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1 T) axons descends in the ventral part of the dorsal column.
2 t collaterals in Lissauer's tract and in the dorsal column.
3 ch express TRPV1 in Lissauer's tract and the dorsal column.
4 3 compared with the contralateral unlesioned dorsal column.
5 3b after complete or partial lesions of the dorsal columns.
6 axonal loss in the corticospinal tracts and dorsal columns.
7 be explained by conduction delays within the dorsal columns.
8 neurons in the corticospinal tracts and the dorsal columns.
9 more similar to peripheral nerve than intact dorsal columns.
10 gray matter rather than in the degenerating dorsal columns.
11 illimeters rostral to a severe lesion of the dorsal columns.
12 ollowing the onset of anoxia in demyelinated dorsal columns.
13 ses in immunostaining in the gray matter and dorsal columns.
14 llum, brainstem, spinocerebellar tracts, and dorsal columns.
15 ining was also prominent along the ascending dorsal columns.
16 cal injections of S-AMPA or NMDA into lumbar dorsal columns.
17 infused with vehicle for two weeks into the dorsal column, 3 mm rostral to the nerve graft, essentia
18 Following a partial section, the remaining dorsal column afferents continue to activate neurons wit
20 er large but incomplete lesions of ascending dorsal column afferents in the cervical spinal cord, the
23 macrophages in axonal retraction within the dorsal columns after spinal cord injury in vivo and foun
24 ter near the crush site and in the ascending dorsal column also exhibited increased Mac1 staining tha
27 eptive fields of spinocervical, postsynaptic dorsal column and spinothalamic tract neurons was used t
28 nkeys with chronic unilateral lesions of the dorsal columns and expanded chin representation, connect
29 he injury, HO-1 was primarily induced in the dorsal columns and occasionally in the lateral white mat
30 lso severely affected, with shrinkage of the dorsal columns and reduction in the number of neurones i
31 f the main dorsal corticospinal tract in the dorsal columns and some damage to the adjacent medial pa
32 expressed in the synaptic terminations from dorsal columns and trigeminal brainstem nuclei, the effe
34 rocesses were seen along fiber tracts in the dorsal columns and, after intracerebral injection, subja
35 important relays for indirect, postsynaptic dorsal column, and direct ascending pathways, respective
37 ted into the superficial dorsal horn and the dorsal columns, as well as across the dorsal commissure.
38 lcium transients within Lissauer's tract and dorsal column at L5 segments when compared to the calciu
39 ipolar ball electrode was placed on the left dorsal column at lumbar spinal cord segments 2-3 in sodi
41 ed to the calcium transients only within the dorsal column at the lumbar 2 (L2) segments, which were
42 ipolar ball electrode was placed on the left dorsal column at the lumbar 2-3 spinal cord segments (L2
43 ipolar ball electrode was placed on the left dorsal column at the lumbar 2-3 spinal cord segments (L2
44 ipolar ball electrode was placed on the left dorsal column at the lumbar 2-3 spinal segments in rats.
46 dorsal hemisection promotes regeneration of dorsal column axons across and beyond the lesion site in
47 This same compound promotes regeneration of dorsal column axons after acute lesions and potentiates
48 hwann cell-mediated remyelination of central dorsal column axons and whether Nrg1 ablation influences
49 lted in the total failure in regeneration of dorsal column axons consequent on the lack of GAP43 upre
50 h wild-type DRG neurons, and regeneration of dorsal column axons does not occur after a conditioning
52 ditioning lesion of the peripheral branch of dorsal column axons is a well-known paradigm enabling th
53 ing cells, suggesting that the retraction of dorsal column axons is not a secondary effect of increas
56 sal column crush resulted in regeneration of dorsal column axons up to and beyond the injury site int
57 utcomes of previous studies, regeneration of dorsal column axons was compared following transplants o
59 n and the electrophysiological properties of dorsal column axons were characterized after transplanta
60 cal doses, both in vivo and in vitro, to rat dorsal column axons which had been experimentally demyel
64 yelinated central axons, particularly in the dorsal columns, become associated with peripheral myelin
65 thin 12 min of the onset of anoxia in normal dorsal columns, but was not abolished until 50 min follo
66 ating lesions were induced in the rat spinal dorsal column by microinjection of lipopolysaccharide, a
67 focal demyelination in the mouse spinal cord dorsal column by microinjection of lysophosphatidylcholi
69 12.2% of control amplitude) in demyelinated dorsal columns compared to controls (30.8 +/- 5.3%) foll
72 el of axonal dieback as well as an adult rat dorsal column crush model of spinal cord injury, we foun
74 to the sciatic nerve 1 week before bilateral dorsal column crush resulted in regeneration of dorsal c
76 or spinal cord corticospinal tract (CST) and dorsal column (DC) axons in both monophasic (C57/BL6 mic
77 mation (MRF) neurons after acute and chronic dorsal column (DC) lesions of the T8 spinal cord and los
79 ctal distension (CRD), whereas lesion in the dorsal column (DC) reverses this neuronal sensitization,
