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1 prevents entry into the S-phase of the cell division cycle.
2 microtubule cytoskeleton throughout the cell division cycle.
3 matin need to be duplicated during each cell division cycle.
4 growth genes to promote entry into the cell division cycle.
5 ch cell growth is controlled during the cell division cycle.
6 nd controlled through the course of the cell-division cycle.
7 e transient interactions throughout the cell-division cycle.
8 coupled to a single complete eukaryotic cell division cycle.
9 centrioles and centrosomes in a single cell division cycle.
10 olume fluctuations that occur over each cell division cycle.
11 icant loss of protocell contents during each division cycle.
12 is not influenced by the status of the cell division cycle.
13 icting DNA replication to once in every cell division cycle.
14 ome replication and does not follow the cell division cycle.
15 companied by a withdrawal from the bacterial division cycle.
16 of cell division starting at the second cell division cycle.
17 on appears to be independent of the cellular division cycle.
18 s important roles in the control of the cell division cycle.
19 t on variation of protein content during the division cycle.
20 of the S phase program in the mammalian cell division cycle.
21 IDR controls entry into S phase of the cell division cycle.
22 on the pattern of DNA replication during the division cycle.
23 ntus that controls an early step in the cell division cycle.
24 fferent patterns of cleavage in their second division cycle.
25 , G1 to S phase progression within the first division cycle.
26 is the last critical decision during a cell-division cycle.
27 is responsible for re-initiation of the cell division cycle.
28 ogression of the eukaryotic cell through its division cycle.
29 lex on chromatin during G1 phase of the cell division cycle.
30 of cells in the committed phase of the cell division cycle.
31 most kinases that are known to regulate the division cycle.
32 ed that assumes expanding volumes and a cell-division cycle.
33 ome synthesis and specific steps in the cell division cycle.
34 d not affect the G1/S transition of the cell division cycle.
35 Cdks) are the central regulators of the cell division cycle.
36 is is likely to be under control of the cell division cycle.
37 e core of the machinery that drives the cell division cycle.
38 thesis of both proteins is required for each division cycle.
39 nto, continuation of, and exit from the cell division cycle.
40 ion of p21, a negative regulator of the cell division cycle.
41 ng progressed beyond mid-S phase in the cell division cycle.
42 in to one round of DNA replication each cell division cycle.
43 nthesis, a pattern similar to the eukaryotic division cycle.
44 attern of DNA synthesis during the bacterial division cycle.
45 her continue to proliferate or exit the cell division cycle.
46 erturbed cells synthesize DNA throughout the division cycle.
47 r link between the cilia life cycle and cell-division cycle.
48 olites change significantly through the cell division cycle.
49 tabolic fluxes are coordinated with the cell division cycle.
50 al activity per DNA copy throughout the cell division cycle.
51 of CDK1 expression had an impact on the cell division cycle.
52 into the regulation of CDK1 during the cell division cycle.
53 t reversible phosphorylation during the cell division cycle.
54 of interacting cells in a model of the cell division cycle.
55 ch other and with progression through a cell-division cycle.
56 ge in a characteristic manner throughout the division cycle.
57 g periodic gene expression in the human cell division cycle.
58 e for cytoplasmic territory and insulate the division cycle.
59 on in cytoskeletal organization and the cell division cycle.
60 nt and, therefore, able to re-enter the cell-division cycle.
61 times that emerge as 12-hr synchronized cell division cycles.
62 the morula stage after only two to four cell division cycles.
63 omatin is gradual and requires multiple cell division cycles.
64 ave traced these defects back to the nuclear division cycles.
65 circadian clock-dependent synchronized cell division cycles.
66 ach other, resulting in circadian-gated cell division cycles.
67 ect chromosome ends from erosion during cell division cycles.
