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1 s of capsaicin-responsive neurons in primate dorsal root ganglion.
2 a the release of substance P in the skin and dorsal root ganglion.
3 ressed in a subset of sensory neurons of the dorsal root ganglion and in cutaneous mechanoreceptors k
4 antly over time, a number of changes, in the dorsal root ganglion and in dorsal horn observed after t
6 NR2B receptor protein expression in both the dorsal root ganglion and spinal dorsal horn ipsilateral
7 mpairs channel slow inactivation within both dorsal root ganglion and superior cervical ganglion neur
8 the peripheral nervous system within sensory dorsal root ganglion and sympathetic ganglion neurons an
9 m channel is preferentially expressed within dorsal root ganglion and sympathetic ganglion neurons an
10 is preferentially expressed in nocioceptive dorsal root ganglion and sympathetic ganglion neurons.
12 be responsible for pathology observed in the dorsal root ganglion and the sensory ganglionopathy docu
13 s: cortical (embryonic rat), embryonic chick dorsal root ganglion, and P-19 (mouse embryonic carcinom
16 ple host axons when co-cultured with primary dorsal root ganglion cells and formed myelin after trans
17 n the plasma membrane of pancreatic beta and dorsal root ganglion cells and link steroid hormone sign
20 contrast to its effect on the hair cell and dorsal root ganglion cells in culture; (iv) SLV recyclin
22 sensory terminals of primary mechanosensory dorsal root ganglion cells, so the presence of such a sy
24 tion of lymphoid cell kinase in Schwann cell-dorsal root ganglion cocultures and dorsal root ganglion
25 hVariant 3 is more abundantly expressed in dorsal root ganglion compared with brain and shows basic
26 The administration of NO donors to primary dorsal root ganglion cultures prevents axonal degenerati
28 y of glial cells and reduced spinal cord and dorsal root ganglion cytokine levels without affecting p
30 oop L1 reduces Na(v)1.6 current density in a dorsal root ganglion-derived cell line, without changing
31 hat CXCL12 and CXCR4 were upregulated in the dorsal root ganglion (DRG) after chronic compression of
32 injury causes down-regulation of MORs in the dorsal root ganglion (DRG) and diminishes the opioid eff
34 ed and manipulated MrgprA3(+) neurons in the dorsal root ganglion (DRG) and found that they exclusive
35 titutive autophagosome biogenesis in primary dorsal root ganglion (DRG) and hippocampal cultures.
36 KChIP2, KChIP3, DPP6, and DPP10 in adult rat dorsal root ganglion (DRG) and spinal cord by immunohist
37 parts in two major pain-related regions, the dorsal root ganglion (DRG) and spinal cord dorsal horn.
38 hannel NaV1.7 is preferentially expressed in dorsal root ganglion (DRG) and sympathetic ganglion neur
39 1.7 which is preferentially expressed within dorsal root ganglion (DRG) and sympathetic ganglion neur
40 tion after axotomy was abolished in both the dorsal root ganglion (DRG) and the distal sciatic nerve.
41 ncreases in osmolality excite isolated mouse dorsal root ganglion (DRG) and trigeminal ganglion (TG)
43 port that stimulated membrane enlargement in dorsal root ganglion (DRG) axons is triggered by intra-a
45 tigate mRNA expression in colonic tissue and dorsal root ganglion (DRG) cells isolated from 3- and 24
49 at a dose of 100 mpk PO due to insufficient dorsal root ganglion (DRG) exposure attributed to poor m
50 ge-diameter (>30 mum) sensory neurons of the dorsal root ganglion (DRG) express distinct combinations
51 pression and functional role of Panx1 in the dorsal root ganglion (DRG) in the development of chronic
52 ion of HIV infection and is characterized by dorsal root ganglion (DRG) inflammation and intraepiderm
54 gated potassium channel subunit Kcna2 in the dorsal root ganglion (DRG) is critical for DRG neuronal
55 d texture is the activation of trigeminal or dorsal root ganglion (DRG) mechanosensory neurons, which
56 elevation, activation of cGMP increases rat dorsal root ganglion (DRG) neurite outgrowth on a polyly
58 nsgenic mice lacking Merkel cells had normal dorsal root ganglion (DRG) neuron numbers, but fewer DRG
60 hondrial trafficking plays a central role in dorsal root ganglion (DRG) neuronal cell survival and ne
62 nduced spontaneous pain and axonal injury of dorsal root ganglion (DRG) neurons and inhibited CCI-evo
63 the Na(+)/K(+)-ATPase (alpha3) in some large dorsal root ganglion (DRG) neurons and large intrafusal
64 um channel that is specifically expressed in dorsal root ganglion (DRG) neurons and peripheral nerve
65 satellite glial cells (SGCs) surrounding the dorsal root ganglion (DRG) neurons appears to play a rol
67 norepinephrine and sensitizes colon-specific dorsal root ganglion (DRG) neurons by increasing express
68 fast-inactivating Kv3.4 potassium current in dorsal root ganglion (DRG) neurons contributes to the hy
70 v5 are expressed by developing TrkA-positive dorsal root ganglion (DRG) neurons during the period of
72 s axon growth from neurons; adult miR-155 KO dorsal root ganglion (DRG) neurons extend 44% longer neu
73 en implicated in the hyperexcitable state of dorsal root ganglion (DRG) neurons following direct inju
74 arization-evoked Ca2+ transient is larger in dorsal root ganglion (DRG) neurons from tumor-bearing mi
76 e firing frequency and spontaneous firing of dorsal root ganglion (DRG) neurons have recently been id
