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1 ntial therapeutic target for protection from disseminated candidiasis.
2 GI tract, a risk factor for haematogenously-disseminated candidiasis.
3 e role of ICAM-1 in the host defense against disseminated candidiasis.
4 ) for the treatment of experimental subacute disseminated candidiasis.
5 were indistinguishable during hematogenously disseminated candidiasis.
6 cule intervention to improve the outcomes of disseminated candidiasis.
7 howed reduced virulence in a murine model of disseminated candidiasis.
8 ortant for host resistance to hematogenously disseminated candidiasis.
9 sted the susceptibility of CCR5(-/-) mice to disseminated candidiasis.
10 roducts were efficacious in a mouse model of disseminated candidiasis.
11 dhesin, Als3p (rAls3p-N), protects mice from disseminated candidiasis.
12 intravascular catheter biofilm infection or disseminated candidiasis.
13 the bloodstream of rabbits with experimental disseminated candidiasis.
14 creased resistance of mice to hematogenously disseminated candidiasis.
15 -N) modestly improves survival during murine disseminated candidiasis.
16 physiological characteristics during murine disseminated candidiasis.
17 ty of human diseases such as oral thrush and disseminated candidiasis.
18 valuated in a murine model of hematogenously disseminated candidiasis.
19 immunization strategies to prevent or treat disseminated candidiasis.
20 prophylactic or therapeutic strategy against disseminated candidiasis.
21 mucocutaneous infections like oral thrush to disseminated candidiasis.
22 virulence factors relevant to hematogenously disseminated candidiasis.
23 ever, highly attenuated in a murine model of disseminated candidiasis.
24 virulence of C. albicans in a mouse model of disseminated candidiasis.
25 esponses in a murine model of hematogenously disseminated candidiasis.
26 ogenesis, adherence, and mortality in murine disseminated candidiasis.
27 the role of the MR in immune response during disseminated candidiasis.
28 nd leukocyte adhesion molecules in mice with disseminated candidiasis.
29 ulence in the mouse model of haematogenously disseminated candidiasis.
30 virulence of C. albicans in a mouse model of disseminated candidiasis.
31 e critical for virulence in a mouse model of disseminated candidiasis.
32 antibody enhanced resistance of mice against disseminated candidiasis.
33 ace mannan epitope, does not protect against disseminated candidiasis.
34 nfection in a murine model of hematogenously disseminated candidiasis.
35 albicans in a murine model of hematogenously disseminated candidiasis.
36 or mice in an intravenous infection model of disseminated candidiasis.
37 ice to produce protective antibodies against disseminated candidiasis.
38 , from superficial mucosal to hematogenously disseminated candidiasis.
39 important determinant of host resistance to disseminated candidiasis.
40 ere combined immunodeficiency SCID mice from disseminated candidiasis (100% survival in BCG-vaccinate
43 cate that the murine model of hematogenously disseminated candidiasis accurately recapitulates the pr
44 tive antibody responses against experimental disseminated candidiasis and Candida vaginal infection.
45 n, leading to disease manifestations such as disseminated candidiasis and chronic mucocutaneous candi
46 the survival of neutropenic mice with lethal disseminated candidiasis and inhalational aspergillosis.
47 Candida parapsilosis is a frequent cause of disseminated candidiasis and is associated with signific
48 virulent in a murine model of hematogenously disseminated candidiasis and less able to adhere to huma
49 also improves survival of outbred mice from disseminated candidiasis and that it is active against m
50 irulence in a murine model of hematogenously disseminated candidiasis and that strains with each gene
51 hat Als protein production was widespread in disseminated candidiasis and that, despite strain differ
53 uired for full virulence in a mouse model of disseminated candidiasis, and Git3 sequence orthologs ar
54 sal of the blood vessel wall by yeast during disseminated candidiasis, and N-WASP may play a key role
56 rs are safe and as effective in experimental disseminated candidiasis as once-daily therapy in neutro
57 ficient mice had increased susceptibility to disseminated candidiasis, as indicated by decreased surv
59 was equally as effective as rAls1p-N against disseminated candidiasis but was more effective than rAl
60 rs did not correlate with protection against disseminated candidiasis, but delayed-type hypersensitiv
61 on gamma (IFN-gamma) in host defense against disseminated candidiasis, but in vivo studies are inconc
63 ation of lymphoreticular malignancy, chronic disseminated candidiasis (CDC), and from a neutropenic-r
64 ch was shown to contribute to hematogenously disseminated candidiasis (DC) after several days in the
65 cate C. albicans NOT5 in the pathogenesis of disseminated candidiasis (DC) induced by intravenous inf
66 ere significantly higher among patients with disseminated candidiasis (DC) or oropharyngeal candidias
68 single large dose of micafungin could clear disseminated candidiasis, even though the micafungin hal
70 analysis of a mouse model of hematogenously disseminated candidiasis (HDC) and episodes of vulvovagi
71 more resistant to experimental hematogenous disseminated candidiasis if they received MAb B6.1 befor
75 E) is a relatively frequent manifestation of disseminated candidiasis in neonates and is associated w
82 orter strains were inoculated into mice in a disseminated candidiasis model, and GFP production was m
83 ant role in murine resistance to gastric and disseminated candidiasis of endogenous (intestinal tract
84 respectively, were found to be resistant to disseminated candidiasis of endogenous origin and to acu
85 ibodies, were as resistant to orogastric and disseminated candidiasis of endogenous origin as were im
86 required for the normal host defense during disseminated candidiasis or for the phagocytosis of C. a
87 BL-A deficiency does not alter resistance to disseminated candidiasis or initial hepatic invasion by
88 and rabbits with persistent neutropenia and disseminated candidiasis, otherwise fatal, demonstrated
90 cells and attenuated mortality during murine disseminated candidiasis, proving that our strategy can
91 the molecular basis of host defense against disseminated candidiasis remains elusive, and treatment
94 released into the bloodstream of hosts with disseminated candidiasis, that phagocytic cells may play
96 ssessed in a murine model for hematogenously disseminated candidiasis, was significantly attenuated c
97 al antibody (MAb) B6.1 protects mice against disseminated candidiasis, whereas the IgM MAb B6 does no
98 ibe here experiments using a murine model of disseminated candidiasis which demonstrate that in three
99 ncidence of life-threatening, hematogenously disseminated candidiasis, which is predominantly caused
100 s mutants was unimpaired in a mouse model of disseminated candidiasis, while M-2 and CA-2 were 2 orde
101 tenuation in a mouse model of hematogenously disseminated candidiasis, while the double mutant was ra
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