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1 ctious mononucleosis and lymphoproliferative disorder.
2 ain regions are implicated in leading to the disorder.
3 ological mechanism for post-traumatic stress disorder.
4 cortical event that underlies a neurological disorder.
5 Ialpha linked to a specific neuropsychiatric disorder.
6 be understood as simply being due to cation disorder.
7 ipidosis type IV, a severe lysosomal storage disorder.
8 mes in mice with moderate amyloid pathologic disorder.
9 logical strategies to treat major depressive disorder.
10 eutic target for the treatment of opioid use disorder.
11 s are influenced by covalent and noncovalent disorder.
12 nsory hyperactivity in post-traumatic stress disorder.
13 de new insights into the pathogenesis of the disorder.
14 (SGAs) versus most other treatments for this disorder.
15 egments of low sequence complexity/intrinsic disorder.
16 ging studies of dopamine function in bipolar disorder.
17 e useful in the treatment of dynamin-related disorders.
18 the pathogenesis and treatment of affective disorders.
19 lying mechanisms involved in KIR2DS1-related disorders.
20 tive medicine for patients with white matter disorders.
21 minants in mPFC for stress-associated mental disorders.
22 re shared mechanisms of pathogenesis between disorders.
23 c shifts, and progression to more aggressive disorders.
24 ) has long been implicated in cardiovascular disorders.
25 g to neurodevelopmental and neuropsychiatric disorders.
26 tibility versus resilience to stress-related disorders.
27 tudies to inform the treatment of these rare disorders.
28 e of protein misfolding in neurodegenerative disorders.
29 G inhibitors targeting chronic inflammatory disorders.
30 mmense potential to treat a range of genetic disorders.
31 icant potential for therapy of neuromuscular disorders.
32 rse health outcomes, including substance use disorders.
33 (AD), as well as in other neurodegenerative disorders.
34 orial ASOs for the treatment of neurogenetic disorders.
35 lay a major role in the etiology of epilepsy disorders.
36 th impairment of cognitive function and mood disorders.
37 cortex (MCCs) are devastating developmental disorders.
38 h numerous diseases, especially neurological disorders.
39 rm-born children suffer from neurobehavioral disorders.
40 acological targets for the treatment of mood disorders.
41 e pathogenesis of B-cell lymphoproliferative disorders.
42 ing is a key feature of various inflammatory disorders.
43 AS) events have been associated with several disorders.
44 ion and its disturbances in neuropsychiatric disorders.
45 athogenesis in a subset of neurodegenerative disorders.
46 vancing our understanding of substance abuse disorders.
47 and predicting progression of mental health disorders.
48 ho are at risk for developing trauma-related disorders.
49 th their association with neurodevelopmental disorders.
50 in the molecular genetic dissection of brain disorders.
51 e the regulation of iron homeostasis and its disorders.
52 are associated with bleeding and thrombotic disorders.
53 and potentially for other neurodegenerative disorders.
54 n with microcephaly and serious neurological disorders.
55 onal diabetes, hypertension, and psychiatric disorders.
56 apeutics for affected patients with bleeding disorders.
57 ypes in zebrafish models of human congenital disorders.
58 cells for treatment of neurogastrointestinal disorders.
59 a non-pharmacological alternative for these disorders.
60 udes new criteria for the diagnosis of these disorders.
61 uman neurodegenerative or neurodevelopmental disorders.
62 f mental disorders, particularly OCD and tic disorders.
63 a series of small probes and the compact but disordered 4E binding protein 2 (4E-BP2) protein in [For
64 Diagnostic and Statistical Manual of Mental Disorders-5 opioid use disorder, and had used non-prescr
65 Health Questionnaire-9, Generalised Anxiety Disorder-7 scale, Alcohol Use Disorder Identification Te
66 ved among adolescents with past-year bipolar disorder (94.2 [1.69]; P = .004), attention-deficit/hype
67 ; P = .004), attention-deficit/hyperactivity disorder (96.3 [0.91]; P = .002), oppositional defiant d
68 nt disorder (97.3 [0.66]; P = .007), conduct disorder (97.1 [0.82]; P = .02), substance use disorders
69 96.3 [0.91]; P = .002), oppositional defiant disorder (97.3 [0.66]; P = .007), conduct disorder (97.1
70 e of Cdc42 bound to ACK via an intrinsically disordered ACK region to guide an analysis of the Cdc42
76 ast-year and lifetime psychiatric disorders, disorder age at onset, and disorder severity in a nation
77 sorder (97.1 [0.82]; P = .02), substance use disorders (alcohol abuse, 96.5 [0.67]; P < .001; drug ab
78 during permeabilization indicated acyl chain disordering along with decreased intensity of the cardio
79 p in high-symptom trajectories of 1) conduct disorder and 2) hyperactivity throughout childhood, iden
80 a feature (eg, neuromyelitis optica spectrum disorder and acute disseminated encephalomyelitis) and f
81 ive evidence that ARPKD is not a homogeneous disorder and further establish DZIP1L as a second gene i
87 4a and 24b) appears to be important for mood disorders and constitutes a neuroanatomical substrate fo
88 individuals with acquired and innate immune disorders and is strongly associated with Hodgkin's lymp
89 contribute to multiple aspects of autoimmune disorders and may play a role in triggering disease.
