ライフサイエンスコーパス: division

1 is a conserved, essential regulator of cell division.

2 ylation in septin filament bundling and cell division.

3 ochondrial fission machinery, and subsequent division.

4 lly associated with DNA replication and cell division.

5 to permit chromosome segregation during cell division.

6 by orienting the mitotic spindle during cell division.

7 glycogen accumulation, and to distorted cell division.

8 sion plane in newborn cells to ensure medial division.

9 a radiation of cellulases implicated in cell division.

10 the hematopoietic cytokine FLT3L during cell division.

11 of DNA damage associated with excessive cell division.

12 essential for DNA replication prior to cell division.

13 rminating embryos, when there is little cell division.

14 ein deposit formation to mother cells during division.

15 in the doubling of the genome prior to cell division.

16 determines the plane and orientation of cell division.

17 on site and were each required for efficient division.

18 ssential processes such as cell migration or division.

19 longed period of time in the absence of cell division.

20 ot transferred to daughter cells during cell division.

21 em cells maintain their identity during cell division.

22 developmental potential following asymmetric division.

23 egulate transcription, translation, and cell division.

24 glycan remodelling enzyme implicated in cell division.

25 e as well as cell cycle progression and cell division.

26 out the cell cycle and is not established at division.

27 tether essential for FtsZ functions in cell division.

28 ls that inhibits the rate of tumor stem cell division.

29 tein deposit formation to aging cells during division.

30 ic cells that play an important role in cell division.

31 tively adapt to osmotic changes and complete division.

32 a continuous process of cell death and cell division.

33 spindle is critical to the fidelity of cell division.

34 to the mammalian host and resumption of cell division.

35 s the fate of daughter cells upon asymmetric division.

36 in a narrow range by coordinating growth and division.

37 A sequence, which are heritable through cell division.

38 correct septal cell wall synthesis and cell division.

39 inates protein phosphorylation and may alter division.

40 ation until the appropriate time during cell division.

41 the rate of peptidoglycan synthesis and cell division.

42 e pole bodies (SPBs)] are essential for cell division.

43 y suggesting roles for such channels in cell division.

44 c precursor, which leads to repeated droplet division.

45 uired to prevent chromosome loss during cell division.

46 RNA chaperone controlling pneumococcal cell division.

47 s with extra centrosomes to avoid multipolar divisions.

48 ably also reduces the number of lateral cell divisions.

49 muscle myosin II (nmy-2) in these asymmetric divisions.

50 phy involving bed nucleus vs central nucleus divisions; (2) CRF content of the CEA-DA path; and (3) s

51 longation (anaerobic conditions) versus cell division (aerobic conditions) in the coleoptiles.

52 uch as zebrafish activate cardiomyocyte (CM) division after tissue damage to regenerate lost heart mu

53 ental changes, regeneration of tissues, cell divisions, aging, and pathological conditions observed i

54 ew that flagella play a central role in cell division among protists that lack myosin II and addition

55 og1 inside the placode, and apical symmetric divisions amplify the specified pool.

56 At the first meiotic division, anaphase-promoting complex/cyclosome associate

57 rol critical cell-fate decisions (e.g., cell division and apoptosis) can function with such low level

58 ading to mitotic catastrophe, defective cell division and apoptosis.

59 kinases (Cdks) are principal drivers of cell division and are an important therapeutic target to inhi

60 ite faces several alternating events of cell division and cell differentiation in which exponential a

61 Although both epithelial cell division and cell extrusion require Piezo1 at the steady

62 indle assembly checkpoint that controls cell division and cell fate.

63 it emerges from Sc-specific versions of cell division and cell merging that are shaped by cell expans

64 displayed symmetric distribution during cell division and could efficiently maintain pluripotency gen

65 lular processes, such as cell motility, cell division and endocytosis.

66 tial for chromosome condensation during cell division and functions in regulating gene expression dur

67 e among cells that had undergone their first division and identified previously unknown molecular det

68 contact with APC was maintained during cell division and led to an unequal inheritance of LFA-1 in d

69 depletion led to a complete block in nuclear division and profoundly inhibited DNA replication.

70 in disk periphery during this window of cell division and that CHMP7 can bind directly to the C-termi

71 (SCR), cooperatively direct asymmetric cell division and the patterning of root cell types by transc

72 Repeated cell divisions and aging impair stem cell function.

73 7 interaction by mAb induced asymmetric cell divisions and differentiation in AML blasts and AML stem

74 species with sensory responses in the dorsal divisions and premotor activity in ventral divisions of

75 nship between the number of normal stem cell divisions and the risk of 17 cancer types in 69 countrie

76 als are absent in the afd1 (absence of first division) and sgo1 (shugoshin) mutants during meiosis II

77 cs, fatty acid and cell wall synthesis, cell division, and cell shape.

