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1 ing the 300 minutes after consumption of the drink.
2 ays (n=14) or 24 days (n=1) after their last drink.
3 arious time points after consumption of each drink.
4 hare of high-sugar, mid-sugar, and low-sugar drinks.
5 erfeiting of Scotch whisky, and other spirit drinks.
6  obtained optimum conditions were applied to drinks.
7  was quite effective in removing Pb(II) from drinks.
8 e before and 2 h after the ingestion of test drinks.
9 enes on a cross-sectional measure of average drinking.
10  is known about the impact of episodic heavy drinking.
11 enrollment) compared to those without recent drinking.
12 blood alcohol levels (BALs) after binge-like drinking.
13 f chronic, dependence-driven, and binge-like drinking.
14 a neuroimmune mechanism of excessive alcohol drinking.
15  genetic risk for high intensity, binge-like drinking.
16 , suggesting pre-systemic feedback gating of drinking.
17                           On day 3, subjects drank 1 x 109 colony-forming units of colonization facto
18                   All patients were asked to drink 1 L of water between early and late scans.
19                          An average consumer drinking 1.5 L of water daily that contains between 2 an
20 g/kg, twice/day, for 7 days; or intermittent drinking 20% ethanol in a two-bottle free choice protoco
21 ents with overweight or obesity who reported drinking 4 cups or less of water per day.
22 volitionally performed self-paced reaches to drink a mug of coffee (successfully completing 11 of 12
23 re less likely to be obese, to smoke, and to drink alcohol on a daily basis and had fewer self-report
24  of rats with a predisposition to prefer and drink alcohol, and was maintained for almost a year.
25 en treatment groups on any other measures of drinking, alcohol craving, or alcohol-related consequenc
26 17 out of 40 samples (42.5%); in some energy drinks also BPF, BADGE and BFDGE were determined.
27 ore anteroventral than those associated with drinking, although there was substantial overlap.
28 otide polymorphism rs112146896 with lifetime drinking and a negative association with anxiety in heal
29 utamatergic MnPO neurons produces effects on drinking and autonomic thermoregulatory mechanisms, prov
30 as associated with an increased frequency of drinking and binge drinking episodes in adolescents.
31 ffect modification between moderate lifetime drinking and binging (relative excess risk due to intera
32 articipates in mechanisms underlying alcohol-drinking and reconsolidation of alcohol-related memories
33 vious findings that naltrexone reduces heavy drinking and reward-related brain activation among treat
34 atus, material deprivation history, smoking, drinking and socioeconomic status, working-age men in fa
35 egative association between lifetime alcohol drinking and superior frontal gyrus volume.
36 dings support a relationship between ethanol drinking and the aldosterone/MR pathway in three differe
37 uroimmune pathways mediate excessive alcohol drinking and these findings will help to prioritize rele
38 e idea that increasing salt intake increases drinking and urine volume is widely accepted.
39 e environments and have potential for use in drinking and wastewater treatment.
40 ing of the price of high-sugar and mid-sugar drinks and activities to increase the market share of lo
41 ulin resistance, and dietary intakes of soft drinks and alcohol and was positively associated with hi
42 edure for the analysis of energy drink, soft drink, and chocolate milk samples was demonstrated.
43 eptions, increases in alcohol use, high-risk drinking, and DSM-IV AUD between 2001-2002 and 2012-2013
44          Twelve-month alcohol use, high-risk drinking, and DSM-IV AUD.
45 nner by which STDP responds to binge alcohol drinking, and its sensitivity to dopamine receptor antag
46 nd automatically measures standing, feeding, drinking, and locomotor activities from 3D trajectories.
47          Cognitive impairments, uncontrolled drinking, and neuropathological cortical changes charact
48 positive loadings for fast foods, carbonated drinks, and refined grains, and high negative loadings f
49  by lay counsellors to patients with harmful drinking attending routine primary health-care settings.
50 ubstantial individual variability in alcohol drinking behaviors in the population, the neural circuit
51 dence for the involvement of FGF2 in alcohol-drinking behaviors.
52  as well as in mechanisms underlying alcohol-drinking behaviors.