91 485 respondents with schizophrenia spectrum disorders and psychotic mood disorders, and associations
92 ed treatment approaches for mood and anxiety disorders and suggest a potentially wider therapeutic ov
93 s a major limitation in the study of biliary disorders and the testing of novel therapeutic agents.
94 d mechanisms involved in the above-mentioned disorders and to help medicinal chemists to design novel
96 ical Manual of Mental Disorders-5 opioid use disorder, and had used non-prescribed opioids in the pas
97 al disorder, or affective-spectrum psychotic disorder, and psychotic symptoms scoring at least 4 on a
98 hrenia spectrum disorders and psychotic mood disorders, and associations of the empirically derived t
99 children are affected by neurodevelopmental disorders, and polybrominated diphenyl ethers (PBDEs) in
100 ns and oligodendrocytes in neurodegenerative disorders, and provide opportunities for the development
101 roach for treating stress-induced behavioral disorders, and that dynamics are a critical axis of mani
103 Many genetic variants associated with sleep disorders are also implicated in neurological disorders
104 kely explains why certain neurodevelopmental disorders are characterized by weak gamma activity despi
106 tribute to many different disorders but most disorders are influenced by many etiologic factors.
107 tinal detachment and vitreoretinal interface disorders, as well as potential future prophylactic and
108 are strongly associated with autism spectrum disorder (ASD), attention deficit hyperactivity disorder
109 e important risk factors for autism spectrum disorder (ASD), developmental delay, and infantile seizu
110 RE) cohorts of children with autism spectrum disorder (ASD), we find strong statistical significance
115 ave therapeutic potential in autism spectrum disorders (ASD), including tuberous sclerosis complex (T
118 ue connect across scales such that patterned disorder at the cellular level leads to the emergence of
119 ns in medication development for alcohol use disorder (AUD) emphasize the need to identify novel mole
122 diagnosis of schizophrenia, schizoaffective disorder, bipolar disorder, or other psychotic illness a
123 the complex interrelationships between sleep-disordered breathing and cardiovascular disease, present
124 al risk factors contribute to many different disorders but most disorders are influenced by many etio
126 polymerizing GTPase domain, an intrinsically disordered C-terminal linker (CTL), and a C-terminal con
127 GA) is an autosomal recessive neurometabolic disorder caused by a mutation in the l-2-hydroxyglutarat
129 Angelman syndrome (AS) is a neurogenetic disorder caused by deletion of the maternally inherited
130 drome (MFS) is a heritable connective tissue disorder caused by mutations in FBN1, which encodes the
131 yndrome (RTT) is a debilitating neurological disorder caused by mutations in the gene encoding the tr
132 e, are rapidly progressive neurodegenerative disorders caused by misfolding followed by aggregation a
134 splasia (CCD) is an autosomal dominant human disorder characterized by abnormal bone development that
135 Celiac disease (CD) is an immune-mediated disorder characterized by an accumulation of immune cell
136 em Atrophy are age-related neurodegenerative disorders characterized by progressive accumulation of a
139 definitive pathophysiologic link with these disorders, confirm that dogs deficient in HACD1 are rele
140 as increased in bipolar and major depressive disorder, consistent with observations in schizophrenia.