78 oning to examine the coordination of growth, division, and chromosome dynamics at a single-cell level

79 w, we osmotically shock the cell during cell division, and find that the cell can actively adapt to o

80 has an important role in plant cell growth, division, and stress response.

81 tissues undergo endoreplication without cell division, and the latest replicating regions occasionall

82 irect fate conversion in the absence of cell division, and their multipotency at the population level

83 ecapitulate known cortical cytoarchitectonic divisions, and greater inter-regional morphometric simil

84 t they complete it, the number of successive divisions, and how cells coordinate proliferation with a

85 gh a signaling cascade leading to multipolar divisions, and its knockout promotes clustering and surv

86 We propose that cell division arrest is programmed in competent pneumococcal

87 appear to be involved in generating physical division asymmetry, but nonetheless is important for spe

88 d, the specific problem caused by asymmetric division at the transcription level has not yet been add

89 es that regulate spindle orientation and the division axis [1, 2].

90 ing cell, rather than the orientation of the division axis, facilitates the onset of this motion.

91 osomes are dependent on the motor for viable division because of its ability to cluster centrosomes a

92 y mature eggs during meiosis II, despite the divisions being separated by just a few hours.

93 The qualitative division between domain-general and domain-specific cogn

94 ally inactive mutant of PBP 2B supports cell division, but in this background the normally dispensabl

95 s into dynamic clusters is critical for cell division, but the interactions between protofilaments an

96 iscontinuous ring that drives bacterial cell division by directing local cell wall synthesis.

97 tain their bond distribution asymmetry after division by rapidly replenishing Ft-Ds bonds at new cell

98 Individual apoptotic enterocytes promote divisions by loss of E-cadherin, which releases cadherin

99 dynamic shape transformations and autonomous division can be activated by spatially confining azobenz

100 trands during DNA replication; however, cell division can reinforce H3K27me3 coverage at target regio

101 Telomeres, which are involved in cell division, carcinogenesis, and aging and constitute impor

102 a crucial structural role in assembly of the division complex, independent of catalysis, and that its

103 Asymmetric cell division, creating sibling cells with distinct developme

104 n of immunohistochemical markers of the cell division cycle (CDC) in 5 of the 16 neurogenic niches of

105 Cell division cycle 42 (CDC42) plays important roles in polar

106 primarily modulates the duration of the cell-division cycle by controlling the G1/S transition known

107 gents of malaria, have evolved a unique cell division cycle in the clinically relevant asexual blood

108 esenting replication initiation, replication-division cycle, and the global biosynthesis rate.

109 r link between the cilia life cycle and cell-division cycle.

110 prevents entry into the S-phase of the cell division cycle.

111 zation of the United Nations [FAO Statistics Division Database (FAOSTAT)], the Australian government,

112 Translational control during cell division determines when cells start a new cell cycle, h

113 We discover that whereas cell divisions drive tissue expansion along one axis, their c

114 from the "symmetry-breaking" periclinal cell divisions during the transition between stage I and stag

115 -449b expression improves the first cleavage division, epigenetic reprogramming and apoptotic status

116 ion, changes that were heritable across cell divisions even after the compound was removed.

117 bacteria Streptomyces undergo a massive cell division event in which the synthesis of ladders of spor

118 ion by adding a constant length between cell division events.

119 Organogenesis occurs through cell division, expansion, and differentiation.

120 a manner that requires the known peroxisome division factor PEROXISOME AND MITOCHONDRIAL DIVISION FA

121 division factor PEROXISOME AND MITOCHONDRIAL DIVISION FACTOR1 (PMD1).

122 complex CusCBA of the resistance-nodulation-division family that is essential for bacterial resistan

123 Genome haploidization involves two meiotic divisions following a single round of DNA replication.

124 Despite evidence of increased Treg cell division, Foxp3 expression was not stably maintained aft

125 for ABCs, were generated after multiple cell divisions from C57BL/6 donors but not from MHC class II-

126 The central player in bacterial cell division, FtsZ, is essential in almost all organisms in

127 acetylated Skp2, resulting in polyploid cell division, genomic instability, and oncogenesis.

128 The pattern of cell division, growth and separation during leaf development

129 x three-dimensional processes involving cell division, growth, migration, and rearrangement, all of w

130 phylum Fermentibacteria (formerly candidate division Hyd24-12).

