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1 revents both the smad2-mediated induction of dorsal mesodermal and endodermal markers and the inducti
5 calization of Xdsh at the membrane of normal dorsal mesodermal cells is consistent with Xdsh controll
6 gion--has a central role in the induction of dorsal mesodermal cells to form the Spemann organizer.
7 prospective neural ectodermal cells, or upon dorsal mesodermal cells, to cause a loss of anterior pat
8 ion in the Xenopus gastrula is essential for dorsal mesodermal development and for Nodal expression i
9 e trunk mesoderm, then refinement to a broad dorsal mesodermal domain, and finally restricted express
10 lar machine separate from MIB that can drive dorsal mesodermal extension exists in the zebrafish gast
12 rises mutations that lead to deficiencies in dorsal mesodermal fates and affect central nervous syste
15 AST-En(R) in intact embryos prevents general/dorsal mesodermal gene expression and axial development.
16 Foxd3 gain-of-function results in expanded dorsal mesodermal gene expression, including the Nodal-r
17 directly induce the same set of general and dorsal mesodermal genes, as well as some endodermal gene
22 nscription factor Tinman (Tin) to induce all dorsal mesodermal tissue derivatives, which include dors
23 e complex (Tin-C) controls the patterning of dorsal mesodermal tissues, including the dorsal vessel o
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