ライフサイエンスコーパス: disseminated intravascular coagulation

1 eiving anticoagulants fulfilled criteria for disseminated intravascular coagulation.

2 was no chemical or microscopic evidence for disseminated intravascular coagulation.

3 ter infection, a full day after the onset of disseminated intravascular coagulation.

4 in-induced thrombosis, and endotoxin-induced disseminated intravascular coagulation.

5 ave value in the treatment of sepsis-induced disseminated intravascular coagulation.

6 l involvement with respiratory compromise or disseminated intravascular coagulation.

7 habdomyolysis, pneumonia, renal failure, and disseminated intravascular coagulation.

8 y including atherosclerosis, vasculitis, and disseminated intravascular coagulation.

9 ay contribute to local fibrin deposition and disseminated intravascular coagulation.

10 , including atherosclerosis, vasculitis, and disseminated intravascular coagulation.

11 vated monocytes in several diseases leads to disseminated intravascular coagulation.

12 There was no evidence of the induction of disseminated intravascular coagulation.

13 nt role in the pathophysiology of sepsis and disseminated intravascular coagulation.

14 antly, arguing against an important role for disseminated intravascular coagulation.

15 e (</= 58%; odds ratio, 0.98; p < 0.05) with disseminated intravascular coagulation.

16 sis and concurrent hepatobiliary dysfunction/disseminated intravascular coagulation.

17 Death was attributed to sepsis and disseminated intravascular coagulation.

18 ivation associated with septic shock-induced disseminated intravascular coagulation.

19 es are relevant biomarkers of sepsis-induced disseminated intravascular coagulation.

20 e lacking TFPI (Tfpi(-/-)) die in utero from disseminated intravascular coagulation.

21 both biochemical and microscopic evidence of disseminated intravascular coagulation.

22 phyxia induces bleeding by hyperfibrinolytic disseminated intravascular coagulation.

23 cally ill patients with sepsis and suspected disseminated intravascular coagulation.

24 in sepsis-associated coagulopathy including disseminated intravascular coagulation.

25 nfections are at a higher risk of developing disseminated intravascular coagulation.

26 During endotoxemia, TF expression leads to disseminated intravascular coagulation.

27 openia, transient leucopenia, and aggressive disseminated intravascular coagulation.

28 lants can be effective in severe sepsis with disseminated intravascular coagulation.

29 dothelial damage, leading to acute death and disseminated intravascular coagulation.

30 iltrates and lung injury, renal failure, and disseminated intravascular coagulation.

31 ated with extreme inflammatory responses and disseminated intravascular coagulation.

32 hagocytosis (70%), leukemia cutis (58%), and disseminated intravascular coagulation (39%) occurred fr

33 oderate to severe renal insufficiency (53%), disseminated intravascular coagulation (45%), and the ac

34 Thirty-five patients had disseminated intravascular coagulation according to Japa

35 several major clinical disorders, including disseminated intravascular coagulation and acute ischemi

36 nt at admission could predict the absence of disseminated intravascular coagulation and allow a bette

37 in inhibition is associated with less severe disseminated intravascular coagulation and better overal

38 ce of thrombotic complications, particularly disseminated intravascular coagulation and deep vein thr

39 inoic acid and chemotherapy, but developed a disseminated intravascular coagulation and died before c

40 coccal sepsis that moderates the severity of disseminated intravascular coagulation and enables host

41 However, except for disseminated intravascular coagulation and hypercalcemia

42 rse the lethal sequelae of sepsis, including disseminated intravascular coagulation and multi-organ f

43 Measurements in disseminated intravascular coagulation and no disseminat

44 ay contribute to the hemostatic imbalance in disseminated intravascular coagulation and other coagulo

45 condary endpoints included reversal of overt disseminated intravascular coagulation and reduction in

46 activation of proteolytic cascades, such as disseminated intravascular coagulation and sepsis, acqui

47 ptoms due to hemorrhage, possibly because of disseminated intravascular coagulation and/or hemophagoc

48 d is associated with venous thromboembolism, disseminated intravascular coagulation, and additional c

49 VTE patients, for diagnosing and monitoring disseminated intravascular coagulation, and as an aid in

50 ogenic mechanisms in infective endocarditis, disseminated intravascular coagulation, and cardiovascul

