

259 This variant is in high linkage disequilibrium with a known functional variant in the UG

260 associates with two polymorphisms in linkage disequilibrium with a known IBD susceptibility variant.

261 sion of HLA-A RNA in vivo, in strong linkage disequilibrium with an HLA-A allele that confers *02:01

262 ariant, rs11556924, is not in strong linkage disequilibrium with any other variant and introduces a c

263 Both SNPs were in strong linkage disequilibrium with asthma-associated 17q21 SNPs previou

264 e trait locus of complex trait is in linkage disequilibrium with at least one marker.

265 didate genes; four SNPs were in high linkage disequilibrium with candidate genes within 366 kb.

266 causal and those that are merely in linkage-disequilibrium with causal mutations.

267 e-nucleotide polymorphisms in strong linkage disequilibrium with causative polymorphisms that can be

268 These polymorphisms were in high linkage disequilibrium with each other (r(2) = 0.96) meaning tha

269 analyses identified multiple SNPs in linkage disequilibrium with each other that were significantly a

270 rCa-risk SNPs, including all SNPs in linkage disequilibrium with each risk SNP, resulting in 100 uniq

271 polymorphism rs1295686 (in complete linkage disequilibrium with functional variant rs20541) is assoc

272 equence a small number of regions in linkage disequilibrium with heading date QTL in thousands of sam

273 2.35 x 10(-9)) which was in moderate linkage disequilibrium with HLA-B*27:05.

274 Inhibitory KIR2DL5A, carried in linkage disequilibrium with KIR2DS1, is expressed by peripheral

275 relevant candidate loci that are in linkage disequilibrium with MICA*008 i.e. HLA-B*08:01, rs3131619

276 s identified, two thirds were not in linkage disequilibrium with nearby SNPs, implicating these varia

277 IL23R, PTGER4, and SNX20 (in strong linkage disequilibrium with NOD2) for CD; and KCNQ2 (near TNFRSF

278 xcept the more than 100 GMAS SNVs in linkage disequilibrium with polymorphisms reported by genome-wid

279 P = 1.4 x 10(-9)), and was in strong linkage disequilibrium with rs10498635.

280 HI, but only rs12243326 is in strong linkage disequilibrium with rs12255372 in our Hispanic populatio

281 Rs3865444 is in linkage disequilibrium with rs12459419 which has been associated

282 intron 8 of the gene, is in complete linkage disequilibrium with rs17026688 and is predicted to affec

283 Marker rs2238448 in ADCY9, in linkage disequilibrium with rs1967309 (r(2)=0.8), was associated

284 m in the ADCY9 gene, the majority in linkage disequilibrium with rs1967309, were associated with the

285 tter residual cognition is in strong linkage disequilibrium with rs1990622A (r2 = 0.66), a previously

286 SNP rs2227473, in linkage disequilibrium with rs2227476, was also associated with

287 9860 polymorphism, which was in high linkage disequilibrium with rs368234815 (R(2) = 0.87).

288 f single-nucleotide polymorphisms in linkage disequilibrium with rs61183828 was located close to two

289 variant-tagging SNP (rs117026326; in linkage disequilibrium with rs73366469), whose minor allele is c

290 olymorphism (SNP) rs2235749 (in high linkage disequilibrium with rs910080) modifies striatal PDYN exp

291 ism is observed in high species-wide linkage disequilibrium with sex.

292 rait-associated SNPs to be in strong linkage disequilibrium with SVs and demonstrate that our panel f

293 thic and soil environments at profound redox disequilibrium with the atmosphere, such as biological s

294 t RNA splicing, but it was in strong linkage disequilibrium with the G allele of the promoter polymor

295 Both were in low linkage disequilibrium with the genome-wide association study si

296 R = 1.63), which is in near complete linkage disequilibrium with the HLA-DRB1*07:01 allele we previou

297 e trait locus (cis-eQTL) variants in linkage disequilibrium with the index variant in 29 of the 41 PD

298 long-range meQTL was found to be in linkage disequilibrium with the most replicated locus of multipl

299 de polymorphism rs6800541 is in high linkage disequilibrium with the nonsynonymous variant in SCN10A,

300 be causal relative to nearby SNPs in linkage disequilibrium with them.