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1 e-GlcCer content, reduced by 50% in the smtB disruptant.
2 thetic pathway, was undetectable in the relA disruptant.
3 used unmodified to obtain the desired single disruptant.
4 The frog MAPK gene also suppressed the pmk1 disruptant.
5 s known to be essential, did not give viable disruptants.
10 at 42 degrees C, the conidia and hyphae from disruptants are chitin deficient, swell excessively, and
12 lition of Gpi11p or Pig-Fp function in GPI11 disruptants blocks GPI anchoring and formation of comple
15 tron microscopy studies showed that the alb1 disruptant exhibited a smooth conidial surface, whereas
16 us role in nocardicin biosynthesis, the nocL disruptant failed to generate the oxime-containing metab
18 ype STE11alpha strain with a ste11alphaDelta disruptant for virulence using the mouse model showed th
19 e of 30 degrees C, gpi1 cells and gpi1::URA3 disruptants form large, round, multiply budded cells wit
24 er observed following membrane feeds of CTRP disruptant lines to Anopheline mosquitoes, despite the d
25 t importantly, the virulence of a ste12alpha disruptant of serotype D strain was significantly reduce
27 f E.coli DExH/D-box protein and DNA helicase disruptants should be useful for analyzing the function
34 in production medium than the wild type or a disruptant to which the intact relA gene was restored.
35 tion by allelic exchange in the case of each disruptant was confirmed by PCR and Southern blot analys
38 nidial surfaces, and the conidia of the alb1 disruptant were ingested by human neutrophils at a highe
39 l of acute infection, wdchs2Deltawdchs3Delta disruptants were considerably less virulent in the same
41 diates in stationary-phase cells, since rev3 disruptants were more sensitive in this phase than in th
42 out selection, and in six other cases, where disruptants were not identified in the initial PCR scree
43 tion of the infection droplets of the double disruptant with either purified enzyme, PLA, or PLD, cau
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