1 d JNK proteins were localized by fluorescent
double labeling.
2 ionation, immunofluorescence, and immunogold
double labeling.
3 method and bisbenzamide/tyrosine hydroxylase
double labeling.
4 he IC and auditory cortex did not reveal any
double labeling.
5 enalidomide treatment using CD138(+)/IRF4(+)
double labeling.
6 ected to both the AI and the LO as judged by
double-labeling.
7 Our
double-labeling analyses of intraneuronal Abeta and CD40
8 A quantitative
double labeling analysis in layers II and III revealed t
9 Finally,
double-labeling analysis of CTb + Fos showed that contex
10 Combining
double labeling and confocal microscopy analyses, we fou
11 ergic neurons, we performed BDNF/parvalbumin
double labeling and discovered that axons from cortical
12 Double labeling and fluorescence-activated cell sorter a
13 Fifteen of these were confirmed with
double labeling and microscopy, including the transcript
14 nd its alpha5beta1 receptor was confirmed by
double labeling and multiple-exposure photomicroscopy.
15 his study, Western blots, immunofluorescence
double labeling,
and the retrograde tracing method were
16 otope probing (Raman-SIP) using a single and
double labeling approach.
17 nstrated by cell lineage tracing and mineral
double-labeling approaches that murine PDL progenitor ce
18 To do this, we used
double-labeling combinations of Fos immunocytochemistry
19 Under the same
double-labeling conditions, however, cones in homozygous
20 Double labeling confirmed up-regulation of CCL2/CCR2 on
21 tochrome c from mitochondria, as revealed by
double-labeling confocal immunofluorescence microscopy.
22 gocytophilum entry into neutrophils by using
double-labeling confocal microscopy.
23 Extensive
double-labeling crossover studies have been conducted.
24 e mechanism was obtained through an isotopic
double-labeling crossover study.
25 Double labeling demonstrated coexpression of JCV T antig
26 Double labeling demonstrated that apoptotic cells includ
27 BrdU staining, providing a valuable tool of
double labeling DNA synthesis, e.g., for tracking the tw
28 Immunogold
double-labeling electron microscopy, using antisera agai
29 abundantly labeled with Fast blue (FB) in a
double-labeling experiment in which FB was co-injected i
30 A
double-labeling experiment with the dimeric model showed
31 ochemical methods to test the feasibility of
double labeling experiments during development.
32 Double labeling experiments indicate that the neurochemi
33 Double labeling experiments revealed that complexes of O
34 Double labeling experiments showed that the location of
35 Confocal scanning laser microscopy and
double labeling experiments with rhodamine 123 suggested
36 Double labeling experiments, by either in situ hybridiza
37 bone formation rates in in vivo fluorochrome
double labeling experiments.
38 Double-labeling experiments (biocytin backfill x seroton
39 Double-labeling experiments colocalizing the bradykinin
40 Double-labeling experiments confirm that NADPH-d, the mo
41 cell the "distal Dab1-immunoreactive cell."
Double-labeling experiments demonstrate that the distal
42 Double-labeling experiments demonstrated that CRN termin
43 However, in immunofluorescent
double-labeling experiments for GABA(B)R1 and GAD, we ne
44 ons affected by such lesions, we carried out
double-labeling experiments for the inhibitory neurotran
45 Double-labeling experiments further demonstrate that V1
46 c gut-associated lymphoid tissue at 14 days;
double-labeling experiments indicated that 93.5% of the
47 Double-labeling experiments reveal NMB is expressed in a
48 Double-labeling experiments revealed several possible li
49 Moreover,
double-labeling experiments revealed that activation of
50 Double-labeling experiments showed striking cellular col
51 Double-labeling experiments showed that most of the prin
52 s of vallate papillae and nasoincisor ducts,
double-labeling experiments showed that syntaxin-1, SNAP
53 Double-labeling experiments showed that the dendritic an
54 of GABA antagonists, uptake inhibitors, and
double-labeling experiments showing that Cr-Aint neurons
55 in the control of food intake, we performed
double-labeling experiments to evaluate the potential ov
56 tly labeled retinal terminals was counted in
double-labeling experiments using confocal microscopy an
57 Double-labeling experiments using probes for previously
58 Additional
double-labeling experiments using the nonspecific neuron
59 Single- and
double-labeling experiments were performed to establish
60 Double-labeling experiments with a subset of modulators
61 Double-labeling experiments with ArINTs and ArINTstBu (T
62 etween caspase activation and NFT formation,
double-labeling experiments with fodrin CCP and PHF-1 we
63 When mAb-GlyRbeta was used in
double-labeling experiments with GlyRalpha1-, alpha2-, a
64 In
double-labeling experiments, antibodies to acetylated hi
65 Also, we performed
double-labeling experiments, injecting BDA in the CRNs a
66 in V1 or V2, and, consistently with previous
double-labeling experiments, some axons can branch to bo
67 t that we identified as the Golgi complex in
double-labeling experiments, while wild type caveolin-3
68 thalamic neurons were compared in retrograde
double-labeling experiments.