68 omplex component, double-parked protein/cell division cycle 10-dependent transcript 1, is not only ne
69 rved dual-specificity phosphatase human cell-division cycle 14A (hCDC14A) associates with the actin c
70 cted a possible phosphorylation site by cell division cycle 2 (Cdc2), which directly phosphorylated r
71 ee genes, two nuclear (beta-tubulin and cell division cycle 2) and a gene from the plastid genome (th
76 wed that induction of expression of the cell division cycle 20 gene (Cdc20), a key regulator of the m
77 MAK associates with CDH1 (FZR1, fizzy/cell division cycle 20 related 1) and phosphorylates CDH1 at
79 over the E3 ubiquitin ligase Cdc20-APC (cell division cycle 20-anaphase promoting complex) as a centr
81 M phase, partially through induction of cell division cycle 25 (Cdc25) isoform C (Cdc25C) degradation
83 ype that is linked to overexpression of cell division cycle 25 (Cdc25)A phosphatase and cell-cycle de
85 lation of p27 at Thr187 was mediated by cell division cycle 25A (Cdc25A), confirmed using Cdc25A inhi
89 also decreased the levels of Cdc25B and cell division cycle 25C (Cdc25C) phosphatases with an increas
91 the let-7 target cell cycle regulators cell division cycle 34 (Cdc34) and E2F transcription factor 5
95 ock protein 90) and its cofactor Cdc37 (cell division cycle 37 protein) are crucial to prevent the ce
97 strate that T cell-specific deletion of cell-division cycle 42 (Cdc42) GTPase causes a profound loss
98 ulated by SRC-like adaptor 2 (Sla2) and cell division cycle 42 (Cdc42) independently of Sla2's role i
103 ses have suggested that reduced Duo and cell division cycle 42 (Cdc42) transcript expression is invol
104 dea, altered expression of genes in the cell division cycle 42 (CDC42)-CDC42 effector protein (CDC42E
106 nascent axon, and the Rho GTPase Cdc42 (cell division cycle 42) activates the mPar6alpha/Par3 (Par fo
110 the Rho family, including Rho, Rac, and cell division cycle 42, regulate the actin cytoskeleton.
112 ing S phase in yeast, and Sld3 recruits cell division cycle 45 (Cdc45) to minichromosome maintenance
113 the replicative helicase containing the cell division cycle 45 (Cdc45)/minichromosome maintenance 2-7
116 he origin-recognition complex (ORC) and cell-division cycle 6 (Cdc6) proteins recognize and encircle
117 xplore the effect of phosphorylation of cell division cycle 6 (Cdc6), a DNA replication initiation fa
121 HPT-JT is caused by mutations of the cell division cycle 73 (CDC73) gene, located on chromosome 1q
124 cells cease proliferation within one or two division cycles after infection by HTLV-1 or transductio
125 te7, thereby favoring the resumption of cell division cycling after pheromone-induced growth arrest.
127 e progression through each stage of the cell division cycle and as such are major targets for deregul
128 s with Cul4A/DDB1 during an unperturbed cell division cycle and both Chk1 phosphorylation and replica
131 Here we show that, when cells leave the division cycle and enter quiescence, telomeres gather in
132 it leads to a reversible exit from the cell division cycle and entry into G0, a cell cycle state cal
133 ission of the midbody at the end of the cell division cycle and for phosphorylation and activation of
134 d Scc2 chromatin association during the cell division cycle and found that the affinity of Scc2 for c
135 RNase enzymes, different phases of the cell division cycle and growth rates, and the existence of no
136 is deposited at centromeres during the cell division cycle and identify an evolutionally conserved p
137 he genome is completely duplicated each cell division cycle and in how the division of cells is spati
138 te aquatic environment, coordinates the cell division cycle and multiple cell differentiation events.
140 ormal centriole numbers within a single cell-division cycle and provide insights into the regulation
141 s of 53 conditional lethal mutations in cell division cycle and RNA synthesis related genes, revealin
143 ing yeast Saccharomyces cerevisiae, the cell division cycle and sporulation are mutually exclusive ce
144 ecific subcellular addresses during the cell division cycle and sporulation, and proper localization
145 destruction of Dup is necessary for the cell division cycle and suggest that Geminin inhibition can r
146 of Epac during different stages of the cell division cycle and the structural features that are impo
147 to explore the relationship between the cell division cycle and the yeast respiratory oscillation, in
148 ls first proliferate via a canonical mitotic division cycle and then enter an endocycle, resulting in
150 Embryos lacking MEK exhibit faster and extra division cycles and fail to undergo timely midblastula t
152 oordination with other processes during cell division cycles and response to environmental cues.
