ライフサイエンスコーパス: dorsolateral

1 difficult tasks, we observed bilaterality in dorsolateral and anterior prefrontal cortex across WM do

2 al connectivity between the thalamus and the dorsolateral and anterior prefrontal cortices.

3 ts suggest that functional integrity of left dorsolateral and dorsomedial prefrontal cortex is critic

4 t anterior insula, left ventral hippocampus, dorsolateral and dorsomedial prefrontal cortex, and caud

5 l MRI (fMRI) experiment we selected the left dorsolateral and dorsomedial prefrontal cortices as targ

6 High labeling densities were found in the dorsolateral and the upper lateral portion of the periaq

7 associated with lower gray matter volume in dorsolateral and ventromedial prefrontal cortex and orbi

8 , and a U-shaped digestive tract ending in a dorsolateral anus.

9 rsal striatal subregion, from dorsomedial to dorsolateral, as skill performance shifts from an attent

10 ntral nucleus of the amygdala (CeAL) and the dorsolateral bed nucleus of the stria terminalis (BNSTDL

11 immediate early gene mapping highlighted the dorsolateral bed nucleus of the stria terminalis (dlBST)

12 ve function, is generally thought to rely on dorsolateral brain regions.

13 eversible inactivation of neural activity in dorsolateral but not dorsomedial striatum using the GABA

14 increased in only Fos-positive neurons from dorsolateral, but not dorsomedial, striatum.

15 r investigating synaptic plasticity in mouse dorsolateral cortico-striatal circuitry and interrogate

16 data demonstrate the pivotal role of a minor dorsolateral corticospinal pathway in mediating spontane

17 recovery, we transiently silenced the minor dorsolateral corticospinal pathway spared by our injury.

18 x; mPFC), a cognitive control network [e.g., dorsolateral (dl)PFC], and a salience network (e.g., ins

19 es if the damage impacts the PCS; lesions to dorsolateral dlPFC that spare the PCS have no effect on

20 e have examined gene expression in the human dorsolateral frontal cortex using RNA- Seq to populate a

21 motor cortex and AAV-expressing Cre in C7/C8 dorsolateral funiculus.

22 mately 3% of CST fibers that project via the dorsolateral funiculus.

23 ected with the primary ventral posterior and dorsolateral geniculate nuclei, respectively, and less w

24 teral posterior nucleus (LP), as well as the dorsolateral geniculate nucleus (dLGN).

25 ll GABAergic neurons that express FoxP2; and dorsolateral glutamatergic neurons that express FoxP2 in

26 ed by SRP sources within the motor system-in dorsolateral hand motor areas for expected hand-related

27 medial, central, centrolateral, dorsomedial, dorsolateral) identifiable throughout development.

28 ves inhibitory inputs from the contralateral dorsolateral IP, and mainly excitatory inputs from the i

29 The donor NCCs migrated along the dorsolateral migration routes in the recipient embryos.

30 We assessed loss of dorsolateral nigral hyperintensity (DNH) on high-field s

31 TD, terminate in specific cell groups in the dorsolateral nTTDo and in PrV.

32 olateral orbital area, lateral orbital area, dorsolateral orbital area, and agranular insular areas.

33 The major pathways are recursive: the dorsolateral pallium (DL) projects strongly to DDi, with

34 otiform fish, Apteronotus leptorhynchus, the dorsolateral pallium (DL) receives diencephalic inputs r

35 lateral pallia, and may belong to a distinct dorsolateral pallium, which extends from rostral to caud

36 e (i.e. cortico-subthalamic) profiles in the dorsolateral part of the subthalamic nucleus (i.e. its s

37 tive melanoblasts were not restricted to the dorsolateral pathway as in birds and mammals but were al

38 demonstrated that VMHdm/c projection to the dorsolateral periaqueductal gray (dlPAG) induces inflexi

39 and increased negative connectivity between dorsolateral PFC (dlPFC) and insula in the controllable

40 c receptor agonist carbachol onto neurons in dorsolateral PFC (DLPFC) of male rhesus macaques perform

41 between the amygdala and ventral PFC (VPFC), dorsolateral PFC (DLPFC), and dorsal anterior cingulated

42 Moreover, the increase in right dorsolateral PFC activity in conjunction with eye blink

43 onally and functionally specific deficits in dorsolateral PFC and hippocampal activation.