69 Finally, we performed
double-labeling,
first by retrograde labeling of HRP inj
70 Double-labeling FISH showed colocalization of tyrosine h
71 pproach to evaluate this hypothesis, we used
double-labeling fluorescence immunohistochemistry to det
72 Double-labeling fluorescence immunohistochemistry using
73 Using
double labeling fluorescent histochemistry with confocal
74 amined using myeloperoxidase (MPO) assay and
double-labeling fluorescent immunohistochemical analysis
75 Combinations of
double-labeling fluorescent immunohistochemistry (D-FIHC
76 As expected,
double labeling for ATF-3, a marker of cell bodies with
77 t macaque monkeys, using immunohistochemical
double labeling for bromodeoxyuridine and cell-type-spec
78 Double labeling for GAD67 and GHRHR in vitro and in vivo
79 tion of the in situ hybridization signal and
double labeling for glial fibrillary acidic protein were
80 The current investigation used
double labeling for NADPHd and Fos-like immunoreactivity
81 Double labeling for NMDA NR1 and c-fos revealed marked a
82 Double labeling for OMP and MAP2 revealed two distinctiv
83 Double labeling for Prox1 and cell-type-specific markers
84 respond to laryngeal afferent stimulation by
double labeling for reduced nicotinamide adenine dinucle
85 nventional retinal synapses was confirmed by
double labeling for synapsin I, a marker for conventiona
86 bserved in dendritic profiles, verified with
double labeling for the dendrite-specific marker microtu
87 ze the phenotype of ER-beta-bearing cells by
double labeling for the GABAergic-associated calcium-bin
88 al cells were identified as keratinocytes by
double labeling for WNV antigen and keratin 10.
89 After
double-labeling for Cx36 and Cx43 by FRIL, neuronal gap
90 Double-labeling for flow cytometric analysis was employe
91 Double-labeling for GAD and synaptophysin confirmed that
92 Double labeling further reveals that the majority of Cck
93 Light and electron microscopic
double labeling further showed that the VGLUT1 subtype o
94 To address this question,
double-labeling histochemical studies were performed for
95 Double labeling identified significant co-localization o
96 and protein immunofluorescence (IF)-RNA FISH
double labeling (
IF/FISH).
97 Double labeling immunocytochemical studies confirmed tha
98 Double-labeling immunocytochemistry revealed m1 receptor
99 By
double-labeling immunocytochemistry, alpha-MSH-IR axon v
100 , cat, and monkey was studied by single- and
double-labeling immunocytochemistry.
101 Double labeling Immunofluorescence was performed using c
102 Using
double-labeling immunofluorescence analysis of intraneur
103 Double-labeling immunofluorescence experiments revealed
104 Double-labeling immunofluorescence histochemistry reveal
105 Double-labeling immunofluorescence microscopic analyses
106 Double-labeling immunofluorescence revealed that approxi
107 from stage 14 (S14) to adulthood by using a
double-labeling immunofluorescence technique.
