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1 was stimulated by slow balloon or rapid air distension.
2 pressure measured before and during gastric distension.
3 ed neuroimaging studies that employed rectal distension.
4 a pH >4) was also measured before and after distension.
5 uced visceral hypersensitivity to colorectal distension.
6 pertension in response to noxious colorectal distension.
7 , NTS) were also activated by proximal colon distension.
8 motility, and hypersensitivity to colorectal distension.
9 e risks and benefits of lung recruitment and distension.
10 astine on the symptom score evoked by rectal distension.
11 nically and statistically recognizable bowel distension.
12 pulmonary hypertension and right ventricular distension.
13 ion of gastric emptying induced by the colon distension.
14 administration, or CCK combined with gastric distension.
15 nges in gastric emptying associated to colon distension.
16 icantly mediate neuronal activation by CCK + distension.
17 in through a mechanism that involves gastric distension.
18 eal represent satiety or feelings of gastric distension.
19 ensory neurons detect gut hormones and organ distension.
20 ferent nerve activity in response to bladder distension.
21 ones, lipid mediators, nutrients and gastric distension.
22 red hemodynamic response to uterine cervical distension.
23 or transverse stretching of the airways, or distension.
24 in compliance was observed during isovolumic distension.
25 alter blood flow and pressure-induced vessel distension.
26 h as heart rate, blood pressure and visceral distension.
27 ion) and during episodes of severe abdominal distension.
28 was measured based on response to colorectal distension.
29 e except perhaps during abnormal ventricular distension.
30 ) and discrete episodes of visible abdominal distension.
31 s strongly associated with the degree of MPA distension.
32 men, with a few months of abdominal pain and distension.
33 attenuates the hypophagic effects of gastric distension.
34 Visceral pain was evaluated with colorectal distension.
35 ere found for abdominal spasms and abdominal distension.
36 ring spontaneously or following intraluminal distension.
37 ed because of poor preparation or inadequate distension.
38 d surfactant administration reduces airspace distension.
39 women were more likely to have asymmetrical distension.
40 les such as posture, volume, and velocity of distension.
41 massive cytoplasmic vacuolization due to ER distension.
42 ables on UES pressure response to esophageal distension.
43 dled mycobiome) restored hypersensitivity to distension.
44 gic IOP had no significant difference in SCS distension.
45 enuated, the multifibre responses to bladder distensions.
46 (45 mmHg) and low (15 mmHg) intensity rectal distensions.
47 to urethral infusion, 81% responded to colon distension, 100% responded to penile stimulation (and DN
52 decerebrate, unanaesthetized ferrets.Gastric distension (30 ml) evoked LOS relaxation (70 +/- 8% decr
53 hicle-treated rats following gastric balloon distension (5 ml), CCK (1 microg/kg) administration, or
55 pressing GLUR2/3 was similar for the gastric distension (59.8-65.6%) and duodenal linoleic acid (60.6
57 e of neurons in the NTS activated by gastric distension (63.9+/-2.9%), linoleic acid (62.8+/-1.4%), a
58 inhibitory responses to intraluminal balloon distension; (7) an absence of reflex activity in respons
60 addition, the cardiovascular variable, pulse distension (a surrogate for stroke volume) was improved
61 tion technique (OP) using pressure-regulated distension, a non-toxic vein marker, and graft storage i
65 ss abundant, with only a small percentage of distension-activated (4.4+/-0.4%) and linoleic acid-acti
67 in IBS, including postprandial bloating and distension, altered motility, visceral hypersensitivity,
69 JCMS virulence correlates with intestinal distension and bacterial accumulation and requires the b
75 y gene c-fos expression following colorectal distension and found increased (P < 0.01) c-Fos(+) cell
78 ulation of the airway epithelium by rhythmic distension and increased FIO(2) may activate NOS II rele
79 plified visceromotor responses to colorectal distension and induced hyperexcitability of colonic noci
80 elease of ATP from urothelium in response to distension and its action on P2X receptors resulting in
81 otransmitters from urothelium in response to distension and its action on receptors on sensory neuron
82 the evoked visceromotor response to bladder distension and led to small but significant changes in b
83 stomach to the brain were thought to detect distension and play little role in nutritional signaling
85 ing gastric stability would increase gastric distension and reduce appetite compared with a control d
86 as rapidly induced in response to epithelial distension and stretch, kidney cyst formation, acute kid
87 ls appear to respond to the level of venular distension and to recruitment of the vascular bed within
88 in cerebral responses to standardized rectal distensions and their expectation, consistent with diffe
89 ) during basal conditions (without abdominal distension) and during episodes of severe abdominal dist
91 d by the visceromotor response to colorectal distension, and colon afferent fiber mechanosensitivity
93 rfactant depletion may contribute to greater distension-and thereby injury-of aerated lung regions; r
95 id, is not normally stimulated by intestinal distension as the meal passes through the gastrointestin
98 was different during isovolumic and isobaric distensions but the passive components were similar.
