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1 er drugs, a phenomenon termed 'incubation of drug craving'.
2 ch can at times be maladaptive (for example, drug cravings).
3 statement of drug seeking, and incubation of drug craving.
4  ensembles in this new form of incubation of drug craving.
5 ned despite adverse consequences and intense drug craving.
6 ous opioids in the neural systems underlying drug craving.
7 entral striatum were associated with intense drug craving.
8 sue that is frequently caused by cue-induced drug craving.
9 sity to engage in drug-seeking behavior, and drug craving.
10 th the conditioned place preference model of drug craving.
11 exposure to drug-associated cues that induce drug craving.
12  cocaine and whether this is associated with drug craving.
13 ion, including those that mediate reward and drug craving.
14  be important for understanding the basis of drug craving, a key factor in the maintenance of substan
15  here may have implications for cue-elicited drug craving after repeated exposure to drugs of abuse.
16 amatergic synapses that lead to endurance of drug craving and addiction are poorly understood.
17 ues that are associated with self-reports of drug craving and appear to be of a greater magnitude tha
18 e findings support the use of stress-induced drug craving and associated hypothalamic-pituitary-adren
19 cortex (PFC) can cause lasting reductions in drug craving and consumption.
20 h stress and drug cue exposure each increase drug craving and contribute to relapse in cocaine depend
21  a novel animal model to study incubation of drug craving and cue-induced drug seeking after prolonge
22                      An insidious feature of drug craving and drug seeking in humans is that it can b
23 entiation that may contribute to cue-induced drug craving and drug-seeking behavior.
24       Addiction-associated behaviors such as drug craving and relapse are hypothesized to result from
25  inhibition of this system may contribute to drug craving and relapse in addicted subjects.
26                      Evidence indicates that drug craving and relapse in humans are often provoked by
27                                              Drug craving and relapse to drug use during abstinence a
28 rly long-lived aspects of addiction, such as drug craving and relapse, and to identify specific genes
29                            In a rat model of drug craving and relapse, cue-induced drug seeking progr
30 o develop medications that effectively treat drug craving and relapse, the core features of addictive
31  the neurobiological factors responsible for drug craving and relapse, with promising therapeutic imp
32 cocaine-seeking behavior, an animal model of drug craving and relapse.
33 ptible to environmental stimuli that trigger drug craving and relapse.
34 lated cues, leading to an intensification of drug craving and relapse.
35 ation by cue-associated activity, triggering drug craving and relapse.
36                                              Drug craving and seeking can increase during a period of
37 ated with previous drug exposure can trigger drug craving and seeking, and form a substantial obstacl
38 tive states, has recently been implicated in drug craving and social stress.
39 ceptor agonists in decreasing stress-induced drug craving and stress-induced initial heroin lapse.
40 s like BP 897 could be used for reducing the drug craving and vulnerability to relapse that are elici
41  and differences in the neural correlates of drug cravings and gambling urges in CD and PG.
42 ncrease the likelihood of relapse by evoking drug craving, and brain-imaging studies have identified
43           Importantly, stress contributes to drug craving, and there is evidence for overlap between
44 hlight sex-specific effects of guanfacine on drug craving, anxiety, and negative mood with significan
45  brain regions involved in the incubation of drug craving, as well as evidence for the underlying cel
46 ere no differences between the two groups in drug craving, depressed mood, anxiety, or Clinical Globa
47 ental procedures used to study incubation of drug craving do not incorporate negative consequences of
48  exposure to drug-associated cues, provoking drug craving even after prolonged abstinence.
49 gaine results in a long-lasting reduction of drug craving (humans) and drug and alcohol intake (roden
50  in synaptic activity related to cue-induced drug craving in 8 crack cocaine-dependent African Americ
51 f regions previously shown to be involved in drug craving in addicted subjects (orbitofrontal cortex,
52    It was suggested in 1986 that cue-induced drug craving in cocaine addicts progressively increases
53 del the altered brain states responsible for drug craving in human drug addicts.
54 anding the neurobiology of the incubation of drug craving in rodents is likely to have significant im
55                The incubation of cue-induced drug craving in rodents provides a model of persistent v
56  of withdrawal from cocaine, suggesting that drug craving incubates over time.
57                        Such an incubation of drug craving is not specific to cocaine, as similar find
58 d animal models and associated with enhanced drug craving, is eliminated in flies mutant for period,
59 itization is proposed to model the increased drug craving observed in human psychostimulant abusers.
60 s was associated with self-ratings of robust drug craving or anger, and comparable alterations in hea
61          The subjects indicated the onset of drug craving or emotional response, allowing comparison
62 ics vs whites: OR, 0.48; 95% CI, 0.24-0.95), drug craving (OR, 2.78; 95% CI, 1.54-5.02), chronic illn
63  drug action and thereby partially alleviate drug craving, or that affect the addiction process per s
64 ked by pathological drug seeking and intense drug craving, particularly in response to drug-related s
65 ther relapse models, including incubation of drug craving, reacquisition and resurgence models, and p
66 luence of ibudilast on subjective (including drug craving), reinforcing, and analgesic effects of oxy
67 ntribute to the impaired decision making and drug craving that characterize cocaine withdrawal.
68 addiction retain some degree of control over drug craving that correlates with neural activity in the
69   Here, we used a rat model of incubation of drug craving, the neuronal activity marker Fos, and the
70 ed frontal lobe contributions to cue-induced drug craving under different intertemporal drug availabi
71               Cocaine abusers may experience drug craving upon exposure to environmental contexts whe
72 l imagery control condition, imagery-induced drug craving was associated with bilateral (right hemisp
73 nger control condition, internally generated drug craving was associated with bilateral activation of
74                         Using a rat model of drug craving, we found that the responsiveness to cocain
75 ips between these measures and self-reported drug craving were examined.
76          Subjective effects of oxycodone and drug craving were measured with visual analog scales (VA

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