108 mbinant, we have been able to demonstrate by
double-labeling immunofluorescence that matrix protein (
109 In
double-labeling immunofluorescent preparations, 61.1 +/-
110 With
double-labeling immunogold electron microscopy (EM), we
111 Double-labeling immunogold experiments showed that appro
112 Using
double labeling immunohistochemistry on mouse and rat sp
113 We demonstrate using
double labeling immunohistochemistry that Group II metab
114 Animals were sacrificed for single and
double labeling immunohistochemistry to identify which c
115 Double labeling immunohistochemistry was used to identif
116 Using
double-labeling immunohistochemistry and confocal micros
117 ne (BrdU) labeling of newly generated cells,
double-labeling immunohistochemistry and TUNEL labeling
118 Double-labeling immunohistochemistry indicated that PR-i
119 By using
double-labeling immunohistochemistry or immunohistochemi
120 Double-labeling immunohistochemistry revealed that all o
121 Double-labeling immunohistochemistry showed NPY-IR axon
122 Double-labeling immunohistochemistry was performed with
123 Double-labeling immunohistochemistry was used to determi
124 Light microscopic
double-labeling immunoperoxidase experiments demonstrate
125 For
double labeling,
immunoreactivity was visualized using i
126 Double-labeling immunostaining and co-immunoprecipitatio
127 ted into the pylorus of mice and fluorescent
double-labeling immunostaining for betaIII-tubulin or PG
128 Protein
double labeling in aqueous buffer at physiological pH, t
129 lateral lemniscus (VNLL and INLL), with some
double labeling in ipsilateral lateral and medial superi
130 tion neurons exhibited a higher incidence of
double labeling in the superficial dorsal horn.
131 Results from
double-labeling in situ hybridization suggest that in Tg
132 The purpose of this study was to use
double-labeling in situ hybridization to identify nAChR
133 By
double-labeling in situ hybridization, a subpopulation o
134 e projecting to accumbens shell, with higher
double-labeling in the ipsilateral projection than in th
135 In both species, lack of overt
double labeling indicated that the ChAT(+) and urocortin
136 verlap of retrograde labeling but negligible
double labeling,
indicating that ventral striatopallidum
137 oxidase staining and using immunofluorescent
double labeling,
infrequent TH-immunoreactive, transplan
138 The advantage of
double labeling is revealed in our demonstration of nove
139 d pre-oromotor neurons, but there was sparse
double-labeling (&
lt;10%).
140 ucleus (XII) motoneurons was studied using a
double labeling method of anterogradely biotinylated dex
141 ural level, we develop and apply a "two-tag"
double-labeling method to label LT11's dendrites and the
142 eparate populations of synapses, we employed
double-labeling methods.
143 We hypothesized that the
double labeling of an adenovirus with fluorescent protei
144 Double labeling of CB2R and glutamine synthetase shows t
145 Double labeling of cells with 12-(N-methyl)-N-[(7-nitrob
146 ent study, this issue was addressed by using
double labeling of descending brain neurons.
147 4-5-day hatchling chicks by using single and
double labeling of fibers and terminals with biocytin co
148 al surgery activated brain CRF neurons using
double labeling of Fos/CRF in naive rats.
149 The
double labeling of ganglion neurons indicates their site
150 Indeed,
double labeling of miR156 showed a meristem-specific pat
151 Double labeling of mRNA and beta-galactosidase immunorea
152 Double labeling of myocilin with other ECM components wa
153 cally for overlap of retrograde labeling and
double labeling of neurons.
154 Transgenesis and
double labeling of NTS and HCRT neurons showed that NTS
155 Double-transgenic mice showed 55%
double labeling of periurethral neuroendocrine cells exp
156 rviving ganglion cells were quantified after
double labeling of retinal tissue with TUNEL and Brn3a.
157 Double labeling of sGC with neuronal nitric oxide syntha
158 Double labeling of sGC with neuronal nitric oxide syntha
159 The C-7,19
double labeling of the A-ring enyne was achieved by the
160 Double labeling of the HCMV(CR208)-infected HF cells dem
161 Double labeling of the N- and C-terminally tagged peripl
162 g context-induced renewal tests by measuring
double labeling of the retrograde tracer cholera toxin s
163 Double labeling of tissue with two different hair cell m
164 Double labeling of TSH4 with the ramosa2, branched silkl
165 Although
double-labeling of microglia with Iba1 and ED1 revealed
166 Double-labeling of neurons in which GFP was driven by C3
167 First, we measured
double-labeling of the neuronal activity marker Fos with
168 process through crossover experiments using
double-labeling (
oxo and phosphine).
169 Double-labeling procedures showed that calretinin-positi
170 ed an iododeoxyuridine and bromodeoxyuridine
double labeling protocol for use in the developing embry
171 Double labeling revealed that 30.2% of nodose neurons ex
172 Double labeling revealed that a large percentage of thes
173 Double labeling revealed that GRIP and GluR2 were coloca
174 versely, m2/choline acetyltransferase (ChAT)
double labeling revealed that m2-positive neurons corres
175 Double labeling revealed that the compartment contained
176 Immunofluorescence
double-labeling revealed a punctate pattern for ENT1 clo
177 Double-labeling revealed that only two regions of the mo
178 Double labeling reveals that all PV+ cells also contain
179 By means of
double-labeling RNA in situ hybridization in mice, we sh
180 alitative abnormalities, with bone turnover (
double labeling)
seen in all specimens.