44 levels were also detected in the postmortem dorsolateral PFC of individuals with depression.

45 raphy, pathways between the amygdala and the dorsolateral PFC, dorsomedial PFC, ventromedial PFC, and

46 c hypoconnectivity in psychosis localized to dorsolateral PFC, medial PFC, and cerebellar areas of th

47 ll positive GABAA receptor subunits from the dorsolateral pole to ventromedial extremities.

48 ced in the Kolliker-Fuse nucleus (KF) in the dorsolateral pons, an important centre for control of re

49 Antidepressants increased activity in the dorsolateral prefrontal (dlPFC), a key region mediating

50 ring processing of neutral faces and reduced dorsolateral prefrontal activity during failed inhibitio

51 EF was uniquely associated with caudal left dorsolateral prefrontal and lateral temporo-parietal cor

52 strengthen the assumption that dysfunctional dorsolateral prefrontal and limbic brain regions are a h

53 ional MRI activity, with thicker cortices in dorsolateral prefrontal brain regions, and with white ma

54 lied 5 days per week for 3 weeks to the left dorsolateral prefrontal cortex (added to the ongoing tre

55 t the executive control circuitry, including dorsolateral prefrontal cortex (cluster-corrected P < .0

56 f 561 immune genes and 20 immune pathways in dorsolateral prefrontal cortex (DLPFC) (144 schizophreni

57 zophrenia, is often associated with aberrant dorsolateral prefrontal cortex (dlPFC) activation.

58 onnectivity seeded from the right IFG to the dorsolateral prefrontal cortex (DLPFC) and anterior cing

59 n distributed neural circuitry including the dorsolateral prefrontal cortex (DLPFC) and appear to ari

60 We applied anodal tDCS over the left dorsolateral prefrontal cortex (DLPFC) and cathodal tDCS

61 onal spike activities of single units in the dorsolateral prefrontal cortex (dlPFC) and the anterior

62 ne density on layer 3 pyramidal cells in the dorsolateral prefrontal cortex (DLPFC) appears to contri

63 and functional alterations of neurons in the dorsolateral prefrontal cortex (dlPFC) are thought to co

64 lation indicated the involvement of the left dorsolateral prefrontal cortex (DLPFC) as well as left M

65 r mechanisms, on the recruitment of the left dorsolateral prefrontal cortex (DLPFC) as well as on the

66 er a temporary neural disruption of the left Dorsolateral Prefrontal Cortex (DLPFC) can improve impli

67 n working memory that reflect dysfunction of dorsolateral prefrontal cortex (DLPFC) circuitry.

68 odes children's own preferences and the left dorsolateral prefrontal cortex (dlPFC) encodes the proje

69 amygdala activation and reduced amygdala and dorsolateral prefrontal cortex (dlPFC) functional coupli

70 od-oxygen-level-dependent (BOLD) signal, and dorsolateral prefrontal cortex (DLPFC) glutamate+glutami

71 l direct current stimulation (tDCS) over the dorsolateral prefrontal cortex (DLPFC) has been effectiv

72 Although the dorsolateral prefrontal cortex (DLPFC) has long been con

73 Here we show that the dorsolateral prefrontal cortex (DLPFC) implements a flex

74 Conflict monitoring theory assumes that the dorsolateral prefrontal cortex (DLPFC) is causally invol

75 We found that damage to the dorsolateral prefrontal cortex (dlPFC) led to a more pos

76 We hypothesized that dorsolateral prefrontal cortex (dlPFC) lesions, due to t

77 curring de novo, in brain-expressed genes of dorsolateral prefrontal cortex (DLPFC) neurons.