53                    Stimulation also produced drinking behaviour that was inhibited as water was inges
54                       We found that repeated drinking bouts modulate differently synaptic plasticity
55 sforms the need for water into the desire to drink, but how this transformation is performed remains
56  in the VTA did not alter binge-like ethanol drinking, but inhibition of VTA-projecting CRF neurons f
57  a distributed thirst circuit that motivates drinking by the common mechanism of drive reduction.
58 mination of five bisphenols in canned energy drinks by UPLC with fluorescence detection, after clean
59 n ounces); untaxed fruit, vegetable, and tea drinks, by 4.37% (p < 0.001); and plain milk, by 0.63% (
60 tion of these neurons is sufficient to drive drinking, cardiovascular responses, and negative reinfor
61 /-2 degrees C for 10min) on whey-grape juice drink characteristics was investigated.
62 h as those generated by other people eating, drinking, chewing, and breathing [1-8].
63 einated coffee and for coffee with additives.Drinking coffee, either caffeinated or decaffeinated, ma
64 -drinker status) and the regular quantity of drinks consumed per week (drinks/week) among drinkers.
65  7 days apart), subjects were given a 500-mL drink containing 40 g of carbohydrate (glucose in the fi
66 d (1:1) to active product (125 mL once-a-day drink containing Fortasyn Connect) or control product.
67 ars of heavy drinking, in which cessation in drinking could prevent the disease.
68 ugh the primary outcome, percentage of heavy drinking days, was lower in participants receiving ABT-4
69                                        Binge drinking, defined as reaching a blood alcohol level of 8
70        Secondary outcomes were the effect of drinking, disability score, days unable to work, suicide
71 ld motor vehicle passengers, speed limit and drunk driving laws, and alcohol ignition interlock use.
72 ion and education, the drinking environment, drink-driving, and brief interventions and treatment.
73                                    Mice that drank during stimulation consumed 912 +/- 163 mul (n = 8
74 pendent rats and the correlates with ethanol drinking during acute withdrawal.
75 dex, pregnancy complications, and smoking or drinking during pregnancy.
76 but not placebo, experienced the least heavy drinking during the following 14 weeks.
77 availability, information and education, the drinking environment, drink-driving, and brief intervent
78 an increased frequency of drinking and binge drinking episodes in adolescents.
79                            The control group drank equal volumes of water and avoided drinking the ju
80 vels can be readily translated into standard drink equivalents per day of different countries, the WH
81  mesostriatal neuroadaptations, resulting in drinking escalation and addiction phenotypes.
82 ositivity were similar across smoking and/or drinking exposure groups: HRfor low exposure, 0.52; 95%
83                             Milk is a common drink for young European children.
84                        Unlike this excessive drinking, for which treatments exist, compulsive alcohol
85 ssociation of rs11720469 with heavy episodic drinking (frequency of consuming 5+ drinks within 24 h).
86 lcohol consumption (units per week and binge drinking) from Scottish Health Surveys done in 1995, 199
87                 Globally, 200 million people drink groundwater contaminated with fluoride concentrati
88  unique neuroadaptations between the alcohol drinking groups.
89 = 0.52; 95% CI: 0.30 to 0.89), whereas those drinking &gt;1 glass/day had significantly higher risk (RR
90 th accelerated fibrosis progression, whereas drinking &gt;14 drinks per week showed increased rates of f
91 VTA dopamine neuron activity in high alcohol drinking (HAD) mice does not differ from alcohol naive m
92                       The results reveal how drinking history alters the association between brain FC
93 s anatomic sites, independent of smoking and drinking history.
94 ria crinita is widely used as a popular folk drink; however, little is known about how the post-harve
95 ong male primary care attendees with harmful drinking in a setting in India.
96  synaptic proteins with novel links to heavy drinking in BXD mice.
97 ed anxiety-like behavior and compulsive-like drinking in dependent rats.
98             For long-term studies of alcohol drinking in mice we used IntelliCages.
99                                         High Drinking in the Dark (HDID-1) mice are a genetic model o
100 Tlr4 knockdown in mouse NAc did not decrease drinking in the two-bottle choice continuous or intermit
101                      The prevalence of binge drinking in the United States is rising.
102 in the past 14 days among those who reported drinking in this period (37.0 g [SD 44.2] vs 31.0 g [27.
