ライフサイエンスコーパス: disorientation

1 in disA-1 cells display aberrant spacing and disorientation.

2 oscope from different perspectives to induce disorientation.

3 nteract during complex movements and spatial disorientation.

4 duct injuries occur as a result of operator disorientation.

5 ng to decrease the morbidity associated with disorientation.

6 to locate objects in a square chamber after disorientation.

7 a novel environment, and slow rotation after disorientation.

8 t timing abnormalities could lead to spatial disorientation.

9 oral disorders (25%), muscle weakness (23%), disorientation (21%), and neck rigidity (17%).

10 % CI=3.67-5.16); for delirium, confusion, or disorientation, 5.14 (95% CI=4.54-5.82); and for panic d

11 auma with loss of consciousness, amnesia, or disorientation and a Glasgow Coma Scale score of 9-15).

12 ntext of the observed forms of topographical disorientation and are found to be in good agreement wit

13 with radar and acoustic sensors and modeling disorientation and attraction with simulations.

14 mature tangles in the entorhinal cortex and disorientation and confusion when navigating familiar pl

15 p (+0.26; P=.04), depression (+0.25; P=.05), disorientation and detachment (+0.23; P= .05), and vital

16 en were at higher risk of delirium/confusion/disorientation and mania, while younger patients were at

17 Simulations revealed a high probability of disorientation and subsequent attraction for nearby bird

18 d review of the literature of "topographical disorientation" and describes several functional MRI stu

19 displacement, and the problems of nostalgia, disorientation, and alienation may ensue.

20 isorder with associated memory loss, spatial disorientation, and other psychiatric problems.

21 isual processing, disorders of topographical disorientation, and the influence of environmental condi

22 n of key perceptual-motor factors leading to disorientation, assessment of their relative impact, and

23 a," characterized by problems with thinking, disorientation, balance disturbances, vertigo, and impot

24 delirium, including cognitive impairment or disorientation, dehydration or constipation, hypoxia, in

25 hallucinations, prosopagnosia, topographical disorientation, disturbance of perception of velocity of

26 ea, fatigue, somnolence, euphoria, vomiting, disorientation, drowsiness, confusion, loss of balance,

27 Individuals with developmental topographic disorientation (DTD) have a life-long impairment in spat

28 Developmental topographic disorientation (DTD) is a life-long condition in which a

29 speech disorder, dizziness, somnolence, and disorientation (each 4%).

30 osed that several varieties of topographical disorientation exist, resulting from damage to distinct

31 Visuospatial disorientation forces Alzheimer's disease patients to ab

32 We show that a disorientation gradient caused by a high density of geom

33 Disorientation has been identified as one of the major b

34 panic disorder, and delirium, confusion, or disorientation) have been reported to occur in 15.7 per

35 ptic flow analysis and contribute to spatial disorientation in Alzheimer's disease.

36 equired for KFs to elongate and to resist BB disorientation in response to ciliary forces.

37 the patients), seizures (in 46 percent), and disorientation (in 42 percent).

38 Such disorientation involved the layer of collagen covering t

39 ge in traumatic brain injury leads to bundle disorientation, loss of axonal viability, and cognitive

40 equencing to identify the mutation in the BB disorientation mutant disA-1.

41 ne causes swelling of unmyelinated axons and disorientation of axonal microtubules at a time when it

42 ral integrity of the shell due to structural disorientation of calcite crystals.

43 eginnings of an explanation for the magnetic disorientation of migratory birds exposed to anthropogen

44 an increase of second-order vasa vasorum and disorientation of normal vasa vasorum spatial pattern.

45 display substantial cell morphology defects, disorientation of septum formation and a significantly p

46 ace to regain their sense of direction after disorientation, often ignoring nongeometric cues even wh

47 ephalopathy (HE) that does not cause obvious disorientation or asterixis (minimal HE [MHE]/grade 1 HE

48 Understanding disorientation requires description of key perceptual-mo

49 maged patients suffering from "topographical disorientation" tentatively support this proposal.

50 lesions involving the PPA cause topographic disorientation, there is little causal evidence linking

51 ons correlated with search performance under disorientation, whereas consistent marking of ground inf