153 that metabolic cycling does not require cell division cycling and that metabolic synchrony does not r
154 ey cellular system coordinated with the cell division cycle, and major efforts in systems biology cur
157 rn division of Drosophila syncytial cortical division cycles, and conventional spindle-directed furro
160 We conclude that mRNA variations during the division cycle, as measured by microarrays, cannot by th
161 kinesin family member 18B (KIF18B) and cell division cycle associated 3 (CDCA3) were of confirmed re
163 cycling occurs during the phases of the cell division cycle associated with mass accumulation in thes
164 ust be able to block progression through the division cycle at key transition points (called "checkpo
165 produces >6,000 nuclei that, during the next division cycle, become encased in plasma membrane in the
167 ow-activity state during an unperturbed cell division cycle but at the same time keeps Chk1 primed to
168 ates the serial events required for the cell division cycle, but no Cdk1 substrate has been identifie
169 Ai treatments does not affect the numbers of division cycles, but the gametophytes exhibit anomalous
170 primarily modulates the duration of the cell-division cycle by controlling the G1/S transition known
171 1 and p27, which regulate the mammalian cell division cycle by inhibiting cyclin-dependent kinases (C
172 xperiment studied DNA replication during the division cycle by labeling an unperturbed culture with a
174 ce DNA damage, but also during aberrant cell-division cycles caused by activated oncogenes and inacti
175 ecrease in protein levels of cyclin B1, cell division cycle (Cdc) 25B, and Cdc25C, leading to accumul
177 the interaction of two oscillators, the cell division cycle (CDC) and the yeast metabolic cycle (YMC)
179 n of immunohistochemical markers of the cell division cycle (CDC) in 5 of the 16 neurogenic niches of
180 n the cell cycle, notably cyclin, E2F1, cell division cycle (CDC), and minichromosome maintenance (MC
181 other hand, cyclins A1, A2, B1 and B2, cell division cycle (CDC)2 and its kinase, CDC25 A and B, bud
182 at it segregated at the very end of the cell division cycle: cells showed a single fluorescent focus
185 lent to that in cells in the G1 phase of the division cycle does not mean there is any particular G1
186 at neighboring nuclei are highly variable in division-cycle duration and that neighbors repel one ano
187 es of oscillations are coupled with the cell division cycle, exhibit period determination by CK1 and
188 on by DDK to form an active CMG [Cdc45 (cell division cycle gene 45), Mcm2-7, GINS (Go, Ichi, Ni, and
189 olved in cellular processes such as the cell division cycle, gene transcription, the DNA damage respo
190 analysis of gene expression during the yeast division cycle has led to the proposal that a significan
191 ot show a change in stiffness throughout the division cycle, implying that enzymatic cell wall remode
192 e program of gene expression during the cell division cycle in a human cancer cell line (HeLa) was ch
193 m measurements of DNA replication during the division cycle in cells growing at different, and more r
195 cycling was detected in the G2 phase of the division cycle in fission yeast, consistent with the ide
196 to mating pheromones by arresting their cell-division cycle in G1 and differentiating into a cell typ
198 orm essential mitotic functions during every division cycle in mammalian cells, they are required in
200 ukaryotic chromosomes occurs once every cell division cycle in normal cells and is a tightly controll
201 remain relatively unchanged during the cell division cycle in primary human T lymphocytes and in mon
202 couples centrosome duplication from the cell division cycle in prostate cancer cells through CEP57, a
203 gents of malaria, have evolved a unique cell division cycle in the clinically relevant asexual blood
204 iardial flagella undergo a multigenerational division cycle in which the parental eight flagella migr
205 es such as cytokinesis and syncytial nuclear division cycles in Drosophila Pseudocleavage furrow memb
208 ubiquitin ligase during an unperturbed cell division cycle, in response to replicative stress and on
209 r of fundamental processes comprise the cell division cycle, including spindle formation, chromosome
210 ssive cascade known to underlie the parasite division cycle indicating that the unique relationship b
211 is transcribed periodically during the cell division cycle, indicating that properly timed gene expr
216 The orderly progression through the cell division cycle is of paramount importance to all organis
217 rganisms, divergence from the canonical cell division cycle is often necessary to ensure the proper g
220 frame and critique hypotheses about how the division cycle is regulated in wild-type and mutant cell
222 n such a state, progression through the cell division cycle is reversibly arrested in an orderly mann
226 ll fates; glucose, which stimulates the cell division cycle, is a potent inhibitor of sporulation.
