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1 de concentrations and causes natriuresis and diuresis.
2 l to show any benefit of dopamine other than diuresis.
3 sport process, which is upregulated by water diuresis.
4  mannitol in an attempt to induce an osmotic diuresis.
5 centration) and lower blood pressure limited diuresis.
6  angiotensin, while inducing natriuresis and diuresis.
7 ary congestion via vasodilation and enhanced diuresis.
8 medullary interstitium, resulting in osmotic diuresis.
9 hrotoxic agents from the kidney by promoting diuresis.
10 sm is the prevention of urea-induced osmotic diuresis.
11 ffective renal plasma flow, natriuresis, and diuresis.
12 nal suppression and enhanced natriuresis and diuresis.
13 in relief, rehydration, and encouragement of diuresis.
14  diuretic therapy but are often resistant to diuresis.
15  water absorption, promoting natriuresis and diuresis.
16 seen in 15% of patients and was treated with diuresis.
17 ro rats, which manifest severe chronic water diuresis, a 117-kD band was seen, in addition to the pre
18 ons of both peptides include natriuresis and diuresis, a decrease in systemic blood pressure, and inh
19                                              Diuresis, a marker of improved kidney function, occurred
20 istration of furosemide alone caused a large diuresis, addition of BG9719 to furosemide increased diu
21 atinine was related to the degree of induced diuresis after controlling for baseline renal function,
22 ized selective kappa agonist U50,488-induced diuresis after sc administration in rats.
23  inner medulla, we tested whether furosemide diuresis alters active urea transport.
24 ther the level of hydration after furosemide diuresis and 22 hr of sodium depletion affects the amoun
25 n of proximal tubule sodium reabsorption and diuresis and abolishes Na,K-ATPase inhibition and NHE-3
26            ECD animals exhibited marked anti-diuresis and anti-natriuresis (40 and 47%), which peaked
27 d for Drosophila kinin-mediated induction of diuresis and chloride shunt conductance, evidenced by ch
28 signed a clinical trial to determine whether diuresis and colloid replacement in hypoproteinemic pati
29  persisted after adjusting for parameters of diuresis and decongestion (odds ratio, 2.0; 95% confiden
30                                              Diuresis and decongestion were examined across a range o
31 eart failure treatment negatively influences diuresis and decongestion.
32 pithelial chloride conductance, resulting in diuresis and depolarization of the transepithelial poten
33 oration of the temporal relationship between diuresis and dyspnea relief and a possible clinical role
34                                          The diuresis and glomerular filtration rate were low and dec
35 pression with increased infection risk, cold diuresis and hypovolemia, electrolyte disorders, insulin
36 us population of KOR underlying KOR-mediated diuresis and illustrates a unique pharmacological profil
37 gh-dose strategy was associated with greater diuresis and more favorable outcomes in some secondary m
38 partly protected GFR, and reduced the marked diuresis and mTAL necrosis in radiocontrast-induced neph
39 enal delivery of BNP resulted in significant diuresis and natriuresis and an increase in GFR.
40                  We further propose that the diuresis and natriuresis seen during air breathing were
41                        F+V and F+T increased diuresis and natriuresis to a similar extent during drug
42 human volunteers reveal that dopamine causes diuresis and natriuresis, as well as some degree of rena
43 ough acute caffeine intake produces moderate diuresis and natriuresis, caffeine increases the blood p
44  failure (CHF) are associated with increased diuresis and natriuresis, preserved glomerular filtratio
45 lthough both CA and Ucn2 infusion produced a diuresis and natriuresis, responses with Ucn2 and Ucn+CA
46 effects of adenosine A1-receptor blockade on diuresis and renal function in patients with acute decom
47       Such combination therapy might enhance diuresis and salt excretion for treatment of hypertensio
48 vity was positively associated with residual diuresis and serum albumin and inversely associated with
49 onal switch in renal K handling during water diuresis and the relevance to renal K homeostasis in cys
50                                   Furosemide diuresis and water diuresis for 1 or 7 d had no effect o
51                                              Diuresis and weight loss over 5 days (5.3 kg more in the
52  other adverse effects, including dysphoria, diuresis, and constipation.
