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1 dical management with heart rate control and diuretic therapy.
2 f changes observed following paracentesis or diuretic therapy.
3 ment of acute decompensated heart failure is diuretic therapy.
4 according to the need for intensification of diuretic therapy.
5 is followed by colloid volume expansion, and diuretic therapy.
6 e especially likely to benefit from low-dose diuretic therapy.
7 s; nocturia can be more safely alleviated by diuretic therapy.
8  body weight, despite the use of intravenous diuretic therapy.
9 atrial fibrillation, pacemakers, and chronic diuretic therapy.
10 ht provide insightful information to titrate diuretic therapy.
11 ion or improved renal function when added to diuretic therapy.
12  ventricular function, renal impairment, and diuretic therapy (adjusted hazard ratio: 3.00; 95% confi
13 n may allow for less intensification of loop diuretic therapy and a lower incidence of worsening rena
14 udy, lower-extremity edema responded to oral diuretic therapy and did not seem to be associated with
15 ave sufficient fluid accumulation to mandate diuretic therapy but are often resistant to diuresis.
16 cardiovascular risk factors, was specific to diuretic therapy but not present for other major antihyp
17  fluid restriction and individually adjusted diuretic therapy by either continuous or bolus infusions
18 ia, or an inadequate response to traditional diuretic therapy despite dose escalation.
19 or mortality benefit from the use of chronic diuretic therapy, diuretics rapidly improve symptoms ass
20 ous ultrafiltration and standard intravenous diuretic therapy for hypervolemic heart failure (HF) pat
21 ltrafiltration is an alternative strategy to diuretic therapy for the treatment of patients with acut
22      The findings were similar for high-dose diuretic therapy (for stroke, RR, 0.49; 95% CI, 0.39-0.6
23 hour intervals, in addition to combinational diuretic therapy in approximately 70% and both oral spir
24 nt systolic and diastolic pressures, and for diuretic therapy, losartan-based therapy was associated
25 atory drugs, fluid management with effective diuretic therapy might be needed.
26  stable after esophageal variceal banding or diuretic therapy of ascites.
27                                The effect of diuretic therapy on arrhythmic death in patients with le
28  this genetic variant modifies the effect of diuretic therapy on the incidence of myocardial infarcti
29 , to 23 of the 93 patients (25%) on ACEI and diuretic therapy (p=0.001) and to 18 of the 46 patients
30 o 18 of the 42 patients (19%) on digoxin and diuretic therapy (p=0.009), to 23 of the 93 patients (25
31 nd chloride excretion, creatinine clearance, diuretic therapy, pH, known diabetes and intensive care
32                                     Low-dose diuretic therapy prevented not only stroke (RR, 0.66; 95
33                 Symptomatic improvement with diuretic therapy supports the presence of HFpEF in patie
34 f metrics of renal function and preadmission diuretic therapy, traditional baseline characteristics,
35                                  Combination diuretic therapy using any of several thiazide-type diur
36 toms or in the change in renal function when diuretic therapy was administered by bolus as compared w
37          In carriers of the adducin variant, diuretic therapy was associated with a lower risk of com
38  385 carriers of the adducin variant allele, diuretic therapy was associated with a lower risk of the
39  carriers of the adducin wild-type genotype, diuretic therapy was not associated with the risk of MI
40                                              Diuretic therapy was superior to beta-blockade with rega
41  potential utility of pendrin inhibitors for diuretic therapy, we tested in mice a small-molecule pen
42 ting to development of gouty attacks such as diuretic therapy, weight gain, and alcohol consumption m
43 of iBNP levels and the timing of intravenous diuretic therapy were documented.
44 ld be managed by modest salt restriction and diuretic therapy with spironolactone or an equivalent in

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