78 levels, have frequently been reported in the dorsolateral prefrontal cortex (DLPFC) of schizophrenia

79 mation relevant for credit assignment in the dorsolateral prefrontal cortex (dlPFC) of two male rhesu

80 ial magnetic stimulation (rTMS) of the right dorsolateral prefrontal cortex (DLPFC) on working memory

81 The extent of dorsolateral prefrontal cortex (DLPFC) plasticity in Alz

82 The dorsolateral prefrontal cortex (DLPFC) plays a pivotal r

83 The dorsolateral prefrontal cortex (DLPFC) plays a pivotal r

84 t the volume of the rostral part of the left dorsolateral prefrontal cortex (DLPFC) predicted an indi

85 othalamus, midbrain, right insula, and right dorsolateral prefrontal cortex (DLPFC) regions supported

86 om nonhuman primate studies, posits that the dorsolateral prefrontal cortex (dlPFC) stores and mainta

87 The primate dorsolateral prefrontal cortex (dlPFC) subserves top-dow

88 activity of neural circuitry in the primate dorsolateral prefrontal cortex (DLPFC) supports a range

89 as closely associated with feedback from the dorsolateral prefrontal cortex (DLPFC) to V1.

90 ical inhibition (LICI) was measured from the dorsolateral prefrontal cortex (DLPFC) using combined tr

91 s in the anterior cingulate cortex and right dorsolateral prefrontal cortex (DLPFC) were compared via

92 We hypothesized that the hippocampus and dorsolateral prefrontal cortex (dlPFC) would play a key

93 direct current stimulation (tDCS) over left dorsolateral prefrontal cortex (dlPFC) yielded a close m

94 ree latent brain factors (amygdala, pACC and dorsolateral prefrontal cortex (DLPFC)) to test the effe

95 anscranial direct current stimulation of the dorsolateral prefrontal cortex (dlPFC), a critical neura

96 sociated with reduced activation in the left dorsolateral prefrontal cortex (DLPFC), a region of the

97 P down-regulates the BOLD signal in the left dorsolateral prefrontal cortex (dlPFC), a risk-integrati

98 ilateral superior parietal lobule, bilateral dorsolateral prefrontal cortex (DLPFC), and bilateral mi

99 monstrated greater fMRI response in caudate, dorsolateral prefrontal cortex (dlPFC), and intraparieta

100 sorders generally involve dysfunction of the dorsolateral prefrontal cortex (dlPFC), but there are fe

101 e frontal lobes [anode electrode at the left dorsolateral prefrontal cortex (DLPFC), cathode electrod

102 ed with decreased gamma activity in the left dorsolateral prefrontal cortex (dlPFC), decreased theta

103 st in anterior cingulate cortex (ACC) before dorsolateral prefrontal cortex (dlPFC), followed by medi

104 e found that threat affected activity in the dorsolateral prefrontal cortex (dlPFC), rather than the

105 In the post-mortem dorsolateral prefrontal cortex (DLPFC), we found strikin

106 ells in layer 3 (L3) and layer 5 (L5) of the dorsolateral prefrontal cortex (DLPFC), we sought to det

107 es is associated with neural activity in the dorsolateral prefrontal cortex (dlPFC).

108 ons in several cortical areas, including the Dorsolateral Prefrontal Cortex (DLPFC).

109 ial direct current stimulation (tDCS) of the dorsolateral prefrontal cortex (DLPFC).

110 cognitive function reliant on area 46 of the dorsolateral prefrontal cortex (dlPFC).

111 linked to a decrease in the activity of the dorsolateral prefrontal cortex (dlPFC).

112 pines on deep layer 3 pyramidal cells in the dorsolateral prefrontal cortex (DLPFC).

113 that flexibility is mainly subserved by the dorsolateral prefrontal cortex (DLPFC).

114 s3749034 and with the expression of GAD25 in dorsolateral prefrontal cortex (DLPFC).

115 a (SZ) is associated with dysfunction of the dorsolateral prefrontal cortex (DLPFC).

116 ts involve dysfunction of the newly evolved, dorsolateral prefrontal cortex (dlPFC).

117 in-positive (PV) interneurons in the primate dorsolateral prefrontal cortex (DLPFC).

118 100 kb of the lead SNP are expressed in the dorsolateral prefrontal cortex (DLPFC): UNC5C, ENC1, and

119 e (Glx), were measured by 1H MRS in the left dorsolateral prefrontal cortex (l-DLPFC) and bilateral h

120 Left dorsolateral prefrontal cortex (L-DLPFC) tDCS induced an

121 prefrontal cortex (Brodmann area 10) and the dorsolateral prefrontal cortex (lateral Brodmann 9) whil

122 atment increased activation in both the left dorsolateral prefrontal cortex (PFC) and supplementary m

123 anterior cingulate (t = 2.27; P = .04), and dorsolateral prefrontal cortex (t = 2.44; P = .03) were

