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1 ferring glucose to an exogeneous acceptor, n-dodecyl beta-D-maltoside.
2 retention of Ste14p activity occurred with n-dodecyl-beta-d-maltoside.
3 spholiposomes from receptor solubilized in n-dodecyl-beta-d-maltoside analogs.
4 gastric H+, K(+)-ATPase was solubilized by n-dodecyl beta-D-maltoside and electrophoresed in blue nat
5 brane channel (Aquaporin-0) solubilized by n-Dodecyl beta-D-Maltoside and from previously published s
6 maintained during membrane solubilization by dodecyl beta-D-maltoside and purification by chromatogra
7            Proteins were solubilized using n-dodecyl beta-D-maltoside and separated using SDS-PAGE.
8 sence of lipids and the common surfactants n-dodecyl-beta-D-maltoside and octyl-D-glucoside, but they
9            The enzyme was solubilized with n-dodecyl-beta-D-maltoside and purified by multistep chrom
10 membranes were solubilized with 0.1% (w/v) n-dodecyl beta-D-maltoside, and the RC complex was purifie
11 s cultured fibroblasts were solubilized with dodecyl-beta-d-maltoside, and the F(1)F(0) was isolated
12 hydrophobic interaction chromatography using dodecyl beta-D-maltoside as the detergent.
13 urified Gt to photoactivated Rho (Rho*) in n-dodecyl beta-D-maltoside (DDM) examined by gel filtratio
14          Here we show that (1) the detergent dodecyl beta-d-maltoside (DDM) mediates the dissociation
15 he modified PDMS with a dynamic coating of n-dodecyl beta-d-maltoside (DDM), which prevents protein s
16 ethylsiloxane) channels using a mixture of n-dodecyl-beta-D-maltoside (DDM) and sodium dodecyl sulfat
17 s provided for the ability of the surfactant dodecyl-beta-D-maltoside (DDM) to prevent charge-induced
18 -octyl beta-D-glucopyranoside (OctGlc) and n-dodecyl beta-D-maltoside (DodGlc2).
19 the HASs were purified to homogeneity from n-dodecyl beta-D-maltoside extracts.
20 ontaining membranes were solubilized in 1% N-dodecyl-beta-D-maltoside in the presence of 0.4% sheep b
21 ecting a membrane protein complex within a n-dodecyl-beta-d-maltoside micelle, we demonstrated a powe
22 ionic sodium dodecyl sulfate than in neutral dodecyl-beta-D-maltoside micelles, and TM2 was disordere
23 n techniques in mild detergents, including n-dodecyl-beta-D-maltoside, n-tetradecyl-beta-D-maltoside,
24 n affect the menaquinone binding site, but n-dodecyl beta-D-maltoside preparations exhibit a relative
25 ue native PAGE and FRET assays revealed 1% n-dodecyl beta-d-maltoside-resistant cis-dimerization for
26  demonstrating that in micelles containing n-dodecyl-beta-D-maltoside, Rho exists as a mixture of mon
27   Receptors solubilized with the detergent n-dodecyl beta-D-maltoside were found to sediment as a sin
28 trifugation of mitochondria solubilized with dodecyl-beta-D-maltoside, with an approximately eightfol

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