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1 l defects, common atrioventricular canal and double outlet right ventricle.
2 y anomalies, such as tetralogy of Fallot and double outlet right ventricle.
3 on of the OFT, and modeling human congenital double outlet right ventricle.
4 s seen ranging from common arterial trunk to double outlet right ventricle.
5 m, membranous ventricular septal defect, and double outlet right ventricle.
6 ital heart diseases, tetralogy of Fallot and double outlet right ventricle.
7 defects, atrioventricular valve defects and double outlet right ventricle.
8 tional semilunar cardiac valve defects and a double-outlet right ventricle.
9 5 (95% confidence interval, -0.58 to -0.31); double-outlet right ventricle, -0.48 (95% confidence int
10 outflow tract malrotation, overriding aorta, double outlet right ventricle, aberrant semilunar valve
11 y Ap2alpha-Cre mediated deletion resulted in double outlet right ventricle alignment heart defects.
12 y (E) 14.5 with cardiac defects that include double outlet right ventricle and a common atrioventricu
14 efects, including ventricular noncompaction, double outlet right ventricles and ventricular septal de
15 ome, 3 with critical aortic stenosis, 2 with double-outlet right ventricle and 1 each with an interru
16 ent defects ranging from overriding aorta to double-outlet right ventricle and dextro-transposition o
17 perinatally with cardiac defects, including double outlet right ventricle, and atrial and ventricula
19 aortic arch type B, aortic arch hypoplasia, double-outlet right ventricle, and ventricular septal de
20 cushion, and conotruncal ridge defects, with double outlet right ventricle, aorticopulmonary window,
21 xtrocardia, transposition of great arteries, double-outlet right ventricle, atrioventricular septal d
22 s of transposition of the great arteries and double-outlet right ventricle can share a common genetic
23 utflow tract, contributing to development of double outlet right ventricle (DORV) and ventricular sep
24 165 induced conotruncal malformation such as double outlet right ventricle (DORV) and ventricular sep
25 os had common atrioventricular canal (CAVC), double outlet right ventricle (DORV), hypoplastic ventri
26 ies included were tetralogy of Fallot (TOF), double outlet right ventricle (DORV), transposition of t
27 r ventricular septal defect (VSD), including double-outlet right ventricle (DORV), before 2001 with a
28 , the most common cardiac malformations were double-outlet right ventricle (DORV), TOF, and endocardi
31 HLHS was present in 45 patients, complex double-outlet right ventricle in 10, unbalanced AV canal
33 ely, transposition of the great arteries and double-outlet right ventricle, in the absence of lateral
34 undum ASD, one each with truncus arteriosus, double-outlet right ventricle, L-transposition of the gr
35 tening and rotation of the OFT, resulting in double-outlet right ventricle morphology, similar to tha
36 ct anomalies identified by micro-CT included double outlet right ventricle (n=36), transposition of t
38 eading to OFT malalignment defects including double-outlet right ventricle, overriding aorta and pulm
39 entricular septal defects, overriding aorta, double-outlet right ventricle, persistent truncus arteri
40 genitors display ventricular septal defects, double outlet right ventricle, semilunar valve hyperplas
41 receded a spectrum of septation defects from double outlet right ventricle to common arterial trunk i
42 diovascular outflow tract defects, including double outlet right ventricle, transposition of the grea
43 ar ventricular septal defects), conotruncus (double-outlet right ventricle, transposition, and membra
44 as a range of heart deformations, including double outlet right ventricle, ventricular-septal defect
45 Cre mice exhibited cardiac defects including double-outlet right ventricle, ventricular septal defect
46 l patterning of the branchial arch arteries, double-outlet right ventricle, ventricular septal defect
47 efect (PMVSD) and only 1 of 20 patients with double outlet right ventricle were found to have a 22q11
48 nd displayed a highly penetrant phenotype of double outlet right ventricle with a concurrent ventricu
49 lar defects in Lp homozygotes, which include double-outlet right ventricle, with obligatory perimembr
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