124 ons important in top-down executive control (dorsolateral prefrontal cortex [dlPFC]), here we test wh

125 Dorsolateral prefrontal cortex activation during inhibit

126 Posttreatment dorsolateral prefrontal cortex activation was reduced in

127 f emotion type and associated with increased dorsolateral prefrontal cortex activity compared with th

128 odulatory effects of trait anger on amygdala-dorsolateral prefrontal cortex and amygdala-lateral orbi

129 igh-level cognitive control functions (i.e., dorsolateral prefrontal cortex and anterior cingulate co

130 ests the striatal afferent connection to the dorsolateral prefrontal cortex and basal ganglia circuit

131 ges in the interhemispheric effects from the dorsolateral prefrontal cortex and dorsal premotor corte

132 al-directed action, such as ventromedial and dorsolateral prefrontal cortex and dorsomedial striatum,

133 her microglial activation is elevated in the dorsolateral prefrontal cortex and hippocampus of untrea

134 )], that span the lateral prefrontal cortex (dorsolateral prefrontal cortex and inferior frontal gyru

135 vortioxetine reduced activation in the right dorsolateral prefrontal cortex and left hippocampus duri

136 er volumes relative to controls in the right dorsolateral prefrontal cortex and left hippocampus, alo

137 ability to regulate the interaction between dorsolateral prefrontal cortex and M1.

138 rior cingulate) and decision-making regions (dorsolateral prefrontal cortex and parietal cortex).

139 terior cingulate, orbitofrontal cortex, left dorsolateral prefrontal cortex and right inferior fronta

140 of structure (decreased grey matter in right dorsolateral prefrontal cortex and right inferior tempor

141 ed functional connectivity between the right dorsolateral prefrontal cortex and right superior occipi

142 de network; 2) hypoconnectivity between left dorsolateral prefrontal cortex and subgenual anterior ci

143 d the most consistent underactivation in the dorsolateral prefrontal cortex and temporal pole, while

144 ddiction-relevant brain regions, such as the dorsolateral prefrontal cortex and the angular and cingu

145 al cortical inhibition [LICI]) from the left dorsolateral prefrontal cortex and the left motor cortex

146 nferior and superior frontal gyri, including dorsolateral prefrontal cortex and ventrolateral prefron

147 gulation despite increased activation of the dorsolateral prefrontal cortex and ventrolateral prefron

148 rts, we tested whether GMV and NAA/Cr in the dorsolateral prefrontal cortex are associated with fatig

149 integrated information is used by the right dorsolateral prefrontal cortex at the time of the decisi

150 ex while recalling resulted in activation in dorsolateral prefrontal cortex bilaterally.

151 EF and DB were associated with the rostral dorsolateral prefrontal cortex bilaterally.

152 We found that neuronal activity in the dorsolateral prefrontal cortex conveyed the necessary in

153 When pain was controllable, the dorsolateral prefrontal cortex downregulated pain-evoked

154 We stimulated over the left dorsolateral prefrontal cortex during a declarative memo

155 ce that NRXN1 expression is highest in human dorsolateral prefrontal cortex during critical developme

156 ignificant differences localized to the left dorsolateral prefrontal cortex during response selection

157 Notably, [oxy-Hb] change in the left dorsolateral prefrontal cortex during SAT showed a posit

158 The optimal target in the dorsolateral prefrontal cortex for treating depression w

159 olymerase chain reaction in human postmortem dorsolateral prefrontal cortex from 286 nonpsychiatric c

160 olymerase chain reaction in human postmortem dorsolateral prefrontal cortex from 286 nonpsychiatric c

161 encing of poly-A RNA derived from postmortem dorsolateral prefrontal cortex from people with BD, alon

162 or calretinin-positive (CR+) neurons in the dorsolateral prefrontal cortex from schizophrenia subjec

163 In the right dorsolateral prefrontal cortex IC cluster, the beta band

164 of the causal influence of the insula on the dorsolateral prefrontal cortex in cocaine users.

165 nal connectivity between the putamen and the dorsolateral prefrontal cortex in OCD patients, as well

166 ted inverted-U response was observed in left dorsolateral prefrontal cortex in patients and was assoc

167 of ErbB4 transcripts is dysregulated in the dorsolateral prefrontal cortex in schizophrenia.