53 n, decreases Na+-K+-ATPase activity, induces diuresis, and exerts a vasodilator effect.
54 ing growth, development, ecdysis, digestion, diuresis, and many more physiological processes.
55 ve cardiac output, stimulate natriuresis and diuresis, and rapidly induce symptomatic relief.
56 ry promptly restored cardiac index, promoted diuresis, and reduced fluid requirements compared with t
57 ter reabsorption, limits natriuretic osmotic diuresis, and results in concurrent extracellular volume
58 d ability to concentrate urine, exhibit mild diuresis, and tend to be blood volume depleted.
59 n includes control of hypertension, cautious diuresis, and, if necessary, ventilatory support.
60 an arterial pressure, lactate clearance, and diuresis are maintained at acceptable levels.
61 e in proximal tubule sodium reabsorption and diuresis associated with inhibition of renal cortex Na,K
62 duces BP without sympathetic activation, but diuresis augments sympathetic activity in elderly hypert
63 ventional therapy, which included aggressive diuresis, blood transfusions to optimize oxygen-carrying
64       We conclude that use of LDD results in diuresis but does not improve other markers of renal or
65 I infusion normally promotes natriuresis and diuresis, but COX2 deficiency blocked this effect.
66 reater hypotension and a loss of the initial diuresis, but no significant change in creatinine cleara
67                                              Diuresis by either protocol was feasible, safe, and effe
68  pathway more strongly, and further enhanced diuresis by exacerbating the downregulation of the Na(+)
69 ockade may improve glomerular filtration and diuresis by exerting a direct beneficial effect on glome
70                           To prevent osmotic diuresis by the large quantity of urea generated by prot
71                                The increased diuresis can be accomplished without increased risk if d
72  OPC 31260 induced a significant increase in diuresis, decrease in urinary osmolality, and rise in pl
73 t the highest infusion rates, they exhibited diuresis, dehydration, and both decreased weight gain an
74                                        Rapid diuresis early in the hospitalization should similarly i
75 e, including increased plasma urea, impaired diuresis, elevated biomarkers, and changes in brain weig
76                Furosemide diuresis and water diuresis for 1 or 7 d had no effect on either basal or A
77 /kg)- and bremazocine (0.0032 mg/kg)-induced diuresis for 20 weeks.
78                                     Two-hour diuresis greatly increased.
79 d 6-10 hours after the fourth BNZ dose, when diuresis had subsided.
80 .3 +/- 6 days) with sodium nitroprusside and diuresis, hemodynamics improved, eNO concentrations fell
81                  A new finding is that water diuresis improves medullary oxygenation (delta R2* = 6.4
82 However, whether or not nesiritide increases diuresis in ADHF patients is unknown.
83  treatment was associated with an equivalent diuresis in both WT and CF mice.
84  sought to determine if nesiritide increases diuresis in congestive heart failure patients.
85 ffective adjunct to furosemide in increasing diuresis in critically ill children with fluid overload.
86                                   The marked diuresis in lopamidol treated rats (630 +/- 125.1%) was
87 that central KOR mediate KOR agonist-induced diuresis in monkeys.
88          Although potentially able to induce diuresis in patients otherwise resistant to high doses o
89 dium transporters, decreased natriuresis and diuresis in response to l-dihydroxyphenylalanine, and de
90 tive renal vasodilation and a marked osmotic diuresis in the innervated kidney.
91 onists was actually associated with improved diuresis in this cohort.
92  data show that activated LK cells stimulate diuresis in vivo, and that LK and IPC signaling affect f
93 f ANP results in substantial natriuresis and diuresis in wild-type mice but fails to cause significan
94 ning 4% BSA resulted in a marked natriuresis/diuresis in wild-type mice but no response in GC-A null
95                 Initial changes included net diuresis, increased ACE inhibitor doses, and frequent ad
96                                              Diuresis induced by frusemide (10 mg) is reported to imp
97  is no conclusive evidence that one means of diuresis is better than another.
