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1 pe was linked to smaller volumes in the left dorsolateral prefrontal cortex.
2 nterior cingulate cortex and to left IFG and dorsolateral prefrontal cortex.
3 hibitory control signal originating from the dorsolateral prefrontal cortex.
4 re associated with reduced activation of the dorsolateral prefrontal cortex.
5  regions within the intraparietal sulcus and dorsolateral prefrontal cortex.
6 anied by recruitment of anterior putamen and dorsolateral prefrontal cortex.
7 d to increased dynamic RSFC between MPFC and dorsolateral prefrontal cortex.
8 ct current stimulation (tDCS) over the right dorsolateral prefrontal cortex.
9 ckness in bilateral precentral gyrus and the dorsolateral prefrontal cortex.
10 perigenual anterior cingulate cortex and the dorsolateral prefrontal cortex.
11 emporo-parieto-occipital junction (TPOJ) and dorsolateral prefrontal cortex.
12 e and decreases during implementation in the dorsolateral prefrontal cortex.
13 al striatum, but a negative association with dorsolateral prefrontal cortex.
14 om a meta-analysis of gene expression in the dorsolateral prefrontal cortex.
15  rate of cortical thinning in the medial and dorsolateral prefrontal cortex.
16 dala to the subgenual anterior cingulate and dorsolateral prefrontal cortex.
17 , precuneus, lateral occipital, temporal and dorsolateral prefrontal cortex.
18  were generated using frozen tissue from the dorsolateral prefrontal cortex.
19 nterior cingulate, medial frontal gyrus, and dorsolateral prefrontal cortex.
20 red, in up to 40 daily sessions, to the left dorsolateral prefrontal cortex.
21  found between perception and GABA levels in dorsolateral prefrontal cortex.
22 ent conventional open-label rTMS to the left dorsolateral prefrontal cortex.
23 ctivity of the right Crus I/II with the left dorsolateral prefrontal cortex.
24 load observed in healthy individuals in left dorsolateral prefrontal cortex.
25  IC sources in or near medial prefrontal and dorsolateral prefrontal cortex.
26 d bilaterally sequentially to left and right dorsolateral prefrontal cortex 750 pulses/side at 20 Hz
27 atum, and (iv) reduced brain activity in the dorsolateral prefrontal cortex (a correlate of willpower
28                      tDCS was applied to the dorsolateral prefrontal cortex, a frequent target for mo
29  reduced functional connectivity between the dorsolateral prefrontal cortex, a region thought to play
30 f high-calorie foods significantly increased dorsolateral prefrontal cortex activation and suppressed
31                         Within the PS group, dorsolateral prefrontal cortex activation correlated wit
32                                              Dorsolateral prefrontal cortex activation during inhibit
33                                Posttreatment dorsolateral prefrontal cortex activation was reduced in
34 d that trauma-exposed youth failed to dampen dorsolateral prefrontal cortex activity and engage amygd
35 family income at age 9 and ventrolateral and dorsolateral prefrontal cortex activity at age 24.
36 f emotion type and associated with increased dorsolateral prefrontal cortex activity compared with th
37 n activity in Brodmann area 10 and the right dorsolateral prefrontal cortex activity when regulating
38 lied 5 days per week for 3 weeks to the left dorsolateral prefrontal cortex (added to the ongoing tre
39 gions, anterior and posterior cingulate, and dorsolateral prefrontal cortex all contributed reliably
40 le patients trait anger positively modulated dorsolateral prefrontal cortex-amygdala coupling.