168 The dorsolateral prefrontal cortex is downregulated, diminis

169 europsychology studies have established that dorsolateral prefrontal cortex is essential in spatial w

170 interneurons were laser microdissected from dorsolateral prefrontal cortex layers 2 and 4, respectiv

171 ng the cognitive deficits and dysfunction of dorsolateral prefrontal cortex observed in this disorder

172 lation and histone acetylation data from the dorsolateral prefrontal cortex of 411 older adults who h

173 lternating current stimulation over the left dorsolateral prefrontal cortex of human participants [n

174 ed, and DNA methylation was increased in the dorsolateral prefrontal cortex of male suicide subjects,

175 n by recording single-unit activity from the dorsolateral prefrontal cortex of monkeys that were perf

176 associated with neuronal alterations in the dorsolateral prefrontal cortex of patients with CFS.

177 predictor of both GMV and NAA/Cr in the left dorsolateral prefrontal cortex of patients with CFS.

178 The CommonMind Consortium sequenced RNA from dorsolateral prefrontal cortex of people with schizophre

179 rom the contribution of interactions between dorsolateral prefrontal cortex of the FPN and precuneus

180 as indexed by [(18)F]FEPPA VT, in either the dorsolateral prefrontal cortex or the hippocampus.

181 ntal state and harm information, whereas the dorsolateral prefrontal cortex plays a crucial, final-st

182 RNA sequencing data of human dorsolateral prefrontal cortex revealed relatively high

183 l anterior cingulate cortex (dACC), and left dorsolateral prefrontal cortex seeds and by relative hyp

184 ted into ventrolateral prefrontal cortex and dorsolateral prefrontal cortex subregions.

185 riven by reduced effective connectivity from dorsolateral prefrontal cortex to anterior prefrontal co

186 rain activation, driven in part by increased dorsolateral prefrontal cortex to midbrain connectivity.

187 ramidal neuron dendrites in Brodmann area 46 dorsolateral prefrontal cortex using the Golgi-Cox techn

188 ced change in connectivity from right IFG to dorsolateral prefrontal cortex was proportional to the c

189 Application of active 10-Hz rTMS to the left dorsolateral prefrontal cortex was well tolerated but wa

190 inhibition (ie, N100 and LICI) from the left dorsolateral prefrontal cortex were selected.

191 s resulted in significant activation in left dorsolateral prefrontal cortex while recalling resulted

192 (BMI) had lower activation in the bilateral dorsolateral prefrontal cortex while viewing unhealthy c

193 iatal and midbrain) and prefrontal cortical (dorsolateral prefrontal cortex) regions during reward an

194 the sgACC and the default mode network, left dorsolateral prefrontal cortex, and insula, and reduced

195 -subcortical circuitry engaging anterior and dorsolateral prefrontal cortex, and midbrain.

196 nique projections from orbitofrontal cortex, dorsolateral prefrontal cortex, and parietal regions.

197 mical connections from orbitofrontal cortex, dorsolateral prefrontal cortex, and posterior parietal c

198 in a network of regions including bilateral dorsolateral prefrontal cortex, anterior cingulate and a

199 y between the posterior cingulate cortex and dorsolateral prefrontal cortex, compared with HC subject

200 Seed-based (bilateral dorsolateral prefrontal cortex, DLPFC) rsFC analysis was

201 long with blunted responses of the bilateral dorsolateral prefrontal cortex, during the processing of

202 In the postnatal dorsolateral prefrontal cortex, expression levels were n

203 nual anterior cingulate cortex [sgACC], left dorsolateral prefrontal cortex, hippocampus, and basolat

204 ed prefrontal regions (orbitofrontal cortex, dorsolateral prefrontal cortex, inferior frontal gyrus,

205 involved in language control, including the dorsolateral prefrontal cortex, inferior parietal lobule

206 ns in contralateral ventral premotor cortex, dorsolateral prefrontal cortex, supramarginal gyrus, and

207 , hypometabolism and/or hypoperfusion in the dorsolateral prefrontal cortex, the anterior and middle

208 work of areas, including the dorsomedial and dorsolateral prefrontal cortex, the intraparietal sulcus

209 s across the two tasks were localized to the dorsolateral prefrontal cortex, where responses were inc

