ライフサイエンスコーパス: diuretic agent

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1 least 3 antihypertensive drugs, including a diuretic agent.

2 ts presenting with AHF requiring intravenous diuretic agents.

3 ty, electrolyte content, and the presence of diuretic agents.

4 d patients with AHF treated with intravenous diuretic agents.

5 class, diastolic blood pressure, and use of diuretic agents.

6 We compared HFrEF patients treated with diuretic agents alone to three treatment arms: 1) ACEI t

7 ive cardiomyopathies are limited to digoxin, diuretic agents and angiotensin-converting enzyme inhibi

8 t impair this mechanism (e.g., thiazide-type diuretic agents and mineralocorticoid receptor antagonis

9 tween neuroblastoma and maternal exposure to diuretic agents, antiinfective agents, estrogens, proges

10 Thiazide and thiazide-like diuretic agents are being increasingly used at lower dos

11 acting agents (e.g., acetazolamide and loop diuretic agents) are preferred.

12 Diuretic agents continue to be used in this setting desp

13 Simultaneous infusion of the diuretic agent furosemide prevented the AVP-induced decr

14 elf impairs delivery of effective amounts of diuretic agent into the urine, the site of action.

15 full-dose antihypertensive drugs including a diuretic agent or >/=4 drugs): control (ABP <125/75 mm H

16 In contrast, compared with diuretic agents, some data suggest that adjustment of ul

17 ephron segments with low water permeability, diuretic agents that impair this mechanism (e.g., thiazi

18 again discontinued all antihypertensive and diuretic agents; they were progressed to a captopril dos

19 se of antihypertensive medication other than diuretic agents was associated with decreased gout risk

20 ronolactone, but administration of all other diuretic agents was discontinued.

21 Antihypertensive and diuretic agents were discontinued 10 days before.

22 The former is caused by diuretic agents, which enhance sodium excretion, often w

23 ated with stable doses of ACE inhibitors and diuretic agents, with or without concurrent digitalis an

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