41 odulatory effects of trait anger on amygdala-dorsolateral prefrontal cortex and amygdala-lateral orbi
42 igh-level cognitive control functions (i.e., dorsolateral prefrontal cortex and anterior cingulate co
43 o-occurring with structural abnormalities in dorsolateral prefrontal cortex and anterior cingulate co
44 ests the striatal afferent connection to the dorsolateral prefrontal cortex and basal ganglia circuit
45 lgesia was associated with activation of the dorsolateral prefrontal cortex and deactivation of senso
46 patients showed significantly increased left dorsolateral prefrontal cortex and decreased left hippoc
47 e show that BICC1 mRNA is upregulated in the dorsolateral prefrontal cortex and dentate gyrus of huma
48 several task-relevant regions, including the dorsolateral prefrontal cortex and DMN regions, was posi
49 l activity in classic 'control' regions (eg, dorsolateral prefrontal cortex and dorsal anterior cingu
50 ges in the interhemispheric effects from the dorsolateral prefrontal cortex and dorsal premotor corte
51 al-directed action, such as ventromedial and dorsolateral prefrontal cortex and dorsomedial striatum,
52          Functional interactions between the dorsolateral prefrontal cortex and hippocampus during wo
53 her microglial activation is elevated in the dorsolateral prefrontal cortex and hippocampus of untrea
54 )], that span the lateral prefrontal cortex (dorsolateral prefrontal cortex and inferior frontal gyru
55                                   Samples of dorsolateral prefrontal cortex and inferior parietal lob
56  lateral prefrontal cortex, particularly the dorsolateral prefrontal cortex and its connections, were
57 vortioxetine reduced activation in the right dorsolateral prefrontal cortex and left hippocampus duri
58 er volumes relative to controls in the right dorsolateral prefrontal cortex and left hippocampus, alo
59  ability to regulate the interaction between dorsolateral prefrontal cortex and M1.
60 y, as well as interactions between the right dorsolateral prefrontal cortex and medial and lateral pa
61 ask performance in ASD, whereas that between dorsolateral prefrontal cortex and parietal cortex (Brod
62 rior cingulate) and decision-making regions (dorsolateral prefrontal cortex and parietal cortex).
63 ve cognitive control requiring processing by dorsolateral prefrontal cortex and parietal cortex.
64                            Abnormal emotion (dorsolateral prefrontal cortex and right inferior fronta
65 terior cingulate, orbitofrontal cortex, left dorsolateral prefrontal cortex and right inferior fronta
66 of structure (decreased grey matter in right dorsolateral prefrontal cortex and right inferior tempor
67 ed functional connectivity between the right dorsolateral prefrontal cortex and right superior occipi
68 de network; 2) hypoconnectivity between left dorsolateral prefrontal cortex and subgenual anterior ci
69 d the most consistent underactivation in the dorsolateral prefrontal cortex and temporal pole, while
70 ddiction-relevant brain regions, such as the dorsolateral prefrontal cortex and the angular and cingu
71 al cortical inhibition [LICI]) from the left dorsolateral prefrontal cortex and the left motor cortex
72 control and addiction-related processes (eg, dorsolateral prefrontal cortex and ventral striatum).
73 nferior and superior frontal gyri, including dorsolateral prefrontal cortex and ventrolateral prefron
74 gulation despite increased activation of the dorsolateral prefrontal cortex and ventrolateral prefron
75 specifically ventromedial prefrontal cortex, dorsolateral prefrontal cortex, and anterior cingulate c
76 the sgACC and the default mode network, left dorsolateral prefrontal cortex, and insula, and reduced
77 ostral and dorsal anterior cingulate cortex, dorsolateral prefrontal cortex, and lateral orbitofronta
78 tal seed regions and the anterior cingulate, dorsolateral prefrontal cortex, and limbic regions such
79 -subcortical circuitry engaging anterior and dorsolateral prefrontal cortex, and midbrain.
80 nique projections from orbitofrontal cortex, dorsolateral prefrontal cortex, and parietal regions.
81 mical connections from orbitofrontal cortex, dorsolateral prefrontal cortex, and posterior parietal c
82 learning-related activation in the striatum, dorsolateral prefrontal cortex, and the MTL during the e
83 la induced by single TMS pulses to the right dorsolateral prefrontal cortex; and 4) greater ventromed
84  in a network of regions including bilateral dorsolateral prefrontal cortex, anterior cingulate and a
85 riatum --> insula --> medial frontal cortex, dorsolateral prefrontal cortex, anterior cingulate corte
86 nction within the frontal lobe involving the dorsolateral prefrontal cortex, anterior cingulate corte
87 ties and volumes of pyramidal neurons of the dorsolateral prefrontal cortex, anterior cingulate corte
88 rts, we tested whether GMV and NAA/Cr in the dorsolateral prefrontal cortex are associated with fatig
89  to quantify GAD67 and Zif268 mRNA levels in dorsolateral prefrontal cortex area 9 from 62 matched pa
90 tural brain marker-volume of left hemisphere dorsolateral prefrontal cortex-associated with the magni
91 were assessed in four frontostriatal tracts (dorsolateral prefrontal cortex-associative striatum, dor
92  integrated information is used by the right dorsolateral prefrontal cortex at the time of the decisi
93 udies should evaluate regions other than the dorsolateral prefrontal cortex, at other time points, an
94   EF and DB were associated with the rostral dorsolateral prefrontal cortex bilaterally.
95 ex while recalling resulted in activation in dorsolateral prefrontal cortex bilaterally.