79 GKIalpha) that regulates axon bifurcation of dorsal root ganglion (DRG) neurons in the spinal cord.
80 hormones in regulating axonal development of dorsal root ganglion (DRG) neurons in the spinal cord.
82 electroporation approach to transfect adult dorsal root ganglion (DRG) neurons in vivo that enables
83 gnitude and properties by voltage clamp from dorsal root ganglion (DRG) neurons in vivo, after classi
84 -expression network analysis, we categorized dorsal root ganglion (DRG) neurons into different subtyp
85 hat the activity of TRPM3 expressed in mouse dorsal root ganglion (DRG) neurons is inhibited by agoni
86 nduced current with activation of ASIC(3) in dorsal root ganglion (DRG) neurons of control rats and r
87 cell patch-clamp recordings from dissociated dorsal root ganglion (DRG) neurons revealed enhanced tet
88 rophysiological characterization of isolated dorsal root ganglion (DRG) neurons revealed that RPRFami
92 t IgG-IC directly excited a subpopulation of dorsal root ganglion (DRG) neurons through the neuronal
93 luence the electrophysiological responses of dorsal root ganglion (DRG) neurons to mechanical stimula
94 ciatic nerve allows the central processes of dorsal root ganglion (DRG) neurons to spontaneously rege
96 nhanced green fluorescent protein-expressing dorsal root ganglion (DRG) neurons transplanted into the
97 after axonal injury, Nogo-A is increased in dorsal root ganglion (DRG) neurons unable to regenerate
98 4-well format axon degeneration assay in rat dorsal root ganglion (DRG) neurons using a trophic facto
99 Depolarization-induced calcium influx in dorsal root ganglion (DRG) neurons was inhibited by both
101 hat voltage-gated sodium channels (VGSCs) in dorsal root ganglion (DRG) neurons were sensitized in a
102 that reduction of GRK2 or increased EPAC1 in dorsal root ganglion (DRG) neurons would promote the tra
103 mature rat retinal ganglion cells (RGCs) and dorsal root ganglion (DRG) neurons, and is essential for
104 s in the up-regulation of alpha(2)delta-1 in dorsal root ganglion (DRG) neurons, and there is a conse
107 hosphatase (PAP) is expressed in nociceptive dorsal root ganglion (DRG) neurons, functions as an ecto
108 expressed in a subpopulation of nociceptive dorsal root ganglion (DRG) neurons, where it acts as a s
109 thereby producing hyperexcitability of small dorsal root ganglion (DRG) neurons, which include nocice
110 ury induces changes in gene transcription in dorsal root ganglion (DRG) neurons, which may contribute
128 with microglia BV-2 cells exposed to G-CSF, dorsal root ganglion (DRG) nociceptors become hyperexcit
129 melanoma and normal neural tissues including dorsal root ganglion (DRG) produce PD-L1 that can potent
130 studied pancreatic nodose ganglion (NG) and dorsal root ganglion (DRG) sensory neurons (identified b
131 ort functional expression of SHANK3 in mouse dorsal root ganglion (DRG) sensory neurons and spinal co
132 pplied to their distal axons, in contrast to dorsal root ganglion (DRG) sensory neurons in which GDNF
141 of rodent embryonic sensory neurons from the dorsal root ganglion (DRG) to demonstrate the role of cG
143 neurons infected in vivo was examined using dorsal root ganglion (DRG) xenografts maintained in mice
144 dent outgrowth and traction forces from PNS (dorsal root ganglion (DRG)) and CNS (hippocampal) neuron
145 set of nonpeptidergic nociceptors within the dorsal root ganglion (DRG), and knockdown of Kv4.3 selec
146 reduction in K(+) channel expression in the dorsal root ganglion (DRG), but little is known about th
148 icular pain model, local inflammation of the dorsal root ganglion (DRG), we observed marked increases
149 in nociceptor (pain-sensing) neurons of the dorsal root ganglion (DRG), where they transmit the larg
155 vivo forepaw muscles/median and ulnar nerves/dorsal root ganglion (DRG)/spinal cord (SC) recording pr
156 n in capsaicin-sensitive nociceptors using a dorsal root ganglion (DRG)/spinal cord neuron co-culture
157 ar CFA injection and in the ipsilateral L4/5 dorsal root ganglions (DRGs) 1 and 3 days after CFA inje
158 ly by small-sized primary sensory neurons in dorsal root ganglions (DRGs) that coexpress the itch sig