99 oesophageal wall compliance during isobaric distension, but no change in compliance was observed dur
106 we examine the effects of 3 weeks of cyclic distension (CD) on fibrin-based tubular tissue construct
108 erior cingulate cortex during noxious rectal distensions, compared to controls and normosensitive pat
109 In contrast, peristaltic waves elicited by distension could be graded in amplitude according to sti
110 reflexia using a model of noxious colorectal distension (CRD) after complete spinal transection at th
112 euronal activities in response to colorectal distension (CRD) were recorded in control, sham-treated
113 e field of neurones responsive to colorectal distension (CRD), and the potential role of ionotropic g
114 With neuroanatomical tracing and colorectal distension (CRD), we sought to identify colonic afferent
115 ses of neurons in the thalamus to colorectal distension (CRD), whereas lesion in the dorsal column (D
116 reported to play a role in tonic colorectal distension (CRD)-induced activation of locus coeruleus (
118 eromotor response (VMR) to graded colorectal distension (CRD; 10-80 mmHg for 30 s with 180 s intersti
123 nephric kidney at 27 h postfertilization and distension/dilation of pronephros at 5 d postfertilizati
124 ammatory cytokine production, excessive lung distension directly affects blood-gas barrier and lung v
126 c reflexes in SCI mice (e.g., via colorectal distension) elicits AD and exacerbates immune suppressio
128 vioral data, these results show that gastric distension enhances CCK-induced neuronal activation in t
129 bleeding disorders presented with abdominal distension, epistaxis, and anemia (hemoglobin 8.2 g/dL).
133 gastro-oesophageal reflux, that is, gastric distension-evoked lower oesophageal sphincter (LOS) rela
134 gastro-oesophageal reflux, that is, gastric distension-evoked lower oesophageal sphincter (LOS) rela
135 ncreased visceromoter response to colorectal distension; expression of NGF increased in colonic muscu
137 n on heart rate, blood pressure and visceral distension from the periphery to the central nervous sys
141 nts with functional gut disorders, abdominal distension has been associated with descent of the diaph
144 rons that did not respond to urinary bladder distension (i.e. out of the 46 remaining neurons, none r
146 osensitivity (elevated thresholds to balloon distension in comparison with age- and sex-matched contr
147 nervous activity generated by localized wall distension in isolated segments of guinea-pig distal col
149 uces visceral hypersensitivity to colorectal distension in response to HeCS by increasing the express
150 eased significantly Fos expression 1 h after distension in selective brain areas, most prominently, t
151 aicin, an effect that is reversed by balloon distension in the duodenum in fasted rats, while balloon
152 se, ondansetron attenuated Fos-LI by gastric distension in the DVC, specifically within the nucleus o
157 nt response to nonpainful and painful rectal distensions in 15 female IBS patients and 12 healthy wom
158 DVC enhanced Fos-LI induced by CCK + gastric distension, in particular at the NTS and AP nuclei.
159 ding from colon sensory neurons during colon distension, in the presence and absence of pharmacologic
160 ceral hyperalgesia in response to colorectal distension, increased stool output and reciprocal change
161 e increased perceptual sensitivity to rectal distension, indicating differences in processing and/or
162 roximal colon distension, compared with sham distension, induced a robust increase in Fos-like immuno
166 03--3 nmol), significantly inhibited gastric distension-induced pyloric relaxation in a dose-dependen
167 cromol) also significantly inhibited gastric distension-induced pyloric relaxation without affecting
170 onditions, such as tissue damage or visceral distension, induces channel opening, membrane depolariza
171 nificantly attenuated the nerve responses to distension, intraluminal acid and bradykinin, as well as
172 nts with functional gut disorders, abdominal distension is a behavioral response that involves activi
175 However, leukocyte recruitment after airway distension is localized to the airways because increased
176 and secretion, which recur rhythmically when distension is maintained above threshold or during mucos
178 pathway may be activated with larger rectal distensions, leading to a more prolonged period of anal
181 Fiber data were analyzed at five different distension loading points (circumferential stretch ratio
183 posterior attachments, with a need for great distension mediolaterally across the intermediate zone.
184 m above the lower border of the LES and with distension moved a median of 1 cm cephalad within the LE
185 medullary vagal nuclei of the stimuli: slow distension (N=10), rapid distension (N=9), and in contro
186 f the stimuli: slow distension (N=10), rapid distension (N=9), and in control animals (N=10) were ide
188 y tract, retention of urine in renal pelvis, distension of bladder, and the development of end-stage
189 loss of DNA-nuclear lamina interactions, the distension of centromeres, and changes in chromatin comp
190 istribution of body fluids and the degree of distension of cranial blood vessels, and these changes i
191 as greater distortion of fiber membranes and distension of interfiber space at the apex of fiber cell
193 (3-oxo-dodecanoyl)homoserine lactone induced distension of mitochondria and the endoplasmic reticulum
194 eflexia, which is often triggered by painful distension of pelvic viscera (bladder or bowel) and cons
196 he afferent nerve firing induced by isotonic distension of the bladder (10-40 cmH(2)O) was increased
197 sed by 33-55% BAN firing induced by isotonic distension of the bladder at 10-40 cmH(2)O pressures.