96 t the executive control circuitry, including dorsolateral prefrontal cortex (cluster-corrected P < .0
97  had increased activity in the contralateral dorsolateral prefrontal cortex compared with patients wi
98 y between the posterior cingulate cortex and dorsolateral prefrontal cortex, compared with HC subject
99       We found that neuronal activity in the dorsolateral prefrontal cortex conveyed the necessary in
100 in gray matter volume in the hippocampus and dorsolateral prefrontal cortex correlate with our abilit
101 onnectivity of the right caudate to the left dorsolateral prefrontal cortex could reflect change in n
102 tary motor area, inferior parietal lobe, and dorsolateral prefrontal cortex despite similar overall p
103 f 561 immune genes and 20 immune pathways in dorsolateral prefrontal cortex (DLPFC) (144 schizophreni
104 ed with the unmedicated patient group in the dorsolateral prefrontal cortex (DLPFC) (MR, 0.26 mm; P =
105 zophrenia, is often associated with aberrant dorsolateral prefrontal cortex (dlPFC) activation.
106  multimodal executive system anchored on the dorsolateral prefrontal cortex (DLPFC) and a salience sy
107 onnectivity seeded from the right IFG to the dorsolateral prefrontal cortex (DLPFC) and anterior cing
108 n distributed neural circuitry including the dorsolateral prefrontal cortex (DLPFC) and appear to ari
109         We applied anodal tDCS over the left dorsolateral prefrontal cortex (DLPFC) and cathodal tDCS
110 onal spike activities of single units in the dorsolateral prefrontal cortex (dlPFC) and the anterior
111 othesis that interactions between regions of dorsolateral prefrontal cortex (dlPFC) and ventromedial
112 ne density on layer 3 pyramidal cells in the dorsolateral prefrontal cortex (DLPFC) appears to contri
113 and functional alterations of neurons in the dorsolateral prefrontal cortex (dlPFC) are thought to co
114 lation indicated the involvement of the left dorsolateral prefrontal cortex (DLPFC) as well as left M
115 r mechanisms, on the recruitment of the left dorsolateral prefrontal cortex (DLPFC) as well as on the
116 er a temporary neural disruption of the left Dorsolateral Prefrontal Cortex (DLPFC) can improve impli
117 rTMS) of Brodmann Area (BA) nine of the left dorsolateral prefrontal cortex (DLPFC) can produce analg
118 n working memory that reflect dysfunction of dorsolateral prefrontal cortex (DLPFC) circuitry.
119               It has been suggested that the dorsolateral prefrontal cortex (DLPFC) contributes in th
120           We found that lesions of the human dorsolateral prefrontal cortex (DLPFC) decreased the eff
121                               Neurons in the dorsolateral prefrontal cortex (DLPFC) encode a diverse
122 odes children's own preferences and the left dorsolateral prefrontal cortex (dlPFC) encodes the proje
123 , dorsal anterior cingulate (ACC), and right dorsolateral prefrontal cortex (DLPFC) evident only in S
124 ment of working memory that relies on normal dorsolateral prefrontal cortex (DLPFC) function.
125 amygdala activation and reduced amygdala and dorsolateral prefrontal cortex (dlPFC) functional coupli
126                       Neurons in the primate dorsolateral prefrontal cortex (dlPFC) generate persiste
127 od-oxygen-level-dependent (BOLD) signal, and dorsolateral prefrontal cortex (DLPFC) glutamate+glutami
128 l direct current stimulation (tDCS) over the dorsolateral prefrontal cortex (DLPFC) has been effectiv
129                                 Although the dorsolateral prefrontal cortex (DLPFC) has long been con
130                        Here we show that the dorsolateral prefrontal cortex (DLPFC) implements a flex
131 ajority of glutamatergic genes tested in the dorsolateral prefrontal cortex (DLPFC) in MDD (F21,59=2.
132       Although prior work has implicated the dorsolateral prefrontal cortex (DLPFC) in norm-based jud
133 rated reduced dendritic spine density in the dorsolateral prefrontal cortex (DLPFC) in schizophrenia.