131 ics and a premature shift to asymmetric cell divisions impairing progenitor cell pool expansion.

132 MAP KINASE17 (MPK17) in affecting peroxisome division in a manner that requires the known peroxisome

133 ding stress responses, development, and cell division in all kingdoms of life.

134 bipolar spindles, but it is not required for division in almost all normal cells.

135 Cell division in Escherichia coli is mediated by a large prot

136 atiotemporal regulation of motility and cell division in M. xanthus and illustrates how the study of

137 Cell division in most bacteria is mediated by the tubulin-lik

138 leads to misorientation of apical-basal cell division in nephron tubules.

139 Cell division in plant cells requires the deposition of a new

140 a phosphatase normally activated during cell division in proliferating cells.

141 on and apoptosis in crowded regions and cell division in sparse regions.

142 pendent HSC mobilization and EMH but not HSC division in the bone marrow.

143 contact-dependent orientation of planar cell division in the mammalian embryonic skin.

144 om the CUP1 array occur at a rate of >10(-3)/division in unselected cells.

145 " cells resulting from the first CD8+ T cell division in vivo exhibited low and high rates of endogen

146 ed this problem in the context of plant cell division in which a large number of TGN-derived membrane

147 Live imaging of meiotic divisions in condensin-depleted cells showed repeated nu

148 ge, antigen-specific cells underwent several divisions in draining lymph nodes (LN; DLNs) while maint

149 the estimated cumulative number of stem cell divisions in the associated tissue (p<0.05), although in

150 TCF1-expressing cells from later divisions in the DLN could self-renew, clonally yielding

151 pression, resulted in randomly oriented cell divisions in the presence of uniaxial stretch.

152 f addition, subtraction, multiplication, and division, including a carryover into multiple cells.

153 resence of antiviral therapy and during cell division induced by immune-mediated lysis of infected he

154 to our knowledge, new insight into how cell divisions influence CAM within a tissue.

155 gmentation by the treatment of mitochondrial division inhibitor 1 (mdivi-1), a mitochondrial fission

156 n regulators MinD and MinE together with the division inhibitor MinC localize to the membrane in conc

157 ical-basal determinants, ensuring asymmetric division into daughter cells of different fates.

158 xcept in the basal glaucophytes, chloroplast division involves two heteropolymer-forming FtsZ isoform

159 Cell division is a complex process that requires precise dupl

160 However, it is unclear how epithelial cell division is controlled to balance cell death at the stea

161 enome into gametes during the second meiotic division is coordinated by a conserved casein kinase 1.

162 Mitotic rounding during cell division is critical for preventing daughter cells from

163 Mitochondrial division is critical for the maintenance and regulation

164 Mean cell size at division is generally constant for specific conditions a

165 ogenitors, and suggesting that an asymmetric division is involved in the acquisition of gonadal cell

166 Cell division is mediated by filaments of FtsZ and FtsA (FtsA

167 developing nephron tubules reveals that cell division is not oriented in the longitudinal (or planar-

168 wly forming basal bodies; and 3) kinetoplast division is observed in G2 after completion of nuclear D

169 Symmetrical division is the best characterized mechanism of stem cel

170 Asymmetric cell division is the primary mechanism to generate cellular d

171 ly restricted following asymmetric stem cell division is unclear.

172 upstream signal to initiate asymmetric cell division is unknown.

173 rast, larval neuroblasts generate longer 50 division lineages, and currently only one mid-larval mol

174 ealed that the dynamins are part of the cell division machinery and that they mediate their effects o

175 In tubules, cell division may be oriented relative to two axes: longitudi

176 Whereas physical asymmetric division mechanisms and cell fate consequences have been

177 indings and compare these strategies to cell division mechanisms elucidated in model organisms.

178 on, as well as active cell behaviors such as division, migration, and tissue development, cells must

179 s in early mitotic entry that distracts cell division mode, leading to exhaustion of the progenitor p

180 mutagenic damage is expressed following cell division, more-rapidly renewing tissues could be at high

181 s also important for efficient mitochondrial division, morphological alterations of the mitochondrial

182 tochastically, a behavior essential for cell division, motility, and differentiation.

183 pacity limit of FTN non-orthogonal frequency-division multiplexing (NOFDM) signal is first demonstrat

184 However, epithelial cell division must be tightly linked to cell death to preserv

185 the CC lining on postnatal day 8 (P8), with division occurring in 19.2% +/- 3.2% of cells.

