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1 -binding fluorochromes and a monoclonal anti-double-stranded DNA antibody.
2 e presence of antinuclear antibodies or anti-double-stranded DNA antibodies.
3 years, is characterized by the production of double-stranded DNA antibodies.
4 he 8.12 idiotype, which is expressed on anti-double-stranded-DNA antibodies.
5 with lupus nephritis, and patients with anti-double-stranded DNA antibodies (adjusted HR approximatel
8 -stranded DNA antibodies, deposition of anti-double-stranded DNA antibody complexes, complement activ
9 immune activation and the generation of anti-double-stranded-DNA antibodies, critically aggravating a
11 ell-B cell collaboration, production of anti-double-stranded DNA antibodies, deposition of anti-doubl
12 (antinuclear antibodies >/=1:80 and/or anti-double-stranded DNA antibodies >/=30 IU/ml) at baseline
13 rcent (at a dilution of 1:120 or more), anti-double-stranded DNA antibodies in 55 percent, anti-Ro an
14 MEK inhibitor was sufficient to induce anti-double-stranded DNA antibodies in a murine model of drug
15 rked female predominance, appearance of anti-double-stranded DNA antibodies in young adulthood, intra
16 tter than C3, plasma C4d, Bb, C5b-9 and anti-double-stranded DNA antibody in distinguishing patients
17 um complement component C3 and elevated anti-double-stranded DNA antibody levels at baseline improved
19 en) PCs and serum IgG antichromatin and anti-double-stranded DNA antibody levels were lower in NZM.Ba
20 reatment did not reduce serum levels of anti-double-stranded DNA antibodies or renal immune complexes
21 ing levels of IgG anti-chromatin and/or anti-double-stranded DNA antibodies or with amounts of these
22 ctivity was associated with anti-Ro and anti-double-stranded DNA antibodies (P = 4.6 x 10(-18) and P
23 sgenic BALB/c donors induced lupus with anti-double stranded DNA antibodies, proteinuria, and immune
24 d between the levels of anti-Scl-70 and anti-double-stranded DNA antibodies (r = 0.558, P < 0.001).
25 renal disease, and had higher titers of anti-double-stranded DNA antibodies than wild-type MRL/lpr mi
26 cations, but correlated positively with anti-double-stranded DNA antibody titers among SLE patients a
27 es from 12 to 5 (P < 0.0001) and median anti-double-stranded DNA antibody titers from 106 to 42 IU/ml
28 Deficiency of CAD resulted in increased anti-double-stranded DNA antibody titers in lupus-prone mice.
35 mplement C3, C4, and CH50 and titers of anti-double-stranded DNA antibodies were normalized after tre
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