134 anscranial magnetic stimulation (TMS) of the dorsolateral prefrontal cortex (DLPFC) is an established
135  Conflict monitoring theory assumes that the dorsolateral prefrontal cortex (DLPFC) is causally invol
136                  We found that damage to the dorsolateral prefrontal cortex (dlPFC) led to a more pos
137                         We hypothesized that dorsolateral prefrontal cortex (dlPFC) lesions, due to t
138 curring de novo, in brain-expressed genes of dorsolateral prefrontal cortex (DLPFC) neurons.
139 levels, have frequently been reported in the dorsolateral prefrontal cortex (DLPFC) of schizophrenia
140 mation relevant for credit assignment in the dorsolateral prefrontal cortex (dlPFC) of two male rhesu
141 ial magnetic stimulation (rTMS) of the right dorsolateral prefrontal cortex (DLPFC) on working memory
142                                The extent of dorsolateral prefrontal cortex (DLPFC) plasticity in Alz
143                                          The dorsolateral prefrontal cortex (DLPFC) plays a pivotal r
144                                          The dorsolateral prefrontal cortex (DLPFC) plays a pivotal r
145                                          The dorsolateral prefrontal cortex (DLPFC) plays an importan
146 t the volume of the rostral part of the left dorsolateral prefrontal cortex (DLPFC) predicted an indi
147 othalamus, midbrain, right insula, and right dorsolateral prefrontal cortex (DLPFC) regions supported
148                                              Dorsolateral prefrontal cortex (DLPFC) retrieval activat
149 om nonhuman primate studies, posits that the dorsolateral prefrontal cortex (dlPFC) stores and mainta
150                                  The primate dorsolateral prefrontal cortex (dlPFC) subserves top-dow
151  activity of neural circuitry in the primate dorsolateral prefrontal cortex (DLPFC) supports a range
152 as closely associated with feedback from the dorsolateral prefrontal cortex (DLPFC) to V1.
153 ical inhibition (LICI) was measured from the dorsolateral prefrontal cortex (DLPFC) using combined tr
154                                          The dorsolateral prefrontal cortex (DLPFC) was chosen as con
155 s in the anterior cingulate cortex and right dorsolateral prefrontal cortex (DLPFC) were compared via
156     We hypothesized that the hippocampus and dorsolateral prefrontal cortex (dlPFC) would play a key
157 le high-frequency rTMS session over the left dorsolateral prefrontal cortex (DLPFC) would reduce cue
158  direct current stimulation (tDCS) over left dorsolateral prefrontal cortex (dlPFC) yielded a close m
159 ree latent brain factors (amygdala, pACC and dorsolateral prefrontal cortex (DLPFC)) to test the effe
160 anscranial direct current stimulation of the dorsolateral prefrontal cortex (dlPFC), a critical neura
161 sociated with reduced activation in the left dorsolateral prefrontal cortex (DLPFC), a region of the
162 P down-regulates the BOLD signal in the left dorsolateral prefrontal cortex (dlPFC), a risk-integrati
163  have implicated orbitofrontal cortex (OFC), dorsolateral prefrontal cortex (DLPFC), and anterior cin
164 ilateral superior parietal lobule, bilateral dorsolateral prefrontal cortex (DLPFC), and bilateral mi
165 igation-guided rTMS was applied to the right dorsolateral prefrontal cortex (DLPFC), and fMRI and fun
166 monstrated greater fMRI response in caudate, dorsolateral prefrontal cortex (dlPFC), and intraparieta
167 sorders generally involve dysfunction of the dorsolateral prefrontal cortex (dlPFC), but there are fe
168 e frontal lobes [anode electrode at the left dorsolateral prefrontal cortex (DLPFC), cathode electrod
169 ed with decreased gamma activity in the left dorsolateral prefrontal cortex (dlPFC), decreased theta
170 st in anterior cingulate cortex (ACC) before dorsolateral prefrontal cortex (dlPFC), followed by medi
171 c input and implicated in schizophrenia (the dorsolateral prefrontal cortex (DLPFC), hippocampus and
172 from a priori regions of interest: bilateral dorsolateral prefrontal cortex (DLPFC), medial frontal/c
173 ween the bilateral hippocampus and the right dorsolateral prefrontal cortex (DLPFC), mirroring clinic
174 e found that threat affected activity in the dorsolateral prefrontal cortex (dlPFC), rather than the
175                           In the post-mortem dorsolateral prefrontal cortex (DLPFC), we found strikin
176 ells in layer 3 (L3) and layer 5 (L5) of the dorsolateral prefrontal cortex (DLPFC), we sought to det
177 ons in several cortical areas, including the Dorsolateral Prefrontal Cortex (DLPFC).
