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1 pathy, manganese intoxication in intravenous drug abusers).
2 progression to HAD are accelerated in opiate drug abusers.
3 s and regions (e.g., prefrontal cortices) in drug abusers.
4 celerated disease observed in HIV-1-infected drug abusers.
5 mercial blood donors, and 17% of intravenous drug abusers.
6 he putamen of 11 self-identified intravenous drug abusers.
7 may underlie decision-making impairments in drug abusers.
8 on to AIDS dementia is accelerated in opiate drug abusers.
10 to develop immune thrombocytopenia than non-drug abusers and have a higher coinfection with hepatiti
12 inal opportunistic infections in intravenous drug abusers and in opioid-medicated postsurgical patien
15 nals in the brains of both animals and human drug abusers, and the cellular mechanisms underlying thi
17 inergic, opioid, and serotonergic systems in drug abusers are seen in positron emission tomography sc
18 ne transporters observed in studies of human drug abusers are the result of the neurobiological effec
20 hromosomal markers whose alleles distinguish drug abusers from control individuals in each of two sam
22 caine doses within the range administered by drug abusers induces cerebral microischemia and that the
24 mong commercial blood donors and intravenous drug abusers, only 1%-3% of the TTV-positive individuals
25 the brain endothelium seen in HIV(+)/cocaine drug abusers paralleled increased CD68 immunostaining co
26 primates, cocaine at doses typically used by drug abusers significantly increased brain glucose metab
27 mental stimuli may be involved in motivating drug abusers to engage in drug seeking after abstinence.
28 duals, the majority of whom were intravenous drug abusers, were analyzed by using PCR with restrictio
29 drug widely prescribed for the treatment of drug abusers with opioid dependence, affects human immun
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