103 nonsignificant increase in estimated monthly drinks in the first year after deployment that regressed
104    Compared to controls, ED had higher total drinks in the past 30 days, higher levels of LPS, sCD14
105 54% of expenditure on food and non-alcoholic drinks) in the lowest SEIFA quintile, a difference of AU
106 s is usually preceded by many years of heavy drinking, in which cessation in drinking could prevent t
107       In mice, (+)-naloxone did not decrease drinking-in-the-dark and only modestly inhibited depende
108 n the VTA CRF system were assessed following drinking-in-the-dark procedures.
109 on-treatment-seeking alcohol-dependent heavy-drinking individuals.
110         Voluntary restraints by the food and drinks industry have had little effect on disease burden
111 exporting much of its production to the soft drinks industry.
112                       The ability to inhibit drinking is a significant challenge for recovering alcoh
113                                     Underage drinking is widely recognized as a leading public health
114 pulsively seeking alcohol, before eventually drinking large amounts.
115 ins with the D allele at the C2cd2l interval drank less alcohol than B allele strains.
116 r deployment that regressed to predeployment drinking levels 2 years after deployment.
117 NTERPRETATION: The health impact of the soft drinks levy is dependent on its implementation by indust
118 < 0.05).In older men and women, whey-protein drinks load-dependently slow gastric emptying and alter
119 ong ED with recent alcohol consumption (last drink &lt;10 days before enrollment) compared to those with
120                           Never-smoker males drinking &lt;/=1 glass/week had significantly lower risk fo
121  parental social class, maternal smoking and drinking, maternal mental health, offspring stressful li
122 d NASH and fibrosis; however, heavy episodic drinking may accelerate fibrosis progression and moderat
123 c strength in DMS D1- and D2-MSNs of alcohol-drinking mice and control mice.
124 vivo microdialysis procedures in MA high/low drinking mice, as well as in isogenic C57BL/6J mice that
125                                           In drinking monkeys, evoked firing of OFC pyramidal neurons
126 ntrol glutamatergic signaling in chronically drinking monkeys.
127                                Compared with drinking no coffee, coffee consumption was associated wi
128 levels from these bisphenols in forty energy drinks of different brands, collected from the market in
129             In addition, the role of alcohol drinking on outcomes in patients with cancer is in its f
130 o placebo, reduced VS activation and bar-lab drinking only among carriers of the DAT1 9-repeat allele
131     Compared to those who neither smoker nor drink, only patients who both smokers and drinkers were
132 roanthocyanidin-rich foods (apples and cocoa drinks; P = 0.04) and, in younger participants (aged <50
133 s extensive epidemiological support for this drinking pattern, a consensus has not been reached.
134 umption, even after accounting for different drinking patterns, obesity, and smoking status at the in
135 d fibrosis progression, whereas drinking >14 drinks per week showed increased rates of fibrosis progr
136 in pairs explained all remaining variance in drink preferences.
137 oped symptoms compatible with botulism after drinking pruno, an illicit, prison-brewed alcoholic beve
138    Inclusion of MTGase into acidified yogurt drinks reduces the serum separation with an improved vis
139 refore, if supported by evidence of benefit, drinking reduction goals could broaden the appeal of tre
140 n the aim of treatment, many drinkers prefer drinking reduction goals.
141 atment goals and clinical recommendations on drinking reduction.
142 d 18+/-3, 4.2+/-0.1, 53+/-5mugL(-1) in apple drinks, respectively.
143 nsing TRCs in thirsty animals induced robust drinking responses toward light even without water.
144                             Reduction in WHO drinking risk level predicted significantly lower odds o
145  test the relationship between change in WHO drinking risk levels between Waves 1 and 2, and alcohol
146  clinical trials, including reduction in WHO drinking risk levels-very high, high, moderate, and low-
147                                In the spirit drinks sector, counterfeiters often 'recycle' used genui
148                              Child food- and drink-sharing behaviors should be included in efforts to
149 cidal behaviour, percentage of days of heavy drinking, Short Inventory of Problems score, WHO Disabil
150 ltage OH processes on whey acerola-flavoured drinks should be performed at low frequencies and voltag
151 n the DMS is a positive regulator of alcohol drinking.SIGNIFICANCE STATEMENT Long-term alcohol intake
152 gative emotional state that drives excessive drinking.SIGNIFICANCE STATEMENT The central amygdala (Ce
153 his new procedure for the analysis of energy drink, soft drink, and chocolate milk samples was demons
154 , and biochemical variables at admission and drinking status during follow-up were obtained.