98            (3) Either mesna or forced saline diuresis is recommended to decrease the incidence of uro
99                            Overly aggressive diuresis leading to intravascular volume depletion has b
100  inhibition promotes natriuresis and osmotic diuresis, leading to plasma volume contraction and reduc
101 SNA through suppressed renin activity, while diuresis may evoke sympathetic activation via the upregu
102 ptor) with improved oral efficacy in the rat diuresis model (ED50=0.01 mg/kg) as well as high oral bi
103 cal properties and in vivo activity in a rat diuresis model (ED50=0.3 mg/kg po).
104            When orally administered in a rat diuresis model, compound 14 promoted sodium excretion (E
105 ed along with their in vivo effects in a rat diuresis model.
106                                              Diuresis, natriuresis, renal excretion, and tissue level
107                                           If diuresis occurs at a rate greater than extravascular flu
108 anifested the expected early natriuresis and diuresis of vasopressin escape.
109            This study tested whether chronic diuresis or antidiuresis regulates facilitated urea tran
110 ver, SBP reduction did not negatively affect diuresis or decongestion (P >/= 0.25 for all parameters)
111 duction was not associated with worsening of diuresis or decongestion.
112 y arteriolar pathology and was responsive to diuresis or dialysis but not to nitric oxide therapy.
113                                              Diuresis or fluid restriction may improve lung function
114 s, indicating that the effect was not due to diuresis or fluid shifts.
115 ndicating a deconditioned state or excessive diuresis, or both.
116                  The SAA-rich diet increased diuresis paralleled by downregulation of aquaporin-2 (AQ
117 sed basal Purea in terminal IMCD in all five diuresis protocols.
118                However, ETx stimulated early diuresis,reduced serum sodium levels, and had more prono
119 canagliflozin doses vs 22 [5%]), and osmotic diuresis-related events (pollakiuria: 12 [3%] for both d
120      The purpose of the study was to compare diuresis renography scan interpretation generated by a r
121                                   The forced diuresis resulted in higher urine flow rate (163.26+/-54
122                                              Diuresis significantly increased basal Purea in terminal
123 sporter are major factors in the natriuresis/diuresis that is one of the hallmarks of ischemic acute
124 ch as mannitol, a safe FDA approved drug for diuresis) through particle size measurement and forster
125 eks later), including vigorous hydration and diuresis to minimize radiation exposure to the fetus.
126 NCC does not cause salt wasting or excessive diuresis under basal conditions, raising the possibility
127 intravenously to saline-loaded rats, induced diuresis via antagonism of renal A1-adenosine receptors.
128 meters, and caused a 50% increase in 24-hour diuresis volume (7.4 +/- 0.9 mL/100g in BDL vehicle vers
129 ffects and produced significant increases in diuresis volume and 24-hour natriuresis (0.08 +/- 0.02 m
130                                         This diuresis was compensated for by a drop in urine volume a
131 edema or fluid overload for which aggressive diuresis was intended.
132               However, furosemide-associated diuresis was reduced only in male, not female, mPGES1-de
133                                          The diuresis was reversible and showed urea > salt excretion
134 ion furosemide, or dual placebo, targeted to diuresis, weight loss, and serum total protein.
135 ological/physiological maneuvers that induce diuresis were studied.
136  result of the use of diuretics or of solute diuresis, whereas only 50% of patients who were hypernat
137 , addition of BG9719 to furosemide increased diuresis, which was significant at the 0.75-microg/mL co
138 c is effective in maximizing natriuresis and diuresis while preserving renal function and inhibiting
139 reserved furosemide-mediated natriuresis and diuresis, while reducing cardiac preload and afterload.
140                             Metal chelation, diuresis with furosemide or chlorothiazide, and competit
141                                       Forced diuresis with intravenous crystalloid, furosemide, and m
142 re 98 participants were randomized to forced diuresis with intravenous crystalloid, furosemide, manni
143 as done to test the hypothesis that a forced diuresis with maintenance of intravascular volume after
144 ned by changes in serum creatinine (SCr) and diuresis with risk/injury/failure/loss/end stage (RIFLE)
145                                        Water diuresis (with or without food) for 3 to 5 d had no effe
146 lexible diuretic regimen after 4.2-liter net diuresis, with counseling also regarding diet and progre

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