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1 e-GlcCer content, reduced by 50% in the smtB disruptant.
2 thetic pathway, was undetectable in the relA disruptant.
3 used unmodified to obtain the desired single disruptant.
4  The frog MAPK gene also suppressed the pmk1 disruptant.
5 s known to be essential, did not give viable disruptants.
6                                      In fau1 disruptants, 5-CHO-THF was elevated 4-fold over wild-typ
7                                         orlA disruptants accumulate trehalose-6-phosphate and have re
8 or the efficient construction of stable gene disruptants and of gene deletions in Streptomyces.
9 not occur in the presence of the aggregation disruptants ANS and CR.
10 at 42 degrees C, the conidia and hyphae from disruptants are chitin deficient, swell excessively, and
11                                 Yeast SPP381 disruptants are severely growth impaired and accumulate
12 lition of Gpi11p or Pig-Fp function in GPI11 disruptants blocks GPI anchoring and formation of comple
13 -related proteins and tested for the desired disruptants by PCR screening of 100 colonies.
14 enting canavanine hypersensitivity of scrheb disruptant cells.
15 tron microscopy studies showed that the alb1 disruptant exhibited a smooth conidial surface, whereas
16 us role in nocardicin biosynthesis, the nocL disruptant failed to generate the oxime-containing metab
17                                         FAT1 disruptants (fat1Delta) fail to accumulate the fluoresce
18 ype STE11alpha strain with a ste11alphaDelta disruptant for virulence using the mouse model showed th
19 e of 30 degrees C, gpi1 cells and gpi1::URA3 disruptants form large, round, multiply budded cells wit
20                      When grown on agar, the disruptants grew more slowly than a control lysogen made
21                        Importantly, the alb1 disruptant had a statistically significant loss of virul
22                                              Disruptants have a nearly-wild-type morphology at 32 deg
23                                     The slm9 disruptant is delayed at the G(2)-M transition as indica
24 er observed following membrane feeds of CTRP disruptant lines to Anopheline mosquitoes, despite the d
25 t importantly, the virulence of a ste12alpha disruptant of serotype D strain was significantly reduce
26                                Growth of the disruptant on low-phosphate medium did not restore actin
27 f E.coli DExH/D-box protein and DNA helicase disruptants should be useful for analyzing the function
28             Furthermore, both rad14 and mag1 disruptants show elevated levels of nitrogen mustard-ind
29                    The resulting wdpks1Delta disruptants showed no morphological defects other than a
30                                   In an aep3-disruptant strain, the shorter ATP8/6 mRNA was undetecta
31 , mismatch repair-defective, rad27 and mre11 disruptant strains.
32                                       Pfs230 disruptants successfully emerge from RBCs and male gamet
33                   In contrast, in the Pfs230 disruptants the expression and localization of other kno
34 in production medium than the wild type or a disruptant to which the intact relA gene was restored.
35 tion by allelic exchange in the case of each disruptant was confirmed by PCR and Southern blot analys
36               The sterol profile of an ERG25 disruptant was consistent with the erg25 allele obtained
37                                Moreover, the disruptant was unable to accumulate ppGpp to the levels
38 nidial surfaces, and the conidia of the alb1 disruptant were ingested by human neutrophils at a highe
39 l of acute infection, wdchs2Deltawdchs3Delta disruptants were considerably less virulent in the same
40                      Furthermore, ste12alpha disruptants were fertile when mated with MATa strains, a
41 diates in stationary-phase cells, since rev3 disruptants were more sensitive in this phase than in th
42 out selection, and in six other cases, where disruptants were not identified in the initial PCR scree
43 tion of the infection droplets of the double disruptant with either purified enzyme, PLA, or PLD, cau
44                Complementation of the double disruptant with pelD gene, or supplementation of the inf

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