51 , liver dysfunction, coagulopathy resembling disseminated intravascular coagulation, and extreme hype

52 plantation, cardiac arrhythmias, presence of disseminated intravascular coagulation, and need for vas

53 ft ventricular failure with pulmonary edema, disseminated intravascular coagulation, and neurologic i

54 e observed changes in vascular permeability, disseminated intravascular coagulation, and systemic inf

55 es to collapse of the circulatory system, to disseminated intravascular coagulation, and to a 30% mor

56 at admission is predictive of the absence of disseminated intravascular coagulation (area under the c

57 of concurrent hepatobiliary dysfunction and disseminated intravascular coagulation as features of ma

58 n in patients with sepsis, septic shock, and disseminated intravascular coagulation associated with i

59 One child died due to disseminated intravascular coagulation associated with p

60 ts had a 13 times higher prevalence of overt disseminated intravascular coagulation at admission (55%

61 Sixty-one had disseminated intravascular coagulation at admission, and

62 so analyzed after exclusion of patients with disseminated intravascular coagulation at admission.

63 se of intrapartum convulsion and one case of disseminated intravascular coagulation (both in the Fole

64 n the setting of sepsis may not be caused by disseminated intravascular coagulation but instead repor

65 nduced systemic proinflammatory response and disseminated intravascular coagulation, by reducing the

66 Intravascular thrombosis or disseminated intravascular coagulation can also occur in

67 -derived microparticles were associated with disseminated intravascular coagulation: CD105-microparti

68 rrogates of cell activation to improve early disseminated intravascular coagulation diagnosis and pat

69 ty of 90.91% and a specificity of 80.60% for disseminated intravascular coagulation diagnosis.

70 newly diagnosed AML, we assessed markers of disseminated intravascular coagulation (DIC) (fibrinogen

71 Pulmonary xenografts develop disseminated intravascular coagulation (DIC) and microva

72 during disease was determined by scoring for disseminated intravascular coagulation (DIC) and recordi

73 inflammation in animal models of sepsis with disseminated intravascular coagulation (DIC) and that co

74 agnosed because of overlapping features with disseminated intravascular coagulation (DIC) and the lac

75 Disseminated intravascular coagulation (DIC) appears to

76 on, prevent ischemia-reperfusion injury, and disseminated intravascular coagulation (DIC) associated

77 Work toward a better definition of disseminated intravascular coagulation (DIC) by characte

78 Disseminated intravascular coagulation (DIC) has been ob

79 n coagulopathy with histological evidence of disseminated intravascular coagulation (DIC) in four.

80 ed using: (1) a carrageenan-induced model of disseminated intravascular coagulation (DIC) in mice; (2

81 activated platelets in inflammation-induced disseminated intravascular coagulation (DIC) in micro-ca

82 Disseminated intravascular coagulation (DIC) is a condit

83 e looked at the utility of a simple evolving disseminated intravascular coagulation (DIC) score that

84 No signs of severe disseminated intravascular coagulation (DIC) were observ

85 However, other processes such as disseminated intravascular coagulation (DIC), hyperfibri

86 Group 1 and 2 animals developed features of disseminated intravascular coagulation (DIC), with reduc

87 Disseminated intravascular coagulation (DIC)-positive ki

88 y followed by vessel obstruction may lead to disseminated intravascular coagulation (DIC).

89 y, mortality, and laboratory consequences of disseminated intravascular coagulation (DIC).

90 The typical laboratory pattern mimics disseminated intravascular coagulation (DIC).

91 omplications of hemoglobinemia, particularly disseminated intravascular coagulation (DIC).

92 days) in platelet counts and fibrinogen with disseminated intravascular coagulation (DIC); after graf

93 ession of human Pdpn-Fc in mouse skin causes disseminated intravascular coagulation due to platelet a

94 receptor moderates the onset and severity of disseminated intravascular coagulation during sepsis and

95 t of treatment, may limit the development of disseminated intravascular coagulation during systemic i

96 r coagulation at admission, and 32 developed disseminated intravascular coagulation during the first

97 The non-hepatobiliary dysfunction/disseminated intravascular coagulation group included pa

98 Although blood coagulation changes such as disseminated intravascular coagulation have been recogni

99 Although septic shock and disseminated intravascular coagulation have been reporte

100 ation syndrome, a similar entity with fever, disseminated intravascular coagulation, hepatobiliary dy

101 Disseminated intravascular coagulation, immune thrombocy

102 one of the significant mechanisms underlying disseminated intravascular coagulation in EHF patients.

