ライフサイエンスコーパス: dominant lethal

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1 function and mutations in that position are dominant lethal.

2 have generated mutants within Orc1p that are dominant lethal.

3 urvive, expression of a single-tailed SSB is dominant lethal.

4 is, and the mutations behaved genetically as dominant lethals.

5 of indel, aneuploidy, environment-dependent dominant lethal, and recessive lethal mutation rates.

6 ants, four were recessive-lethal, three were dominant-lethal, and six had no discernible phenotype.

7 4 strains resistant to exogenously expressed dominant lethal D genes were experimentally evolved.

8 amino-terminally deleted parB alleles has a dominant lethal effect resulting in the inhibition of ce

9 missing different C-terminal fragments have dominant lethal effects because they block cell division

10 findings provided a rough measure of induced dominant-lethal frequencies.

11 h based on mosquitoes carrying a conditional dominant lethal gene (release of insects carrying a domi

12 lity, or both; the release of Aedes carrying dominant lethal genes, such as the OX513A strain of A. a

13 e release of transgenic mosquitoes harboring dominant lethal genes, the introduction of arbovirus-blo

14 nsects that are homozygous for a repressible dominant lethal genetic construct rather than being ster

15 race amounts of nonphosphorylatable Cdc6 are dominant lethal in strains bearing nonphosphorylatable O

16 pletely stabilize the protein, we isolated a dominant lethal mutant, CSE4-351, that was stable.

17 ives protein translocation, we purified four dominant lethal mutants of BiP.

18 turnover, a genetic screen was conducted for dominant lethal mutants.

19 spatially distant intragenic suppressor of a dominant lethal mutation in the guanine nucleotide-bindi

20 also placed in cis with the plasmid-encoded dominant lethal mutation.

21 Screening for suppressors of dominant lethal mutations of essential genes is challeng

22 Suppressors of the dominant lethal phenotype have led to the identification

23 sm of translation initiation and explain the dominant lethal phenotype of the H69 mutation.

24 The dominant lethal phenotype of these mutant ORC complexes

25 ion of the cysteine mutant pair results in a dominant lethal phenotype that requires the presence of

26 the universally conserved A2451 conferred a dominant lethal phenotype when expressed in E. coli.

27 give Mi-DS3-induced necrosis, but produced a dominant lethal phenotype when introduced into Mi-1.2.

28 Mutations at either position confer a dominant lethal phenotype when the mutant 23 S rRNA is c

29 ng them, the severely affected mutants had a dominant lethal phenotype, and even the intermediate mut

30 ith a nonaromatic amino acid gives rise to a dominant lethal phenotype, and the altered gp2.5 has red

31 were found in genetic screening to exhibit a dominant lethal phenotype, resulting in drastic loss in

32 rmation, but many of these mutations cause a dominant lethal phenotype, which prevents production of

33 th such proposals, deletion of H69 confers a dominant lethal phenotype.

34 co-expressed wild-type ATPase and lead to a dominant lethal phenotype.

35 variants whose expression in vivo leads to a dominant lethal phenotype.

36 This dominant-lethal phenotype was attributed to error catast

37 s with side chains larger than valine confer dominant lethal phenotypes.

38 Unlike the dominant lethal proteins, these proteins do not require

39 i tolerates at least low-level expression of dominant lethal Ran mutants.

40 t lethal gene (release of insects carrying a dominant lethal, RIDL) is being developed to control the

41 RIDL((R))(1) (Release of Insects carrying a Dominant Lethal) technology is a proposed modification o

42 We select for phage encoding a dominant lethal version of gene 2.5, whose viability is

43 of these endpoints, as well as for presumed dominant lethals, were found among various postspermatog

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