178 ial direct current stimulation (tDCS) of the dorsolateral prefrontal cortex (DLPFC).
179 cognitive function reliant on area 46 of the dorsolateral prefrontal cortex (dlPFC).
180  linked to a decrease in the activity of the dorsolateral prefrontal cortex (dlPFC).
181 pines on deep layer 3 pyramidal cells in the dorsolateral prefrontal cortex (DLPFC).
182  this response could also be evoked from the dorsolateral prefrontal cortex (dlPFC).
183 by dorsomedial prefrontal cortex (DMPFC) and dorsolateral prefrontal cortex (DLPFC).
184  that flexibility is mainly subserved by the dorsolateral prefrontal cortex (DLPFC).
185  of neurons within Brodmann's Area 46 of the dorsolateral prefrontal cortex (dlPFC).
186 s3749034 and with the expression of GAD25 in dorsolateral prefrontal cortex (DLPFC).
187 a (SZ) is associated with dysfunction of the dorsolateral prefrontal cortex (DLPFC).
188 ts involve dysfunction of the newly evolved, dorsolateral prefrontal cortex (dlPFC).
189 in-positive (PV) interneurons in the primate dorsolateral prefrontal cortex (DLPFC).
190 es is associated with neural activity in the dorsolateral prefrontal cortex (dlPFC).
191  100 kb of the lead SNP are expressed in the dorsolateral prefrontal cortex (DLPFC): UNC5C, ENC1, and
192 to examine the expression of 58 genes in the dorsolateral prefrontal cortex (DLPFC, comprised of Brod
193 nd insula but increased BOLD activity in the dorsolateral prefrontal cortex (dlPFC; local maxima corr
194 ons important in top-down executive control (dorsolateral prefrontal cortex [dlPFC]), here we test wh
195                        Seed-based (bilateral dorsolateral prefrontal cortex, DLPFC) rsFC analysis was
196              When pain was controllable, the dorsolateral prefrontal cortex downregulated pain-evoked
197                  We stimulated over the left dorsolateral prefrontal cortex during a declarative memo
198 ce that NRXN1 expression is highest in human dorsolateral prefrontal cortex during critical developme
199 e observed both reduced 1) activation of the dorsolateral prefrontal cortex during encoding and 2) de
200 ignificant differences localized to the left dorsolateral prefrontal cortex during response selection
201         Notably, [oxy-Hb] change in the left dorsolateral prefrontal cortex during SAT showed a posit
202         tDCS was administered over the right dorsolateral prefrontal cortex during the retrieval-prac
203 long with blunted responses of the bilateral dorsolateral prefrontal cortex, during the processing of
204 evance of the findings and the proposal that dorsolateral prefrontal cortex dysfunction represents a
205                             In the postnatal dorsolateral prefrontal cortex, expression levels were n
206 e efficacy of 10-Hz rTMS applied to the left dorsolateral prefrontal cortex for the treatment of pred
207                    The optimal target in the dorsolateral prefrontal cortex for treating depression w
208 ain transcriptome in high-quality postmortem dorsolateral prefrontal cortex from 11 individuals diagn
209 olymerase chain reaction in human postmortem dorsolateral prefrontal cortex from 286 nonpsychiatric c
210 olymerase chain reaction in human postmortem dorsolateral prefrontal cortex from 286 nonpsychiatric c
211 encing of poly-A RNA derived from postmortem dorsolateral prefrontal cortex from people with BD, alon
212 n basket cell (PVBC) inputs in area 9 of the dorsolateral prefrontal cortex from schizophrenia and ma
213  or calretinin-positive (CR+) neurons in the dorsolateral prefrontal cortex from schizophrenia subjec
214 agnetic stimulation (rTMS) targeted over the dorsolateral prefrontal cortex has been shown to modulat
215 nual anterior cingulate cortex [sgACC], left dorsolateral prefrontal cortex, hippocampus, and basolat
216       Mean diffusivity (MD) was computed for dorsolateral prefrontal cortex, hippocampus, parahippoca
217                                 In the right dorsolateral prefrontal cortex IC cluster, the beta band
218 of the causal influence of the insula on the dorsolateral prefrontal cortex in cocaine users.