160 ann cell-dorsal root ganglion cocultures and dorsal root ganglions from Lck(-/-) mice show a reductio
162 at the Ascl1 promoter, isolated from murine dorsal root ganglion (hypermethylated) and striated cell
163 plotted global expression changes in the rat dorsal root ganglion in three peripheral neuropathic pai
164 permeant cAMP analog, 8-bromo cAMP, into the dorsal root ganglion induced mechanical hyperalgesia and
165 that signals emanating from within the mouse dorsal root ganglion mediated partly by early-born neuro
166 imulated oligodendrocytes was validated in a dorsal root ganglion microfluidics chamber platform.
167 city, nerve amplitude, and sciatic nerve and dorsal root ganglion morphology at 0.25 x MTD, 0.5 x MTD
168 rsal column lesions and reduced in models of dorsal root ganglion neuron (DRGN) axon regeneration.
169 n of the effects of the Del-L955 mutation on dorsal root ganglion neuron hyperexcitability with those
170 ng a CD4-Na(v)1.2/L2 reporter protein in rat dorsal root ganglion neuron-Schwann cell myelinating coc
172 adapting, mechanically activated currents in dorsal root ganglion neuronal cultures are absent in Pie
174 that TLRs 3, 7, and 9 are expressed by human dorsal root ganglion neurons (DRGNs) and in cultures of
176 , PKCdelta, and PKC was also observed in the dorsal root ganglion neurons after chronic treatment wit
177 receptor that increases cAMP, in a subset of dorsal root ganglion neurons and also within neurons of
179 ase ion transporter in both cultured primary dorsal root ganglion neurons and injured peripheral nerv
180 for PKCepsilon was found in the majority of dorsal root ganglion neurons and intensely labeled lamin
181 e-collapsing molecule than Nogo-66 for chick dorsal root ganglion neurons and mature cortical neurons
182 t channels enhance resurgent currents within dorsal root ganglion neurons and show by current-clamp t
185 and biochemical modulation of T-channels in dorsal root ganglion neurons as measured by a large incr
187 f3r gene expression could not be detected in dorsal root ganglion neurons by single-cell RT-PCR.
188 diated local expression of erythropoietin in dorsal root ganglion neurons can protect in vivo as well
189 that in small PLCdelta4(-/-) TRPM8-positive dorsal root ganglion neurons cold, menthol and WS-12, a
190 n; and (5) electrophysiology recordings from dorsal root ganglion neurons collected during remission
191 tion of the alpha2delta-1 subunit in sensory dorsal root ganglion neurons contributes to the generati
193 iated inhibition of M current in nociceptive dorsal root ganglion neurons did not reduce the efficacy
195 Here, we show that only 43% of CQ-excited dorsal root ganglion neurons expressed TRPA1; as expecte
197 pic screen, we find that CAST/Ei mouse adult dorsal root ganglion neurons extend axons more on CNS my
198 tion of NFATc3 and NFATc4 in hippocampal and dorsal root ganglion neurons following electrically evok
200 m photoactivated axonal segments in cultured dorsal root ganglion neurons from DLS/LeJ and dl20J dilu
202 between TRPV1 and Pirt, and that dissociated dorsal root ganglion neurons from Pirt knock-out mice ha
203 ne also acts on endogenous TRPA1 in cultured dorsal root ganglion neurons from rats and in the entero
204 erior cervical ganglion neurons, and renders dorsal root ganglion neurons hyperexcitable and superior
205 ons in sodium channel Na(V)1.7, which render dorsal root ganglion neurons hyperexcitable, are present
206 neonatal-short, isoform of Na(v)1.7 renders dorsal root ganglion neurons hyperexcitable, reducing th
209 from synaptic vesicles along axons of mouse dorsal root ganglion neurons in culture promotes myelin
212 We now find that treatment of adult rat dorsal root ganglion neurons in vitro with LRP1 agonists
213 c or optogenetic depolarization of GABAergic dorsal root ganglion neurons in vivo reduced acute and c
214 activation does not depend on the age of the dorsal root ganglion neurons in which the mutant is expr
216 K-2 channels was also demonstrated in native dorsal root ganglion neurons indicating that heterodimer
218 -alpha2A cross-desensitization was absent in dorsal root ganglion neurons lacking beta-arrestin 2.