141 type consisting of short, twisted roots with disordered cortical microtubule arrays that are hypersen
144 a diagnosis of ICD-10 schizophrenia-spectrum disorder, delusional disorder, or affective-spectrum psy
146 gent music reward therapy for social anxiety disorder designed to reduce attention dwelling on threat
147 ence with past-year and lifetime psychiatric disorders, disorder age at onset, and disorder severity
148 days of life, while diagnoses of opioid use disorder during this period were uncommon in both groups
149 ome STRs was associated with various genetic disorders (e.g., the Huntington disease), and thus was u
151 DNA repair underlie other neurodegenerative disorders (eg, ataxia-telangiectasia), and DNA double-st
152 y dyskinesia (PCD) is an autosomal recessive disorder frequently caused by non-assembly of dynein arm
154 y of inflammation in fear- and anxiety-based disorders has gained interest as growing literature indi
156 R, 1.15 [95% CI, 1.06-1.25]; autism spectrum disorder hazard ratio [HR], 2.02 [95% CI, 1.80-2.26]; at
157 basis of structural brain alterations in the disorder, highlighting impediments to progress, recent d
158 es with a reduced risk of atopy and allergic disorders; however, limited data are available from low-
159 ation increase the risk and severity of mood disorders; however, only recently have the importance of
161 , 1.01 [95% CI, 0.81-1.25]), autism spectrum disorder (HR, 0.83 [95% CI, 0.62-1.13]), or attention-de
163 alised Anxiety Disorder-7 scale, Alcohol Use Disorder Identification Test (AUDIT), and self-reported
165 with Parkinson's disease and impulse control disorders, impairments are observed across subtypes of d
172 isorders are also implicated in neurological disorders in which sleep abnormalities are common; for e
174 gregates, which are implicated in neurologic disorders including Alzheimer disease and traumatic brai
175 ed with a range of increasingly common human disorders, including Alzheimer and Parkinson diseases, t
177 tion in several neurological and psychiatric disorders, including cognitive decline associated with A
178 ated with pathways implicated in psychiatric disorders, including dopamine neuron differentiation and
179 tments is tightly linked to severe metabolic disorders, including obesity, metabolic syndrome, lipody
180 odomain region was found to be intrinsically disordered independent of the activation state of caspas
182 umatoid arthritis, and devastating monogenic disorders (interferonopathies) characterized by expressi
183 echanism of a novel risk gene for major mood disorders involved in synaptic function and related inte
184 tration and are associated with inflammatory disorders involving marked eosinophilia (e.g. asthma), w
185 idiopathic rapid eye movement sleep behavior disorder (IRBD) patients at risk for short-term developm
186 behavior.SIGNIFICANCE STATEMENT Alcohol use disorder is a chronic relapsing disorder that is associa
190 phatase that is mutated in the developmental disorders Joubert and MORM syndromes, is essential for t
192 ults suggest that cognitive abnormalities in disorders like schizophrenia in which gluatamatergic sig
193 ering new insight into the etiology of human disorders linked to loss of endosomal NHE function.
195 erials form via low-valence linkages between disordered loops protruding from the protein surface.
199 ess disorders, particularly major depressive disorder (MDD), highlighting insights from positron emis
200 ause the neurodegenerative lysosomal storage disorder mucolipidosis type IV, and a gain-of-function m
201 cyte loss, which occurs in the demyelinating disorder multiple sclerosis (MS), contributes to axonal
202 h noncarriers (n=389), children with genomic disorders (n=31) scored significantly poorer on all meas
203 ectrum disorder and other neurodevelopmental disorders (NDDs) are behaviorally and medically heteroge
204 Propagation of coherent light through a disordered network is accompanied by randomization and p
206 al-directed behavior in obsessive-compulsive disorder (OCD) are caused by impaired frontostriatal fun
207 mong Tourette syndrome, obsessive-compulsive disorder (OCD), and attention deficit hyperactivity diso
210 tic protoporphyria (EPP) is a rare inherited disorder of the heme biosynthesis pathway resulting in t
211 t nephrotic syndrome (SRNS), a heterogeneous disorder of the renal glomerular filtration barrier, res
212 December 2014 in 84 eyes of 84 patients with disorders of anterior corneal stroma were correlated to
213 g twice a day), and metabolism and nutrition disorders (one [4%] at 200 mg twice a day and one [7%] a
214 to reduce the treatment gap for alcohol use disorders, one of the leading causes of the global burde
216 patients with schizophrenia, schizoaffective disorder, or a mood disorder who had moderate or severe
217 schizophrenia-spectrum disorder, delusional disorder, or affective-spectrum psychotic disorder, and
218 zophrenia, schizoaffective disorder, bipolar disorder, or other psychotic illness according to the Sw
219 order, specific phobias, generalized anxiety disorder, or separation anxiety and who received CBT, ph