186 after reaching a size threshold), and timer (division occurs after a fixed time from birth).

187 radigms of cell-size control, such as adder (division occurs after adding a fixed size from birth), s

188 fter adding a fixed size from birth), sizer (division occurs after reaching a size threshold), and ti

189 Mitochondrial division requires division of both the inner and outer mitochondrial membr

190 g cell size asymmetry during asymmetric cell division of Drosophila neuroblasts (NBs).

191 ientation to polarity during asymmetric cell division of Drosophila neuroblasts.

192 We gained genetic insights to the division of labor among mycobacterial RNases H by deleti

193 e undergone apoptosis in situ has revealed a division of labor among the tissue resident phagocytes t

194 n", representing the first institutionalized division of labor beyond age and sex.

195 Social division of labor enables the collection of stores of fo

196 In mutualistic symbioses, such as division of labor, both parties can gain, but they might

197 and tribal regions, such as the Nagarkurnool division of Mahabubnagar district (which became Nagarkur

198 t has a pivotal role in directing asymmetric division of mammalian stem cells to sustain the stem cel

199 tural barrier has resulted in strong genetic division of populations into two sectors, with no detect

200 ng synergies, suggesting that this classical division of reach and grasp in PMd and PMv, respectively

201 involved in cell wall biosynthesis and cell division of Streptococcus pneumoniae.

202 example of a completely ossified otoccipital division of the braincase in a stem lobe-finned fish.

203 ity cell-autonomously control the asymmetric division of the first ground tissue cells.

204 e field properties of neurons in the ventral division of the medial geniculate body (MGBv).

205 s of propranolol into either the infralimbic division of the medial prefrontal cortex (mPFC) or the b

206 ction in growth rate and termination of cell division of the underlying epidermal cells.

207 is leaf epidermis [5], polarized, asymmetric divisions of stomatal stem cells (meristemoid mother cel

208 l divisions and premotor activity in ventral divisions of the SC.

209 During development, cell division often generates two daughters with different de

210 With division on the basis of the patients' phenotype, fibrob

211 transcription (STAT) protein to promote cell division only in OB-ISCs.

212 The question of how cell division orientation is determined is fundamentally impo

213 ts, and define a mechanism that couples cell division orientation to intercellular adhesion.

214 ty and cancer by limiting the number of cell divisions, our findings suggest that extending the lifes

215 tion between phyllotaxis and the apical cell division pattern indicating a position-dependent pattern

216 and yet the manner and degree to which cell division patterns affect airspace networks and photosynt

217 Our results indicate that cell division patterns influence the photosynthetic performan

218 ulated a mutation rate of 1.3 mutations per division per cell.

219 cells exhibited greater variability in cell division placement and exponential growth rate across in

220 spindle pole bodies, transient MTOCs in the division plane (eMTOCs) and nuclear-envelope associated

221 Vesicles are putatively delivered to the division plane by transport along microtubules of the bi

222 hrough this new function, the SIN resets the division plane in newborn cells to ensure medial divisio

223 These data indicate that division plane orientation, together with proper cell-cy

224 Proper division plane positioning is crucial for faithful chrom

225 We show that MapZ is important for proper division plane selection; thus, the question remains as

226 s how and when cells effectively place their division plane with respect to their neighbours.

227 mechanism(s) may concentrate the CPC at the division plane.

228 hus specifically inactivated by CYK-4 at the division plane.

229 uilt at discrete sites that moved around the division plane.

230 During bacterial division, polymers of the tubulin-like GTPase FtsZ assem

231 e frequency of symmetric and asymmetric cell divisions producing daughter cells capable of self-renew

232 Our data are complemented by cell division profiles and expression patterns of key genes,

233 first reported link between a GpsB-like cell division protein and factors important for escape from t

234 ntain a disulfide bond is the essential cell division protein FtsN, but the importance of this bond t

235 icillin-binding protein PBP 2B is a key cell division protein in Bacillus subtilis proposed to have a

236 Recently, the early division protein MapZ was identified and implicated in p

237 During our studies on GpsB, a late cell division protein that controls activity of the bi-functi

238 All other division proteins are thought to require the Z-ring for

239 li is one of the essential components of the division proto-ring that provides membrane tethering to

240 nkage' and 'QTL effect', makes a fine genome division, provides a comprehensive understanding of the

241 5 in reprogramming and may increase the cell division rate and result in an accelerated kinetics of i

242 The bacterial cell division regulators MinD and MinE together with the divi

243 thful segregation of chromosomes during cell division relies on multiple processes such as chromosome

244 n actomyosin dynamics during asymmetric cell division remain unclear.