220 examined micro-receptor coupling to VDCCs in dorsal root ganglion neurons of delta(+/+), delta(+/-),
222 channels in rat endocrine GH(3) cells, mouse dorsal root ganglion neurons or cardiac myocytes, and re
224 .2/Kv7.3 heteromers and native M currents in dorsal root ganglion neurons suggest the following concl
225 at dorsal root injury model, transduction of dorsal root ganglion neurons to express kindlin-1 promot
227 teers, sensitized TRPV1 in mouse nociceptive dorsal root ganglion neurons via HRH1; this effect could
228 ratching behavior and activation of cultured dorsal root ganglion neurons was dependent on Mrgprs rat
229 a(2+) channels expressed in HEK293 cells and dorsal root ganglion neurons were abolished by blocking
230 two distinct types of cutaneous nociceptive dorsal root ganglion neurons were identified as respondi
232 n prostate cells, PC3 prostate cancer cells, dorsal root ganglion neurons, and hippocampal neurons.
233 eurons, chemotherapy-induced cytotoxicity of dorsal root ganglion neurons, and retinal ganglion cells
234 similar expression levels in spinal cord and dorsal root ganglion neurons, and that both kinases part
235 is transiently expressed in a wide range of dorsal root ganglion neurons, and that its expression is
236 PV1 current (IC(50) = 0.4 nm) in dissociated dorsal root ganglion neurons, and this IC(50) is approxi
237 s hTRPM8, and rTRPM3, which are expressed in dorsal root ganglion neurons, are insensitive toward apo
238 1 x 10(12) vg of AAV-PHP.S transduced 82% of dorsal root ganglion neurons, as well as cardiac and ent
239 age-gated K(+) channel robustly expressed in dorsal root ganglion neurons, becomes dysfunctional upon
240 el, depolarize resting membrane potential of dorsal root ganglion neurons, enhance spontaneous firing
241 including altered ion channel expression in dorsal root ganglion neurons, enhanced dorsal horn gluta
244 ed sustained ASIC currents in both groups of dorsal root ganglion neurons, independent of mu opioid r
245 llular signal-regulated kinase expression in dorsal root ganglion neurons, induced by co-cultured MRM
247 that maintain the position of postmigratory dorsal root ganglion neurons, neural crest derivatives f
249 physiology and Ca(2+) imaging experiments on dorsal root ganglion neurons, NGF- and IL-6-induced incr
250 nic spinal commissural neurons, motoneurons, dorsal root ganglion neurons, retinal ganglion cells, an
251 ppressed spontaneous activity in dissociated dorsal root ganglion neurons, reversed hypersensitivity
252 g calcium imaging in cultured primary murine dorsal root ganglion neurons, the response of neurons af
253 een TLR4 and TRPV1 is shown in rat and human dorsal root ganglion neurons, TLR4/TRPV1-coexpressing HE
254 tion and inhibits neurite outgrowth in adult dorsal root ganglion neurons, validating Slit2 signaling
256 tion in both male and female embryonic mouse dorsal root ganglion neurons, we show that MAP4K4, MINK1
258 duced robust neurite outgrowth by cocultured dorsal root ganglion neurons, which was prevented by neu
281 currents recorded from small neurons in the dorsal root ganglion of normal rats are potentiated by e
284 ore variable across cultures than in primary dorsal root ganglion, particularly for genes related to
287 y processes characterized by spinal cord and dorsal root ganglion production of proinflammatory cytok
288 fer elevated intracellular calcium levels in dorsal root ganglion pruriceptors, and (iii) injection o
291 P that binds and regulates multiple mRNAs in dorsal root ganglion sensory neurons and thereby promote
292 ated virus (AAV) has been shown to transduce dorsal root ganglion sensory neurons following direct in
294 ta from an in vitro preparation of small rat dorsal root ganglion somata showing a reduction in the m
297 peripheral immune cell infiltration into the dorsal root ganglion, suggesting that adaptive immune re
298 We show here that synaptic contacts from dorsal root ganglions to a small number of dorsal column
299 evidence indicates that gene transfer to the dorsal root ganglion using replication-defective herpes
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