155 , smokers (SMK), drinkers (DRN), smoking-and-drinking subjects (SAD), marijuana users (MAR), smoking-
156 ted with medication in predicting subsequent drinking, such that individuals with greater reduction i
157  than comparison groups and less problematic drinking than classic psychedelic users.
158 discovered several proteins related to heavy drinking that have potential as novel targets for treati
159                                         Test drinks that contained various amounts of cocoa flavanols
160 oup drank equal volumes of water and avoided drinking the juices.
161 agement was associated with changes in heavy drinking, treatment attendance, drug use, cigarette smok
162 ocessed foods, and ultra-processed foods and drinks (UPFDs).
163 ing-and-marijuana users (SAM), marijuana-and-drinking users (MAD), and users of all three substances
164                            Lactating mothers drank vegetable, beet, celery, and carrot juices for 1 m
165                       Whey acerola-flavoured drink was treated using ohmic heating (OH) at 65 degrees
166                                       Coffee drinking was associated with reduced risk for death from
167 nse to naltrexone, whereas individuals whose drinking was driven by negative reinforcement (ie, relie
168           PREDICT proposed individuals whose drinking was driven by positive reinforcement (ie, rewar
169 ptogenetic activation of these neurons, mice drank water and performed an operant lever-pressing task
170 mental Protection Agency limit for Cu(2+) in drinking water (20 muM).
171 e fed a high fat diet with 5% sucrose in the drinking water (HFS) for 7 months and then were fed for
172 fat diet supplemented with 30% d-fructose in drinking water (obesogenic diet) for 25-33 weeks.
173 ed PI-IBS following exposure to contaminated drinking water 7 years ago.
174 ronmental Protection Agency (EPA)'s National Drinking Water Advisory Council (NDWAC) recommended esta
175 to two groups: controls provided with normal drinking water and DOCA provided with DOCA pellets and s
176 EDS) model estimated mean iAs exposures from drinking water and rice were 4.2 mug/day and 1.4 mug/day
177 EDS) model estimated mean iAs exposures from drinking water and rice were [Formula: see text] and [Fo
178 uvial flood-risk management and forecasting, drinking water and sewer network operation and managemen
179 n potential cumulative endocrine activity in drinking water and to inform prioritization of future mo
180  most commonly identified causative agent in drinking water associated with disease outbreaks, can be
181 h-based, household action level" for lead in drinking water based on children's exposure.
182 breaks, can be harbored by and released from drinking water biofilms.
183 bsequent accumulation of cadmium in food and drinking water can result in accidental consumption of d
184                    GenX was also detected in drinking water collected from 3 out of 4 municipalities
185     Ongoing exposures to even relatively low drinking water concentrations of long-chain PFAAs substa
186 uation was conducted based on representative drinking water conditions to determine a minimal model (
187 el (3 reactions, 8 constants) applicable for drinking water conditions.
188 and therefore they may pose greater risks to drinking water consumers given their widespread occurren
189                                              Drinking water containing the highest level of GenX also
190 s by bloom-forming cyanobacteria can lead to drinking water crises, such as the one experienced by th
191 nogenic N-nitrosodimethylamine (NDMA) during drinking water disinfection is a major challenge.
192                              In conventional drinking water disinfection, N-chloroisobutyraldimine ca
193  bacterial and fungal taxa commonly found in drinking water distribution systems through the treatmen
194 rently installed treatment processes at U.S. drinking water facilities to be on the order of $500 mil
195 s) since the U.S. EPA analysis suggested few drinking water facilities would be affected by bromide d
196 lm-associated L. pneumophila under simulated drinking water flow containing a disinfectant residual w
197  of 2014, when the city was rendered without drinking water for >2 days.
