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1 f fiber in the pathogenesis and treatment of diverticular disease.
2 d with a greater and not lower prevalence of diverticular disease.
3 nary evidence that it might be effective for diverticular disease.
4 ctors appear to contribute to development of diverticular disease.
5 known about the role of heritable factors in diverticular disease.
6 matory bowel disease, pouchitis, and colonic diverticular disease.
7 comitant rise in the number of patients with diverticular disease.
8 harmacological abnormalities associated with diverticular disease.
10 S) of 15,220 Icelanders for association with diverticular disease (5,426 cases) and its more severe f
11 ion of M3 receptors in diverticular disease (diverticular disease 7.8% [1.9-20.4], controls 1.7% [0.8
12 e area was 13.2% (6.0-23.3) in patients with diverticular disease and 2.5% (1.6-3.7) in controls (p<0
13 was 17.5% (range 10.0-37.0) in patients with diverticular disease and 47.0% (29.0-54.0) in controls (
14 rs7609897-T: P=1.5 x 10(-10), OR=0.87) with diverticular disease and in FAM155A (family with sequenc
15 cohort study using patients with perforated diverticular disease and population controls identified
16 ons of sigmoid colons from ten patients with diverticular disease and ten controls, after resections
17 ad surgery for colorectal cancer, 14,019 for diverticular disease, and 7222 for inflammatory bowel di
20 nts with primary diagnoses for colon cancer, diverticular disease, benign colonic neoplasm, and ulcer
21 represents the most serious complication of diverticular disease, but little is known regarding its
22 rcinoma is best differentiated from masslike diverticular disease by the absence of diverticula in th
23 nd pain in the left quadrant of the abdomen, diverticular disease causing mortality and morbidity doe
24 receptors were upregulated in patients with diverticular disease compared with controls: the median
25 of laparoscopy in the elective treatment of diverticular disease decreases rates of complications co
26 ies, we identified 142,123 incident cases of diverticular disease diagnosed at a hospitalization (197
27 n of heritable factors to the development of diverticular disease diagnosed at a hospitalization or o
28 .0001), with upregulation of M3 receptors in diverticular disease (diverticular disease 7.8% [1.9-20.
33 n's disease in 95, ulcerative colitis in 18, diverticular disease in 17, carcinoma in 25 (16 after ra
34 essionals (40-75 years old) who were free of diverticular disease in 1986 (baseline) was performed.
41 he use of laparoscopy in colon resection for diverticular disease is feasible and results in fewer co
44 oline did not differ between the two groups (diverticular disease mean 5.6% [SD 0.3], controls 5.2% [
45 acetylcholine was increased in patients with diverticular disease (mean -log EC(50) 5.6 [SD 0.3]) com
46 nsferase activity was lower in patients with diverticular disease (median 19.5%, range 12.0-30.0) tha
47 lon at CT colonography, representing chronic diverticular disease (n = 97) or sigmoid carcinoma (n =
49 arge, prospective study of men without known diverticular disease, nut, corn, and popcorn consumption
50 gy, clinical presentation, and management of diverticular disease of the colon and its complications.
52 ned as surgical intervention at any time for diverticular disease or presence of computed tomography
54 gastritis, gastric cancer, diarrhea, colonic diverticular disease, polyps, cancer, liver disease incl
57 y, 16 sequence variants are followed up in a diverticular disease sample from Denmark (5,970 cases, 3
59 de inflammation in symptomatic uncomplicated diverticular disease (SUDD), and the role of anti-inflam
60 laboratory investigations into the causes of diverticular disease suggests that disturbances in choli
61 acetyltransferase was lower in patients with diverticular disease than in controls: median percentage
62 cular disease in one twin when the other had diverticular disease was 14.5 (95% CI, 8.9-23) for monoz
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