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1 ans for augmenting the GVL effect of delayed donor lymphocyte infusion.
2 the graft-versus-leukemia effects of delayed donor lymphocyte infusion.
3 by day 200 after cyclosporine withdrawal and donor lymphocyte infusion.
4 rdizing the graft-versus-leukaemia effect of donor lymphocyte infusion.
5 sease occurred in 5 of 14 RIC patients after donor lymphocyte infusion.
6 5 days post-transplantation and responded to donor lymphocyte infusion.
7 resolved after treatment with rituximab and donor lymphocyte infusion.
8 nd one was reinduced into continuous CR with donor lymphocyte infusion.
9 omplete donor chimerism without the need for donor lymphocyte infusion.
10 of the anti-leukemia response in vivo after donor lymphocyte infusion.
11 a complete cytogenetic remission after CD4+ donor lymphocyte infusion.
12 se (GVHD), and the toxic effects of adjuvant donor-lymphocyte infusion.
13 GVHD and seven (14%) had chronic GVHD before donor-lymphocyte infusion.
14 unotherapy of hematologic malignancies using donor lymphocyte infusions.
15 found to inhibit the GVL response of delayed donor lymphocyte infusions.
16 l transplantation (alloTCD-HSCT) followed by donor lymphocyte infusions.
17 e response with reduced immunosuppression or donor lymphocyte infusions.
18 pse either prior to or during treatment with donor lymphocyte infusions.
19 ent was accomplished only following multiple donor lymphocyte infusions.
20 s a platform for adoptive immunotherapy with donor lymphocyte infusions.
23 xperienced a complete remission after CD4(+) donor lymphocyte infusions also developed high-titer ant
25 ere isolated from peripheral blood after CD4 donor lymphocyte infusion and recognition of donor-deriv
33 c cells, we assessed whether GVHD induced by donor lymphocyte infusion (DLI) affects the persistence,
35 Ten dogs also received escalating doses of donor lymphocyte infusion (DLI) alone or with pentostati
36 LVL (mean, 11 L) to collect lymphocytes for donor lymphocyte infusion (DLI) and other therapies was
38 geneic stem cell graft followed by postponed donor lymphocyte infusion (DLI) can experience graft-ver
41 In the current study, we examined whether donor lymphocyte infusion (DLI) could be used as adoptiv
42 raft-versus-leukemia-based therapies such as donor lymphocyte infusion (DLI) for chronic myelogenous
43 te for patients who achieved remission after donor lymphocyte infusion (DLI) for progression, was 65%
46 atched, nonmyeloablative allogeneic SCT with donor lymphocyte infusion (DLI) in patients with lymphoi
48 geneic bone marrow transplantation (BMT) and donor lymphocyte infusion (DLI) provide valuable treatme
56 ulations play a critical role in response to donor lymphocyte infusion (DLI), an established and pote
57 lant may achieve durable remission following donor lymphocyte infusion (DLI), showing the potency of
59 their phenotype, and their role in governing donor lymphocyte infusion (DLI)-mediated alloresponses.
64 performed of the response to treatment with donor lymphocyte infusions (DLI) and the survival in 66
68 disease (GVHD) by delayed administration of donor lymphocyte infusions (DLI) to established mixed ch
70 cal immunosuppression on the in vivo fate of donor lymphocyte infusions (DLI)-derived T cells, their
73 lymphocytes from the original marrow donor (donor lymphocyte infusions [DLI]) is remarkably effectiv
75 tem cell transplantation (HSCT) and received donor lymphocyte infusions (DLIs) after transplantation.
76 after reduced-intensity allografts and while donor lymphocyte infusions (DLIs) can be effective salva
77 of this strategy remains of interest because donor lymphocyte infusions (DLIs) can induce a potent gr
79 nancies often are treated with unmanipulated donor lymphocyte infusions (DLIs) from the transplant do
80 We describe the toxicity and efficacy of donor lymphocyte infusions (DLIs) given to 81 patients (
83 y the mechanisms governing the activation of donor lymphocyte infusions (DLIs) manifesting as lymphoh
88 ent of animals with NST, posttransplantation donor lymphocyte infusions (DLIs), and a vaccine, compri
90 ronic myeloid leukemia patients who received donor lymphocyte infusions following transplant relapse.
91 ntation is being explored through the use of donor lymphocyte infusions for patients who have relapse
95 n proven by the demonstration of response to donor lymphocyte infusion in patients relapsing after al
97 evidence for its presence is the efficacy of donor lymphocyte infusions in generating anti-tumor resp
100 hematopoietic stem cell transplantation and donor lymphocyte infusion is essential for acute graft v
103 osuppression, antiviral therapy, unprocessed donor lymphocyte infusion, mobilized peripheral blood pr
105 nt research is focusing on methods of making donor lymphocyte infusions more effective in the nonchro
106 ant cells or mixed chimerism could receive a donor lymphocyte infusion of 0.5 to 2 x 10(8) mononuclea
107 by cytoreductive therapy, followed either by donor lymphocyte infusion or second HSCT for consolidati
108 eduction of immune suppression plus or minus donor lymphocyte infusion or stem cell boost, which stab
112 eated for relapsed MM after alloTCD-HSCT and donor lymphocyte infusions, supporting the development o
113 All types of cell-based therapies, from donor lymphocyte infusion to dendritic cell vaccines, an
114 chimerism in the presence of GVHD after CD4 donor lymphocyte infusion was observed in a patient, HLA
118 nd can preclude the administration of graded donor lymphocyte infusions, which may optimize the thera
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