198 y addition of 2.5% dextran sodium sulfate to drinking water for 5-9 consecutive days.
199             Mice were given (13)C-acetate in drinking water for measurement of cholesterol synthesis.
200 bent with the potential to improve access to drinking water for millions living in developing countri
201           Groundwater is the major source of drinking water for people living in rural areas of India
202  population at risk from elevated arsenic in drinking water from private wells.
203  for direct determination of free cyanide in drinking water has been reported.
204 onmental Protection Agency to issue lifetime drinking water health advisories for perfluorooctanoic a
205                         Arsenic exposure via drinking water impacts millions of people worldwide.
206  detectable incidence of waterborne AGI from drinking water in the systems and time periods studied.
207 ndicator of microbiological contamination of drinking water in time-series studies attempting to disc
208 reported MCL violations to the national Safe Drinking Water Information System (SDWIS).
209                                        Daily drinking water intake and rice consumption rate distribu
210  the distribution of iAs exposure rates from drinking water intakes and rice consumption in the U.S.
211                               Access to safe drinking water is a human right, crucial to combat inequ
212 etection marker for mercury ions (Hg(2+)) in drinking water is of great interest for toxicology asses
213  approach that can be used to determine what drinking water lead concentrations keep children's blood
214 han the US Environmental Protection Agency's drinking water limit (2 ppb), within 10 min.
215                                              Drinking water maximum contaminant levels (MCL) are esta
216      Long-term increased lithium exposure in drinking water may be associated with a lower incidence
217                                              Drinking water microbial communities impact opportunisti
218 ng water treatment processes shape the final drinking water microbial community via selection of comm
219                                Comparison of drinking water PFOA concentrations to those study findin
220 N-Cl-DCAM tends to deprotonate under typical drinking water pH conditions, and the anionic form of N-
221 ses in disinfection byproducts at downstream drinking water plants.
222                 Complexities associated with drinking water plumbing systems can result in undesirabl
223 ) adsorbed to granular activated carbon in a drinking water production plant, which cannot be labeled
224 ts both carbon cycling in surface waters and drinking water production.
225 ty in surface waters and treatability during drinking water production.
226 he classical disinfection treatments used in drinking water production.
227                              Chlorination of drinking water protects humans from water-born pathogens
228                       In the USA, impacts to drinking water quality, biogeochemical cycles, and aquat
229                            Lead exposure via drinking water remains a significant public health risk;
230 ate change to hydraulic fracturing, and from drinking water safety to wildfires, environmental challe
231 tform for measurements of fluoride levels in drinking water samples.
232 e higher exposures than adults from the same drinking water source.
233 pical de facto potable reuse scenario, where drinking water sources are located downstream of treated
234  research was key to the nation's first-ever drinking water standard for CrVI adopted by California i
235 nd groundwaters at levels above health-based drinking water standards.
236 rtant role in the removal of impurities from drinking water supplies.
237 ill identify the dangers hidden in America's drinking water supply and redirect attention to ensure s
238 m these regions contribute up to half of the drinking water supply for some European countries.
239                            The two municipal drinking water systems of New Orleans, LA, U.S.A. were s
240                       Twenty-two (22) public drinking water systems serving 2.5 million people were i
241 tormwater systems differ from wastewater and drinking water systems to which LCA is more frequently a
242 e children chronically exposed to Mn through drinking water to investigate the effect of Mn exposure
243 n potential in Lake Michigan, which provides drinking water to over 10 million people.
244 entrations of 2,6-dichlorobenzamide (BAM) in drinking water treatment plants (DWTPs).
245       Study results suggest that centralized drinking water treatment processes shape the final drink
246  water distribution systems, and centralized drinking water treatment represents a potential control
247 city and chemical consumption for individual drinking water unit processes are used to estimate embed
248 nated dipeptides as chlorination products in drinking water using complementary high-resolution quadr
249 cation systems are easy ways to obtain clean drinking water when there is no large-scale water treatm
250 illimolar sodium chloride level (freshwater, drinking water, and aquarium water, as well as dechlorid
251 he existence of a variety of heavy metals in drinking water, and the four-electrode sensor can distin
252 lication of our strategy in aqueous samples (drinking water, apple juice, and skim milk).