219 nal connectivity between the putamen and the dorsolateral prefrontal cortex in OCD patients, as well
220 ted inverted-U response was observed in left dorsolateral prefrontal cortex in patients and was assoc
221 e 226 genes, 9 differed in expression in the dorsolateral prefrontal cortex in patients with BP: CACN
222 tribute to lower levels of GAD67 mRNA in the dorsolateral prefrontal cortex in schizophrenia.
223  of ErbB4 transcripts is dysregulated in the dorsolateral prefrontal cortex in schizophrenia.
224 ed prefrontal regions (orbitofrontal cortex, dorsolateral prefrontal cortex, inferior frontal gyrus,
225  involved in language control, including the dorsolateral prefrontal cortex, inferior parietal lobule
226 inputs (lower-order), with less weighting of dorsolateral prefrontal cortex inputs (higher-order).
227                                          The dorsolateral prefrontal cortex is downregulated, diminis
228 europsychology studies have established that dorsolateral prefrontal cortex is essential in spatial w
229 ing the intraparietal sulcus, precuneus, and dorsolateral prefrontal cortex is involved in selecting
230 mory activation in PMDD, specifically in the dorsolateral prefrontal cortex, is related to PMDD sever
231 e (Glx), were measured by 1H MRS in the left dorsolateral prefrontal cortex (l-DLPFC) and bilateral h
232                                         Left dorsolateral prefrontal cortex (L-DLPFC) tDCS induced an
233 prefrontal cortex (Brodmann area 10) and the dorsolateral prefrontal cortex (lateral Brodmann 9) whil
234  interneurons were laser microdissected from dorsolateral prefrontal cortex layers 2 and 4, respectiv
235 tal evidence that increasing activity in the dorsolateral prefrontal cortex leads to greater evidence
236  in the alcohol-dependent group included the dorsolateral prefrontal cortex, medial prefrontal cortex
237 default (posterior cingulate) and executive (dorsolateral prefrontal cortex) networks.
238 ng the cognitive deficits and dysfunction of dorsolateral prefrontal cortex observed in this disorder
239 lation and histone acetylation data from the dorsolateral prefrontal cortex of 411 older adults who h
240 lternating current stimulation over the left dorsolateral prefrontal cortex of human participants [n
241 67), a key enzyme for GABA synthesis, in the dorsolateral prefrontal cortex of individuals with schiz
242 ed, and DNA methylation was increased in the dorsolateral prefrontal cortex of male suicide subjects,
243 n by recording single-unit activity from the dorsolateral prefrontal cortex of monkeys that were perf
244 predictor of both GMV and NAA/Cr in the left dorsolateral prefrontal cortex of patients with CFS.
245  associated with neuronal alterations in the dorsolateral prefrontal cortex of patients with CFS.
246 The CommonMind Consortium sequenced RNA from dorsolateral prefrontal cortex of people with schizophre
247 idal cell volumes in layers III and V in the dorsolateral prefrontal cortex of post-stroke and vascul
248 rom the contribution of interactions between dorsolateral prefrontal cortex of the FPN and precuneus
249 onotonically decreased BOLD signal change in dorsolateral prefrontal cortex on Day 1 as a function of
250 anial direct current stimulation of the left dorsolateral prefrontal cortex or a sham stimulation con
251 as indexed by [(18)F]FEPPA VT, in either the dorsolateral prefrontal cortex or the hippocampus.
