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1 erent RF centers that lie within the RF of a dorsal horn cell.
2 gence between cutaneous mechanoreceptors and dorsal horn cell are constant was examined.
3  primary afferent mechanoreceptive axons and dorsal horn cells are invariant over skin location and d
4 the postsynaptic activity evoked in neonatal dorsal horn cells by cutaneous afferents differs conside
5 , and form nonselective synapses with nearby dorsal horn cell dendrites, establishing the initial dor
6                    The parallel variation of dorsal horn cell dendritic domain width and primary affe
7 bition takes place when the sensitization of dorsal horn cells develops, and PKC may play a significa
8 vo extracellular recordings were made of 171 dorsal horn cells in both superficial and deep laminae i
9 onsiderable sensitization in a population of dorsal horn cells in the neonate.
10 ing the spatial and modality organization of dorsal horn cell inhibitory receptive fields (RFs) in de
11 neous afferent ingrowth, suggesting that the dorsal horn cells might provide important cues guiding s
12 ields was used to simulate RFs of the entire dorsal horn cell population in order to estimate RF area
13 In contrast, following neonatal skin injury, dorsal horn cell receptive field sizes were significantl
14 d a quantitative model of the development of dorsal horn cell receptive fields (RFs) and somatotopic
15                       A descriptive model of dorsal horn cell receptive fields was used to simulate R
16                        The initial embryonic dorsal horn cell RF assembly hypothesis was supported by
17 orn cell dendrites, establishing the initial dorsal horn cell RFs.
18 nset neuropathic pain behavior and increased dorsal horn cell sensitivity to cutaneous mechanical and
19  previously proposed model of development of dorsal horn cell somatotopy and RF geometries must be re
20        However, the response pattern of deep dorsal horn cells to heat has not been well described.
21 ld properties and evoked activity of newborn dorsal horn cells to single repetitive and persistent in
22 us mechanism for altering the sensitivity of dorsal horn cells to substance P, potentially via the ac
23 cannabinoids in the descending inhibition of dorsal horn cells via a supraspinal site of action.
24  First, although contralateral inhibition of dorsal horn cells was well established by postnatal day
25          Spatial divergence is the number of dorsal horn cells whose RFs overlap the RF center of a p
26 ) in vivo extracellular recordings of single dorsal horn cells with plantar cutaneous receptive field

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