80 y by forming neuronal relays between injured dorsal column (DC) sensory axons and the denervated dors
82 collaterals of dorsal root axons within the dorsal column (DC) was studied after neonatal spinal ove
84 atter (VGM), ventral white matter (VWM), and dorsal columns (DC) at levels ranging 15 mm rostral and
85 sneuronal atrophy occurred in neurons of the dorsal column (DCN) and ventral posterior lateral thalam
86 that delivers electrical stimulation to the dorsal column (DCS) of the spinal cord to suppress epile
88 In NG2 knock-out mice, sensory axons in the dorsal columns dieback further than their control counte
90 set blindness, cerebellar ataxia, nystagmus, dorsal column dysfuction, and spasticity with upper moto
92 my, DTI revealed a lesion in the ipsilateral dorsal column extending from the lumbar to the cervical
94 ere used to record field potentials from the dorsal columns following distal local surface stimulatio
96 and behavioural consequences of cutting the dorsal column have been reported to range from mild and
97 l cord of adult rats or the injury site in a dorsal column hemisection model of spinal cord injury, a
99 on reduced immunostaining of the ipsilateral dorsal columns in segments rostral to the lesion and in
101 ave re-examined this issue by sectioning the dorsal columns in the cervical region and recording the
102 that epidural electrical stimulation of the dorsal columns in the spinal cord restores locomotion in
103 ripheral nerve graft bridging the transected dorsal columns in the thoracic spinal cord resulted in a
104 d axons, we studied axonal conduction in rat dorsal columns in vitro following a 60-min period of ano
105 o examine the spinal cord in rats with focal dorsal column inflammatory or demyelinating lesions to d
106 what extent this brain reorganization due to dorsal column injuries is related to axonal sprouting is
107 ndergo severe atrophy 4 weeks after thoracic dorsal column injury and that ChABC is neuroprotective f
108 onditioning lesion 1 or 2 weeks prior to the dorsal column injury results in growth into the spinal c
109 l pattern of callosal connections depends on dorsal column input and not on normal interhemsipheric i
110 sal root lesion (DRL), (2) or a combined DRL/dorsal column lesion (DRL/DCL), or (3) a DRL/DCL where t
111 , the relationship between the extent of the dorsal column lesion and the amount of cortical reactiva
112 luation of total soluble CSPGs 2 weeks after dorsal column lesion in the rat demonstrated that NG2 is
113 on across the monkeys shows that even if the dorsal column lesion is partial, preserving most of the
121 cortex were more widespread after incomplete dorsal column lesions (DCLs) than after a complete DCL.
122 ntly higher acutely than those of LINGO-1 in dorsal column lesions and reduced in models of dorsal ro
123 eorganization of digit representations after dorsal column lesions and reveals differences between fu
125 mus are affected by long standing unilateral dorsal column lesions at cervical levels in macaque monk
126 cuneate nucleus that survives high cervical dorsal column lesions by traveling in the lateral funicu
127 itory protein, CD59 (hCD59), into transected dorsal column lesions of the spinal cord of the immunosu
128 the near-completeness of the more extensive dorsal column lesions we studied, we suggest that altern
130 In three other squirrel monkeys, unilateral dorsal column lesions were placed at the cervical segmen
132 nucleus through pathways that circumvent the dorsal column lesions, contributing to cortical reactiva
133 on and ethidium bromide-induced demyelinated dorsal column lesions, with or without co-transplantatio
138 the few spared primary axon afferents in the dorsal columns, likely have a major role in the reactiva
140 are found: 1) ascending ipsilaterally in the dorsal column near the dorsal intermediate septum or the
141 that distinguish ventral, intermediate, and dorsal column neuroectoderm and neuroblasts, and a cell
142 nts have a transformation of intermediate to dorsal column neuroectoderm fate, and only 10% of the in
143 m dorsal root ganglions to a small number of dorsal column neurons, a caudal extension of nucleus gra
146 Tactile receptive fields (RFs) of brainstem dorsal column nuclei (DCN) neurons rapidly enlarge when
147 ance P receptor binding was localized in the dorsal column nuclei (DCN) of the rat, cat, monkey, and
148 sizes and plasticity of RFs in the brainstem dorsal column nuclei (DCN) when peripheral inputs were n
150 cluding excitatory projection neurons of the dorsal column nuclei and for vital components of the int
154 on of labeled neurons in the spinal cord and dorsal column nuclei following injections of Fluoro-Gold
155 Based on their anatomical connections, the dorsal column nuclei may contribute to thalamic changes
156 luded the effects of TEA and 4-AP in NTS and dorsal column nuclei neurones, but not in dorsal vagal n
158 axonal accumulation of neurofilaments in the dorsal column nuclei of the cat by using immunohistochem
160 t in mammals, involving projections from the dorsal column nuclei to the thalamus, and thence to soma
161 ed with somatosensory function, although the dorsal column nuclei were also labelled above control.