222 of shared environmental factors to the cross-disorder overlap was not statistically significant (11%,
223 ple sclerosis, neuromyelitis optica spectrum disorders, Parkinson disease, Alzheimer disease, Hunting
224 Here, we describe mGluR5 findings in stress disorders, particularly major depressive disorder (MDD),
226 E STATEMENT Anxiety and posttraumatic stress disorder patients generalize fear to nonfearful fear sti
227 etween the coexisting phases by ordering the disordered phase, which could explain their role in cell
229 e proportion meeting criteria for depressive disorder (PHQ-9 score >/=10) at 4- and 12-month follow-u
230 olving a folded protein and an intrinsically disordered protein (IDP), are prevalent in the cell, inc
234 show that tardigrade-specific intrinsically disordered proteins (TDPs) are essential for desiccation
235 protein interactions involving intrinsically disordered proteins are important for cellular function
237 red a state similar to that of intrinsically disordered proteins, resulting in a predominant form of
238 to traumatic stress on post-traumatic stress disorder (PTSD), this study examined longitudinal change
244 , which has important implications for human disorders related to primary cilia dysfunctions, such as
249 derstanding the pathophysiology of epileptic disorders, seizures remain poorly controlled in approxim
250 iatric disorders, disorder age at onset, and disorder severity in a nationally representative sample
251 shed light on MAP7 function in neurological disorders.SIGNIFICANCE STATEMENT Neurons communicate wit
252 l changes in learning, attention, and speech disorders.SIGNIFICANCE STATEMENT We characterized the pr
253 dolescents with confirmed diagnoses of panic disorder, social anxiety disorder, specific phobias, gen
254 novel tool to study dynamics in ordered and disordered solids and provides new perspectives on elect
256 diagnoses of panic disorder, social anxiety disorder, specific phobias, generalized anxiety disorder
257 multimodal meta-analysis assessed shared and disorder-specific neuroanatomy and neurofunction of inhi
258 sheets will contribute to the exploration of disordered structures from earth-abundant elements for e
259 Emerging evidence suggests impulse control disorder subtypes have dissociable correlates, which ind
261 ncephalomyelitis) and from non-demyelinating disorders such as chronic small vessel disease and other
263 icroglia are implicated in neurodegenerative disorders such as Parkinson's disease (PD), it is import
264 Modern functional neurosurgery for movement disorders such as Parkinson's disease, tremor, and dysto
266 in a number of psychiatric and neurological disorders, such as schizophrenia and Alzheimer's disease
268 , even under native conditions, was markedly disordered, suggesting that the beta-annulus had been di
269 ed temperature dependence, is omnipresent in disordered superconductors - ranging from high-temperatu
271 holocarboxylase deficiency, a rare metabolic disorder that can be life-threatening if left untreated.
272 Alcohol use disorder is a chronic relapsing disorder that is associated with compulsive alcohol-seek
273 CM-AVM) is a blood and lymphatic vessel (LV) disorder that is caused by inherited inactivating mutati
275 ment of Adult Patients with Major Depressive Disorder." The evidence review done for the guideline fo
277 3 disorders included hepatobiliary and renal disorders (three [13%] at 200 mg twice a day), asthenia
278 troversial topics in the management of these disorders to highlight areas of importance for future cl
280 component cancer of Cowden syndrome (CS), a disorder typically associated with germline mutations in
282 omotional effects of structural water on the disordered vanadium oxide nanosheets will contribute to
284 of patients with neurological or psychiatric disorders, we investigated missense variants identified
285 HOD: Adult outpatients with major depressive disorder were randomly assigned to open or placebo-contr
286 th Lewy bodies and genetic neurodegenerative disorders were alternative diagnoses in more than half o
287 gs (AMDs) on clinical features of autoimmune disorders were discovered by chance during World War II.
289 contribute to the pathogenesis of autoimmune disorders when an abnormal immune response targets norma
290 nd indicate that sporadic FTD is a polygenic disorder where multiple pleiotropic loci with small effe
291 ction shared across the spectrum of autistic disorders - whether caused by rare highly penetrant muta
292 phrenia, schizoaffective disorder, or a mood disorder who had moderate or severe tardive dyskinesia.
294 ofibromatosis type 1 (NF1), a common genetic disorder with a birth incidence of 1:2,000-3,000, is cha
295 factor X (F10) in humans is a rare bleeding disorder with a heterogeneous phenotype and limited ther
296 to reconcile the tissue specificity of these disorders with the ubiquitous requirement for ribosomes
299 rototype of heritable ectopic mineralization disorders, with phenotypic overlap with generalized arte
300 increase the risk of childhood externalizing disorders, yet no large cohort study has investigated th
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