210 e associated with hypoactivation of the left dorsolateral prefrontal cortex, whereas behavioral sympt

211 fortful DM) leading to activation across the dorsolateral prefrontal cortex, whereas experts are expe

212 fortful DM) leading to activation across the dorsolateral prefrontal cortex, whereas experts are expe

213 the primary visual cortex and highest in the dorsolateral prefrontal cortex, whereas the GABA measure

214 novices showed significant activation of the dorsolateral prefrontal cortex, whereas this activation

215 novices showed significant activation of the dorsolateral prefrontal cortex, whereas this activation

216 Suppressing imagination engaged the right dorsolateral prefrontal cortex, which modulated activati

217 le patients trait anger positively modulated dorsolateral prefrontal cortex-amygdala coupling.

218 tural brain marker-volume of left hemisphere dorsolateral prefrontal cortex-associated with the magni

219 were assessed in four frontostriatal tracts (dorsolateral prefrontal cortex-associative striatum, dor

220 nd fewer normalized streamlines in the right dorsolateral prefrontal cortex-sensorimotor striatum and

221 hermore, normalized streamlines in the right dorsolateral prefrontal cortex-sensorimotor striatum neg

222 rmalized streamlines in the right-hemisphere dorsolateral prefrontal cortex-sensorimotor striatum pre

223 eral prefrontal cortex-associative striatum, dorsolateral prefrontal cortex-sensorimotor striatum, ve

224 found between perception and GABA levels in dorsolateral prefrontal cortex.

225 ent conventional open-label rTMS to the left dorsolateral prefrontal cortex.

226 ctivity of the right Crus I/II with the left dorsolateral prefrontal cortex.

227 load observed in healthy individuals in left dorsolateral prefrontal cortex.

228 IC sources in or near medial prefrontal and dorsolateral prefrontal cortex.

229 pe was linked to smaller volumes in the left dorsolateral prefrontal cortex.

230 nterior cingulate cortex and to left IFG and dorsolateral prefrontal cortex.

231 re associated with reduced activation of the dorsolateral prefrontal cortex.

232 regions within the intraparietal sulcus and dorsolateral prefrontal cortex.

233 anied by recruitment of anterior putamen and dorsolateral prefrontal cortex.

234 d to increased dynamic RSFC between MPFC and dorsolateral prefrontal cortex.

235 hibitory control signal originating from the dorsolateral prefrontal cortex.

236 ct current stimulation (tDCS) over the right dorsolateral prefrontal cortex.

237 were generated using frozen tissue from the dorsolateral prefrontal cortex.

238 nterior cingulate, medial frontal gyrus, and dorsolateral prefrontal cortex.

239 red, in up to 40 daily sessions, to the left dorsolateral prefrontal cortex.

240 la induced by single TMS pulses to the right dorsolateral prefrontal cortex; and 4) greater ventromed

241 ne composite score was negatively related to dorsolateral prefrontal cortical function during risky d

242 Here, we show that tRNS over the bilateral dorsolateral prefrontal cortices (dlPFCs) improved learn

243 uding insula and orbitofrontal, rostral, and dorsolateral prefrontal cortices (p < .05, corrected), i

244 er activity in the lateral orbitofrontal and dorsolateral prefrontal cortices compared with healthy m

245 adults received real or sham tDCS over their dorsolateral prefrontal cortices during two 30-minute ma

246 h posterior parietal, anterior temporal, and dorsolateral prefrontal cortices emerged as the most ext

247 associated with the responses of insula and dorsolateral prefrontal cortices to the receipt of large

248 blind and sighted participants, the IPS and dorsolateral prefrontal cortices were more active during

249 ed regions contributed more to occipital and dorsolateral prefrontal cortices.

250 nal control in dorsal anterior cingulate and dorsolateral prefrontal cortices.

251 ciative-limbic subthalamic nucleus and right dorsolateral prefrontal functional connectivity in healt

252 Language-switching in production recruited dorsolateral prefrontal regions bilaterally and, importa

253 x, including visual, posterior parietal, and dorsolateral prefrontal regions.

254 tructural connectivity strength in the right dorsolateral prefrontal tract were related to increased

255 ng had regional specificity in the bilateral dorsolateral prefrontal, precuneus, left postcentral, an

256 peractivation in cognitive control mediating dorsolateral prefrontal-dorsal and striatal regions.