204 d: 17 +/- 7 versus 26 +/- 12 mm Hg; P<0.01), distension of the carotid artery, and carotid arterial w
206 homeostasis can result in either collapse or distension of the endolymphatic compartment in the cochl
207 ed insulin secretion accompanied by abnormal distension of the ER lumen, defective trafficking of pro
212 in mitochondrial morphology, which included distension of the outer unit membrane and bloating of th
213 nuclei activated during isovolumetric phasic distension of the proximal colon (10 ml, 30 s on/off for
214 d with sham distension, isovolumetric phasic distension of the proximal colon (10 ml, 30 s on/off for
215 n nuclei activated during noxious mechanical distension of the proximal colon in conscious rats, usin
217 rred following WLST, and was associated with distension of the right ventricle (RV) and reduced cardi
221 ted nutrients (GPR65 neurons) and mechanical distension of the stomach and intestine (GLP1R neurons).
223 administration of adenosine, suggesting that distension of vascular smooth muscles does not explain b
225 properties, by combining mechanical testing (distension) of excised porcine coronary arteries with si
226 d selectin inhibitors blocked the effects of distension on leukocyte recruitment, suggesting their in
227 ess in normal subjects the effect of gastric distension on the LES length and pressure and its exposu
228 (e.g., ATP and UTP), released during bladder distension or from damaged cells after tissue insult, ar
230 en of the rat urinary bladder in response to distension or stimulation with bacterial endotoxins.
231 bably by conveying feedback about intestinal distension or transit to the brain, (2) are not essentia
232 ndicate that pannexin channels contribute to distension- or LPS-evoked ATP release into the lumen of
234 of ileus in the form of nausea, vomiting and distension, pain as assessed by analgesic consumption an
236 ession coefficient, -1.3% change in arterial distension per 10% increase in leptin; 95% CI, -1.9% to
237 s reduced in EoE patients (P = .02) with the distension plateau of EoE patients substantially lower t
238 essively opened the esophageal lumen until a distension plateau was reached such that the narrowest c
243 s indicates that colonic hypersensitivity to distension, rather than excessive gas production, produc
246 riding responses to esophageal water and air distension, respectively, in a volume-dependent fashion
249 , 10 mM) responses could be dissociated from distension responses by adjusting the infusion rate and
251 mice identified two distinct populations of distension-responsive colon afferents: high-firing frequ
252 In high-altitude studies, retinal venous distension (RVD) was ophthalmoscopically assessed in 24
256 pends on the initial orientation angle at no-distension state (lambda(theta) = 1.0 and lambda(axial)
258 cumbent positioning, minimization of gastric distension, subglottic suctioning, avoidance of ventilat
263 d to pathological cyclic stretch (CS) at 18% distension to test the hypothesis that FA protein paxill
264 onses of thoracic spinal neurons to duodenal distension, to determine the afferent pathway and to exa
265 corded in response to graded urinary bladder distension (UBD) in rats pretreated with intravesical re
266 y were to examine effects of urinary bladder distension (UBD) on T(3)-T(4) spinal neurons receiving c
272 at the chance of having intraoperative bowel distension was increased about two fold in patients rece
275 rity (21/22, 95.5 %) of HT fibres to bladder distensions was enhanced with a significantly reduced th
276 f swallows evoked by upper airway/pharyngeal distensions was not significantly reduced by RLN transec
277 ems were noted in bowel functions other than distension, we recommend avoiding nitrous oxide administ
281 ncreased GUSB activity and reduced lysosomal distension were found in regions of the thalamus and tec
283 defecatory desire volumes to rectal balloon distension were normalized in 10 of 13 patients (PRE: me
285 Responses of most LF afferents to colon distension were sensitized by luminal application of cap
289 oreflexes, however, responses to ventricular distension were small and seem unlikely to be of importa
292 at cilia in the corpus responded to acid and distension, whereas cilia in the antrum responded to foo
293 iving adult animals had moderate ventricular distension, whereas pups of the early lethal phenotypic
294 ty in inflamed lungs causes excessive tissue distension, which triggers stretch-induced pathological
295 associated with cerebral and retinal venous distension, whose magnitude correlates with HAH burden.
297 echanical stimuli (wide range of oesophageal distension with pressure up to 100 mmHg) and detect noxi
300 ut mice were significantly less sensitive to distension, with an average response magnitude only 58 a
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