253 inc in various samples including well water, drinking water, black tea, rice, and milk.
254 d on phenol addition and recovery studies in drinking water, obtaining recoveries rates between 90% a
255 re, for each 100 mg/L reduction in sodium in drinking water, systolic/diastolic BP was lower on avera
256  Given that groundwater is a major source of drinking water, the main objective of this work was to i
257 ore than a billion people lacking accessible drinking water, there is a critical need to convert nonp
258 senite (As(III)) that contaminates crops and drinking water.
259 or microbial community structure in finished drinking water.
260 ovided with DOCA pellets and sodium chloride drinking water.
261 dipeptides as new disinfection byproducts in drinking water.
262 e resources for recreation and as sources of drinking water.
263 mmunities from the impact of contaminants in drinking water.
264 aking groundwater as their primary source of drinking water.
265 or the removal of E. coli and turbidity from drinking water.
266 can be modulated by providing the inducer in drinking water.
267 he concentration of this chemical present in drinking water.
268 ated arsenical, or arsenobetaine exposure in drinking water.
269 ffects in communities where they contaminate drinking water.
270 al and anthropogenic barriers, even reaching drinking water.
271  and analyze fluoride concentration level in drinking water.
272 wo-day summit to identify options to improve drinking-water quality for N.C. residents served by priv
273 cal levels of D-mannose safely achievable by drinking-water supplementation suppressed immunopatholog
274 val of BMAA in the chlorination process of a drinking-water system.
275 dicators to investigate the sources of Mo in drinking-water wells from shallow aquifers in a region o
276 tion byproducts (N-DBPs) whose occurrence in drinking waters has recently been reported in several DB
277 e broadly speaking, N-Cl-HAMs in chlorinated drinking waters is of significance because they are orga
278 tual DCAN degradation product in chlorinated drinking waters.
279 radation are generally atypical for finished drinking waters.
280 rate use, and 19.7% heavy use (6.7% had 8-14 drinks/week and 13.0% had >14 drinks/week) at cohort ent
281  rs7686419 (beta=-0.04, P=3.41 x 10(-10) for drinks/week and OR=0.96, P=4.08 x 10(-5) for drinker sta
282 nd rs4665985 (beta=0.04, P=2.26 x 10(-8) for drinks/week and OR=1.04, P=5 x 10(-4) for drinker status
283 egular quantity of drinks consumed per week (drinks/week) among drinkers.
284 (6.7% had 8-14 drinks/week and 13.0% had >14 drinks/week) at cohort entry.
285 week), moderate (4-7 drinks/week), heavy (>7 drinks/week), and very heavy (>14 drinks/week).
286 nent, light (1-3 drinks/week), moderate (4-7 drinks/week), heavy (>7 drinks/week), and very heavy (>1
287 and use categorized as abstinent, light (1-3 drinks/week), moderate (4-7 drinks/week), heavy (>7 drin
288  status and beta=-0.19, P=1.91 x 10(-35) for drinks/week), which replicated in Hispanic/Latinos (OR=0
289  heavy (>7 drinks/week), and very heavy (>14 drinks/week).
290 he rural area, cigarette smoking and alcohol drinking were associated with insomnia.
291  sweetened, artificially flavored milk-based drinks were associated with the %FGV with girls who cons
292                     Mineral water and energy drinks were chosen for removal of lead since the latter
293                             Units of alcohol drunk were positively correlated with the amount of retr
294 d the effects of self-supplementation with a drink with omega-3s, antioxidants, and resveratrol on Mi
295 ugar-sweetened beverages (SSBs; high tax for drinks with >8 g of sugar per 100 mL, moderate tax for 5
296 d for the determination of Cd and Pb in soft drinks with different brands and flavours.
297 ial for the production of digestible protein drinks with good consumer visual appeal owing to their c
298  determination of the dyes in processed soft drinks with satisfactory results (recovery>95% and RSD%<
299 episodic drinking (frequency of consuming 5+ drinks within 24 h).
300  who never binged, moderate drinkers (60-229 drinks/year) who binged had a higher risk (HR = 1.25, 95

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