257 at a frontal-subcortical loop, the so-called dorsolateral prefrontal-striatal circuit, underlies the

258 tients showed disorder-specific reduced left dorsolateral-prefrontal activation and reduced posterior

259 s of injured dorsal projecting versus spared dorsolateral projecting corticospinal neurons, we establ

260 nervate one class of dLGN neurons within the dorsolateral shell, the primary terminal domain of direc

261 or correlates with broad reconfigurations of dorsolateral striatal (DLS) circuit properties that incr

262 These state correlates were not observed in dorsolateral striatal CINs recorded in the same rats.

263 tic and chemogenetic approaches to show that dorsolateral striatal PV interneurons influence the init

264 Cdk5 conditional knockout, or viral-mediated dorsolateral striatal-specific loss of Cdk5 all impaired

265 Here, we studied the role of dorsolateral striatum (DLS) and dorsomedial striatum (DM

266 utions of the striatopallidal A(2A)Rs in the dorsolateral striatum (DLS) and the dorsomedial striatum

267 The dorsolateral striatum (DLS) is implicated in habit forma

268 neurotrophic factor (BDNF) signaling in the dorsolateral striatum (DLS) keeps alcohol intake in mode

269 wiring to dorsomedial striatum (DMS) versus dorsolateral striatum (DLS), and natural activity patter

270 rophic factor and its receptor, TrkB, in the dorsolateral striatum (DLS), are part of an endogenous s

271 Interneuron ablation in the dorsolateral striatum (DLS), corresponding roughly to th

272 of striosome compartments restricted to the dorsolateral striatum (DLS), where less dopamine release

273 ypes, and ischemic stroke was induced in the dorsolateral striatum (DLS).

274 ntly, D-serine within the NAcore but not the dorsolateral striatum also selectively reduced aversion-

275 ch for studying synaptic plasticity in mouse dorsolateral striatum and critically implicate D1-dopami

276 Consistent with our predictions, the dorsolateral striatum contained information about respon

277 In particular, the dorsolateral striatum contributes to motor learning.

278 Thus, while the recruitment of dorsolateral striatum dopamine-dependent control over co

279 Meanwhile, DREADD-mediated inhibition of the dorsolateral striatum enhanced response-outcome conditio

280 Selective loss of Cdk5 in dorsolateral striatum increased locomotor activity and a

281 The dorsolateral striatum mediates habit formation, which is

282 ssion of Zif268 in the nucleus accumbens and dorsolateral striatum of LgA rats.

283 rn after brief training, whereas activity in dorsolateral striatum suppresses unrewarded turns after

284 e core of the nucleus accumbens (but not the dorsolateral striatum).

285 e also assessed spine density on WD36 in the dorsolateral striatum, a region that is not implicated i

286 n direct pathway medium spiny neurons in the dorsolateral striatum, and is strengthened by serial ord

287 162 into the nucleus accumbens core, but not dorsolateral striatum, selectively reduced cue-controlle

288 ms involved in habitual control, such as the dorsolateral striatum, should contain stimulus and respo

289 and sensorimotor inputs from motor cortex to dorsolateral striatum, we show that associative and sens

290 lasticity mechanisms in both the ventral and dorsolateral striatum.

291 served in both the nucleus accumbens and the dorsolateral striatum.

292 of the molecular basis of plasticity in the dorsolateral striatum.

293 ver behaviour shifts from the ventral to the dorsolateral striatum.

294 in the indirect, striatopallidal pathway in dorsolateral striatum.

295 terminals forming asymmetric synapses in the dorsolateral subthalamic nucleus was reduced by 55.1% an

296 superior occipital (cue-alpha) and the left dorsolateral superior frontal gyri (item-gamma) on permu

297 A common tonotopy with a dorsolateral to ventromedial gradient of low to high fre

298 utputs from these four quadrants of the SPZ (dorsolateral, ventrolateral, dorsomedial, and ventromedi

299 an increase 88%, P=0.032) across prefrontal (dorsolateral, ventrolateral, orbital), anterior cingulat

300 electrosensory lateral line lobe (ELL), the dorsolateral zone (DLZ), and the medial zone (MZ).