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1  only a low density of receptors below 1,000 dpm/mg in a significant number of tumors.
2 ceptor density corresponding to 1,000-10,000 dpm/mg of tissue; conversely, single-ligand binding, alt
3 mits of quantitation were 175 pg and 0.00175 dpm injected.
4 f 3.0 x 10(-15), which is equivalent to 0.02 dpm tritium/mg of material.
5                                  Notably, <1 dpm (0.45 pCi) of (14)C-labeled compound was used in eac
6 tent had risen to values in the range of 100 dpm per liter.
7  high binding (mean density, 4,447 +/- 1,128 dpm/mg of tissue) with the antagonist.
8 mpared to the PE-Free group (2011.9+/-174.14 dpm/mg) (p<0.05).
9 had a low binding (mean density, 844 +/- 168 dpm/mg of tissue) with the agonist whereas 12 had a high
10 -1) x min(-1) (19.1 +/- 3.1 vs. 36.5 +/- 7.2 dpm/micromol) glucagon infusions, implying that a greate
11 bis-pyridine adducts of (dpm)Mn(II)(py)(2), (dpm)Fe(II)(py)(2), and (dpm)Co(II)(py)(2) reveal each di
12 tural samples with activities as low as 0.20 dpm above background (2sigma, integration time = 2 hr).
13 g) compared to the PE group (1907.32+/-136.3 dpm/mg) in the lateral septum (p<0.05).
14 ), and PAF (from 790 +/- 108 to 3380 +/- 306 dpm).
15 2 with the agonist (mean density, 348 +/- 49 dpm/mg of tissue) whereas all cases had a high sst2 bind
16 ve content, in the mid-1940s, of less than 5 dpm per liter of krypton.
17 her (4.50 +/- 0.29 vs. 3.16 +/- 0.21 x 10(5) dpm/ml per 5 h; P < 0.05) in the diabetic subjects than
18  the antagonist (mean density, 3,777 +/- 582 dpm/mg of tissue).
19  had higher receptor binding (2550.9+/-63.59 dpm/mg) when compared to the PE-Free group (2011.9+/-174
20 5) and phospholipase C (control 478.5 +/- 67 dpm/well, +PDGF 1049.3 +/- 93, n = 4, p = 0.003), while
21 -1) x min(-1) (19.0 +/- 3.9 vs. 41.4 +/- 5.7 dpm/micromol) and 3.0 ng x kg(-1) x min(-1) (19.1 +/- 3.
22 els of V1A receptor binding (2689.93+/-254.8 dpm/mg) compared to the PE group (1907.32+/-136.3 dpm/mg
23                       The specific activity (dpm/nmol DHA) in ER-enriched fraction was similar or hig
24 (dpm)Mn(II)(py)(2), (dpm)Fe(II)(py)(2), and (dpm)Co(II)(py)(2) reveal each divalent ion to be high-sp
25       Chemical oxidation of the deprotonated dpm framework results in the four-electron oxidation of
26 d, 1,9-dimesityl-5,5-dimethyldipyrromethane (dpm), have been prepared.
27 le uptake of EBD and (125)I-labeled insulin (dpm per gram dry tissue) with 0.1-Hz stimulation (n = 6)
28 (SQUID, EPR) of the bis-pyridine adducts of (dpm)Mn(II)(py)(2), (dpm)Fe(II)(py)(2), and (dpm)Co(II)(p
29    Site-specific fat specific activity (SA) (dpm/g lipid) decreased as a function of fat mass in both
30           The PARP-1 activities (mean +/- SD dpm/10(6) cells) were 18,554 +/- 9,070 (n=257), 14,847 +
31                    Palmitate tracer storage (dpm/g adipose lipid) and calculated palmitate storage ra
32 lly populated ligand-based orbitals from the dpm construct lie above partially filled metal 3d orbita
33                             Arylation of the dpm ligand alpha to the pyrrolic nitrogen donors limits
34 bound, its geometry or spin state within the dpm framework.
35 lectrochemical or chemical oxidation of the (dpm)M(II)(py)(2) complexes.
36       Differential pulse voltammetry on the (dpm)M(II)(py)(2) series reveals a common two-electron ox
37 tetrahedral in the solid state, whereas the (dpm)Ni(II)(py)(2) is low-spin and adopts a square-planar

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