227 rive photosynthetic cell growth and the cell division cycle; it also exhibits a highly choreographed
228 lpha is phosphorylated on Ser123 by the cell division cycle kinase Cdk2 beginning early in S phase an
231 ulatory nodes that govern both the host cell division cycle machinery and the initiation of centrosom
233 23, homolog of mammalian D123, is a new cell division cycle mutant with a G2 delay at permissive temp
235 individual cells were followed over multiple division cycles, no direct correlation was observed betw
238 nt work demonstrating similarity between the division cycle of C. crescentus and that of A. tumefacie
241 sitions of microtubule arrays throughout the division cycle of cells lacking a defined centrosome.
245 associated with the G0/G1 phase of the cell division cycle of slowly growing budding yeast, transcri
246 oposed pattern of DNA replication during the division cycle of the K12 cells analysed is not consiste
249 ication, dMyc is dispensable for the mitotic division cycles of both germline and somatic components
250 at this "consistency test" prevents repeated division cycles of normal cells but might become defecti
252 king advantage of the rapid synchronous cell division cycles of Xenopus eggs and cell-free systems de
256 Large T stabilization domain region to cell division cycle protein 20 (Cdc20) and, possibly, cdc20 h
264 activated protein kinase (MAPK) and the cell-division cycle protein kinase Cdc2, are known to be orga
266 uivocally that the roles of CycE/Cdk2 in GSC division cycle regulation and GSC maintenance are separa
269 Cytokinesis, the final stage of the cell division cycle, requires the proper placement, assembly
270 ysis, we found that MYC even within one cell division cycle resulted in a several-magnitude increase
271 Z-ring disassembly occurs at the end of the division cycle, resulting in the complete degradation of
274 duction pathways that regulate an early cell division cycle step and the gain of motility later in th
275 oincide exactly with the S phase of the cell division cycle, suggesting that oxidative metabolism and
276 found that, during the G2 phase of the cell division cycle, TFAP4 is targeted for proteasome-depende
280 cleavage cycles are replaced by longer cell division cycles that include gap phases and checkpoints.
281 embryos display defects in the rapid nuclear division cycles that precede gastrulation in nuclear mig
282 of nutrients triggers an exit from the cell division cycle, the induction of autophagy, and eventual
283 one and only one daughter centriole per cell division cycle, the prevailing view is that centriole ov
286 DNA checkpoint kinase that couples the cell division cycle to the circadian cycle abolishes synchron
287 amily A) and the AAA-type ATPase Cdc48 (cell division cycle), Ubr1 directs the substrate to proteasom
288 /DNA synthesis)-phase transition of the cell division cycle, ultimately resulting in decreased cell p
289 ion of membranous structures during the cell division cycle underlie the cell division defects in fbl
292 Cdk2-wt had no apparent effect on the cell division cycle, whereas Cdk2-dn inhibited progression th
293 that uncouples DNA replication from the cell division cycle, which is reinforced in metazoans by the
294 Cytokinesis is the last step of the cell-division cycle, which requires precise spatial and tempo
295 tes transition between G1 and entry into the division cycle, while CDKB is essential specifically for
296 nd is essential for coordinating the nuclear division cycle with cytokinesis through the cytokinesis
297 eplication occurred during a fraction of the division cycle with gaps before and after DNA synthesis,
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