252 positive valence tasks; and hyperactivity in dorsolateral prefrontal cortex (P < .001) and superior t
253 atment increased activation in both the left dorsolateral prefrontal cortex (PFC) and supplementary m
254 ntal state and harm information, whereas the dorsolateral prefrontal cortex plays a crucial, final-st
255 lly defined network includes portions of the dorsolateral prefrontal cortex, posteromedial prefrontal
256                               The changes in dorsolateral prefrontal cortex pyramidal cells were not
257 ctive regional pyramidal cell atrophy in the dorsolateral prefrontal cortex-rather than neuronal dens
258 tion of activation in the striatum and right dorsolateral prefrontal cortex (rDLPFC; ie, the degree t
259 iatal and midbrain) and prefrontal cortical (dorsolateral prefrontal cortex) regions during reward an
260                 RNA sequencing data of human dorsolateral prefrontal cortex revealed relatively high
261 zation in the 6-12 Hz range within the right dorsolateral prefrontal cortex, right frontal eye-fields
262 l anterior cingulate cortex (dACC), and left dorsolateral prefrontal cortex seeds and by relative hyp
263 nd fewer normalized streamlines in the right dorsolateral prefrontal cortex-sensorimotor striatum and
264 hermore, normalized streamlines in the right dorsolateral prefrontal cortex-sensorimotor striatum neg
265 rmalized streamlines in the right-hemisphere dorsolateral prefrontal cortex-sensorimotor striatum pre
266 eral prefrontal cortex-associative striatum, dorsolateral prefrontal cortex-sensorimotor striatum, ve
267 ted into ventrolateral prefrontal cortex and dorsolateral prefrontal cortex subregions.
268 between these systems by disruption of right dorsolateral prefrontal cortex, such that participants m
269 ns in contralateral ventral premotor cortex, dorsolateral prefrontal cortex, supramarginal gyrus, and
270  anterior cingulate (t = 2.27; P = .04), and dorsolateral prefrontal cortex (t = 2.44; P = .03) were
271 ontrols, among three brain regions including dorsolateral prefrontal cortex, temporal cortex and cere
272 , hypometabolism and/or hypoperfusion in the dorsolateral prefrontal cortex, the anterior and middle
273 PD had reduced activation in the ipsilateral dorsolateral prefrontal cortex, the globus pallidus inte
274 t PD had reduced activity in the ipsilateral dorsolateral prefrontal cortex, the globus pallidus inte
275 work of areas, including the dorsomedial and dorsolateral prefrontal cortex, the intraparietal sulcus
276 evels at individual CpG sites generated from dorsolateral prefrontal cortex tissue using a bead assay
277 riven by reduced effective connectivity from dorsolateral prefrontal cortex to anterior prefrontal co
278 rain activation, driven in part by increased dorsolateral prefrontal cortex to midbrain connectivity.
279 sorders in the microarray-based "BrainCloud" dorsolateral prefrontal cortex transcriptome.
280 ramidal neuron dendrites in Brodmann area 46 dorsolateral prefrontal cortex using the Golgi-Cox techn
281 ate cortex (dACC), and the ventrolateral and dorsolateral prefrontal cortex (VLPFC, DLPFC).
282  anticipatory and outcome-locked activity in dorsolateral prefrontal cortex was greater for safe than
283      Response to high-probability rewards in dorsolateral prefrontal cortex was inversely associated
284 onnectivity of the right caudate to the left dorsolateral prefrontal cortex was negatively associated
285 ced change in connectivity from right IFG to dorsolateral prefrontal cortex was proportional to the c
286  covariates, cortical thickness of the right dorsolateral prefrontal cortex was significantly thinner
287 Application of active 10-Hz rTMS to the left dorsolateral prefrontal cortex was well tolerated but wa
288      In further morphometric analysis of the dorsolateral prefrontal cortex, we showed that neither d
289 inhibition (ie, N100 and LICI) from the left dorsolateral prefrontal cortex were selected.
290 s across the two tasks were localized to the dorsolateral prefrontal cortex, where responses were inc
291 e associated with hypoactivation of the left dorsolateral prefrontal cortex, whereas behavioral sympt
292 fortful DM) leading to activation across the dorsolateral prefrontal cortex, whereas experts are expe
293 fortful DM) leading to activation across the dorsolateral prefrontal cortex, whereas experts are expe
294 the primary visual cortex and highest in the dorsolateral prefrontal cortex, whereas the GABA measure
295 novices showed significant activation of the dorsolateral prefrontal cortex, whereas this activation
296 novices showed significant activation of the dorsolateral prefrontal cortex, whereas this activation
297            The antisaccade task requires the dorsolateral prefrontal cortex, which is part of the pre
298    Suppressing imagination engaged the right dorsolateral prefrontal cortex, which modulated activati
299 s resulted in significant activation in left dorsolateral prefrontal cortex while recalling resulted
300  (BMI) had lower activation in the bilateral dorsolateral prefrontal cortex while viewing unhealthy c

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