181 Double labeling showed that all protein kinase C-immunor
182 Immunofluorescent
double labeling showed that in aged vessels IL-1beta and
183 in regions (e.g. hypothalamus and amygdala),
double labeling showed that most CBP was not co-localize
184 Double labeling showed that neuronal nitric oxide syntha
185 Double labeling showed that the thalamic valop populatio
186 Double labeling showed that type 5b cone bipolar cells e
187 Double labeling showed these to be both astrocytes and o
188 r, immunofluorescence of BRCA1 and nucleolin
double-labeling showed colocalization in both nucleoli a
189 Double labeling shows coincidence of STIM1 and STIM2 wit
190 Immunofluorescence
double labeling shows that approximately 80% of the c-ne
191 Immunofluorescence
double-labeling staining showed no significant differenc
192 Here, we used a
double-labeling strategy (varying both the distance betw
193 This
double-labeling strategy provides a simple method to rel
194 Immunofluorescent
double labeling studies additionally reveal the prominen
195 Double labeling studies confirmed that the alpha-syn::GF
196 Furthermore,
double labeling studies for cytochrome c/alpha-synuclein
197 Double labeling studies revealed (EGFP)hHsp27 and actin
198 Double labeling studies showed that nicotine induced c-f
199 Immunohistochemical
double labeling studies with YFP and serotonin antisera
200 Double-labeling studies and examination of preclinical c
201 Double-labeling studies combining DAT and tyrosine hydro
202 Double-labeling studies demonstrated colocalization of T
203 Double-labeling studies demonstrated that NGF mRNA was e
204 Double-labeling studies demonstrated the localization of
205 Double-labeling studies examining CARTp with tyrosine hy
206 Double-labeling studies revealed that approximately 50%
207 Double-labeling studies revealed that early OL progenito
208 The
double-labeling studies revealed that mGluR5 immunoreact
209 Double-labeling studies show that lMAN(shell) contains t
210 Double-labeling studies showed colocalization of the two
211 Double-labeling studies showed that BrdU-labeled cells t
212 Double-labeling studies using confocal microscopy showed
213 In
double-labeling studies using confocal microscopy, fluor
214 Double-labeling studies with antibodies to phosphorylate
215 on of all the major septal receptor genes in
double-labeling studies.
216 he present results and those from our recent
double-labeling study suggest that following spinal cord
217 In this
double-labeling study, we placed a green axonal tracer i
218 d with the region that showed FluoroGold/Fos
double labeling,
suggesting reciprocal connections betwe
219 retrogradely labeled neurons and significant
double labeling,
suggesting that cortical projections sp
220 of markers were correlated, and an antibody
double labeling technique was employed.
221 Using a new
double-labeling technique we have colocalized antibodies
222 Immunohistochemical
double-labeling technique with Fos and markers for norad
223 nd thus the complete covalent structure, the
double-labeling technique, along with mass spectrometry,
224 Y-NB (Lucifer yellow-Neurobiotin) retrograde
double-labeling technique, in conjunction with specific
225 by employing electron spin and nuclear spin
double-labeling techniques did not yield unambiguous cha
226 This was investigated with immunofluorescent
double-labeling techniques to coregister PV- and CB-expr
227 In these studies, we used
double-labeling techniques to examine whether Cal conten
228 d that intracellular flora by using the same
double-labeling techniques to identify Fusobacterium nuc
229 bers in the IPL, standard immunocytochemical
double-labeling techniques were applied, using antibodie
230 Accordingly, immunocytochemical
double-labeling techniques were used to identify GABA an
231 BA-type neurons using retrograde marking and
double-labeling techniques, are profoundly excited by mu
232 Using
double-labeling techniques, some presynaptic terminals w
233 Using
double-labeling techniques, we established that these in
234 Double labeling the brainstem sections revealed that irI
235 Double labeling the hypothalamic sections with NPB antis
236 Double-labeling the hypothalamic sections with guinea-pi
237 Double-labeling the sections with PrRP antisera and tyro
238 ons of the RVM and caudal pons and performed
double labeling to evaluate the expression of alpha-7 an
239 However,
double-labeling uncovered no coexpression of FRU(M) and
240 born neurons using doublecortin, as well as
double labeling using an additional antibody to glial fi
241 Fluorescent
double labeling using antibodies to bNOS and glutamic ac
242 En face
double labeling using Ki-67 and progenitor markers revea
243 ions were processed for immunohistochemistry/
double labeling using patient sera/cerebrospinal fluid a
244 myocilin with ECM components was examined by
double labeling,
using different-sized gold particles.
