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1 opamine receptor agonist) and haloperidol (a dopamine receptor antagonist).
2 ncreases were not inhibited by a D1 subclass dopamine receptor antagonist.
3 oselective synthesis of (+)-sonepiprazole, a dopamine receptor antagonist.
4 mate receptor antagonist, or SCH 23390, a D1 dopamine receptor antagonist.
5 ion and depression-like symptoms, than other dopamine receptor antagonists.
6 nge alcohol drinking, and its sensitivity to dopamine receptor antagonists.
7 domisation was stratified by baseline use of dopamine receptor antagonists.
8 SCH 23390) or D2-like (100 microM sulpiride) dopamine receptor antagonists.
9 dopamine neuron stimulation and resistant to dopamine receptor antagonists.
10 phrenia have in common the property of being dopamine-receptor antagonists.
14 the effect of co-administering SCH23390 (D1 dopamine receptor antagonist) and quinpirole (D2 dopamin
17 bute to the pathophysiology of migraine, and dopamine receptor antagonists are prescribed in the trea
21 l history of tardive dystonia resulting from dopamine-receptor antagonist (DRA) treatment in 107 pati
22 Prolixin(R)) is a potent phenothiazine-based dopamine receptor antagonist, first introduced into clin
23 ion, was attenuated by pretreatment with the dopamine receptor antagonist, flupenthixol, into the cor
25 f systemic administration of the nonspecific dopamine receptor antagonist flupentixol on response inv
26 intra-accumbens microinjection of either the dopamine receptor antagonist flupentixol or the nicotini
27 n determined; in addition, the effect of the dopamine receptor antagonist fluphenazine (0.1-1.0 mg/kg
35 the increase in dopamine turnover and by the dopamine receptor antagonists, haloperidol, clozapine, a
36 iously, systemic administration of selective dopamine receptor antagonists has been shown to reduce a
38 ipsychotic potential of a highly specific D4 dopamine receptor antagonist (ie, L-745,870) in patients
40 ng/ml PTX or with 1 microM L745,870, a D(4) dopamine receptor antagonist, indicating that activation
42 the nucleus accumbens core and infusion of a dopamine receptor antagonist into the contralateral dors
43 perant tasks and determined how injection of dopamine receptor antagonists into the accumbens influen
44 Like VTA baclofen injection, injection of dopamine receptor antagonists into the NAc profoundly re
47 y D2 receptor blockade determines whether D2 dopamine receptor antagonist-mediated gene expression is
48 ors, hopes have run high that a selective D4 dopamine receptor antagonist might improve the pharmacol
50 yperactivity was abolished by treatment with dopamine receptor antagonists or catecholamine biosynthe
52 en injected systemically with flupentixol, a dopamine receptor antagonist, or vehicle before testing
53 nit activity was unaffected by D1 or D2-like dopamine-receptor antagonists, or by inhibition of evoke
54 s prolonged by perfusion of the selective D2 dopamine receptor antagonist raclopride (100 microM) int
56 pical antipsychotic drug that is primarily a dopamine receptor antagonist, reduced 5-HT2C VNV isoform
58 ion of haloperidol (0.5 or 1.0 mg/kg, sc), a dopamine receptor antagonist, reversed both the behavior
59 bilateral intracranial injections of the D-1 dopamine receptor antagonist SCH 23390 HCl (0, 0.8, 1.6,
60 luence of bilateral VTA injections of the D1 dopamine receptor antagonist SCH 23390 or the 5-HT2 rece
62 hold was significantly increased by the D(1) dopamine receptor antagonist SCH23390 (10 microg kg(-1),
63 d persistent activity was enhanced by the D1 dopamine receptor antagonist SCH23390 [R(+)-7-chloro-8-h
64 t-LTP was completely blocked by the D1-like dopamine receptor antagonist SCH23390, but not by the D2
65 e, although, for a minority of units, the D1 dopamine-receptor antagonist SCH23390 attenuated neural
66 ed" methamphetamine seeking, using selective dopamine receptor antagonists (SCH39166 or raclopride) a
67 reatment of cells with 10 muM SCH23390, a D1 dopamine receptor antagonist, significantly inhibited th
68 electrophysiological action of droperidol, a dopamine receptor antagonist, since these cultures conta
70 Tardive dyskinesia results from exposure to dopamine receptor antagonists, such as typical and atypi
71 and can be blocked in rats and monkeys with dopamine receptor antagonists, suggesting that increased
73 ts, whereas administration of D1- or D2-like dopamine receptor antagonists to further reduce dopamine
74 by measuring the ability of sulpiride, a D2 dopamine receptor antagonist, to increase the depolariza
76 ed reinstatement by all compounds, whereas a dopamine receptor antagonist was effective only in block
78 dyskinetic clozapine (1) is a low-potency D2 dopamine receptor antagonist which appears to act select
79 se in the striatum, and haloperidol (hal), a dopamine receptor antagonist with high affinity for D2-l
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