ライフサイエンスコーパス: double infection

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1 ation, little is known about the dynamics of double infection.

2 o examined the nature of cell-mediated HIV-1 double infection.

3 sequence types, raising the possibility of a double infection.

4 ot the only factor contributing to nonrandom double infection.

5 fered additional survival costs when hosting double infections.

6 nt types of JCV at the same time, indicating double infections.

7 ymorphism and infection type suggesting that double infections are the stable type, with singly infec

8 Results show that double infections can reinforce interspecies reproductiv

9 We observed a decreased frequency of double infection compared with experiments in which DCs

10 However, the frequency of double infection differed significantly, depending on th

11 lysed datasets of cell-free HIV-1 single and double infection experiments in cell culture.

12 of previously uninfected matrilines, (ii) a double infection in a matriline already bearing a differ

13 is is the first direct evidence of nonrandom double infection in HIV-1.

14 measured and were used to calculate whether double infection occurred at frequencies expected from r

15 r CXCR4-tropic HIV-1 Env and have found that double infection occurred more frequently than random ev

16 e found that regardless of the protein used, double infection occurred more frequently than random ev

17 In primary CD4(+) T cells, double infection occurred most frequently when both viru

18 We found that double infection occurred significantly more frequently

19 Therefore, our results indicate that HIV-1 double infection occurs more frequently than it would at

20 pe 1 (HIV-1) infection is nonrandom and that double infection occurs more frequently than predicted f

21 evious study, we also demonstrated nonrandom double infection via dendritic cell (DC)-mediated HIV-1

22 Increased double infection was observed in all experiments regardl

23 from random distribution; increased rates of double infection were observed in both a T cell line and

24 asma cpn60 UT sequences, while identifying a double infection with SbGP/MPV and aster yellows (16SrI)

25 Each species have double infections with a representative from both the A

26 Both triple (wNlonAwNlonB1wNlonB2) and double infections (wNlonAwNlonB1, wNlonAwNlonB2) have be

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