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1 er drugs, a phenomenon termed 'incubation of drug craving'.
2 ch can at times be maladaptive (for example, drug cravings).
3 statement of drug seeking, and incubation of drug craving.
4 ensembles in this new form of incubation of drug craving.
5 ned despite adverse consequences and intense drug craving.
6 ous opioids in the neural systems underlying drug craving.
7 entral striatum were associated with intense drug craving.
8 sue that is frequently caused by cue-induced drug craving.
9 sity to engage in drug-seeking behavior, and drug craving.
10 th the conditioned place preference model of drug craving.
11 exposure to drug-associated cues that induce drug craving.
12 cocaine and whether this is associated with drug craving.
13 ion, including those that mediate reward and drug craving.
14 be important for understanding the basis of drug craving, a key factor in the maintenance of substan
15 here may have implications for cue-elicited drug craving after repeated exposure to drugs of abuse.
17 ues that are associated with self-reports of drug craving and appear to be of a greater magnitude tha
18 e findings support the use of stress-induced drug craving and associated hypothalamic-pituitary-adren
20 h stress and drug cue exposure each increase drug craving and contribute to relapse in cocaine depend
21 a novel animal model to study incubation of drug craving and cue-induced drug seeking after prolonge
28 rly long-lived aspects of addiction, such as drug craving and relapse, and to identify specific genes
30 o develop medications that effectively treat drug craving and relapse, the core features of addictive
31 the neurobiological factors responsible for drug craving and relapse, with promising therapeutic imp
37 ated with previous drug exposure can trigger drug craving and seeking, and form a substantial obstacl
39 ceptor agonists in decreasing stress-induced drug craving and stress-induced initial heroin lapse.
40 s like BP 897 could be used for reducing the drug craving and vulnerability to relapse that are elici
42 ncrease the likelihood of relapse by evoking drug craving, and brain-imaging studies have identified
44 hlight sex-specific effects of guanfacine on drug craving, anxiety, and negative mood with significan
45 brain regions involved in the incubation of drug craving, as well as evidence for the underlying cel
46 ere no differences between the two groups in drug craving, depressed mood, anxiety, or Clinical Globa
47 ental procedures used to study incubation of drug craving do not incorporate negative consequences of
49 gaine results in a long-lasting reduction of drug craving (humans) and drug and alcohol intake (roden
50 in synaptic activity related to cue-induced drug craving in 8 crack cocaine-dependent African Americ
51 f regions previously shown to be involved in drug craving in addicted subjects (orbitofrontal cortex,
52 It was suggested in 1986 that cue-induced drug craving in cocaine addicts progressively increases
54 anding the neurobiology of the incubation of drug craving in rodents is likely to have significant im
58 d animal models and associated with enhanced drug craving, is eliminated in flies mutant for period,
59 itization is proposed to model the increased drug craving observed in human psychostimulant abusers.
60 s was associated with self-ratings of robust drug craving or anger, and comparable alterations in hea
62 ics vs whites: OR, 0.48; 95% CI, 0.24-0.95), drug craving (OR, 2.78; 95% CI, 1.54-5.02), chronic illn
63 drug action and thereby partially alleviate drug craving, or that affect the addiction process per s
64 ked by pathological drug seeking and intense drug craving, particularly in response to drug-related s
65 ther relapse models, including incubation of drug craving, reacquisition and resurgence models, and p
66 luence of ibudilast on subjective (including drug craving), reinforcing, and analgesic effects of oxy
68 addiction retain some degree of control over drug craving that correlates with neural activity in the
69 Here, we used a rat model of incubation of drug craving, the neuronal activity marker Fos, and the
70 ed frontal lobe contributions to cue-induced drug craving under different intertemporal drug availabi
72 l imagery control condition, imagery-induced drug craving was associated with bilateral (right hemisp
73 nger control condition, internally generated drug craving was associated with bilateral activation of
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