245 Double labeling was carried out using anti-CD3, anti-CD6
246 No
double labeling was found when using a fluorescent trace
247 la, and midbrain as CRH neurons, although no
double labeling was found.
248 Double labeling was observed in the cell bodies of Purki
249 c and any or all of the ipsilateral targets,
double labeling was rare, suggesting that contralateral
250 Immunofluorescence
double labeling was used to determine cellular localizat
251 Much less
double-labeling was associated with injections into eith
252 function (bone formation) after tetracycline
double-labeling was performed by fluorescence microscopy
253 The same pattern of
double-labeling was seen from injections at each spinal
254 Commissural divergence, assessed by
double labeling,
was less than 3% in each area.
255 abeling and with preembedding immunogold for
double labeling,
was localized in cell bodies with ultra
256 By using
double labeling,
we found no evidence for mGluR1alpha or
257 Double labeling with a caveolin antibody indicated that
258 Cell death was quantified by
double labeling with a membrane impermeable dye and 4',6
259 AII amacrine cells, positively identified by
double labeling with an antibody against calretinin, wer
260 Double labeling with anti-PECAM1 antibody and anti-place
261 Double labeling with anti-PECAM1 antibody and one of thr
262 TULP1-positive cells were identified by
double labeling with antibodies specific for cones, rods
263 Double labeling with antibodies to calretinin and gamma-
264 Double labeling with antibodies to each amino acid and t
265 ype of BrdU-positive cells was identified by
double labeling with antibodies to neuronal or glial mar
266 transgenic mice were confirmed as neurons by
double labeling with antineurofilament antibody.
267 Double labeling with BDA and (3)H-leucine signifies that
268 Double labeling with BrdU and the cell cycle marker Ki-6
269 Double labeling with c-FLIP antibody and terminal deoxyn
270 In
double labeling with calretinin and parvalbumin, few neu
271 Double labeling with ChAT and synapsin antibodies showed
272 Double labeling with confocal microscopy of DLB midbrain
273 Moreover,
double labeling with Fluoro-Jade B and glial fibrillary
274 Double labeling with glial fibrillary acidic protein (as
275 Double labeling with isolectin B4 (IB4) showed that all
276 ricular nucleus of the hypothalamus (PVH) by
double labeling with markers expressed in viruses inject
277 Double labeling with mGluR4a antibodies and antibodies t
278 Double labeling with neuropeptide Y (NPY), a marker for
279 Double labeling with OMP and synaptophysin demonstrated
280 Double labeling with other molecular markers confirmed t
281 Double labeling with parvalbumin antibodies in monkey re
282 Double labeling with Plk3 and gamma-tubulin, the latter
283 By
double labeling with rhodopsin, we demonstrate that earl
284 Double labeling with several other markers for amacrine
285 Double labeling with SMI-32 and parvalbumin confirmed th
286 Double labeling with SNAP25 and calbindin antibodies dem
287 Cone cell death was analyzed by
double labeling with TdT-dUTP terminal nick-end labeling
288 al apoptosis was determined by histochemical
double labeling with terminal deoxynucleotidyl transfera
289 Double labeling with the 275 kD hair cell antigen (HCA)
290 Double labeling with the ganglion cell marker RBPMS demo
291 esence of small, clear synaptic vesicles and
double labeling with the presynaptic markers synaptophys
292 Double labeling with the vesicular glutamate transporter
293 o functional recovery, sequential retrograde
double labeling with two fluorescent dextran amines was
294 Double labeling with various glutamate receptor subunit
295 Double-labeling with a monoclonal antibody against micro
296 Double-labeling with anti-CD34 antibodies demonstrated t
297 Immunohistochemistry was performed by
double-labeling with anti-human MMP1 and collagen type I
298 rrested in the G2 phase of the cell cycle by
double-labeling with BrdU and a mitosis-specific marker;
299 Also,
double-labeling with synaptotagmin showed that Kv1.2 col
300 Double labeling (
with NeuN and GFAP) immunohistochemistr