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1 , tacrolimus formulations were switched (1:1 dose ratio).
2 acy by increasing the tumor-to-normal tissue dose ratio.
3 ted to the alternate formulation at a 1:1 mg dose ratio.
6 ned-agent therapy as a function of the tumor-dose ratio and the fraction of activity contributed by e
7 is a promising approach to improve absorbed dose ratios and achieve high durable remission rates wit
8 stimates of dose profiles and peak-to-valley dose ratios at the position of the targeted and traverse
11 iodide, therefore, yielding a lower absorbed dose ratio between NIS-transfected and -nontransfected c
14 residence time, and improved tumor-to-kidney dose ratio compared with (177)Lu-DOTATATE and (90)Y-OPS2
15 se was calculated as a function of the tumor-dose ratio, defined as the (90)Y-DOTATOC tumor dose per
19 evaluated to compute the predicted-to-actual dose ratio ([Formula: see text]) in tumor volumes (TVs)
24 or-dose increase of 68% occurred for a tumor-dose ratio of 2.57, using 92% of the maximum tolerated (
26 ncreased activity in ALL cells in vitro, the dose ratio of the two drugs that exerted equivalent cyto
27 l dose profiles and calculate peak-to-valley dose ratios of 30-40 for cell cultures and approximately
28 ed dose of (90)Y, with tumor-to-normal organ dose ratios of 7:1 for liver and 15:1 for lung and kidne
29 ttenuating the cardiomyopathy caused by DOX, dose ratios of DZR:DOX capable of providing total or nea
30 nnitol reactivity, expressed as the response dose ratio (RDR: max % fall in FEV1 /cumulative dose), w
32 o-dose-limiting-organ (bone marrow) absorbed dose ratio, that is, the therapeutic index, was higher i
38 Tumor-to-lung, -kidney and -liver radiation dose ratios were 7.4:1, 5.3:1 and 2.6: 1, respectively.
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