162 ory inputs from the somatosensory cortex and dorsal column nuclei were found to terminate almost excl
164 ory pathways originating in the dorsal horn, dorsal column nuclei, and trigeminal nuclei, and the oth
165 in segments rostral to the lesion and in the dorsal column nuclei, presumably from the loss of ascend
166 uded neurons in sensory pathways such as the dorsal column nuclei, the area postrema, the spinal trig
172 n to examine the physiological properties of dorsal column nucleus neurons during persistent pain.
173 cord send long ascending projections to the dorsal column nucleus that are important in the transmis
174 bulbs, optic tectum, octavolateral area, and dorsal column nucleus, and it innervates, apart from the
175 and medial nuclei of the octavolateral area, dorsal column nucleus, solitary tract nucleus, motoneuro
176 (SpVi) and subnuclei caudalis (SpVc) and the dorsal column nucleus-based lemniscal and cortical pathw
177 tion potential conduction in the spinal cord dorsal column of adult rats in which focal demyelinating
178 progressive loss of myelin in the brain and dorsal column of the spinal cord associated with increas
181 igalacto-cerebroside, are implanted into the dorsal columns of 4-8 d rat spinal cord, some of the mye
182 ult CNS, ES cells were transplanted into the dorsal columns of adult rat spinal cord 3 days after che
186 in ventral, ind in intermediate, and msh in dorsal columns of fly neurectoderm, and of homologous ge
187 d severely impaired axonal conduction in the dorsal columns of global Nrg1 mutants (where Schwann cel
191 ound demyelinating phenotype observed in the dorsal columns of Nrg1 mutant mice is related to conduct
192 ins of adult monkeys with chronic lesions of dorsal columns of spinal cord at cervical levels undergo
195 cells were transplanted into the transected dorsal columns of the rat spinal cord to induce axonal r
196 the ascending afferents from the hand in the dorsal columns of the spinal cord in monkeys is followed
197 ucleus in adult monkeys after lesions of the dorsal columns of the spinal cord or therapeutic amputat
200 after section of ascending afferents in the dorsal columns of the spinal cord, it reorganizes to ove
201 imb in monkeys because of transection of the dorsal columns of the spinal cord, therapeutic amputatio
204 ty is achieved by ipsilateral lesions of the dorsal columns or lidocaine microinjection into the nucl
205 Rats with incomplete lesions that spared the dorsal column pathway on the left side but not the right
206 own that the gracile nucleus in postsynaptic dorsal column pathway plays an important role in conveyi
212 on of long axons in corticospinal tracts and dorsal columns, resulting in spasticity and difficulty w
213 axons in mice with selective lesions of the dorsal column revealed that DLCST axons arborize extensi
214 sh motor output longitudinally after a C3/C4 dorsal column SCI that bilaterally ablated the dorsal co
217 Previous studies have shown that injured dorsal column sensory axons extend across a spinal cord
218 infusions through a lumbar puncture rescued dorsal column sensory axons innervating the nucleus grac
219 s underwent C3 lesions to transect ascending dorsal column sensory axons, and autologous bone marrow
221 eration of several spinal systems, including dorsal column sensory, regionally projecting propriospin
222 etrated control fibroblast grafts, including dorsal column sensory, rubrospinal, and nociceptive axon
224 stepwise multiple regression showed that the dorsal column signal and diagnosis subtype alone explain
227 dorsal half, Ia fibers entering through the dorsal columns still grew ventrally within the host dors
232 spinal cord lesion, even when <1% of labeled dorsal column terminations in the cuneate nucleus remain
233 he central branch of the same neurons in the dorsal column, these central axons will regenerate and,
236 s of axon injury and regeneration, including dorsal column transection, dorsal root rhizotomy and per
239 c organization of the gracile nucleus of the dorsal column-trigeminal complex, neuroanatomical tracer
240 ascin-C-rich dorsal root entry zone into the dorsal column up to C1 level and above (>25 mm axon leng
241 jury from both sides including epicenter and dorsal column was microdissected and used for further st
242 ion, inflammatory cell infiltration into the dorsal columns was reduced in phenytoin-treated mice wit
246 phosphorylated NFs of the small axons in the dorsal columns were widely spaced, suggesting that phosp
247 ndary lesion extending rostrocaudally in the dorsal columns (where long ascending and descending fibe
248 of the graft had grown up to 0.5 mm into the dorsal column white matter, 30+/-2% up to 1 mm, 19+/-3%
249 d injury, sensory neurons that course in the dorsal columns will regenerate, presumably because their
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