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1 jections have a crucial role in coordinating drinking behavior.
2 te to brain function and behavior, including drinking behavior.
3 i (CSs), need to be dissociated from alcohol drinking behavior.
4 l function, blood pressure, food intake, and drinking behavior.
5 n COAs who have not demonstrated any problem drinking behavior.
6 ons that may be encountered when engaging in drinking behavior.
7 ng of genetic mechanisms influencing alcohol drinking behavior.
8 ereby 5-HT(1A) receptor activation modulates drinking behavior.
9 er that appears to play a role in eating and drinking behavior.
10 f 15-year-old adolescents assessed for risky drinking behavior.
11 contributes to mechanisms underlying alcohol drinking behavior.
12 lies high anxiety-like and excessive alcohol-drinking behavior.
13 r many individuals with harmful or hazardous drinking behavior.
14 tionally involved in, mediating high alcohol drinking behavior.
15 ed in alcohol reinforcement and high alcohol drinking behavior.
16 dence for the involvement of FGF2 in alcohol-drinking behaviors.
17 nship between core ADHD symptoms and smoking/drinking behaviors.
18 art of the brain (DMS) that controls alcohol-drinking behaviors.
19 as well as in mechanisms underlying alcohol-drinking behaviors.
20 he development and/or maintenance of ethanol drinking behaviors.
21 tend to exhibit heterogeneous postdeployment drinking behaviors.
22 al role of HDAC2 in anxiety-like and alcohol-drinking behaviors.
23 oadaptations that underlie excessive alcohol-drinking behaviors.
24 l circuit responsible for individual alcohol drinking behaviors.
25 ion is associated with healthier smoking and drinking behaviors.
26 r to molecular mechanisms underlying alcohol-drinking behaviors.
27 3beta, that in turn drives excessive alcohol-drinking behaviors.
28 st the actions of GDNF in the VTA on ethanol-drinking behaviors.
29 dence and comorbidity of anxiety and alcohol-drinking behaviors.
30 ar mechanisms underlying anxiety and alcohol-drinking behaviors.
31 burst activity in HAD mice decreases alcohol drinking behaviors.
32 EB gene is associated with increased alcohol-drinking behaviors.
33 us Arf6 in the PFC to modulate human alcohol-drinking behaviors.
34 lateral ventricle, thereby suppressing water-drinking behavior after hyperosmotic shock, similar to S
35 l subset of alcoholic transplant recipients, drinking behavior after liver transplantation is associa
36 anic, and they lend support to the view that drinking behavior among those with panic disorder is rei
38 area (LHA) might be important in modulating drinking behavior and fluid balance has led to numerous
39 brain neuropeptide that controls feeding and drinking behavior and gastric emptying and elicits neuro
40 ian preoptic nucleus, involved in initiating drinking behavior and salt appetite), neuroendocrine sys
41 rtance of assessing multiple levels of binge drinking behavior and their predictors among youth to ta
43 es over 6 years after the sessions to assess drinking behaviors and alcohol use disorder (AUD) sympto
44 lysis, we replicated prior associations with drinking behaviors and identified multiple novel phenome
45 blood pressure, body mass index, smoking and drinking behaviors, and exercise were examined in an ong
47 al sessions was measured on both seeking and drinking behaviors, as well as on drinking in the 2-bott
48 not significantly associated with a person's drinking behavior, but the behavior of relatives and fri
49 rgic neurons in the subfornical organ drives drinking behavior, but the brain targets that mediate th
50 ing promise in the prevention of problematic drinking behavior, but their effect on illicit drug use
52 s well as slightly increased risk of smoking/drinking behaviors by an indirect effect of depression.
53 r findings suggest that GDNF reduces alcohol-drinking behaviors by reversing an alcohol-induced allos
54 dicate that the hPer1 gene regulates alcohol drinking behavior during stressful conditions and provid
55 er1 were tested for association with alcohol drinking behavior in 273 adolescents and an adult case-c
62 The present investigation evaluated alcohol-drinking behaviors in mice that are haplodeficient in CR
64 ubstantial individual variability in alcohol drinking behaviors in the population, the neural circuit
65 reports showing increased KOR regulation of drinking behaviors induced by ethanol exposure, the stro
69 bic dopamine system have been shown to alter drinking behavior, presumably because this dopaminergic
72 enetic predisposition to anxiety and alcohol-drinking behaviors using alcohol-preferring (P) and -non
76 l drinking, alcohol-seeking, or relapse-like drinking behavior, we demonstrate that N-acetylhomotauri
77 ed using high-throughput RNA sequencing, for drinking behavior were dominated by neurophysiological t
80 m of ADHD had a robust impact on smoking and drinking behaviors, while being mediated by anxiety and
81 ng mechanism in the establishment of alcohol drinking behavior with changing the DA-5-HT balance as a
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