103 Characterization of null fish revealed disseminated intravascular coagulation in larvae seconda

104 al growth factor, complement activation, and disseminated intravascular coagulation in one or more he

105 be a causative agent in the pathogenesis of disseminated intravascular coagulation in sepsis.

106 is sometimes encountered in association with disseminated intravascular coagulation in the donor, and

107 al sampling experiments revealed evidence of disseminated intravascular coagulation in the livers of

108 g4) mice, mainly reflecting earlier signs of disseminated intravascular coagulation in this latter co

109 t microvascular endothelial stress (nonovert disseminated intravascular coagulation) in patients who

110 r, splenomegaly, bleeding, fever, infection, disseminated intravascular coagulation, increasing heigh

111 In primate models of sepsis, APC blocks disseminated intravascular coagulation initiated by Esch

112 tress in which there is no evidence of overt disseminated intravascular coagulation, injury of the en

113 Disseminated intravascular coagulation is a prominent ma

114 These results indicate that disseminated intravascular coagulation is an early and i

115 of coagulation, which can culminate in overt disseminated intravascular coagulation, is a prominent f

116 ble thrombomodulin, activated protein C, and disseminated intravascular coagulation markers.

117 ing times, and tissue factor- or LPS-induced disseminated intravascular coagulation models, no signif

118 ssed children with septic shock and signs of disseminated intravascular coagulation, more than 60% of

119 fever, neither delayed blood coagulation and disseminated intravascular coagulation nor death from sh

120 bility, excessive bleeding in the setting of disseminated intravascular coagulation or other states o

121 There was no evidence of disseminated intravascular coagulation or renal dysfunct

122 ts with only hepatobiliary dysfunction, only disseminated intravascular coagulation, or neither.

123 ng patients develops overt hyperfibrinolytic disseminated intravascular coagulation, partly caused by

124 y survival rate in hepatobiliary dysfunction/disseminated intravascular coagulation patients (65.4% a

125 lacebo-treated non-hepatobiliary dysfunction/disseminated intravascular coagulation patients (71.4% v

126 isseminated intravascular coagulation and no disseminated intravascular coagulation patients showed t

127 deep venous thrombosis, pulmonary embolism, disseminated intravascular coagulation, portal vein thro

128 ch as leukostasis, tumor lysis syndrome, and disseminated intravascular coagulation put the patient a

129 ebrile neutropenia, transient and reversible disseminated intravascular coagulation related to rituxi

130 e interval 1.2-4.7; p = .035) and have overt disseminated intravascular coagulation (relative risk 4.

131 e serious sequelae including hemoglobinemia, disseminated intravascular coagulation, renal failure, a

132 Despite comparable disseminated intravascular coagulation scores, acute pro

133 oinflammatory condition that can manifest as disseminated intravascular coagulation, septic shock, an

134 tions were sicker and more likely to develop disseminated intravascular coagulation than patients wit

135 One procedure was complicated by disseminated intravascular coagulation that necessitated

136 lect a vascular injury during sepsis-induced disseminated intravascular coagulation that precedes obv

137 of a fVII immunoconjugate to TF might cause disseminated intravascular coagulation, the active site

138 levels of PC predispose mice to early-onset disseminated intravascular coagulation, thrombocytopenia

139 Prediction of disseminated intravascular coagulation was also analyzed

140 Disseminated intravascular coagulation was diagnosed acc

141 Concurrent hepatobiliary dysfunction/disseminated intravascular coagulation was noted in 43 p

142 Evidence of disseminated intravascular coagulation was noted; howeve

143 In two baboons, disseminated intravascular coagulation was successfully

144 th drowning-induced asphyxia developed overt disseminated intravascular coagulation within 24 hours.

145 epsis may therefore limit the development of disseminated intravascular coagulation without increasin