245 Mitochondrial division requires division of both the inner and outer m

246 ng polarity in CE cells, and that asymmetric divisions resulting from CE polarity are required for co

247 NE cells undergoing proliferative, symmetric divisions retract their basal processes, and both daught

248 nts treadmilled circumferentially around the division ring and drove the motions of the peptidoglycan

249 The cytokinetic division ring of Escherichia coli comprises filaments of

250 nced by antibiotic susceptibility, misplaced division septa and cell lysis.

251 illation to help ensure that the cytokinetic division septum forms only at the mid-cell position.

252 We found that the division septum was built at discrete sites that moved a

253 uilding a peptidoglycan-like sacculus and/or division septum.

254 eprophase band and persisted at the cortical division site afterwards.

255 ZapA and ZauP co-localized at the division site and were each required for efficient divis

256 ation of Rga1 from the immediately preceding division site and, consequently, abnormal bud-site selec

257 as fully rescued by introduction of cortical division site localized TANGLED1-YFP.

258 stion remains as to what drives pneumococcal division site selection.

259 as identified and implicated in pneumococcal division site selection.

260 ore frequent Cdc42 repolarization within the division site when the first temporal step in G1 is assu

261 ire the Z-ring for recruitment to the future division site.

262 al peptidoglycan-synthesizing enzymes to the division site.

263 lizes to the immediately preceding and older division sites by interacting with Nba1 and Nis1.

264 l origin regions localize to the future cell division sites, before FtsZ.

265 During cell division, spindle fibers attach to chromosomes at centro

266 likely due to the fact that their growth and division takes place within an osmotically protected env

267 Meiosis is a specialized cell division that generates gametes, such as eggs and sperm.

268 ck' for the rapid molecular changes and cell division that result upon re-oxygenation.

269 first detected shortly after the asymmetric division that segregates the germ cell lineage.

270 from founder cells, triggering new formative divisions that generate lateral root primordia (LRP).

271 ll PLT genes can activate the formative cell divisions that lead to de novo meristem establishment an

272 During cell division, the mitotic kinesin-5 Eg5 generates most of th

273 Following asymmetric division, the mother cell engulfs the forespore, surroun

274 nical stretch itself rapidly stimulates cell division through activation of the Piezo1 channel.

275 TS167, suggesting that this drug blocks cell division through an off-target mechanism.

276 Healthy enterocytes inhibit stem cell division through E-cadherin, which prevents secretion of

277 l processes, ranging from germline stem cell division to epithelial tissue homeostasis and regenerati

278 n of progenitor cells from asynchronous cell division to G1 arrest and neuronal specification at the

279 h in some cell types it is possible for cell division to occur in the absence of centrosomes, these d

280 hastic conversion of normally symmetric cell divisions to asymmetric and vice versa during developmen

281 mation is complete before resumption of cell division, to provide this pathogen with the maximum pote

282 ngal xerophiles, metabolic activity and cell division typically halts between 0.700 and 0.640 water a

283 o occur in the absence of centrosomes, these divisions typically result in defects in chromosome numb

284 from differences in the number of stem-cell divisions underlying each tissue, leading to different m

285 l order reflecting preceding cell growth and division variations.The quiescence-exit process is noisy

286 A link between the epigenomic state and cell division versus cell elongation is suggested, as no diff

287 ility during developmentally controlled cell division via a network of protein-protein interactions i

288 ular portion of the ventral posterior medial division, VPMpc) of mice and the thalamic terminal field

289 countries within each age-specific analytic division, we multiplied randomly selected mean annual EM

290 When we disrupted feedback, apoptosis and divisions were uncoupled, and the organ developed either

291 gs suggest that mechanisms for rescuing cell division when the biochemical activity of PBP 2B is pert

292 viding a mechanism for cells to trigger cell division when they reach a threshold concentration of Cd

293 e controls the outcome: stretch induces cell division, whereas crowding induces extrusion.

294 ors remain internalized by cells during cell division, which enables tagging several generations of c

295 velopment is characterized by rapid cleavage divisions, which impose significant constraints on metab

296 clades plays a highly conserved role in cell division, while the distribution of a second subclade su

297 ation in processes related to cell cycle and division; while migration, adhesion and cell-to-cell com

298 cycle, while preserving the coupling of cell division with cell growth, remain uninvestigated.

299 epigenetic states are inherited through cell division, with intriguing mechanistic links to histone v

300 ltiple rounds of DNA replication and nuclear division without cytokinesis, resulting in a multinuclea